Visual pathway

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VISUAL PATHWAY Dr.S.Soundari Consultant Ophthalmologist Dr Agarwal’s Eye Hospital Chennai

description

Dr. Soundari from Dr. Agarwals Eye Hospital presenting a presentation on Visual pathway , in Kalpavriksha 2012 - Chennai

Transcript of Visual pathway

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VISUAL PATHWAY

Dr.S.SoundariConsultant Ophthalmologist

Dr Agarwal’s Eye HospitalChennai

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Optic nerve is an outgrowth of the brain

Its fibers posses no neurolemmal cells

Surrounded by meninges unlike any peripheral nerves

Both the primary and second order neurons are in the retina.

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VISUAL PATHWAY

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Optic nerve

Intraocular part

Intraorbital part

Intracanalicular part

Intracranial part

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OPTIC NERVE LESION &

FIELD DEFECTS

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Optic nerve field defects

Central scotoma

Enlargement of blind spot

Arcuate field defects

Altitudinal field defects

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Paillomacular bundle

Macular fibres enter the temporal aspect of the disc. Defect can lead to

Central scotoma

Centrocecal scotoma

Paracentral scotoma

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Causes for central scotoma

Demylineation[retrobulbar neuritis]

Leber’s hereditary optic neuropathy

Toxins- tobacco,lead,alcohol,methanol

Vitamin B12 deficiency

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Enlargement of blind spot

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Altitudinal field defect

Ischaemic optic neuropathy

Branch retinal artery occlusion

Inferior retinal coloboma

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CHIASMAL LESION &

FIELD DEFECTS

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Chiasma

Lower nasal fibres cross low and anteriorly

Upper nasal fibres cross high and posteriorly

Macular fibres also cross in the posterior part of the chiasm.

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Location of chiasma

Central fixation -80%- above the sella

Pre fixed chiasm-10%-located anteriorly-

so pitutary tumour involves the optic tract first [lower temporal fields first]

Post fixed chiasm-10%-located posteriorly- so optic nerve gets involved first[upper temporal fields first]

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Pitutary adenoma

Visual fields ; bitemporal hemianopia,junctional scotoma,

bitemporal hemianopic scotomaColour vision; early red deficitVisual acuity tends to reduceOptic disc- bow tie atrophy rarely papilloedemaExtraocular movements: cranial nerve palsies,see saw nystagmus,spasm nutans.

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hemifield slip- due to the failure of controlling phoria by fusion.

Post fixation blindness.

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Pseudo bitemporal hemianopia

Bilateral sectoral retinitis pigmentosa

Tilted disc

Bilateral inferotemporal retinoschsis.

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OPTIC TRACT LESIONS &

ITS FIELD DEFECTS

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OPTIC TRACT

Carries ipsilateral temporal fibres and controlateral nasal fibres and pupillary fibres.

So right optic tract lesion will cause left homonymous hemianopia

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ASSOCIATIONS

Controlateral pyramidal signs.

Incongruous homonymous hemianopia.

Wernicke's hemianopic pupil

Optic atrophy

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OPTIC RADIATION AND ITS FIELD DEFECTS

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OPTIC RADIATIONS

The corresponding retinal elements lie progressively closer, so congruous hemianopia.

Passes through the temporal lobe and pareital lobe and ends in the visual cortex.

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TEMPORAL LOBE

Controlateral congruous homonymous superior quadrantanopia[pie in the sky]

Controlateral hemisensory disturbance

Mild hemiparesis

Paraxysomal olfactory and uncinate fits.

Formed visual hallucinations

Seizures and receptive dysphasia.

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Pie in the sky

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PAREITAL LOBE

Controlateral congruous homonymous inferior quadrantanopia[pie on the floor]

Visual perception difficulties

Right-left confusion

Acalculia

Assymmetric OKN.[OKN response diminished towards the side of the lesion.]

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Pie on the floor

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Striate calcarine cortex

Congruous homonymous hemianopias with macular sparing, macular involvement alone.

Formed visual hallucinations.

Anton's syndrome[ denial of blindness]

Riddoch phenomenon

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THANK YOU