ECG. Electrocardiography It is a voltage difference, record the electrical activity of the heart as...
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Transcript of ECG. Electrocardiography It is a voltage difference, record the electrical activity of the heart as...
ElectrocardiographyElectrocardiography It is a voltage
difference, record the electrical activity of the heart as well as valuable information about the heart function and structure.
Willem Einthoven 1924
Leads (Leads (lead x electrodelead x electrode))LIMB LEADSIIIIII
AVFAVLAVR
CHEST LEADSV1V2V3V4V5V6
bipolar
unipolar
P waveP waveRepresent the
electrical activity of both atria ( atrial depolarization)
The depolarization slow within the AV node, there is a brief delay or PAUSE before the depolarization conducted to the ventricles
Normal duration <0.12 sec
Absent P wave:Atrial fibrillation SA BlockAV Rhythm
Peak P wave:Atrial hypertrophy
QRS ComplexQRS ComplexRepresent the
electrical activity of both ventricles.
Ventricular depolarization( initiation of the ventricular contraction
QRS ComplexQRS ComplexQQ wave wave
RR wavewave::
SS wave : wave :
: : Normal QRS duration < 0.12 secNormal QRS duration < 0.12 sec
QRS ComplexQRS ComplexQQ wave wave
first first downward downward deflection deflection ..
septal septal depolarization.depolarization.
0.04sec0.04sec..
RR wavewave: : first upwardfirst upward deflection.deflection. height: 5-8 mm.height: 5-8 mm. early ventricular
depolarization
ST - SegmentST - SegmentST segment: the
plateau phase of ventricular repolarization.
Isoelectric or> or<1mm.
If the ST segment elevated or depressed beyond the normal baseline this usually sign of serious pathology. (MI)
T- WaveT- WaveT-wave :represent rapid
phase of ventricular repolarization.
peaked T wave: early MI hyperkalemia Black races
Inverted : MI . Ventricular hypertrophy. Hypokalemia Digoxin
U waverepolarization of the interventricular
septum. low amplitude Prominent: suspect hypokalemia,
hypercalcemia or hyperthyroidism
J wave represents the approximate end of
depolarization and the beginning of repolarization
camel-hump sign .Hypothermia hypocalcemia.
AXISAXISAt any point during depolarization
and repolarization electrical potential are being propagated in different directions.
Most of these cancel each other out and only the net force is recorded. This net is called AXIS or cardiac VECTOR
Principles of ECG recording
Explain the indication and the procedure for the patient. (assurance )
Ask the patient to take off any metals he/she wears.
Expose the wanted sites.Cleaning of skin and shaving if necessary. Place the electrodes in the correct
positions . Instruct the patient to remain still (should
not talk during the test ) and relax their shoulders and legs while the recording takes place (1 min)
RAWIHIR: rate, regularity,rhythm(sinus
or asinus),A: axis.W:waves.I :intervals.H: hypertrophy.I: ischemia
Normal Sinus RhythmRate = 60-100 beat / minute. The rhythm is regular All intervals are within normal limits There is a P for every QRS and a QRS
for every P. P : QRS ratio = 1 : 1.The P waves all look the same Presence of P, QRS, T in each cycle.Normal shape, time of waves,
segments and intervals
Interfering factors Inaccurate placement of the electrodes Electrolyte imbalances Poor contact between the skin and the electrodes Movement or muscle twitching during the test
Drugs that can affect results include digitalis, quinidine, and barbiturates
MIWhen myocardial blood supply is abruptly reduced toa region of the heart, a sequence of injurious eventsoccur :
Ischemia ( subendocardial or transmural)
InjuryNecrosis, and eventual fibrosis
(scarring) if the blood supply isn't restored in an appropriate period of time
Inferior border leads
◦II, III and aVF◦the Inferior wall
of the RV◦ Posterior
Descending Branch of the RCA.
Posterior MINo leads look at the posterior wall.
usually associated with inferior and/or lateral wall MI. The changes of posterior myocardial infarction are seen
indirectly in the anterior precordial leads. Leads V1 to V3 face the endocardial surface of the posterior wall of the left ventricle. As these leads record from the opposite side of the heart instead of directly over the infarct, the changes of posterior infarction are reversed in these leads. The R waves increase in size, becoming broader and dominant, and are associated with ST depression and upright T waves. This contrasts with the Q waves, ST segment elevation, and T wave inversion seen in acute anterior myocardial infarction.
ST depression is considered reciprocal ECG changes in what should be ST elevation for acute posterior wall injury.
ECG Leads - Views of the Heart
lead border Arterial supply
V3 & V4 anterior Right Ventricle
RCA
V1 & V2 Septum LAD
a VL,V5 & V6
Lateral Left Ventricle
LCX
II+III+AVF inferior borderof right ventricle
RCA