Tests in Pediatric Neurology(2)
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Transcript of Tests in Pediatric Neurology(2)
7/23/2019 Tests in Pediatric Neurology(2)
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Direction of Pediatric Neurology of Pediatric Department of Tbilisi
State Medical University
Tests in Pediatric Neurology
//// The cause of seizures in early childhood is:
// Deficiency of Piridoxine
/// Deficiency of Calcium Panthothenate
/// Excessive amount of Riboflavine
/// Deficiency of Folium acid
//// The most frequent reason of eilesy in childhood is:
/// !eurosis
// Residual encehaloathy
/// "ller#y/// Poor feedin#
//// The tri##erin# factors of eilesy in early childhood is follo$in#% excet:
/// &nfection
/// Trauma
/// Radiation
// Poor nursin#
//// The simle absence is defined as:
// 'rief loosin# of consciousness
/// "brut droin#
/// Tonic(clonic seizures $ithout losin# of consciousness
/// &nvoluntary urination and defecation
)//// *est syndrome usually occurs in:
/// !eonate eriod
// + to , year of a#e
/// -chool eriod/// Pre(school eriod
.//// The transient Todds aresis is associated $ith:
/// 0eneralized eilesy
/// -imle absence
/// Comlex absence
// Partial eilesy
1/// 'y means of electroencehalo#rahy $e can not:
/// Establish the dia#nosis of eilesy
/// Define the localization of eiletic foci
,
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/// "ssess the efficiency of theraeutic mana#ement
// Define the etiolo#y
2//// The follo$in# dru#s are recommended for the treatment of absences:
// -uccinimides and 3alroic acid
/// 4idantoins
/// 'arbiturates
/// Carbamazeine
5//// 6ennox(0astaut -yndrome is defined as:
// Frequent% sometimes serial olymorhic seizures $ith mental retardation7
/// 8nly febrile seizures
/// 8nly artial seizures/// Disease $ith beni#n course
//// The follo$in# dru#s are recommended for the treatment of *est syndrome:
// 4ormones and 3alroic acid or 3i#abatrine
/// Phenobarbital
/// Dihenine
/// Diazeame
//// 0eneralized eilesies are the follo$in#% excet:
/// "bsences
/// 9yoclonic seizures
/// Tonic(clonic seizures
// ac;sons motor seizures
//// Partial seizures are the follo$in#% excet:
/// &dioathic < a#e(related=
/// -ymtomatic
/// Cryto#enic// *est syndrome
//// EE0 attern of tyical absences is:
/// Temoral shar $aves
// > 4z7 -i;e($ave comlexes
/// 4i#h amlitude #eneralized activity
/// )(. 4z7 -i;e($ave comlexes
//// The tyical EE0 attern of *est syndrome is:
// 4ysarrhythmia
?
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/// Focal slo$ $ave activity
/// > 4z7 -i;e($ave comlexes
/// 8nly shar $aves
//// The tyical EE0 attern of uvenile absence eilesy is:
/// Diffuse slo$ $ave activity
// 0eneralized bilateral synchronous >%)(@ 4z7 -i;e($ave comlexes7
/// 6o$ amlitude activity
/// 4ysarrhythmia
//// The tyical EE0 attern of atyical absence is the follo$in#:
/// Diffuse slo$ $ave activity// A%)(?%) 4z7 -i;e($ave comlexes
/// 4ysarrhythmia
/// Focal slo$ $ave activity
//// The simle artial seizures are the follo$in#% excet:
/// 9otor
/// Clonic
// "bsences
/// 3e#etative
//// The reserved consciousness is the si#n of:
/// 6ennox(0astaut syndrome
/// *est syndrome
/// "bsences
// -imle artial seizures
//// The follo$in# are not related to #eneralized seizures:
/// "brut onset/// Pro#ressive seizures
// Preserved consciousness
/// "mnesia of seizure eisode
//// The follo$in# are related to the tyical absences:
// "brut onset
/// The #radually onset
/// A%)(?%) 4z7 -i;e($ave comlexes
/// Duration of seizures >A seconds and more
>
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//// The follo$in# is related to atyical absences:
/// "brut onset
// The #radually onset
/// >(>%) 4z7 -i;e($ave comlexes
/// Duration of seizures less than ,)seconds//// "ura can be:
/// 3isual
/// "uditory
/// 0ustatory
// "ll of them
//// Febrile seizure can occur:
// 'efore . years of a#e
/// .(,? years of a#e/// Puberty eriod
/// + to ?A years of a#e
//// -imle febrile seizure is not characterized by:
/// Duration B ,) minutes
// Reetition rate ?@ hours
/// Rare eisodes
/// "bsence of neurolo#ical symtoms after seizure
//// -imle febrile seizure is characterized by:
/// Duration ,) minutes
/// Focal aroxysm
/// Reetition rate ?@ hours
// Rare eisodes
//// Comlex febrile seizure is characterized by:
/// Duration B ,) minutes// Duration ,) minutes
/// Rare eisodes
/// Preserved consciousness in ictal eriod
//// Comlex febrile seizure is not characterized by:
// Rare eisodes
/// Duration ,) minutes
/// Reetition rate ?@ hours
/// Focal aroxysms
@
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//// "bsence seizure is:
/// "dversive seizure
/// Tonic seizure
/// Tonic(clonic seizure
// *est syndrome
//// "bsence seizure is not:
/// *est syndrome
// "dversive seizure
/// 6ennox(0astaut syndrome
/// -alaami seizures
//// Partial seizures can not be:
/// "dversive/// Tonic
/// Clonic
// *est syndrome
//// &nfantile sasms can occur:
/// > years of a#e
// >(1 months of a#e
/// &n neonatal eriod
//// &n uberty eriod
//// &nfantile sasms first $as described by:
/// 6ennox
/// 0astaut
// *est
/// ac;son
//// &nfantile sasm is defined as:
// Flexor(extensor sasms% mental retardation% hysarrhythmia on EE0
/// !ormal sychomotor develoment
/// -imle artial seizures
/// > 4z7 -i;e($ave activity on EE07
//// &nfantile sasms occur durin# the follo$in# conditions:
// -evere dama#e of C!-
/// 4yerthermia
/// &ntoxication
/// &nfection
)
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//// The recommended daily dosa#e of "CT4 for infantile sasm is:
/// ?A(>A unit
// 2A(,AA unit
/// 6ess than ,A unit
/// ) unit
//// The initial dosa#e of Prednisolone used for the treatment of infantile sasms is:
// ? m#/;#/d
/// A%) m#/;#/d
/// @ m#/;#/d
/// , m#/;#/d
//// The reduction of dosa#e of hormones in infantile sasms can be started:
/// *hen seizures become rare
// "fter cessation of seizures and hysarrhythmia on EE0
/// "fter imrovin# the sychomotor functionin#
/// "fter imrovin# the EE0 atterns
//// The dru# used in infantile sasms is:
// 4i#h doses of Piridoxine
/// 3itamin C
/// 3itamin D
/// 3itamin E
7//// The dru# not recommended for the treatment of infantile sasms is:
/// "CT4
/// &mmuno#lobulin
/// Piridoxine
// !ivalin
//// The dru# not recommended for the treatment of infantile sasms is:
/// !ivalin
// "CT4
/// Phenobarbital
///Piracetam
@, The comlex absence is not characterized by:
// "brut onset
/// Tonic comonent
/// Clonic comonent/// 0radual onset
.
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@? The simle absence is characterized by:
/// Fixation of #aze
// 3e#etative manifestation
/// "brut be#innin# and finishin#
/// d7 "bsolutely imaired consciousness
@> The simle absence is characterized by:
/// 0radual be#innin# and finishin# of aroxysms
/// Duration of seizure more t)han >A seconds
/// 9ioclonic comonent
// "brut cessation of motor activity
@@ The comlex artial seizure is characterized by:
/// Preserved consciousness
// Comlete or incomlete loss of consciousness/// 8nly motor comonent
/// "bsence of amnesia
@) 0eneralized tonic(clonic seizure is not characterized by:
/// 6oss of consciousness
/// "brut droin#
// Preserved consciousness
/// Tonic(clonic sta#e
@. !eonatal seizure occurred:
/// "fter , month of a#e
// ,(@ $ee;s of a#e
/// ?(@ months of a#e
/// "fter , years of a#e
@1 The cause of neonatal seizures are the follo$in#% excet:
/// 4yoxic(ischemic dama#e of C!-
// 8enheims myotonia/// &ntracranial hemmora#e
/// 9icroelements imbalance
@2 The cause of neonatal seizures are the follo$in#% excet:
/// &ntrauterinal infections
/// Piridoxine deficiency
// 3erdin#(4offmans sinal amyotrohy
/// &nherited metabolic disorders
1
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@5 !eonatal seizures can not be:
/// !ot manifested
/// Tonic
/// 9ioclonic
// "tonic
)A The reason of seizure syndrome caused by calcium deficiency are the follo$in#% excet:
/// The lo$ level of arathyroid hormone
// 4i#h level of bilirubin in blood
/// Disorder of vitamin D metabolism
/// 4yoma#nemia
), Eiletic status is defined as:
/// !ormal consciousness bet$een aroxysms
// Permanent aroxysms $ith duration of >A minutes or more/// "mnesia of seizure eisode
/// "rterial hyertonia
)? Eiletic status is not defined as:
/// Permanent aroxysms $ith duration of >A minutes or more
/// "mnesia of seizure eisode
// !ormal consciousness bet$een aroxysms
/// Eiletic atterns on EE0
//// The mana#ement of eiletic status does not imly:
/// 9onitorin# of vital functions
/// +r#ent anticonvulsive theray
/// Exress dia#nosis
// Per os administration of anticonvulsants
//// The follo$in# dru#s are recommended for the mana#ement of eiletic status:
// Diazeam or 6orazeam
/// -uccinimedes/// 6amotri#ine
/// 3i#abatrine
//// The dru# not recommended for the mana#ement of eiletic status is:
/// Phenobarbitale ,Am#/;#
/// Pentobarbitale .(2 m#/;#
/// 6idocaine ,(? m#/;#
// 6amotri#ine ?%) m#
2
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//// The dru# not recommended for the mana#ement of eiletic status is:
/// Diazeame or 6orazeam
/// Phenitoine
// 3i#abatrine
/// Phenobarbitale
//// The dru# administration in eilesy lasts for:
/// ,(> months
/// >(. months
// >() years
/// , years
)2//// The anticonvulsive theray is conducted:
/// "s a short eriod course for ,(? years
// Permanently% $ith administration of dru# for several years/// 'y usin# of broad sectrum anticonvulsant for . months
/// 'y administration of dru# for , year
)5//// The anticonvulsive theray is recommended to start by:
// -in#le anticonvulsant
/// T$o or three anticonvulsants
/// 3itamins
/// "ntibiotics
.A //// The dru#s of first choice in febrile seizure are the follo$in#:
/// Phenobarbitale
// 3alroic acid
/// 6amotri#ine
/// Toiramate
.,//// The dru# recommended to use for the treatment of artial seizures is:
// Carbamazeine
/// -uccinimide/// ' #rou vitamins
/// 4ormone
.?//// The dru# not recommended to use for the treatment of artial seizures is:
/// Carbamazeine
/// 3alroic acid
// -uccinimnides
/// Phenobarbitale
5
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.> The dru# recommended to use for the treatment of absence seizures is:
/// Carbamazeine
/// Phenobarbitale
// 3alroic acid
/// Phenitoine
.@ The dru# not recommended to use for the treatment of is:
/// -uccinimides
/// Clonazeame
/// 3alroic acid
// Carbamazeine
.) The follo$in# dru#s are used for the treatment of 6ennox(0astou syndrome:
// 6amotri#ine
/// Phenobarbitale/// Piridoxyne
/// "ntibiotics
.. The dru#s not used for the treatment of 6ennox(0astaut syndrome is:
/// 9onotheray $ith 6amictal
// Phenobarbitale
/// 3alroic acid
/// Clonazeame
.1 The anticonvulsants of ne$ #eneration is:
/// Phelbatole
/// 8xcarbamazeine
/// "cetazolamide
// Phenobarbitale
//// The basic anticonvulsant is not:
// 0abaentine
/// Phenobarbitale/// Carbamazeine
/// 3alroic acid
.5 The basic anticonvulsant is :
/// Toiramate
/// 3a#abatrine
// Carbamazeine
/// 9idazolam
,A
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1A 'esides the dru#s the aroach also used for the treatment of intractable eilesy is:
/// Physiotheray
// eto#enic diet
/// 8ccuational theray
/// Psychotheray
//// For the treatment of intractable eilesy is used:
/// Electrohoresis
/// 8ccuational theray
// -timulation of 3a#us
/// Psychotheray
1? For the treatment of intractable eilesy is used:
// !eurosur#ical oerations on brain
/// 8ccuational theray/// Psychotheray
/// Electrohoresis
1> Porencehaly often causes the follo$in# form of cerebral alsy:
/// -astic dile#ia
/// -astic tetraaresis
// 4emiaretic form
/// 4yer;inetic form
1@ "lasia of basal #an#lia and nuclear aundice cause the follo$in# form of cerebral alsy:
// 4yer;inetic form
/// 4emiaretic form
/// -astic tetraaresis
/// -astic dile#ia
1) 6ittly disease is resented as the follo$in# form of cerebral alsy:
/// -astic tetraaresis
// -astic dile#ia/// 4yer;inetic form
/// "tonic(astatic form
1. The disorders of extremities in sastic dile#ia are exressed:
/// 8n one side
// Disorder is symmetric
/// +er extremities are affected more
/// +er and lo$er extremities are affected on one side
//// &n cerebral alsy the symmetric reflex from head to trun; and extremities is:
,,
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/// &ncreased
// &s increased in neonate eriod and inhibited in re(school eriod
/// &s absent
/// &s al$ays chan#ed
12 &n cerebral alsy of aralitic form the muscle atrohy is exressed by the follo$in# $ays:
/// &s absent
/// 8ccurred only in later residual eriod
/// 8ccurred only durin# muscle hyotonia
// &s mildly exressed and has diffuse manifestation
15 The revalence of cerebral alsy er ,AAA is:
/// A%) and less
// ,%) and ?
/// ) and more/// ,A and more
2A &n cerebral alsy of sastic tetrale#ia form is exressed:
// Tetraaresis more affected the uer extremities
/// 4emile#ia on one side and hemiaresis on other side
/// Tetraaresis occurred only in severe cases
/// Disorders only in lo$er extremities
2, &n cerebral alsy of tetrale#ia form the muscle tone is:
/// "l$ays decreased
/// -li#htly increased
// &s increased si#nificantly
/// &s increased only in later residual eriod
?//// &n cerebral alsy of sastic dile#ia form are affected:
/// 8nly internal casule on one side
/// Cerebral eduncles
// Cerebral hemisheres% esecially cortex/// 'rainstem
2> &n cerebral alsy of sastic tetrale#ic form atient start $al;in#:
// &n very rare cases
/// 8n the third year of life
/// "fter )(. year of a#e
/// 8nly in the case of intact mental field
@//// &n cerebral alsy of hyer;inetic form is affected the follo$in# structures:
,?
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/// The uer arts of brainstem
/// The cerebral cortex
/// Cerebellum
// -trioaidal system
//// 4earin# more frequently is imaired durin# the follo$in# forms of cerebral alsy :
/// -astic dile#ia
/// -astic tetrale#ia
/// 4emiaretic form
// 4yer;inetic form occurrin# after nuclear aundice
2.//// The sychiatric disorders in cerebral alsy of atonic(astatic form occur:
/// never occur
// in case of frontal lobes disorders
/// in case of cerebellum disorders/// in case of disorders of cerebellum ath$ays
1 //// The muscle tone in cerebral alsy of atonic(astatic form is chan#ed by the follo$in# $ays:
/// -astic
/// Ri#id
// 4yotonia
/// Distonia
22 The course of cerebral alsy is:
/// Pro#redient
// Re#redient in case of adequate treatment
/// Re#redient only in early eriod of life
/// Pro#redient only in reschool a#e
25 The ro#nosis of cerebral alsy deends on the follo$in# issues:
/// 8nly eriod of dama#e
/// 8nly eriod of startin# of treatment
/// The somatic condition of child// Time and de#ree of dama#e and eriod of startin# of treatment
5A Contractures and deformation in cerebral alsy :
// 9ay increase to#ether $ith a#e
/// &s stable
/// 9ay decrease to#ether $ith a#e
/// 9ay increase only in case of sastic tetrale#ia
5, The eilesy of childhood is often caused by:
/// &nfection and hyotrohy
// 0enetic redisosition and ne#ative exo#enous factors
,>
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/// 9inimal cerebral dysfunction
/// Defects of carin#
//// The bloody saliva in eiletic seizure is the result of:
/// !asal bleedin#
/// Pharyn#eal bleedin#/// Disorder of blood a##re#ation system
// Dama#e of ton#ue and chee;s
5> The reventive vaccination in case of eilesy can be conducted only in the follo$in#
cases:
/// "fter ,(? years from the last seizure
// "fter . months from the last seizure to#ether $ith increasin# the anticonvulsant
theray
/// "fter ? years from the last seizure
/// 8nly $hen the EE0 atterns are normal
5@ The #radual decreasin# of antieiletic treatment is ossible in the follo$in# cases:
/// "fter ,(? years from the last seizure
/// *hen there is imrovement on EE0
/// 8nly in uberty eriod
// "fter >() years from the last seizure and in case of normal EE0 atterns
5) The etiolo#ical factors of neurosis is:
/// -omatic condition
/// "#e
/// Premorbid bac;#round
// Psychotrauma
5. &n re( and uberty eriod fear is often accomanied by:
/// 4allucinations
/// -eizures
// Exressed ve#etative reactions
/// &nvoluntary urination
//// 6o#oneurosis is often occurred in the follo$in# eriods:
// 'efore ) years of a#e
/// early school eriod
/// 6ate school eriod
/// Puberty eriod
52 The neuro#enic tics often occur in the follo$in# eriods:
/// 'efore > years
,@
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/// >() years
// )(,? years
/// "fter ,. years
55 The main aroach in the treatment of neurosis is:
/// Physiotheray/// Dru#s
/// Comlex aroach containin# dru#s and hysiotheray
// Psychotheray
,AA The inherited unconditioned automatism reflexes are:
/// The reflex of suc;in#
/// The reflex of automotive climbin#
/// The reflex of #rasin#
// "ll of them
//// The follo$in# are not the reflexes of oral automatisms:
/// The reflex of suc;in#
/// The reflex of see;in#
/// The reflex of Gelehant trun;H
// The reflex of defense
,A? The follo$in# are not the inherited myelencehalic reflexes of neonatal eriod:
/// The tonic symmetric nec; reflex
/// The tonic asymmetric nec; reflex <9a#nus(leins=
// The reflex of #rasin#
/// The symmetric and asymmetric tonic reflexes
//// The suc;in# reflex doesnt disaear:
/// +ntil ? months
/// +ntil . months
// +ntil ,2 months
/// +ntil ,? months
//// The #rasin# <Robisons= reflex in normal neonates disaears:
/// "t ? months of a#e
/// "t >(@ months of a#e
// "t , year of a#e
/// "fter , year
,A) The diffuse decrease of muscle tone in neonates can occur in the follo$in# cases:
/// Dama#e of cerebellum
/// *erdni#(4offmanns sinal amyotrohy
/// 8enheims myotonia
// Cerebral alsy of hemiaretic form,A. The feature of *erdni#(4offmanns amyotrohy is:
,)
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/// Disaearance of ;nee reflex
/// The ro#redient course of disease
/// Diffuse hyotonia
// 4yereflexia
,A1 The inherited unconditioned reflexes of neonates disaears at the follo$in# a#es:
/// 'efore , month
/// 'efore ,(? months
// 'efore >() moths
/// "fter , years
//// 6andau inherited reflex in neonates disaears:
/// "t , month of a#e
/// "t ,(> months of a#e
/// "t . months of a#e// &t never disaears
,A5 The follo$in# are not T8RC4 infection:
// Rubella infection
/// Cytome#alovirus infection
/// 4eres -imlex infection
/// Toxolasmosis
//// &n con#enital rubella infection microcehaly occurs:
/// &n ,AAI of cases
// &n ,A(?AI of cases
/// &n )AI and more
/// &t never occurs
,,, 4ydrocehaly% enohthalmy% sastic aresis% eiletic seizures% chorioretinitis associated
$ith fetal heatitis in neonates are the features of:
/// Rubella infection
// Toxolasmosis
/// Cytome#alovirus infection/// 6isteriosis
//// The most frequent form of develomental defect of brain is:
/// 9yelocele
/// 9acrocehaly
// 9icrocehaly
/// Dysostosis of s;ull and face
,,> 9icrocehaly is divided by the follo$in# $ays:
// Primary and secondary
/// Con#enital and acquired
,.
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/// Postinfectious and osttraumatic
/// Diffuse and focal
,,@ &n microcehaly occur the follo$in# rocesses:
/// The brain is si#nificantly small in size comared to s;ull
// The small size s;ull is correlated to reduced size brain/// 8nly brain art of s;ull is reduced comared to face art
/// The face and brain art of s;ull are roortional
,,) 9orholo#ical determinant of microcehaly is:
/// The roortionally reduction of the $hole brain
// The severe defects of develoment
/// 8nly macro( and micro#yria
/// 8nly disorder of s;ull
//// The secondary microcehaly occurs in:
/// Prenatal eriod
/// 8nly ostnatal eriod
// Perinatal eriod and in the first months of life
/// "ny a#e
,,1 The sizes of s;ull in microcehaly at the moment of birth is:
/// "l$ays reduced by >(@ cm
/// Reduced by ) cm and more
/// 9ay be increased
// 9ay be normal
//// The motor develoment in microcehaly is:
/// "l$ays normal
// &s al$ays delayed
/// &s delayed in case of combination $ith cerebral alsy
/// &s delayed only in rimary microcehaly
5 The ro#nosis of microcehaly can be redicted by:
/// The rate of increasin# of s;ull sizes
/// -;ull sizes
// The level of mental retardation
/// The time of adequate treatment
The increasin# of intracranial ressure is the si#n of:
// Craniostenosis
/// Primary microcehaly
/// -econdary microcehaly/// 9icrocehaly associated $ith eilesy
,1
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,?, The sontaneous comensation of con#enital hydrocehaly may occur:
/// 'y imrovin# the cerebrosinal fluid absortion
// Durin# the atrohy of choroidal lexuses and by reducin# the cerebrosinal fluid
roduction
/// 'y movin# a$ay of s;ull sutures
/// Durin# the liquorrhea
,?? 4ydrocehaly:
/// &s al$ays secondary and is the result of brain atrohy
/// &s al$ays rimary
// 9ay be rimary or secondary
/// "l$ays occurs durin# orencehaly
//// &n con#enital hydrocehaly the follo$in# chan#es occur on the fundus of eye:
/// Con#estion/// "n#ioathy
// The decoloration of otic dis;
/// The neuritis of otic nerve
,?@ The non(comensatin# hydrocehaly occurs:
/// 8nly durin# the viral infection
/// 8nly durin# the enterovirus infection
/// 8nly durin# the neuroinfection
// Durin# the C!- trauma or infection
//// The sur#ical treatment of hydorcehaly is recommended in order to:
/// &mrove venous circulation
/// !ormalize the cerebral hemodinamics
// 9a;e the collateral circulation of cerebrosinal fluid
/// Reduce the secretion of cerebrosinal fluid
//// The erinatal eriod is defined as the eriod:
/// From the first movement of fetus to moment of birth// From ?2 $ee;s of #estation to first 1 days of life
/// From ?2 $ee;s of #estation to moment of birth
/// The first 1 days of life
,?1 The traumatic inury of sinal cord in neonates often occurs in the follo$in# se#ments:
/// The uer cervical
// &ntumenscentia cervicalis
/// Thoracal
/// &ntumenscentia lumbaris
//// The obstetrical aralysis of hand of total tye occurs durin# the dama#e of the follo$in#
se#ments:
,2
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/// C)(C.
/// C1(C2
/// D,(D?
// C)(D,
//// The obstetrical aralysis of hand of roximal tye occurs durin# the dama#e of the follo$in#
se#ments:
// C)(C.
/// C1(C2
/// D,(D?
/// C)(D,
,>A The obstetrical aralysis of hand of distal tye < Deerin(lum;e= occurs durin# the
dama#e of the follo$in# se#ments:
/// C)(C.
/// C1(C2// D,(D?
/// C)(D,
131 The structural defect of the brain in cerebral alsy affects the follo$in# functions:
/// 8nly motor develoment
/// 8nly seech develoment
// The develoment of the $hole brain
/// &t doesnJt affect on brain develoment
//// The cerebral alsy in relation to hyoxic(ischemic encehaloathy is:
/// &ndeendent disease
/// 8rdinary outcome
/// The have no relation
// 8ne ossible variant of outcome
,>> &n cerebral alsy of sastic dile#ia form occurs:
/// +er sastic araaresis
/// -astic hemiaresis
// 6o$er araaresis/// Tetraaresis
//// The sastic tetraaresis form of cerebral alsy is defined as:
/// Tetraaresis $ith mild mental retardation
// Tetraaresis $ith severe mental retardation
/// The eriheral aresis in hands and sastic aresis in le#s
/// The sastic aresis in hands and eriheral aresis in le#s
,>) &n cerebral alsy of atonic(astatic form are imaired the follo$in# structures:
/// 'oth internal casules
/// 'asal #an#lias
,5
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// Cerebellum
/// The brain hemisheres
//// The ossible reason of traumatic inury of facial nerve in neonates is not:
/// Face resentation
/// Difficult labor /// The fractures of temoral bone
// -ubarachnoidal hemmora#e
//// The reason of sinal cord trauma can be:
// Pelvic resentation
/// Cross birth
/// !ormal cehalic resentation
/// 'ro$ resentation
//// The sinal inury in neonates occurs in the follo$in# cases:
/// &ntrauterine hyoxia
/// !atal eriod ashyxia
/// !atal eriod infection
// !atal eriod trauma
//// The severe dama#e of C!- can be defined by "#arJs scale by the follo$in# scores:
/// A(,
// ,(@
/// )(.
/// .(1
,@A The moderate dama#e of C!- can be defined by "#arJs scale by the follo$in# scores:
/// A(,
/// ,(@
// )(.
/// .(1
//// The mild dama#e of C!- can be defined by "#arJs scale by the follo$in# scores:
/// A(,
/// ,(@
/// )(.
// .(1
,@? The etiolo#ical factor of hyoxic(ischemic encehaloathy is not:
/// 4yoxia
/// Trauma
/// Toxic rocess// Con#enital factor
?A
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//// The #ood ro#nosis has the follo$in# form of hydrocehaly:
/// 8cclusive
// 8en% $ith slo$ rate of ro#ression
/// 8ccurrin# durin# intrauterine infections
/// 8ccurrin# after delivery trauma
,@@ The mental retardation in severe hydrocehaly is resented by the follo$in# $ays:
/// -evere retardation
// Ran#in# from mild form to idiotia
/// 8ften in normal ran#e
/// -li#htly exressed retardation
//// The reason of 0reheJs symtom in hydrocehaly is the follo$in#:
/// The irritation of cilliosinal symathic nucleus
/// Defects of vision/// &ncreasin# of intraorbital ressure
// Deformation of orbitas and exohthalmus
,@. The head circumference starts to increase in the follo$in# eriods:
// The first month of life
/// >() months of life
/// "fter . months of life
/// "t , year of life
//// The follo$in# conditions occur in severe hydrocehaly:
/// 8tical nerve neuritis
// 4ydroanencehaly
/// Pyramidal deficiency
/// 6esion of cerebellum and its connections
//// The most frequent reason of con#enital hydrocehaly is:
/// Porencehaly
/// "#enesia of corus callosum
/// "lasia of cerebellum
// 8cclusion of cerebral aquaductus
,@5 The child $ith con#enital hydrocehaly may has:
// !ormal or sli#htly increased head circumference
/// &ncreased head circumference by @() cm
/// Reduced circumference of head
/// &ncreased head circumference by )(2 cm
//// The most frequent form of hydrocehaly is:
?,
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/// Traumatic
/// Toxic
/// 4yoxic
// Con#enital
,), Dilatation of subarachnoid saces occurs in the follo$in# forms of hydrocehaly:
/// &nternal
// External
/// 'asal
/// Convexital
//// Dilatation of brain ventricles and subarachnoid saces occurs in the follo$in# forms of
hydrocehaly:
/// &nternal
/// External
// 8en
/// Convexital
,)> Dilatation of only ventricles occurs in the follo$in# forms of hydrocehaly:
// &nternal
/// External
/// 'asal
/// Convexital
//// 'y the eriod of develoment hydrocehaly can be divided in the follo$in# forms:
/// Primary and secondary
// Con#enital and acquired
/// Pre( and ostnatal
/// Early < before . month= and late < after . month=
//// Passive hydrocehaly occurs in the follo$in# cases:
// 'rain atrohy or ro#ressive sclerosis of brain
/// Disorder of cerebrosinal fluid absortion
/// 3enous stasis
/// Excessive roduction of cerebrosinal fluid
,). The basic si#n of hydrocehaly is:
/// &ncreasin# of intracranial ressure
// &mbalance bet$een the roduction and absortion of cerebrosinal fluid
/// Dilatation of ventrical system and cerebrosinal circulation $ays
/// &ncreased head circumference
//// The clinical feature of hyertensive(occlusive crisis imlies:
??
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// Common cerebral hyertensive si#ns and focal neurolo#ical symtoms
/// 9enin#eal si#ns
///0eneralized seizures
/// 8nly focal si#ns
//// The reason of hyertensive(occlusive crisis is:
/// Excessive roduction of cerebrosinal fluid
// 8cclusion of cerebrosinal fluid circulation $ays
/// -ubarachnoid hemmorra#e
/// Difficulty of cerebrosinal fluid absortion
//// The K(ray si#ns of hydrocehaly are not:
/// "trohy of s;ull bones
/// Flattenin# of s;ull base bones
/// Dilatation of sella turcica// Existence of intracranial calcification
4ead of !eurolo#ical Direction
of Pediatric Deartment of
Tbilisi -tate 9edical +niversity%
Professor !ana 0eladze