RJPO Paediatric
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Herlien H. Megawe Dept. of Anesthesiology & Reanimation
Airlangga University School of MedicineSurabaya
No matter what time it is, wake me up, even if its in the middle of a cabinet meeting
Ronald Reagan
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I. BASIC PRINCIPLES :
a. Urgency of childs problem
b. Ordering of priorities (e.g. epidural hematoma, closed femoral fracture)
c. Critically ill and profound hypotensive condition, an immediate operationneeded resuscitation & anesthesia are provided simultaneously
d. Establish : - clear airway : A- provide ventilation : B- support hemodynamics : C
e. Hypnosis & analgesia, as condition will allow
f. Titrated doses of : - Hypnotics (Benzodiazepine, for amnesia Ketamine)
- Opioids for pain- Neuromuscular blocking agent for immobility
g. As patient stabilizes, inhalation agent are added as tolerated
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II. RAPID CLINICAL ASSESSMENT OF THE SERIOUSLY ILL INFANT/CHILD
A = AIRWAY : - Obstruction
- Partial- Total
B = BREATHING : - Respiratory rate
- Flare- Recession : - sternal- intercostal- subcostal
- Silent chest C = CIRCULATION : - Pulse : volume
- Blood pressure- Capillary refill time- EKG- Skin colour : - dry, red, warm
- greyish, cold, wet, clammy
D = DISABILITY : - Unresponsive to voice/pain- Posture- Pupils : size/reaction- Conscious level- Convulsions
E = EXPOSSURE : - Rash - Purpura- Swelling - Urticaria
- Fever - Angio-edema
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ASSESSMENT OF PROGRESS AND DETECTION OF
DETERIORATION :Re-assessment of ABCDE at frequent intervals
LEVEL OF CONSCIOUSNESS SHOULD BE RECORDED USING
THE AVPU SCALE : A : Alert V : Responds to voice
* P : Responds to pain (GCS < 8)U : Unresponsive
* Pinching a digit, pulling frontal hair
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III. PATHWAYS LEADING TO CARDIO. RESPIRATORYARREST :
FLUIDLOSS
BLOOD LOSS
GASTROENTERITIS
BURN
LOSSMALDISTRIBUTION
SEPTIC SHOCK
CARDIAC DISEASE
ANAPHYLAXIS
RESPIRATORYDISTRESS
FOREIGN BODY
CROUP
ASTHMA
RESPIRATORYDEPRESSION
CONVULSIONS
RAISED ICP
POISONING
CIRCULATORY
FAILURE
RESPIRATORY
FAILURE
CARDIAC ARREST
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Indication for intubation and ventilation :
Inadequate oxygenation via bag-and-mask technique
Prolonged ventilation required
Flail chest
Inhalational burn injury
Shock
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VASCULAR ACCESSPreferred options :
A. Intra venousB. Intra osseous
A. Preferable via the superior v. cava Via the inferior v. cava takes longer to reach the heart Via the periph. route fluid flush
First priority : accurate safety rapidlyB. Intra osseous :
- Easy & safe- Reach the heart = periph. ven. access- Also in older age & adults
C. Tracheal :Third placeFor first drug adrenaline
D. Intracardiac : not recommended
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IV. RESUSCITATION BEFORE ANESTHESIA
Weight in kg = 2 (age in years + 4)
Estimated blood volume = 80 ml/kg body weight
Assess
response
Assessresponse
F lu id vo lum e and typ e An initial fluid bolus of 20 ml/kg is given as fast
as possible
This should be repeated after assessment if thereis no improvement in vital signs
The most common mistake in the treatment ofhypovolaemic shocked children is failure to giveenough fluid
Crystalloid20 ml/kg
Colloid20 ml/kg
Blood
UrgentSurgical opinion
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V. OTHER VITAL PROCEDURES CARRIED OUT BEFORE
RESUSCITATION AND ANESTHESIA : HISTORY :
Vomit (quantity & quality) Last urination/defaecation) Bloody stool/profuse
BLOOD TESTS (taken immediately after venous access)
NASOGASTRIC TUBE PLACEMENT
RADIOGRAPHS
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VI. PLAN1. Weight (kg) = 2 (year + 4) kg2. Estimated Blood Volume : 80 ml/kg
3. Internal diameter endotracheal tube : Year + 4 = one size smaller, actual size , one size bigger, e.g. :
4 4 - 4,5 - 54. Fluid bolus : 20mg/kg
- Crystalloid
(Colloid)- Blood5. Defibrill. Dose I : 2 Joule/kg
2 Joule/kg in 90 seconds4 Joule/kg
Defibrill. Dose II : 4 Joule/kg4 Joule/kg in 90 seconds4 Joule/kg
6. Between defibrill. dose I and II : Adrenalin dose I : 1 ml/10.000 sol. (10 Ug/kg) Adrenalin dose II : 1 ml/1000 sol. (100 Ug/kg)
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VII. PREMEDICATION, ANXIETY & FEAR
I.V. anticholinergics benefits :1. Maintenance of cardiac output by increasing heart rate2. Prevention of reflex bradycardia :
Airway manipulation Scoline/halothane
ANXIETY & FEAR
Calm appearance Reassuring Of the anesthesiologist is of greatbenefit for patients & parents
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VIII. URGENT SITUATION WITH COMPROMISED AIRWAY
Foreign body aspiration Epiglottitis Croup Bleeding tonsil Facial/laryngeal trauma
Compromised airway & struggle during intubation attempt,choices are : Awake intubation Volatile agent (Sevoflurane/Halothane) in oxygen with gentle
cricoid pressureThis approach is favoured = The patient continues
breathing ! Slight head down position : pulmonary aspiration is less likely
when patient regurgigates
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FULL STOMACH
Postpone surgery for > 4 hours
Reduce the mean gastric residual volume by 50% (does notguarantee empty)
If there is no specific airway for difficult intubation Anesthesia of choice : rapid sequence of induction
Pre-oxygenation + sulfas atropine Rapid induction agent : - Ketamine
- Propofol- Thiopenthal
Muscle relaxant = Recuronium 1,2 mg/kg The smaller the child, the more rapid he will desaturate (< FRC) Newborns can become hypoxic in less than 1 minute
Difficult to pre-oxygenate & denitrogenate a struggling toddler
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HYPOVOLEMIA
Ongoing bloodloss
Pending blood availability
Type specific un-crossmatched low incidence oftransfusion reaction
Ketamine : induction agent of choice in small
dosis 1 2 mg/kg I.V. (within one minute) Atropine 0,02 mg/kg or scopolamine 0,01 mg/kg
administered before
5% Albumine
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IX. PAIN MANAGEMENT
Injured children are in pain on arrival Potentially hypovolemic : Fentanyl preferable
Must be titrated in small increments (0,5 1,0 Ug/kg) to avoid
chestwall or glottic rigidity
Unstable & neurologic dysfunction : opioids with caution
Regional nerve blocks = - femoral nerve
- axillary
Providing analgesia
As a primary anesthetic
(avoid risks of general anesth. = aspiration)
Supplement for postop analgesia
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CAUTIONS
Titrate sedative in small increments to avoid lossof airway reflexes
Appear alert & Sedation sleepy After the block : painfull stimuli is removed
Close communication with surgeons = ability toperform sensory and motor examinations
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X. SPECIAL PROBLEMS & MANAGEMENT A. AIRWAY
Acute airway obstruction : Inspiratory stridor Tachypnoea Sternal & intercostal retractions Agitation (due to hypoxia) Cyanosis Tachycardia
Few of these symptoms & signs are manifested, yet their
condition may rapidly become life-threateningInitial respons :O2 & calm, to prevent dynamic collapse of the airway associated with agitation
- Not cyanotic- Sable vital signs
- Upright position- Supine position further airway obstruction
Blood gas analysis is not vitalPaO2 = 80 torr ; orPaO2 = 60 torr
In Contrast : pulse oxymetry Non-invasive Immediate & continuous means for assessing oxygenation
Recommended as a modality in all airway emergencies
X-ray is helpful clarifying the cause of obstruction
does not alter the response of the anesthesiologist
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B. UPPER AIRWAY OBSTRUCTION1. Epiglotitis Skill
Knowledge Urgent diagnosis & treatment Additional signs : - drooling
- difficulty in swallowing Favoured approach worldwide : Endotracheal intubation
Avoid = inspection increase obstruction (dynamic airway collapse) Radiographic = - Only when stable- Skilled personnel- Adequate resuscitation equipment
In the operationg room : Calm, sitting on the lap o/t mother Induction overface : - Halothane
- Sevoflurane Looses consciousness supine Head up slightly
Essential for the anesthesiologist
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Intubation tehnique
Lifting the base of the tongue
Without touching the epiglottis
Exposure of the rimaglottidis
Partially obstructed orifice
0,5 mm ID smaller choosen sprayed beforehand
A stylet within the endotracheal tube Failure : - tracheostomy
- cricothyorotomy
Adequate sedation to prevent extubation
* Titrated opioids
- Breathe spontanneously
- ETT remains in place for 24 48 hours, until swelling decreased
- Extubation
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2. Foreign body aspiration History of choking
Cyanosis
A wheezing child : Not always asthmatic May be foreign body aspiration
Agitation : due to seriously underlying hypoxemia Radiographic examination
If the child is stable Helpful to localize & identify Mostly are not radiopaque
Hyperinflation Atelectasis Principle of anesthetic management = epiglottitis
While eating suspicious
(peanut, popcorn)
Clues of presence of foreign body
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C. LOWER AIRWAY OBSTRUCTION1. Bronchiolitis
Tachypnea Retractions Wheezing Hyperinflated chest & diffuse crepitations Progressive exhaustion hypercarbia respiratory failure (silent chest)
Focus of treatment = correct hypoxemia Pulse oxymetry : - degree of hypoxemia
- respons to therapy Nebulized mist Bronchodilators
Titrated I.V, fluids not able to drink Ribavirin : antiviral agentCaution : Particles tend to disk, obstructing the ventilator
No absolute P aCO2 value that dictates the course of action
Not proven beneficial
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2. Asthma, status asthmaticus
A cardinal feature : reversibility spontaneuous or with therapy Wheezing sinonymous with bronchospasm Predominant expir. wheezing With increasing fatique slight air movement wheezing no
longer audible
Management :a. Support oxygenationb. Reduce airway obstruction
c. Support ventilationd. Prevent complication (e.g. pneumothorax)e. Inhaled & I.V. drugs
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Anesthesia : Optimize oxygenation, control brochospasm Standard agents Ketamine : - Bronchodilating
- Hypersecretion Avoid histamine release agents : - Morphine
- Curare- Thiopental
Drying of secretions intra-operatively : - Atropine- Glycopyrrolate
May exacerbate mucous plugging post-operatively Wheezing during anesthesia mechanical problem :
- endobronchial intubation- plugging, kinking- cuff herniation
Mechanical ventilation is difficultHigh Airway Pressure: - air trapping
- pneumomediastinum- pneumothorax
Controlled mechanical ventilation: - Degree of hypercarbia is pernitted/accepted- Adequate oxygenation- Adequate cardiac output
No sciencetific data yet
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CIRCULATION :
1. Hypovolemia : Most common cause of shock Crystalloid solutions are effective
No scientific studies of superiority of colloid solutions Blood as soon as crossmatching is carried out, or
O neg.Type spec. uncrossmatched
Urgent
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XI. OPERATING ROOM MANAGEMENT
1. Acute blood loss Secure large bore venous access is of higher priority than arterial access Arterial catheters in :
Arterial blood gas for adequate ventilationFrequent blood sampling : metabolic derangements
Hemodynamic instabilityNeed to alter blood pressure rapidly
Central venous access, only when hemodynamic stability returns Body temperature, hypothermia :
Potentiates neuromuscular blockade
Exacerbates coagulopathy
2. Anesthesiologists vigilance continued : until care is transferred to the appropriate physicians and nurses
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