Cerebral Palsy Infographic- Cerebral Palsy: A Gude For Parents
Principles of Cerebral Palsy - Add …docshare01.docshare.tips/files/18793/187938777.pdf · ·...
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Principles of Cerebral Palsy
Mamoun Kremli
Associate Professor
Consultant Pediatric Orthopedics College of Medicine & King Khalid University Hospital
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cerebral palsy
Definition
None progressive disease of the CNS secondary to a perinatal insult, resulting
in varying degrees of motor milestone delay and dysfunction
Incidence 2 – 5 % of live births
2 per thousand children at school age
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Cerebral palsy
Classification
According to Type of motor dysfunction
Spastic 65 %
Athetoid 10 %
Ataxic 5 %
Mixed 12 %
Hypo tonic 1 %
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Cerebral palsy
Classification
According to Pattern of involvement
Monoplegia : one limb / rare
Diplegia : both LL >> UL / good intelligence / prematurity
Hemiplegia : unilateral usually UL > LL / 33 % seizures
50 % mentally retarded
Triplegia : rare / usually both LL + one UL
Quadriplegia : total body / often mentally retarded /
with seizures / severe hypoxia
Double hemiplegia : bilateral UL > LL
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Cerebral palsy
Spastic Diplegia
• The most common type
• Speech / intellect: normal – slightly impaired
• UL : gross motor OK
minor incoordination of fine motor skills
• LL : spastic :
hip: flexion, adduction, int. rotation
knee: flexor / extensor spasticity /or equal
ankle: equinus
foot: pes valgus
• Most walk independently by 4 years
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Cerebral palsy
Spastic Hemiplegia
• 30 % of all CP
• One side affection
upper > lower extremity
• 50 % mentally retarded
• 33 % seizures
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Cerebral palsy
Spastic Quadriplegia
• All four limbs involved – and trunk
• Often mentally retarded
• With seizures
• Most ( 80 % ) non walkers
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Cerebral palsy
Clinical Assessment
Goals of Physical Examination
• Determine grades of muscle strength and selective
control.
• Evaluate muscle tone and determine type.
• Evaluate degree of deformity / contracture at each joint.
• Assess linear, angular and torsional deformities of spine,
long bones, hands and feet.
• Appraise balance, equilibrium and standing / walking
posture.
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Cerebral palsy
Clinical Assessment
Hip Flexors
• Ilio-psoas
( the main and most powerful )
• Sartorius
• Tensor fascia lata
• Rectus femoris
• Adductors
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Cerebral palsy
Clinical Assessment
Hip Flexors
Thomas test
• easy & simple, well
known
•problem : depends on how much is the
other hip flexed
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Cerebral palsy
Clinical Assessment
Hip Flexors
With fixed knee flexion, Thomas test should
be performed with knee outside at table
edge to prevent false positive results
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Cerebral palsy
Clinical Assessment
Hip Flexors
Staheli Test
Prone position Pelvis over table edge
More accurate
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Cerebral palsy
Clinical Assessment
Hip Flexors
Ely / Rectus Femoris
Test
• Well known
• Significance ?
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Cerebral palsy
Hip Adductors
Superficial layer
- Pectineus
- Adductor longus
- gracilis
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Cerebral palsy
Hip Adductors
Intermediate layer
- adductor brevis
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Cerebral palsy
Hip Adductors
Deep layer
- adductor Magnus
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Cerebral palsy
Clinical Assessment
Hip Adductors
Hip abduction / knees extended Hip abduction / knees flexed
The Gracilis
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Cerebral palsy
Hip Rotators
Internal Rotators
• Gluteus Medius
anterior portion is the main
internal rotator
• Gluteus Minimus
• Semitendinosis
• Adductors
• Tensor fascia lata
External Rotators
• Piriformis
• Gemilli
• Obturator internus
• Obturator externus
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Cerebral palsy
Clinical Assessment
Hip Rotation
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Cerebral palsy
Clinical Assessment
Knee Flexion
Hamstring Tightness
The Hamstring Test
Holt’s method
• Hip flexed 90 degrees
• Popliteal angle
degrees less than full extension
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Cerebral palsy
Clinical Assessment
Knee Flexion
( Hamstring Tightness )
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Cerebral palsy
Clinical Assessment
Ankle
Silfverskiold – 1923
Gastroc. Vs T. Achilles ( Soleus )
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Cerebral palsy
Clinical Assessment
Rotational Profile
• Foot propagation angle
• Femoral rotation
int. / extr. Rotation
• Tibial rotation
Foot – Thigh Angle
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Cerebral palsy
Clinical Assessment
Posture / Gait
• Lying
• Sitting
• Standing
• walking
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Cerebral palsy
Clinical Assessment
Upper Limb Superficial layer Middle layer Deep layer
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Cerebral palsy
Clinical Assessment
Upper Limb
• Elbow flexion
• Forearm pronation
• Wrist flexion
• Finger flexion
• Thumb in palm
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Cerebral palsy
Clinical Assessment
Upper Limb
Wrist dorsi-flexed Wrist palmar-flexed
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Cerebral palsy
Clinical Assessment
Upper Limb
Test for spastic wrist flexors
1. Flex wrist
2. Gradually extend
3. Palpate contracted /
spastic tendon
( FCU or FCR )
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Cerebral palsy
Clinical Assessment
Upper Limb
Test for contracted FDS / FDL
Extend and stabilize wrist and metacarpal-phalangeal joints
Stabilize DIP &
attempt to extend PIP
Stabilize PIP &
Attempt to extend DIP
FDS FDP
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Cerebral palsy
Goals of Management
(Treatment)
Achievable goals should be set
The child with CP becomes the adult with CP
Goals based on needs of adults
• Communication : verbal / nonverbal
• Activity of daily living (ADL)
feeding, dressing, toileting, bathing …
• Mobility
• walking
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Cerebral palsy
Goals of Management
(Treatment)
• Turn focus of parents from the disease to
the goal-oriented approach
needs time and a lot of discussion
• Physician and Physiotherapist must have the
same perspective
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Cerebral palsy
Types of Management
(Treatment)
• Physical therapy
• Orthotics
• Control of spasticity
• Orthopedic surgery
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Cerebral palsy
Spasticity
• Present in most patient with CP (65 % )
• When reduced patients may :
- perform integrated muscle movement
- develop muscle strength
- function at a higher level
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Cerebral palsy
Spasticity
• Approaches :
Selective dorsal rhizotomy
Intrathecal baclofen
Botulinum-A toxin
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Cerebral palsy
Selective Dorsal Rhizotomy
Cut 30 – 50 % of abnormal dorsal rootlets L2 - S1
• Followed by intensive physiotherapy
• Results encouraging
• May cause hyperlordosis / hip subluxation
• Best for : spastic diplegia, 4-8 yrs, no previous surgery,
no contractures, no extra pyramidal signs
• ? Not enough alone
• Orthopedic procedures obtain similar results
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Cerebral palsy
Baclofen
GABA agonist – inhibits release of excitatory
neurotransmitter at level of spinal cord
• Oral : mixed reports/ side effects/ not selective
• Continuous intrathecal – implantable pump
• Good results in releasing spasticity, and
improving function
• Complications of pump and catheter
• Needs specialized centers
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Cerebral palsy
Botulinum-A toxin
Acts at myo-neural junctions
Inhibits exocytosis of Acetylcholine
• Inject selected muscles at multiple sites
• Spasticity reduction may last up to 6 months
• Reversible , painless , minimal side effects
• Most patients still require lengthening for permanent correction
• Role : - Facilitates physiotherapy and mobilization
- Delays surgical management
- Trial to determine effects of specific
proposed surgical treatment
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Cerebral palsy
Types of Physical Therapy
• CNS modifications :
applying external stimuli / effectiveness not proven
e.g. - Neuro-developmental therapy
- Sensory integration therapy
- Patterning
- Conductive education
- Pressure point stimulation
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Cerebral palsy
Types of Physical Therapy
• Conventional PT :
works peripheral on muscles, tendons, and ligaments
- Active exercises
- Passive ROM exercises
- Passive stretching
- Bracing
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Cerebral palsy
Physical Therapy
Involve parents as much as possible
(even if they resist)
Do not raise false hopes
which could increase frustration
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Cerebral palsy
Physical Therapy
Post operative PT is essential to maximize benefit of surgical procedures
Goals :
- maintain / improve ROM
- regain pre-operative muscle strength
- maximize ambulation
- improve function – if possible
How frequent ? : as necessary
For how long ? : usually for a limited period
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Cerebral palsy
Physical Therapy
Maintenance Physical Therapy
Goals :
- balance and gait training
- maximize voluntary muscle usage
- prevent joint contractures
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Cerebral palsy
Physical Therapy
Maintenance Physical Therapy
Prevention of joint contracture – potential benefit
- by supervision of a daily range of joint motion program (can be applied by parents at home)
- for patients who lack the motor strength and voluntary control to maintain joint ROM
no real proof of benefit
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Cerebral palsy
Physical Therapy
Maintenance Physical Therapy
How often ?
For how long ?
some parents demand
regular physical therapy sessions
at least twice per week
for life !
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Cerebral palsy
Physical Therapy
There is no evidence that any type of physical
therapy can have a beneficial lasting effect on
motor function beyond early to middle
childhood (age 4-8 years).
Thomas S. Renshaw
( Lovell & Winter’s Pediatric Orthop.)
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Cerebral palsy
Physical Therapy
Children older than this no doubt benefit more
by devoting their time (and their families’ and
society’s resources) to the development of
communication, cognitive, and recreational
skills instead of endless therapy sessions.
Thomas S. Renshaw
( Lovell & Winter’s Pediatric Orthop.)
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Cerebral palsy
Orthotics
• Immobilization may cause atrophy
• Night splints :
- Do not prevent nor reduce deformity
- may cause irritation, pain or stimulate reflexes in spastic muscles and relaxes the weaker apponents – thus may increase deformity rather than reduce it !
• May be useful only in Athetoid
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Cerebral palsy
Orthotics
“ Experience in the management of cerebral-
palsied children with or without bracing leads to
the conclusion that a brace has very little place
in the management of cerebral palsy, provided
the child is given adequate physiotherapy and
the fixed deformity is prevented by good
combined physiotherapy and surgery “
Sharrard 1976
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Cerebral palsy
Orthotics
• In a review of 204 cerebral-palsied children Lee (1982) concluded :
Regardless of spastic hemiplegia, diplegia or athetosis, bracing versus non-bracing was ineffective in :
- preventing the need for surgery to correct
deformities
- preventing recurrences after surgery
- improving the gait.
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Cerebral palsy
Orthopedic Procedures
Usually multiple deformities at different joints
Knowledge of complex effects each deformity has
on other lower extremity joints
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Cerebral palsy
Orthopedic Procedures
primary deformity : needs treatment
compensatory deformity :
can improve without intervention
Distinguish between
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Cerebral palsy
Prerequisites for effective
surgery • Type : spastic
• Extent : hemiplegics / diplegics : good results
quadriplegics : minimal improvement
• Age : 3- 12 years
• IQ : good
• Good upper limb function : for walking
• Underlying muscle power : not weak
• Walker / non-walker :
surgery hardly changes state but improves gait
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Cerebral palsy
Goals of Treatment
Golden Roles
• Turn focus of parents from disease to the
goal-oriented approach
• Surgeons should spend time with patient at
least as much as operation time !
• Involve physiotherapists during all stages of
management – and
listen to their suggestions and comments
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Cerebral palsy
Ambulation
“ Factors “
• Balance
• Muscle control
• Strength
• Functional joint motion
• Sensory input
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Cerebral palsy
Goals of Treatment
Walking
• Community walker : free in community
• Household walker :
free at home / wheelchair outside
• Physiological walker :
walks only in PT dept. or at home between
parallel bars / with assistance
• Non-walker : wheelchair
independent / assistive transfer /dependant
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The Hip in Cerebral palsy
Indications For Orthop Surgery
• Prevent structural changes
usually early
• Improve function
usually later
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Cerebral palsy
Timing For Orthop Surgery
• For structural changes : Early
e.g. Hip subluxation , usually <5 years
• To improve function ( gait ) :
defer until walking ( independently / with aids )
until gait pattern develops and could be assessed
walking : 18 – 21 months in hemiplegia
3 – 4 years in spastic diplegia
• Optimum time of lower extremity surgery
5 – 7 years: can analyze and observe gait pattern
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Cerebral palsy
Timing For Orthop Surgery
Surgery is NOT “The Last Resort”
( not after all other methods failed !)
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Cerebral palsy
Timing For Orthop Surgery
Surgery should not be unduly staged one by one
( with each birthday )
?ETA ? Hams ? Psoas ? Rectus Femoris
Equinus Crouch Flexion Stiff Knee Ok
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Cerebral palsy
Orthop Surgery
Post Operative Management
• Analgesia :
- do not be mean !
- child should not cry to get the dose !
- sedation (valium) is not enough !
• Immobilization :
- minimal time / mobilize rapidly !
- can ambulate in cast one week post oper.
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Mamoun Kremli
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Cerebral palsy
Hip Deformities
• Dynamic deformities
• Tight adductors – scissoring
• Tight flexors – with pelvic inclination
compensatory knee / ankle/ trunk deformities
• Hip subluxation / dislocation :
not common in walking patients with diplegia
• Wind-swept deformity : in quadriplegics
• Internal rotation : in spastic hemi & diplegics
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Cerebral palsy
Hip Deformities
• Structural deformities :
- contractures of flexors, adductors, or
extensors - with ligs. And capsule
- acetabular dysplasia
- femoral ante version
- coxa valga
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Cerebral palsy
Knee Deformities
• Knee flexion deformity:
Tight Hamstrings
medial > lateral
Structural - Functional
• Stiff extended knee
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Cerebral palsy
Ankle & Foot Deformities
• Equinus
• Equino-valgus
• Equino-varus
• Pes cavus
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Cerebral palsy
Spastic Diplegia
• The most common type
• Speech / intellect: normal – slightly impaired
• UL : gross motor OK
minor incoordination of fine motor skills
• LL : spastic :
hip: flexion, adduction, int. rotation
knee: flexor / extensor spasticity /or equal
ankle: equinus
foot: pes valgus
• Most walk independently by 4 years
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Cerebral palsy
Spastic Diplegia
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Cerebral palsy
Spastic Diplegia - Hip Adductor Spasm
• Normally base of gate :
5 –10 cm ( heel to heel )
• Indications for surgery :
- scissoring
- hip abduction in supine < 10o
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Cerebral palsy
Spastic Diplegia - Hip Adductor Spasm
• Types of adductor “release” : ( open – not closed )
adductor longus tenotomy
sliding proximal end & suturing to brevis
adductor longus & gracilis myotomy
• Leave adductor brevis ( the major hip stabilizer )
• No anterior branch obturator neurectomy
(n. to adductor brevis)
• Release brevis -partially-if can not abduct 45o UGA
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Cerebral palsy
Spastic Diplegia
Hip Adductor Spasm
Post Operative after Adductor Release
Bilateral Long-leg cast connected with stick
Keeping each hip in 30o –40o abduction only
For 3 weeks
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Cerebral palsy
Spastic Diplegia - Hip Flexion Deformity
Indication for surgery
• Hip flexion deformity never decrease by
passive stretching, physiotherapy, orthotics,
sleeping prone … Eugene E. Bleck 1987
• Hip flexion deformity > 20o needs surgery
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Cerebral palsy
Spastic Diplegia - Hip Flexion Deformity
• Iliopsoas is the main and most powerful hip flexor
• Function of Iliopsoas not compensated by other flexors
• Without Iliopsoas :
- can not lift foot more than few inches from floor
- can not climb stairs
• Iliopsoas is the main contributor to FFD of hip
• Need to lengthen / preserving strength
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Cerebral palsy
Spastic Diplegia - Hip Flexion Deformity
What not to do !
• Yount’s fasciotomy of Tensor F. Lata – not enough
• Souter’s muscle sliding of Sartorius, Rectus Femoris,
and Tensor Fascia Lata – 66% recurrence
• Proximal Rectus Femoris tenotomy
- if it feels tight intraop. it is normally tight
- if hamstrings spastic :
overpowers the weekend Quad.in 6m – 1 year
• Myotomy of ant. Fibers of Gluteus Medius
very important pelvic stabilizer in stance phase
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Cerebral palsy
Spastic Diplegia - Hip Flexion Deformity
Iliopsoas tenotomy / lengthening / recession
• Tenotomy : not in ambulatory patients
in non-ambulatory OK
• Recession : good for subluxated hip
might weaken it too much
• Lengthening (z plasty) : best / easy
satisfactory in ambulating patients
no risk of too much weakening of flexion power
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Cerebral palsy
Spastic Diplegia - Hip Flexion
Deformity
• Iliopsoas tenotomy / lengthening / recession
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Cerebral palsy
Spastic Diplegia – The Knee
• Crouched gait :
- tight hamstrings : needs hamstring release
- could be secondary to weak triceps surae
• Type of surgery :
- tenotomy: too risky, causes knee extension
- transfer : results not as good as thought
- lengthening : best ( medial first , ? then lateral)
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Cerebral palsy
Spastic Diplegia – The Knee
• Lengthening hamstrings :
reduces hip extension power
increases hip flexion
• Add hip flexor ( Psoas ) lengthening if
concomitant hip flexion is present
• Hamstring transfer to femur instead of complete release
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Cerebral palsy
Spastic Diplegia – The Knee
Tight Hamstrings
• Surgery when popliteal angle 45o and above
• Start medially, ? then laterally if needed
• Lengthening better than release
• Add distal rectus femoris release
if concomitant cospasticity
• Add psoas lengthening
if preexisting hip contracture
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Cerebral palsy
Spastic Diplegia – The Knee
stiff knee gait
- mainly caused by Rectus Femoris
- treat by:
Rectus Femoris release:
proximal ?
distal – better
Distal transfer (medially to Sartorius)
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The Hip in Cerebral palsy
Effect of Knee Surgery on The Hip
Hip flexion Deformity increases after hamstring release
Better to transfer hamstring insertion to keep hip extended
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Cerebral palsy
Spastic Diplegia – The Foot & Ankle
Equinus deformity
( a little equinus is better than calcaneus)
• Velocity of muscle growth :
early : rapid ( birth – 4 years : doubles )
late : slow ( 4 years – maturity : doubles )
• ETA If done early returns more commonly
Recurrence rate : 75 % if done at 2 years
25 % if done at 4 years
14 % if done at 7 years
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Cerebral palsy
Spastic Diplegia – The Foot & Ankle
Toe Walking
• Dynamic :
Treat by :
Bracing
Spasticity reduction
Surgery ( careful ! )
• Fixed :
Treat by :
Serial casting
Surgery
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Cerebral palsy
Toe Walking
Gastrocnemius Release
Indications:
• isolated gastroc. Contractures
• dynamic toe walking
problems :
• reduces knee flexion power
• might not be effective
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Cerebral palsy
Toe Walking
ETA
Indications :
- fixed deformity
- tight TA
Problems :
over lengthening
Types :
z -plasty
sliding : - percutaneous
- open
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Cerebral palsy
Spastic Diplegia – The Foot & Ankle
Equino - valgus
• More frequently seen
• Tight TA and Talonavicular subluxation
• No perfect muscle balancing procedure
• Treatment :
ETA and subtalar arthrodesis
Osteotomies
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Cerebral palsy
Spastic Diplegia – The Foot & Ankle
Equino - varus
• Not very common
• Treatment :
- split transfer of tib. post. tendon
lateral half, posterior to interosseous memb.,
to peroneus brevis laterally
- elongation of tib. Post.
and split transfer of tib. ant. Laterally
prerequisite : - passively correctable foot
- good tib. ant.
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Cerebral palsy
Spastic Hemiplegia
• 30 % of all CP
• One side affection
upper > lower extremity
• 50 % mentally retarded
• 33 % seizures
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Type 1
• Weak tib ant. / triceps surae not tight
• Functional drop foot in swing phase
• Plantar flexion disappears in stance phase
• Treatment :
AFO with 90 degree plantar stop
Cerebral palsy
Spastic Hemiplegia
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Cerebral palsy
Spastic Hemiplegia
Type 2
• Weak tib ant / spastic triceps surae and tib post
• Equinus in all phases of gait – and varus
secondary knee hyperextension ( in late stance )
• Treatment :
ETA + elongation or split transfer tib post
AFO needed
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Cerebral palsy
Spastic Hemiplegia
Type 3
• Same like type 2 + tight hamstrings
often with cospasticity of Rectus Femoris
• Ankle equinovarus, knee crouch and stiff
• Treatment : same like type 2 +
hamstring lengthening (medial )
+distal Rectus Femoris release/transfer
AFO important
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Cerebral palsy
Spastic Hemiplegia
Type 4
• Same like type 3 +
hip flexor and adductor spasticity
• Equinovarus, crouch stiff knee, in addition to
hip adduction and flexion
• Treatment : same like type 3 +
hip adductor release and Psoas lengthening
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Cerebral palsy
Intoeing
• Usually caused by femoral ante-version
• Internal tibial torsion adds to intoing
• If severe : Derotation osteotomy
Delay to late childhood if possible
• Derotation osteotomy of femur might cause tightening of medial hamstrings
( might need lengthening )
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Cerebral palsy
In-toeing
Why?
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Cerebral palsy
In-toeing
Why ?
• Not related to spasticity of internal rotators
• Usually caused by femoral ante-version
• Femoral ante-version caused by:
Iliopsoas (flexor) spasticity – which causes
FFD & lack of hip extension - which
reduces torque on proximal femur - thus
normal derotation of physiological ante-version
does not occur
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Cerebral palsy
Why Does Anteversion Cause
In-toeing ?
• Anteversion rotates Greater Trochanter
posteriorly
• On stance phase need Abductor power
• Abductor power optimized when Greater
Trochanter is lateral not posterior
• Internal rotation in stance phase brings
Greater Trochanter laterally
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Cerebral palsy
In-toeing Pitfalls in derotation osteotomy
• Failure to recognize concomitant excessive
external torsion of tibia
• Over correction with loss of all internal
rotation of hip – causing gait problems:
pelvis can not externally rotate during gait
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Cerebral palsy
In-toeing Indication for derotation osteotomy
• Severe femoral anteversion, with loss of all
external rotation.
• Dynamic in-toeing causing gait abnormalities.
• Dynamic in-toeing causing secondary foot
deformity.
• Usually not before 8-10 years.
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Cerebral palsy
Spastic Quadriplegia
Management
Most are non walkers ( 80 % )
Aim for comfortable sitting on wheelchair
Specific objectives :
• Back : Scoliosis : fixation – straight spine
• Hips : good sitting – level pelvis - ROM (30o-90o)
good lying
hygiene
not painful
• Knees : reasonable ROM (20o-90o) – for sitting / lying
• Feet : plantigrade - rest comfortably on foot rest
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Cerebral palsy
Spastic Quadriplegia
Walkers: 20%
• Good head control
• Good trunk control
• Can attempt to stand
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Cerebral palsy
Hip Problems in
Spastic Quadriplegia
• Adduction and flexion deformity
• Wind swept deformity
• Internal rotation deformity
• Hip thrust and extension deformity
• Hip at risk
• Hip subluxation
• Hip dislocation
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Cerebral palsy
Hip Problems in
Spastic Quadriplegia
• Adduction and Flexion
deformities
• Wind-swept deformity
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Cerebral palsy
Hip Problems in
Spastic Quadriplegia
Hip Adduction and Flexion
• Scissoring, uncomfortable sitting, hygiene
• Hip adductor release
? Obturator neurectomy
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Cerebral palsy
Hip Problems in
Spastic Quadriplegia
Hip at risk
(valgus, anteverted, acet. dysplasia)
Femoral Anteversion more than true Valgus
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Cerebral palsy
Spastic Quadriplegia
Hip Management
Hip at risk
Often progresses to subluxation or dislocation
unless treated
• Hip adductor release
• Hip flexor release
• ? Obturator neurectomy
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Cerebral palsy
Hip Problems in
Spastic Quadriplegia
Hip Subluxation and Dislocation
Orthotics and physical therapy methods have
not been demonstrated to have any effect on
prevention of subluxation or dislocation of
the hip in CP.
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Cerebral palsy
Hip Problems in
Spastic Quadriplegia
Hip subluxation ( partially out )
( > 30 % uncoverage / broken Shenton’s line )
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Cerebral palsy
Hip Problems in
Spastic Quadriplegia
Hip subluxation ( partially out )
Dislocated Subluxated
50 %
Reimer’s migration index
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Cerebral palsy
Hip Problems in
Spastic Quadriplegia
Hip at Risk Hip subluxation
At 2.5 years At 7 years
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Cerebral palsy
Hip Problems in
Spastic Quadriplegia
High risk of hip subluxation and dislocation in
non-walking diplegics and quadriplegics
Should obtain a pelvic x-ray annually
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Cerebral palsy
Hip Problems in
Spastic Quadriplegia
• The independently walking child with spastic diplegia rarely dislocates, if ever, and only those who have partial weight bearing with crutches or walkers subluxate.
• Full wt. bearing = internally rotate
• Partial wt. bearing = subluxate
• Non ambulatory sitting = dislocate
Eugene Bleck
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Cerebral palsy
Spastic Quadriplegia
Hip Management
Hip subluxation
• Hip adductor release
• Hip flexor release
• ? Obturator neurectomy
• ? Femoral varus / ?derotation osteotomy
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Cerebral palsy
Hip Problems in
Spastic Quadriplegia
Hip dislocation is painful in 50 %
Hip dislocation increases deformity
Should prevent / treat hip dislocation
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Cerebral palsy
Spastic Quadriplegia
Hip Management
Hip Dislocation
• If detected early ( < one year ) :
Surgery: soft tissue
femoral osteotomy
? Acetabuloplasty (secondary Dysplasia)
• If detected late ( > One year ) :
painless: leave alone
painful : surgical resection
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Cerebral palsy
Hip Problems in
Spastic Quadriplegia
Hip Dislocation
( completely out )
Surgical resection Painful dislocated
> 1 year
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Cerebral palsy
Spastic Quadriplegia
Hip Management
Extension Contracture
• Rare
• Caused by tight contracted Hamstrings
• Interferes with comfortable sitting
• Does not allow hip flexion
• Treat by lengthening proximal Hamstrings
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Cerebral palsy
Spastic Quadriplegia
Hip Management
Extension Thrust
• More common than extension contracture
• Rigid and sustained hip extension
• Throws child out pf wheelchair
• Treated by proximal lengthening of Hamstrings
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Cerebral palsy
Spastic Quadriplegia
Management
• Scoliosis : Fixation – straight spine
• Hips : ( dislocation painful in 50 % )
- hip at risk : (valgus, anteverted, acet. dysplasia)
hip adductor and flexor release, ? Obturator neurect.
- hip subluxation :
same + ? Derotation / varus osteotomy
- hip dislocation :
if detected early: surgery
if detected late : no pain – leave
pain – proximal resection
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Cerebral palsy
Errors in Surgical Planning
• Frequent surgical procedures for “new”
deformities – were not identified initially
• Planning over-correction
- to avoid quick recurrence
- to compensate for another deformity
e.g. excessive ext. rot. of femur when
int. tibial as well as femoral anteversion
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Cerebral palsy
Errors in Surgical Planning
• Planning significant under-correction of bony and joint deformities
e.g. hip dislocation with ligament Contracture does not correct with little muscle
lengthening
e.g. significant anteversion or dislocated hip can not benefit by muscle surgery alone
Slight under-correction is better than over-correction e.g. intoing
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Cerebral palsy
Errors in Surgical Planning
• Recurrence of deformity
- very early surgery often ends in recurrence
- after muscle lengthening need aggressive
stretching and splinting to avoid
recurrence
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Principles in Cerebral palsy
Conclusion
• Set goals for management - not treatment
• Same perspective by surgeon and
physiotherapist – both indispensable!
• Look at multiple-level joint problems
• Know indications and limitations of various
management options
• Keep dynamic thinking – re-evaluate
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Mamoun Kremli
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Mamoun Kremli