Peripheral Nerve-blok Neuromuscular

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    PERIPHERAL NERVE

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    BRACHIAL PLEXUS LESION

    Brachial plexus is a complex structure originatingfrom C5-T1 nerveroots.

    Trauma : most common cause of brachialplexopathy.

    Closed injuries typically produce injury due totraction on the plexus, such as after a fall.

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    ERB PALSY

    In injuries that cause head & shoulder to bestretch apart such as in newborn deliveries orfalls on shoulder, result in upper trunkplexopathies with involvement ofC5-C6-C7erb palsy.

    Waiters tip deformity :Shoulder : adduction, internal rotation

    Elbow : extention, supination

    Wrist : flexion

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    KLUMPKE PALSY

    Injury in which there is forceful upward tractionon arm, result in lower trunk plexopathies affect

    C8-T1 fibers

    klumpke palsy.

    Clinical presentation : hand weakness

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    DIAGNOSE

    Anamnesa : Trauma ?

    Physical Exam. : MMTSensory

    Reflexes

    Supporting Exam. : NCV / EMG

    MRI (tumor ?)

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    TREATMENT

    Positioning

    ROM exercise

    Strengthening exercise

    Orthoses : shoulder sling, splint

    Electro Stimulation : MMT < 3

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    CARPAL TUNNEL SYNDROME

    Contents of carpal tunnel include :

    -Median nerve.

    -Flexor pollicis longus-Four tendons each from flexor digitorumsuperficialis & profundus muscles.

    Swelling or increased pressure inside carpaltunnel lead to compression ofmedian nerve,producing symptoms of CTS

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    Clinical presentation

    Paresthesias (numbness, tingling, and burning)involving median nerve distribution (first 3digits).

    Deep aching pain in hand & wrist. Pain willradiate info forearm, even rarely to shoulder.

    Patient might report subjective swelling of

    hand but on inspection, no swelling is usuallyapparent.

    Nocturnal worsening of symptoms is common

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    Clinical presentation

    Flick sign : patient often report awakening withhand paresthesias, which are then relieved by

    shaking involved hand.

    Advance CTS : sensory symptoms last longer orbecome persistent, & thenar weakness develops.

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    CTS is often associated with repetitive hand &wrist movements or the use of vibrating

    machine. The great majority of cases are idiopathicin

    origin.

    Predispose / risk factor :

    -DM -RA

    -Amyloidosis -Obesity

    -Hypothyroidism -Pregnancy

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    PHYSICAL EXAMINATION

    Abnormality in median sensory testing.

    MMT is more advance cases show weakness and

    atrophy of Abductor Pollicis Brevis & OpponensPollicis Muscles.

    Provocative test : Tinels sign, Phalens sign,Prayer sign

    Flick sign

    Supporting examination : NCV / EMG

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    TREATMENT

    ROM exercise

    Strengthening exercise

    Nocturnal wrist splint : hold the wrist in 0-30 of extension.

    USD / Phonoporeses / Laser

    Medication : NSAID, Corticosteroidinjection

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    POLIOMYELITIS

    is a viral infection involving the anterior horncells (LMN), producing weakness in affectedbulbar and/or spinal myotomes.

    Clinical presentation :

    -Initial symptoms : fever, malaise, headache,

    gastrointestinal or upper respiratory tracksymptoms.

    -Weakness was typically asymmetric, morecommon in the lower extremities

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    Patient with history of polio infection maydevelop fatigue and worsening muscle weakness

    severe years or even decades after initialinfection.

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    POST POLIO SYNDROME

    Criteria for establishing a diagnosis :

    Confirmed history of paralitic polio

    Partial to fairly complete neurologic & functionalrecovery

    Periode of neurologic & functional stability for atleast 15 years.

    No other medical explanation for the newhealthproblems

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    Onset of 2 or more of following symptoms sinceachieving stability :

    -Unaccustomed fatique.-New weakness in muscle previously affectedand/or unaffected

    -Muscle and/or joint pain

    -Functional loss

    -Cold intolerance

    -New atrophy

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    TREATMENT

    ROM exercise

    Strengthening exercise

    Endurance exercise Orthoses : HKAFO

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    BELLS PALSY

    idiophatic peripheral fascial nerveparalysis.

    Risk factor :

    -DM -Hyperacusis

    -HT -Lack of lacrimalis

    -Pregnancy -Schizophrenia

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    TREATMENT

    Acute (day of 0-4) :

    HEP : warm compress, fascial massage.

    HE : sunglasses, Y-plester Day of > 4 :

    -SWD / Laser

    -ES

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    HERNIATED NUCLEUS PULSOSUS

    A disk injury in which the nuclear pulposusmigrates through the annular fibers.

    A higher prevalence occurs for the lumbarregion at the L4-L5 or L5-S1 followed bythe C5-C6 disc.

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    CLASSIFICATION

    Buldging disc

    Prolapsed disc

    Extruded disc

    Sequestered disc

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    ETIOLOGY

    Spontaneous

    Lifting activities

    Coughting / sneezing

    Bending / twisting activity

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    CLINICAL PRESENTATION

    Acute neck or back discomfort radiatingdown the upper or lower limb

    Weakness, numbness, paresthesias or painsecondary to chemical or mechanical stimuli tothe disc or nerve root.

    Exacerbation occurs with lumbar motion

    (forward flexion, extention), sitting, sneezing,coughing, valsava manuver

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    DIAGNOSTIC STUDY

    X rays

    MRI

    Myelography

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    TREATMENT

    Acute : relative rest

    Diathermi : MWD, SWD, USD

    Laser TENS

    Traction : cervical, lumbal

    Orthoses : Lumbar corset HEP : William Flexion Exercise

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    LEPROSY (HANSENS DISEASES)

    Etiology : Mycobacterium Leprae

    Onset : Immune status dependent

    Clinical presentation :-Most common world-wide neuropathy

    -Sensory abnormality

    -Drop hand, drop foot-Fascial palsy

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    SIGN OF LEPROSY

    A slowly growing patch on the skin that does notitch or hurt

    Tingling numbness, or some loss of feeling inthe hand & feet. Or definite loss of feeling inskin patches.

    Slight weakness or deformity in hand & feet

    Enlargement of certain nerves, with or withoutpain or tenderness.

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    DIAGNOSE

    Definite loss or change of feeling in skinpatches.

    Definite enlargement of nerves. Presence of leprosy bacilli in a skin smear

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    TREATMENT

    Long term medical treatment : to control the leprosyinfection as early as possible Dapsone, Rifampisin,Clofazimine.

    Emergency treatment (when necessary to control) &prevent further damage (from leprosy reaction).

    Safety measures, aids, exercise, & education toprevent deformities (sores, burns, injuries, contractures).

    Social Rehabilitation. Work with individual, parent,schools, & community to create a better understandingof leprosy, to lessen peoples fear, & to increaseacceptance, so child & adult with leprosy can lead a full,happy, meaningful life

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    REHABILITATION OF LEPROSY

    ROM exercise

    Stretching exercise

    Strengthening exercise ES

    Orthesa : Knuckle bender, dorsal spring

    HEP : Soaking-Oiling-Scrapping HE : sunglasses, glove, shoes/sandal

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    THANK YOU