Ophthalmology 5th year, 1st lecture (Dr. Tara)

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The lecture has been given on Oct. 11th, 2010 by Dr. Tara.

Transcript of Ophthalmology 5th year, 1st lecture (Dr. Tara)

  • The eye ball & the orbit.The lateral orbital rim is considerably recessed compared with the medial orbit, which continues anteriorly to end at the nasal bridge. This leaves approximately one half of the globe unprotected. At its closest distance to the bony orbit the globe is about 4 mm from the roof, 4.5 mm from the lateral wall, 6.5 mm from the medial wall, and 6.8 mm from the floor.It is slightly closer to the lateral orbital wall than the medial wall and is nearer the roof than the floor of the orbit.The globe is positioned in the anterior portion of the orbit and constitutes about 20% of the entire volume of the orbit.

  • The bony orbit.*

  • *

  • Bony orbitMedial wall = Maxilla, lacrimal, ethmoid, sphenoid (anterior to posterior).

    Inferior (floor) = Maxilla (medial), zygoma (lateral), palatine (posterior).Lateral wall = Zygoma , Sphenoid (greater wing). Orbital Wall Bones Superior (roof) = Frontal , Sphenoid (lesser wing).

  • Blood supply*

  • Superior orbital fissure &optic foramenOF=optic foramen.SOF=superior orbital fissure.IOF=inferior orbital fissure.IV=trochlear nerveIII=third cranial nerveVI=sixth cranial nerve (the abducent)L=lacrimal nerveF=frontal nerve.N=nasocillary nerve*

  • Nerve supply*

  • Extraocular muscles*

  • ACUTE ORBITAL INFLAMMATIONS2. Deep Periostitis :Marked constitutional symptoms.Deep-seated pain in the orbit.Swelling and redness of the eyelids and the conjunctiva.Proptosis of the eyeball which may be deviated to one side.Pain and swelling at the orbital margin.Swelling and redness of the eyelids and the conjunctiva.An abscess may form at the site of the lesion.Clinical Picture1. Periostitis at the Orbital Margin :ORBITAL PERIOSTITISInflammation of the orbital periosteum is most often due to injuries, extension of inflammation from the neighbouring parts, tuberculosis or syphilis.

  • Complications 3. Orbital cellulitis. 4. Infection may spread to the cranial cavity, thus causing meningitis, cavernous sinus thrombosis or cerebral abscess.1. Sinus formation may occur in tuberculous periostitis. 2. Superior orbital fissure syndrome, i.e. various ocular motornerve palsies, trigeminal neuralgia and occasionally amaurosis due to involvement of the optic nerve.

  • The treatment consists of systemic antibiotic therapy and local application of heat. If an abscess forms, it should be incised. In deep periostitis, an exploratory operation may be necessary.

    Treatment.

  • ORBITAL CELLULITISMetastatic infection via the blood stream, e.g. in cases of pyaemia.Rarely direct infection by a penetrating wound, especially if a foreign body is retained within the orbit. Etiology Commonly caused by The spread of infection from the neighbouring areas, erysipelas of the face, ethmoidal sinusitis, lacrimal abscess, stye or suppurating chalazionOrbital cellulitis is an acute inflammation of the fatty-cellular tissue of the orbit.

  • Clinical Picture General.Fever, malaise and prostration are common symptoms. Sometime cerebral symptoms supervene, namely, delirium, coma and convulsions.Complications of Orbital Cellulitis 1. Thrombosis of the cavernous sinus. 3. Panophthalmitis. 2. Meningitis. 4. Optic neuritis. (d) Proptosis which is axial and irreducible. (e) Limitation of ocular movements usually in all directions causing diplopia. (f) Fundus examination may reveal engorged retinal veins and sometimes papillitis. Abscess formation may occur. It may burst through the skin of the eyelids near the orbital margin or in the conjunctival fornix.Ocular.The following signs are most important : (a) Severe pain in the orbit which increases during ocular movements. (b) Lid oedema with redness of the skin (c) Chemosis of the conjunctiva.

  • A swab is taken from the conjunctival sac for culture and sensitivity test to available antibiotics. Vigorous systemic and local use of antibiotic drugs to which the causative micro-organisms are sensitive, e.g. sulphonamide or any broad spectrum antibiotic. Local heat by frequent hot bathing is very beneficial. If abscess formation is suspected, early incision is recommended.

    Treatment of Orbital Cellulitis

  • CAVERNOUS SINUS THROMBOSIS2. Marked chemosis of the conjunctiva. 3. Rapidly increasing proptosis.Ocular.The following signs are most important : 1. Severe supraorbital pain due to irritation of the ophthalmic nerve.Clinical Picture General.There are severe systemic effects, e.g. high fever often associated with rigors, headache, vomiting and drowsiness.is caused by spread of infection from a focus of sepsis in the area drained venous sinus, e.g. orbital cellulitis, erysipelas or septic wounds of the face, middle ear or thrombophlebitis from infection in the mouth, pharynx, paranasal sinuses in the mastoid region. Rarely, cavernous sinus thrombosis may occur from metastatic infection.

  • Limitation of ocular movements due to paralysis of the third, the fourth and the fifth cranial nerves, which run in the lateral wall of the cavernous sinus. Congestion of the retinal veins and sometimes papilloedema. Complications 1. Meningitis. 2. Pyaemia. 3. Pulmonary abscess caused by a septic embolus. Presence of oedema in the mastoid region due to congestion of the emissary vein.

  • 1. Intensive antibiotic and sulphonamide therapy. 2. Intravenous anticoagulants. 3. Treatment of the causative focus of sepsis.

    Treatment

  • CHRONIC ORBITAL INFLAMMATIONS Clinical Picture.Proptosis and pain are the cardinal features. This is usually associated with edema of the lids, chemosis of the conjunctiva and vascular engorgement often at the insertion of the rectus muscles. Limitation of vertical ocular movements are more frequent than the horizontal ones.Chronic Inflammatory Granuloma of the Orbit Orbital pseudotumours comprises a group of space occupying lesions in the orbit resulting from a non-specific granulomatous inflammation, which are difficult to differentiate clinically from orbital tumours.

  • Conservative therapy is the rule. Systemic steroids, 40 - 60 mg daily for 1 - 2 weeks usually give dramatic response. The dose is tapered when the condition becomes under control with resolution of pain, proptosis and visual defect. Radiation therapy may shrink the lymphocytic infiltration.

    Treatment.

  • The diagnosis depends clinically on the rapid proptosis with pain in a healthy young subject (between the ages of 10-30 years). In suspicious cases, aspiration of the cyst with a fine needle should be made. The presence of hooklets or scolices is diagnostic, but many orbital cysts are sterile.Ocular complications include proptosis, neuro-keratitis, glaucoma, papilloedema, optic neuritis, congestion of the retinal veins and haemorrhages.

    PARASITIC ORBITAL INFECTION Hydatid Cyst

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