Management of patients with multiple trauma

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Management of patients with multiple trauma . Prof. M K Alam MS; FRCS. ILO’s. Incidence of trauma Causes and types of trauma Timing and mode of death in trauma patients and its effect on trauma management. Pre-hospital care and triage Hospital care Primary survey and initial management - PowerPoint PPT Presentation

Transcript of Management of patients with multiple trauma

Page 1: Management of patients with multiple trauma
Page 2: Management of patients with multiple trauma

Management of patients with multiple trauma

Prof. M K Alam MS; FRCS

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ILO’s Incidence of trauma

Causes and types of trauma 

Timing and mode of death in trauma patients and its effect on trauma management.  

Pre-hospital care and triage

Hospital care 

Primary survey and initial management 

Secondary survey

Pathophysiology of common injuries 

Investigations during primary and secondary survey

A brief outline of management of major injuries.

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Epidemiology

• Trauma remains the most common cause of death between the ages of 1 and 44 years.

• Affects a disproportionate number of young people- the burden to society in terms of lost productivity, premature death, and disability is considerable.

• A major public health issue.

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Arab News 16th Feb. 2014

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Arab News 3rd March 2014

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Arab News 16th Feb 2014• 20 deaths daily on the Kingdom's roads.

• Last year- 707 amputations due to RTA.

• Accidents increased by 78% in the KSA recently

• Affecting mostly young between 18 and 22 years

• Around 30% of those injured are permanently disabled.

• The state has spent SR21 billion treating such patients

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Causes of trauma• RTA or MVA• Pedestrian trauma• Fall from height• Assault• Firearm injuries• Knife• Industrial accidents• Natural disasters• Explosions

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Types of trauma

• Blunt trauma results of an impact from blunt object

• Penetrating trauma results from an object piercing the body

• Assessment and diagnosis of blunt injuries are more difficult than of penetrating injuries

• Multi-trauma- injury affecting simultaneously different organ and body system

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Trimodal death in trauma• Immediate: Within seconds or minutes after injury- 50% of

deaths due to injury to the aorta, heart, brainstem, or spinal cord or by acute respiratory distress.

• Early: Within hours of injury- approximately 30% of deaths. Half of these deaths are caused by hemorrhage and the other half by central nervous system (CNS) injury. These patients can be saved by appropriate treatment (golden hour).

• Late: peaks from days to weeks, mortality due to infection and multiple organ failure.

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Improvement in mortality

• Early deaths: Prevention and control program by legislation and behavior modification

• Later deaths: • Trauma centers providing better care.• Better understanding of pathophysiology of

multiple organ failure and brain injury

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Pre-hospital care• Delivery to the hospital for definitive care as rapidly

as possible- scoop and run

• Only critical interventions at the scene

• Airway established, hard collar, spine board, control any external hemorrhage

• Infusion on way to the hospital

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Triage

• Definition: Prioritizing victims into categories based on

their severity of injury, likelihood of survival, and urgency of care.

• Goals:– Identify high-risk injured patients who would benefit

from the resources available in a trauma center. – Limit the excessive transport of non-severely injured

patients so that the trauma center is not overwhelmed.

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Hospital care

• ATLS approach

• A well defined order

• Primary survey- initial assessment and management

• Treat the greatest threat to life

• Immediate intervention as the threat to life is identified

• Detailed history not essential

• Re-evaluation of initial management

• Secondary survey- a head to toe evaluation

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Primary survey

• A B C D E• Airway & cervical spine protection

• Breathing

• Circulation

• Disability (neurologic assessment)

• Exposure and Environmental control

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Primary survey- a team approach• Simultaneous diagnosis and treatment by multiple

providers

• Reduces the time to assess and stabilize a multiple trauma patients

• Team should be organized before patient arrival.

• Leadership and unity of command are essential

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Primary survey-one clinician

Do not perform subsequent steps in the primary survey until after addressing life-threatening conditions in the earlier steps.

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Part II

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A

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Airway & cervical spine

• Verbal response: Salam! How are you?

Airway is compromised if:

• No response- unconscious , airway obstruction

• Noisy breathing

• Severe facial trauma

• Oropharyngeal bleeding or foreign body

• Patient agitated - hypoxia

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Airway & Cervical spine• Adequacy of airway- completed within seconds

• Open the front of the collar for airway manipulation

• Maintain manual stabilization by an assistant

• Oropharyngeal airway/ bag valve mask ventilation

• Oxygen supplement + pulse oximetry

• Rapid-sequence endotracheal intubation

• Frequent reassessment for airway compromise

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Difficult airway

• Surgical airway when oral intubation cannot be accomplished:

– Cricothyroidotomy –Surgical– Percutaneous needle technique- only temporary – Tracheostomy (laryngeal injury)

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B

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BREATHING

Life threatening injuries to look for:

• Tension pneumothorax

• Open pneumothorax (open chest wound)

• Flail chest with underlying pulmonary contusion

• Massive hemothorax

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BREATHING

• Dyspnoea

• Unilateral diminished chest expansion

• Bruising/ abrasion

• Distended neck vein

• Trachea deviated to the opposite side

• Percussion: dull - haemothorax

Hyper resonant - Pneumothorax

• Diminished/ absent breath sound

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Tension pneumothorax Pathophysiology

• Collapsed lung acts as a one-way valve

• Each inhalation- additional air accumulate in the pleural space. • Normal negative intrapleural pressure becomes positive,

depressing the ipsilateral hemidiaphragm, pushing the mediastinal structures into the contralateral chest

• Contralateral lung is compressed, the heart is rotated about the superior and inferior vena cava, decreasing venous return and cardiac output while distending the neck veins

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Tension pneumothoraxClinical features & treatment

• Respiratory distress• Tracheal deviation away from the affected side• Lack of or decreased breath sounds • Distended neck veins or systemic hypotension• Subcutaneous emphysema, hyper resonance• Treatment: x-ray confirmation not required• Wide bore needle in 2nd inercost. space, mid clavicular• Chest tube in 5th intercost. space, ant. axillary line

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Open pneumothorax or sucking chest woundPathophysiology

• Full-thickness loss of the chest wall: free communication between the pleural space and the atmosphere.

• Collapse of the lung on the injured side

• If the diameter of the injury is greater than the narrowest portion of the upper airway, air will preferentially move through the injury

• impair ventilation on the contralateral side

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Open pneumothorax Management

• Complete occlusion of the injury may result in converting an open pneumothorax into a tension pneumothorax.

• Initial treatment: occlusive dressing, which is taped on three sides over the wound

• Dressing permits effective ventilation, while the untaped side allows accumulated air to escape from the pleura

• Definitive treatment: wound closure and tube thoracostomy

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Flail chest with pulmonary contusion Pathophysiology

• Four or more ribs fractured in at least two locations

• Paradoxical movement of free-floating segment may

occasionally compromise ventilation.

• More importantly, an underlying pulmonary contusion

may compromise oxygenation or ventilation

• Initial chest x-ray underestimates the degree of contusion.

• The lesion evolve with time and fluid resuscitation.

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Flail chest with pulmonary contusionManagement

• Respiratory failure in these patients may not be immediate

• Frequent re-evaluation is needed.

• Intubation and mechanical ventilation is required

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Massive hemothorax

• Accumulation of >1.5L of blood• Disruption of large vessel• Flat neck vein• Dullness on percussion• No breath sound• Shock• Management: Chest tube in 5th space, fluid resuscitation.

Thoracotomy if significant bleeding continues.

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Part III

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C

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Circulation

• Assessment of cardiovascular compromise and

management

• Is the patient in shock?

• Is there any external bleeding source?• Any internal hemorrhage?

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CirculationPathophysiology

• Shock is secondary to hemorrhage in most trauma patients

• Patient can be in shock before developing hypotension

• Hypotension- a sign of decompensation (class III )

• 5 locations for major blood loss:

ChestAbdomenPelvis and retroperitoneumMultiple long bone fractures ( lower limb)External hemorrhage

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Pathophysiology of blood loss• Responses are compensatory• Progressive vasoconstriction- skin, muscle, viscera• Tachycardia to preserve cardiac output• Increased peripheral resistance- catecholamines• Venous return preserved in early stage by reduced

blood volume in venous system• Continued bleeding- shock develops • Inadequate tissue perfusion, metabolic acidosis

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Classes of hemorrhagic shock

Class I Class II Class III Class IV

Blood loss (ml)

Up to 750 750- 1500 1500- 2000 > 2000

Pulse <100 >100 >120 >140

BP Normal Normal Decreased Decreased

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CirculationIndicators of shock in trauma patients

• Tachycardia*• Agitation• Tachypnea• Sweating• Cool extremities

• Weak peripheral pulse• Decreased pulse

pressure• Hypotension• Oliguria

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CirculationCardiogenic shock

• Tension pneumothorax- most common cause, Pericardial tamponade(penetrating trauma), Myocardial contusion

• Beck’s triad- hypotension, distended neck vein (raised CVP >15 cm H2O), muffled heart sound

• CVP: Hemorrhagic <5 cmH2O• Dysrhythmias in contusion• Ultrasonography : helpful in diagnosis • Treatment: fluid resuscitation, pericardiocentesis

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CirculationNeurogenic shock

• Loss of sympathetic tone due to cord injury

• Hypotension, warm well perfused limbs,

diminished/absent motor function

• Bradycardia

• Management: IV fluid, vasopressor, corticosteroids

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CirculationSeptic shock

• Delayed arrival• Penetrating abdominal injuries• Early septic shock- normal circulating volume• Tachycardia• Warm skin• Systolic close to normal, • Wide pulse pressure

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Circulation Initial management

• External haemorrhage- compression dressing

• IV access- two peripheral catheters

• ECG monitoring

• Blood sample- typing and lab. investigations

• Initial resuscitation:1-2L of Ringer's lactate or NS

• Packed red blood cells if no response

• Foley’s catheter: urine output is .5 mL/kg/hour in adult

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Circulation Initial management

Search for any source of blood loss:

• CXR, X-ray pelvis, FAST (focused abdominal

sonography in trauma)

• If fracture pelvis is found pneumatic antishock

garment or a bed sheet wrapped around the pelvis

may be applied

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Evaluation of fluid resuscitation

• BP and pulse rate• Urine output (0.5ml/kg/hour)• Mental status and skin color/temperature• CVP• Acid/base status

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Management decisionsRapid responders

• Hemodynamics return to normal after fluid resuscitation

• Hemodynamics remain stable even after reducing infusion to maintenance rate.

• Probably bleeding has stopped spontaneously• Continued evaluation for source of bleeding• May still need surgery

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Management decisionsTransient responders

• Decompensate once fluid resuscitation is slowed

down

• There is ongoing bleeding or inadequate resuscitation

• Increase fluid resuscitation and blood transfusion

(type specific or O negative)

• ?Surgical intervention

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Management decisionsNon-responders

• Fail to respond to fluid and blood resuscitation• Major blood loss (>40%) & ongoing loss• Immediate surgical intervention

• ? Non-hemorrhagic shock (cardiogenic)• Echocardiography• CVP

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Part IV

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D

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Disability Neurologic evaluation

• Level of consciousness measured by the

Glasgow Coma Scale (GCS)

• If the GCS is used in intubated and paralyzed

patients, record should be made

• Pupillary response can still be assessed in a

paralyzed patient

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Glasgow Coma Scale (GCS), Total = 15

Eye response Vocal response Motor responseSpontaneous 4 Oriented 5 Obeys commands 6

To voice 3 Confused 4 Purposeful movement to pain 5

To pain 2 Inappropriate words 3

Withdraw from pain 4

None 1 Incomprehensible words 2

Flexion to pain 3

*** None 1 Extension to pain 2

*** *** None 1

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Head injury severity

• Mild GCS ≥ 13• Moderate GCS 9- ≤ 12• Severe GCS ≤ 8

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E

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Exposure/ Environment control• Completely undress the patient

• Perform a rapid head-to-toe examination

• Identify any injuries to the back, perineum, or other

areas that are not easily seen in the supine position

• Unexpected injuries may be discovered

• Once assessment completed, cover the patient with

blanket ( prevent cold exposure)

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Secondary Survey

• Only after completion of primary survey(ABCDE)

• Life threatening injuries have been dealt

• Normalization of vital signs

• A head to toe evaluation

• Detailed history and examination

• Continuous reassessment of vital signs

• Additional lab. & radiological tests and collecting results

• Additional tubes, lines and monitoring devices

• Priorities and plan definitive management of all injuries

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Head injury

• Traumatic brain injury (TBI)- the leading cause of death in

trauma patients- 50% of all traumatic deaths.

• Primary injury- the anatomic and physiologic disruption that

occurs as a direct result of trauma

• Secondary injury- extension of the primary injury, result from

local swelling, increased ICP, hypoperfusion, hypoxemia, or other

factors.

• Aim- detection and treatment of primary injury and prevention of

secondary injury

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Head injury- management

• Maintain BP >90 mmHg, PaO2 >60 mmHg

• Assess GCS and lateralizing signs- pupil and motor function

• Pupillary asymmetry >1 mm suggests intracranial injury

• Larger pupil is on the side of the mass lesion

• Extremity weakness- detected by testing motor power

• CT scan head- accurate localization of the lesion

• Epidural or subdural hematoma causing mass effect evacuated

• Diffuse axonal injury- maintain cerebral perfusion and prevent rise in ICP

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Spinal cord injuries

• Intensive hospital care, long-term rehabilitation, life-long care.

• Initial care- strict immobilization of the spine

• Complete neurologic assessment

• Steroid therapy must be initiated within a few hours of injury

• Injuries above C3- are apneic, need intubation

• between C3 and C5 – may need intubation later

• Complete transection- poor prognosis

• Preservation of remaining function

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Thoracic injuries• Life-threatening : tension pneumothorax, massive

hemothorax, open pneumothorax, flail chest, and cardiac tamponade

• Rib fractures, sternal fracture, lung contusion, Injuries to trachea, bronchi, heart, diaphragm, esophagus, thoracic aorta

• Diagnostic modalities: CXR, ultrasonography, chest CT, esophagography, esophagoscopy, bronchoscopy, and angiography

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Part V

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Abdominal injuries

• 25% of all trauma victims require abdominal exploration.

• Physical examination- inadequate to identify intra-

abdominal injuries

• Diagnostic modalities- CXR, FAST, DPL,CT & laparoscopy

• Blunt trauma: • Hemodynamically stable- CT scan , • Hemodynamically unstable- FAST

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Diagnostic peritoneal lavage (DPL)

• Insert catheter below umbilicus under LA and full asepsis and saline (1L NS) infusion into peritoneum

• Returning fluid is bloody- +ve lavage• Rapid and safe• Bloody aspirate- laparotomy• Do not determine origin of blood• Too sensitive• Does not evaluate retroperitoneal injury• Replaced by FAST and CT scan

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FAST- focused abdominal sonography in trauma

• Superseded DPL in assessment of abdominal trauma

• 98% sensitivity for hemoperitoneum

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Abdominal injuries (penetrating)

• All gun shot injuries- urgent surgery

• Stab (knife) injury:

Hemodynamically stable- CT scan, surgery only if intra-abdominal injuries found

Hemodynamically unstable- surgery

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Splenic injury

• Most frequently injured in blunt trauma (personal series)

• History of injury to the left side of the chest, flank, or left upper part of the abdomen

• Bruising, pain tenderness- lower chest and upper abdomen on left side

• Diagnosis- CT in hemodynamically stable patients FAST or exploratory laparotomy in an unstable patients

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Splenic injury Non-surgical management (70%)

• Hemodynamically stable patients:

• FAST, CT for diagnosis

• No other intra-abdominal injury requiring operation

• Admission to ICU for continuous monitoring

• Serial Hb. , & repeated abdominal assessment

• If hypotension develops - taken for surgery

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Splenic injury Surgical management

• Hemodynamically unstable

• FAST: splenic injury, free fluid (hemoperitoneum)

• Surgery- splenectomy

• Polyvalent pneumococcal vaccine (pneumovax)

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Liver injury- pathophysiology• Susceptible to injury due to large size(1200-1600 g)

• Covered by bony thoracic cage

• Injury frequency - only 2nd after spleen( personal series)

• Highly vascular- only 4% of body weight but 28% of total body blood flow

• Double blood supply- portal vein & hepatic artery

• Draining hepatic veins- short and thin walled

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Liver injury• Spontaneous hemostasis- 50% of small lacerations

• Profuse bleeding from deep hepatic lacerations-

a formidable challenge

• Mortality rate 8%- 10%, morbidity rate from 18%-30%,

• Diagnosis: FAST in hemodynamically unstable,

CT scan in hemodynamically stable

• Management based on hemodynamic status

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Liver injuryNon-operative management

• Hemodynamically stable patients• CT scan• No other indications for abdominal exploration• ICU admission for close observation• Serial hemoglobin estimation• Transfusion requirements of <2 units of blood• Surgery- if become unstable

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Liver injury Surgical management

• Principles of surgical management: control of bleeding,

removal of devitalized tissue, and adequate drainage.

• Bleeding vessels & biliary radicles are individually ligated

• Pringle’s maneuver

• Perihepatic packing- when fail to control hemorrhage

• Packs removed in 48 hours

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Pancreatic injuries

• Pancreatic injury is rare• Caused by penetrating injury or direct blow • Diagnosis is difficult to make• CT scan, elevated serum amylase may help• No duct injury: simple drainage• Ductal injury: distal resection

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Bowel injuries

• Mostly due to penetrating trauma

• Also seen after blunt trauma

• Features of peritonitis

• CT scan free air in peritoneum/ contrast leak

• Small bowel: Suture repair

• Colon: suture repair± proximal colostomy

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Renal injuries

• Minor- renal contusion (85%) Conservative management

• Major: Deep medullary injuries with extravasation

Vascular injuries Surgical repair

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Thank you!

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Part VI

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Case for discussion

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• An ambulance is bringing a young man who was riding a motor bike. He was thrown from the speeding motor bike on a bending road. He was not wearing a safety helmet. His left leg appears grossly deformed.

• The ambulance has informed ER before bringing him.

• You are the only doctor in ER• What to do?

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Preparation before patient arrival

• Airway equipment, cervical collar, pulse oximetry, ECG monitor, oxygen

• Laryngoscope, Needles, chest tubes, under-water seal,• Minor op. set, local anaesthetic, • IV fluids at room temp.• Blood sample tubes• Splints• Radiologist and technician• Foley catheter and urine bag

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Management in a hospital

• Patient arrives in hospital

• Patient is on a spinal board

• Deformed left lower limb with blood stain on cloth?• What to do next?

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Primary survey

• A• B• C• D• E

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Assessment of airway

• Talk to the patient

Danger signs• Not talking • Oro-facial bleeding• Confused• Agitated• Neck hematoma

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Airway management

• Clearing oral cavity• Oropharyngeal / bag valve mask• Chin lift / jaw thrust• Oral endotracheal intubation• Surgical methods• Adjuncts: oximetry, oxygen• Cervical collar if not applied during transport• Manual in-line support by an assistant

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Breathing

• Patient continues to be dyspnoeic?• Oxygen saturation not improving?• Chest injuries to look for and manage

1. Tension pneumothorax2. Massive hemothorax3. Flail chest4. Open chest wound

pO2 and respiratory rate improves

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Circulation• Pulse, BP,RR• Any external bleeding? Look at his deformed limb • 2 IV line, blood samples• RL or NS 1-2 L as bolus rapidly• Quick response: slow down iv to maintenance• Transient response: BT ?bleeding• No response: ?Major bleeding ? Inadequate

resuss. ?non- hemorrhagic shock ( cardiogenic, spinal, septic)

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Hemorrhagic vs Non-hemorrhagic shock

• Neck vein• Pulse (rhythm, volume, rate)• Heart sound• ECG• CVPLater : Spinal injury Late presentation with abdominal injury

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Major bleeding sources

• Chest: massive hemothorax

• Abdomen: hemoperitoneum

• Pelvis: pelvic & retroperitoneal hematoma

• Lower limb fractures

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Investigations for bleeding source

• CXR*• FAST• DPL• CT• X-ray pelvis*

*X-ray c spine- the only other x-ray allowed during Primary survey

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Disability & Exposure

• GCS• Full exposure including the blood mark on his

lower limb. • Splint the limb- if not already done during

assessment for external hemorrhage • Cover patient with a blanket• Reassess ABCD

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Secondary survey• Only after completion of primary survey(ABCDE)

• Life threatening injuries have been dealt

• Normalization of vital signs

• A head to toe evaluation

• Detailed history and examination

• Continuous reassessment of vital signs

• Additional lab. & radiological tests and collecting results

• Additional tubes, lines and monitoring devices

• Priorities and plan definitive management of all injuries

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Thank you!