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Clemson University TigerPrints All Dissertations Dissertations 5-2014 IMPACT OF PHYSICAL AND PSYCHOSOCIAL WORKPLACE HAZARDS ON EMPLOYEE HEALTH: AN IRISH T ALE OF CIVIL SERVANT WORKERS Kyle R. Stanyar Clemson University Follow this and additional works at: hps://tigerprints.clemson.edu/all_dissertations Part of the Psychiatry and Psychology Commons is Dissertation is brought to you for free and open access by the Dissertations at TigerPrints. It has been accepted for inclusion in All Dissertations by an authorized administrator of TigerPrints. For more information, please contact [email protected]. Recommended Citation Stanyar, Kyle R., "IMPACT OF PHYSICAL AND PSYCHOSOCIAL WORKPLACE HAZARDS ON EMPLOYEE HEALTH: AN IRISH T ALE OF CIVIL SERVANT WORKERS" (2014). All Dissertations. 1401. hps://tigerprints.clemson.edu/all_dissertations/1401

Transcript of IMPACT OF PHYSICAL AND PSYCHOSOCIAL WORKPLACE HAZARDS …

Page 1: IMPACT OF PHYSICAL AND PSYCHOSOCIAL WORKPLACE HAZARDS …

Clemson UniversityTigerPrints

All Dissertations Dissertations

5-2014

IMPACT OF PHYSICAL ANDPSYCHOSOCIAL WORKPLACE HAZARDSON EMPLOYEE HEALTH: AN IRISH T ALEOF CIVIL SERVANT WORKERSKyle R. StanyarClemson University

Follow this and additional works at: https://tigerprints.clemson.edu/all_dissertations

Part of the Psychiatry and Psychology Commons

This Dissertation is brought to you for free and open access by the Dissertations at TigerPrints. It has been accepted for inclusion in All Dissertations byan authorized administrator of TigerPrints. For more information, please contact [email protected].

Recommended CitationStanyar, Kyle R., "IMPACT OF PHYSICAL AND PSYCHOSOCIAL WORKPLACE HAZARDS ON EMPLOYEE HEALTH: ANIRISH T ALE OF CIVIL SERVANT WORKERS" (2014). All Dissertations. 1401.https://tigerprints.clemson.edu/all_dissertations/1401

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IMPACT OF PHYSICAL AND PSYCHOSOCIAL WORKPLACE HAZARDS ON EMPLOYEE HEALTH: AN IRISH TALE OF CIVIL SERVANT WORKERS

____________________________________

A Dissertation Presented to

the Graduate School of Clemson University

____________________________________

In Partial Fulfillment of the Requirements for the Degree

Doctor of Philosophy Industrial-Organizational Psychology

____________________________________

by Kyle R. Stanyar

May 2014 ____________________________________

Accepted by:

Dr. Robert R. Sinclair, Committee Chair Dr. Patrick Rosopa

Dr. Paul Merritt Dr. James McCubbin

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ABSTRACT

Obesity, mental health problems, and absenteeism are both economic and health burdens

for employers and employees. Research suggests that physical and psychosocial hazards

in the workplace contribute to health risks and health problems among employees. There

is a need for researchers to examine how exercise, diet, and age interact with the negative

effects of workplace hazards upon health. Hypotheses 1a through 3b predicted that

physical and psychosocial workplace hazards would negatively impact body mass index

(BMI), general mental health, and sickness absences. Further, hypotheses 4a through 9b

predicted that exercise and diet would buffer stress from occupational hazards upon BMI,

mental health, and sickness absences. Finally, hypotheses 10a through 11b predicted that

age would act as a moderator between occupational hazards and employee health

outcomes. A sample of 16, 651 civil servant workers from the Northern Ireland Civil

Service Workforce were examined. The data was split into two groups based on salary-

senior level pay grade and lower level pay grade. The results confirmed hypotheses 1a,

1b, 3a, 3b, 11a, and 11b for the lower level pay grade, but failed to support hypotheses 2

and 4a through 10b. Additionally, the results confirmed hypotheses 1b for the senior level

pay grade; however the results failed to confirm hypotheses 1a and hypotheses 2 through

11b. Importantly, physical activity was related to a lower BMI, improved mental health,

and less sickness absences, while a healthy diet was related to a lower BMI for both pay

grades. Promoting physical activity and a healthy diet are viable methods for improving

employee health.

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ACKNOWLEDGEMENTS

Several people have played a vital role in helping to complete this dissertation. I

am extremely grateful and indebted to each and every one of them. Without their support

and guidance I would not have been able to complete this paper.

First, I would like to thank my advisor and mentor Dr. Robert Sinclair. Without

his support and guidance, my dissertation would not be what it is today. His expertise in

Occupational Health Psychology and his high standards for work quality have helped

improve my own work. Without his help I would not have made it into graduate school,

and I am greatly appreciative of that.

In addition to my advisor and mentor, I also have committee members who I

deeply respect. Dr. James McCubbin has helped expand my expertise in health

psychology and allowed me to apply my knowledge of health psychology to workplace

settings. Further, Dr. Paul Merritt has allowed me to gain valuable experience in

designing and implementing research studies related to physical fitness. Finally, Dr.

Patrick Rosopa has been a great mentor for teaching me statistics and research design.

I must also thank Dr. Jonathan Houdmont, who allowed me to use his data for my

dissertation. Without the generosity of Dr. Houdmont, I would not have a dissertation at

all. I greatly appreciate his generosity.

Finally, I want to thank my family and friends for giving me support throughout

the years, allowing me to get to this point in my life. I want to especially thank my

parents Dave and Susan Stanyar, as well as my sister, who have always supported me in

my choice to pursue a Ph.D. in Industrial-Organizational Psychology. Thank you!

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TABLE OF CONTENTS

Pages

TITLE ............................................................................................................................... i

ABSTRACT ..................................................................................................................... ii

ACKNOWLEDGEMENTS ............................................................................................ iii

TABLE OF CONTENTS ................................................................................................ iv

CHAPTER 1: INTRODUCTION .................................................................................... 1

Obesity ...................................................................................................................... 15

Mental Health ............................................................................................................ 17

Immune System ........................................................................................................ 20

Physiological Process of Stress ................................................................................. 21

Workplace Hazards ................................................................................................... 23

Workplace Hazards and Stress .................................................................................. 25

Physical Hazards and Health .................................................................................... 25

Psychosocial Hazards and Health ............................................................................. 29

The Current Study ..................................................................................................... 32

Workplace Hazards and Obesity ............................................................................... 37

Psychosocial Hazards and Mental Health ................................................................. 41

Physical Hazards and Mental Health ........................................................................ 44

Physical Workplace Hazards and Employee Absences ............................................ 48

Psychosocial Workplace Hazards and Employee Absences ..................................... 53

Physical Activity and Exercise ................................................................................. 57

Diet and Eating Habits .............................................................................................. 59

Health Behaviors and Habit Formation .................................................................... 61

Physical Activity and Stress ...................................................................................... 62

Physical Activity and Obesity ................................................................................... 66

Physical Activity and Mental Health ........................................................................ 70

Physical Activity and Absences Due to Sickness ..................................................... 75

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Table of Contents (Continued)

Pages

Diet and Stress .......................................................................................................... 81

Diet and Obesity ....................................................................................................... 83

Diet and Mental Health ............................................................................................. 85

Diet and Sickness Absence ....................................................................................... 88

Aging Workforce ...................................................................................................... 91

Age and BMI ............................................................................................................. 93

Age and Sickness Absence ....................................................................................... 96

CHAPTER 2: METHOD ............................................................................................... 98

Participants ................................................................................................................ 98

Measures ................................................................................................................... 99

Procedures ............................................................................................................... 107

CHAPTER 3: RESULTS ............................................................................................. 110

Data Screening ....................................................................................................... 110

Descriptive Statistics of Measured Variables ........................................................ 110

Correlation Analyses ............................................................................................... 112

Regression Analyses ............................................................................................... 115

Supported Interactions ............................................................................................ 118

Non-Supported Interactions .................................................................................... 120

Alternative Analyses ............................................................................................... 122

CHAPTER 4: DISCUSSION ....................................................................................... 124

Theoretical Implications ......................................................................................... 125

Organizational Implications .................................................................................... 138

Strengths of Current Study ...................................................................................... 148

Weaknesses of Current Study ................................................................................. 151

Future Research ...................................................................................................... 152

Conclusions ............................................................................................................. 158

REFERENCES ............................................................................................................ 159

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Table of Contents (Continued)

Pages APPENDICES ............................................................................................................. 207

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FIGURES

Figure Page

Figure 1 .......................................................................................................................... 33

Figure A1 ...................................................................................................................... 214

Figure A2 ...................................................................................................................... 215

Figure A3 ...................................................................................................................... 216

Figure A4 ...................................................................................................................... 217

Figure A5 ...................................................................................................................... 218

Figure A6 ...................................................................................................................... 219

Figure A7 ...................................................................................................................... 220

Figure A8 ...................................................................................................................... 221

Figure A9 ...................................................................................................................... 222

Figure A10 .................................................................................................................... 223

Figure A11 .................................................................................................................... 224

Figure A12 .................................................................................................................... 225

Figure A13 .................................................................................................................... 226

Figure A14 .................................................................................................................... 227

Figure A15 .................................................................................................................... 228

Figure A16 .................................................................................................................... 229

Figure A17 .................................................................................................................... 230

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TABLES

Table Page

Table B1 ....................................................................................................................... 231

Table B2 ....................................................................................................................... 232

Table B3 ....................................................................................................................... 233

Table B4 ....................................................................................................................... 234

Table B5 ....................................................................................................................... 235

Table B6 ....................................................................................................................... 236

Table B7 ....................................................................................................................... 237

Table B8 ....................................................................................................................... 238

Table B9 ....................................................................................................................... 239

Table B10 ..................................................................................................................... 240

Table B11 ..................................................................................................................... 241

Table B12 ..................................................................................................................... 242

Table B13 ..................................................................................................................... 243

Table B14 ..................................................................................................................... 244

Table B15 ..................................................................................................................... 245

Table B16 ..................................................................................................................... 246

Table B17 ..................................................................................................................... 247

Table B18 ..................................................................................................................... 248

Table B19 ..................................................................................................................... 249

Table B20 ..................................................................................................................... 250

Table B21 ..................................................................................................................... 251

Table B22 ..................................................................................................................... 252

Table B23 ..................................................................................................................... 253

Table B24 ..................................................................................................................... 254

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Table Page

Table B25 ..................................................................................................................... 255

Table B26 ..................................................................................................................... 256

Table B27 ..................................................................................................................... 257

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CHAPTER 1

INTRODUCTION

People are spending increased amounts of time at work, as organizations compete

to stay profitable in a globalized economy. Mexico tops the list, with Mexicans spending

2,250 average annual hours of work per person, recently surpassing South Korea at 2,193

average annual hours of work per person (OECD, 2012). In comparison, U.S. employees

average 1,787 annual hours and the Dutch logged the least amount of working hours per

person, averaging 1,379 hours of annual work. The United Kingdom averaged 1,625

hours of annual work per person, ranking 23rd among 32 nations surveyed by the

Organization for Economic Co-operation and Development (OECD, 2012).

In light of the fact that people are spending increased amounts of time at work,

health promotion in the workplace is becoming recognized as an important area of

research. Psychologists have argued that psychology needs to take a more active role in

applying research to practice, in order to prevent occupational stress, illness, and injury

(NIOSH, 2013). Occupational health psychology can help bridge the gap between

research and practice. According to NIOSH (2013) Occupational Health Psychology

concerns the application of psychology to improving the quality of work life, and to

protecting and promoting the safety, health, and well-being of workers. Considering the

fact that people are spending increased amounts of their daily lives at work, Occupational

Health Psychology (OHP) has become recognized as a critical area of study for

investigating the causes and consequences of employee health and well-being.

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One of the challenges of workplace health promotion is that it involves

encouraging behaviors outside of work, which may be perceived as an action beyond an

employer’s authority or responsibility (Eakin, 1992). Further, some researchers fear that

placing an emphasis on individual employee behaviors outside of work may create an

environment conducive to employers discriminating against their employees who suffer

from health problems (Khatri, Brown, & Hicks, 2009). For example, discrimination

against obese employees and employees with mental health problems is quite common

(Roehling, Roehling, & Pichler, 2007; Stuart, 2006). However, many researchers and

practitioners now acknowledge that it is necessary to promote employee health both

within and outside the organization, while at the same time protecting the privacy and

rights of employees (NIOSH, 2013).

NIOSH (2013) has coined the term “total worker health” as a strategy to integrate

occupational safety and health protection with health promotion. The goal of total worker

health promotion is to simultaneously prevent worker injury and illness and to advance

health and well-being. Recent research demonstrates that both work-related factors and

health factors outside of the workplace contribute to the health and well-being of

employees (NIOSH, 2013). In the past, workplace health and safety programs have been

segmented. For example, health protection programs have been solely targeted towards

reducing employee exposure to risk factors arising in the workplace. In contrast, most

workplace health promotion programs have focused on health behaviors outside of the

workplace (NIOSH, 2013). Many health promotion programs tend to omit organizational

factors from consideration when creating programs and many health protection programs

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from an organizational framework tend to omit employee health behaviors outside of the

workplace (Griffiths, 1999). According to NIOSH (2013), the most effective health and

safety promotion programs combine both work-related factors and health factors beyond

the workplace.

When taking total worker health into account, it is necessary to understand the

prevalence and impact of health issues among the workforce in order to illustrate how

important the topic of employee health is. First and foremost, obesity is a major health

concern among the working population. The CDC defines being overweight as a body

mass index (BMI) of 25 or higher and obesity is defined as a BMI of 30 or higher.

According to the OECD (2013), as of 2009, the United States leads the world in obesity,

with 33.8% of adults being categorized as obese. Not far behind the U.S. in obesity rates

were Mexico (30%), New Zealand (26.5%), Chile (25.1%), Australia (24.6%), Canada

(24.2%), and the United Kingdom (23%). In contrast, the least obese countries were

Korea (3.8%) and Japan (3.9%). Shockingly, at least 1 in 2 people are now overweight or

obese in half of the 32 countries surveyed by the OECD.

The high rates of obesity worldwide are cause for concern because obesity is

linked to many unfavorable health outcomes. According to the CDC (2012), overweight

and obese individuals are at an increased risk for several health problems such as heart

disease, type 2 diabetes, cancer, hypertension, stroke, and sleep disturbances. Obesity can

be caused by a person’s environment, genetics, diseases, drugs, or a combination of all

four.

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There are also occupational safety concerns for obese workers. Obese employees

suffer from increased occupational injuries compared to non-obese employees. Ostbye,

Dement, and Krause (2007) report that obese workers suffer from increased rates of

lower extremity pain and back pain, falls or slips, and injuries caused by exertion and/or

lifting. Polack et al. (2007) also found that employees who were classified as obese by the

body mass index scale were almost twice as likely to sustain traumatic workplace

injuries. Importantly, the combination of obesity and high-risk occupations that require

heavy lifting were found to be particularly detrimental to employee injuries (Ostbye,

Dement, & Krause, 2007).

Obesity is also a large financial burden for individuals, organizations, and

countries. Healthcare expenditures for obese people are 25% higher than for a person of

normal weight (OECD, 2013). Further, obesity is responsible for 1-3% of total healthcare

expenditure among OECD countries surveyed and 5-10% for the U.S. Interestingly,

obese people also earn up to 18% less in wages compared to people of normal

bodyweight (OECD, 2013). The reduced earning potential of obese employees is likely

linked to increased absences. For example, obese employees miss more work days due to

short-term absences, long-term disability, and premature death compared to non-obese

employees (Harvard School of Public Health, 2011). Obese employees suffer from

increased occupational injuries compared to non-obese employees as well. Finkelstein et

al. (2010) estimate that the average annual cost of obesity related productivity losses and

medical expenditure for employers is 73.1 billion dollars annually. Employers must also

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pay higher life insurance premiums and pay out more worker compensation for

employees who are obese (Harvard School of Public Health, 2011).

Specifically, obesity is expected to have a huge economic impact on the U.S. and

the U.K. Wang, McPherson, Marsh, Gortmaker, and Brown (2011) project that by 2030

there will be 65 million more obese adults in the U.S. and 11 million more obese adults in

the U.K. Obesity is predicted to cost 48-66 billion dollars annually for the U.S. and 1.9-2

billion dollars annually for the U.K. in medical costs associated with obesity. Given the

projected increase in obese individuals in the workplace, organizations will need to turn

to occupational health psychology for answers about how to improve employee health

and how to keep healthcare and insurance costs of employees to a minimum.

Another important factor to consider when promoting “total worker health” is the

mental health of the working population. The World Health Organization (2001)

estimates that approximately 450 million people worldwide suffer from a mental health

problem. Mental health disorders vary by country, but are a particular problem for the

U.S. According to the World Mental Health Consortium Survey (2004), the U.S. suffers

from the highest rates of anxiety and mood disorders among 14 countries surveyed. The

U.S. rates of anxiety disorders are 18.2% and 9.6% for mood disorders. Not far behind

the U.S. in anxiety and mood disorders are France (12%; 8.5%) and Lebanon (11.2%;

6.6%). In contrast, the countries with the lowest rates of anxiety and moods disorders are

China (2.4%) and Japan (5.3%). Although the United Kingdom was not included in the

Mental Health Consortium Survey, a study by Wiles et al (2006) found that 4.4% of the

U.K. population suffers from mental health problems.

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Importantly, poor mental health is associated with several negative outcomes,

which can impact an individual’s well-being. Complications associated with poor mental

health can include unhappiness, decreased enjoyment of life, social conflicts, self-harm or

harm to others, weakened immune system, heart disease, and other medical conditions

(Mayo Clinic, 2012). Risk factors for mental illness can include having a biological

relative with a mental illness, experiences in the womb, experiencing stressful life events,

use of drugs, and having a lack of social support (Mayo Clinic, 2012).

There are also occupational safety concerns for employees who have mental

health problems. Employees who suffer from poor mental health may not be able to cope

with job stress as well as mentally healthy people (Gabriel, 2000). Further, when

employees are unable to cope with job stress, they are at an increased risk for workplace

accidents and illnesses (Gabriel, 2000). Suzuki et al. (2004) report that occupational

errors are more prevalent among employees with poor general mental health compared to

employees who reported good general mental health. In addition, employees who take

medication for mental health problems do not anticipate that the side-effects of the

medication may make them feel worse initially. Consequently, the negative side-effects

of medication have been cited as a contributing factor to lowered work performance and

accidents (Haslam, Atknson, Brown, & Haslam, 2005). However, Gabriel (2000) notes

that workers with mental disorders vary in their response to stress. In addition, a good

match between the employees’ needs and working conditions can be considered more

important to employee health, more so than mental health conditions (Gabriel, 2000).

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The negative effects of mental health problems extend beyond the individual who

may suffer from a mental health disorder. Although the financial cost of mental disorders

can be difficult to precisely calculate, the estimated annual cost of mental illness is

approximately 2.5 trillion dollars worldwide (Bloom et al., 2011). The Agency for

Healthcare Research and Quality estimates mental healthcare costs approximately 57.5

billion dollars annually in the U.S (Soni, 2009). In comparison, the adverse effects of

poor mental health in the U.K. cost approximately 77 billion in terms of welfare benefits

and lost productivity at work (National Mental Health, 2011). The main economic burden

of mental illness for most countries does not stem from healthcare costs, but from a loss

of income due to unemployment and indirect costs due to chronic disability (National

Institute of Mental Health, 2013).

When examining the health of the working population it is also important to

review factors in the workplace that can impact employee health. Since employees spend

many hours at their job, an individuals’ work environment can potentially play a

significant role in their physical and mental health. In light of the fact that a growing

proportion of people are working indoors, in the post-industrial era, new workplace

hazards have shifted from an outdoor environment to indoor environments such as office

buildings (Jaakkola & Jaakkola, 2007). These work environments present employees with

new workplace hazards.

NIOSH (2013) states that several elements contribute to a healthy office

environment for employees. Attention to chemical hazards, office equipment, work-

station design, task design, and physical environment is needed. Physical environment

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factors can include, but are not limited to temperature, lighting, noise, and ventilation. In

addition, psychological factors can contribute to a healthy office environment. These

factors can include personal interactions with peers and supervisors, work pace, and job

control. Employees may develop stress and be prone to injury and illness if the demands

of the job exceed their psychological or physical abilities and resources (NIOSH, 2013).

NIOSH (2013) also describes several situations which can lead to injury or illness

in the workplace. For example, physical hazards such as leaving extension cords across

walkways or unsecure objects falling from an overhead shelf can compromise worker

health. Job-task related injuries can also occur, if the speed, repetition, or duration of an

activity exceeds one’s physical or psychological capabilities. Environmental hazards such

as exposure to chemicals or biological sources can also lead to acute or long term health

problems. Finally, design-related hazards can contribute to employee ill-health. For

example, non-adjustable furniture or equipment can be hazardous for employees who

have physical health problems such as chronic or acute lower back pain (NIOSH, 2013).

Workplace injuries are a serious and prevalent occurrence, which can be highlighted by

examining the rates of workplace accidents worldwide.

Worldwide accident rates are difficult to estimate because countries vary in their

workplace safety standards and accident reporting. According to Hӓmӓlӓinen, Saarela,

and Takala (2009), the worldwide total of occupational accidents has increased; however

fatality rates per 100, 000 workers has decreased. Worldwide, there were approximately

360, 000 fatal occupational accidents in 2003 and almost 2 million fatal work-related

diseases in 2002. Each day, more than 960, 000 people are injured on the job and each

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day 5, 330 people die due to work-related disease (Hӓmӓlӓinen, Saarela, & Takala, 2009).

On average, the African countries suffer from higher rates of workplace accidents and

injuries leading to fatalities whereas Western Europe tends to show lower rates of

workplace accidents and fatalities.

Although less developed countries tend to suffer from high levels of occupational

accidents, workplace accidents are a serious problem in developed countries as well.

According to the Bureau of Labor Statistics (2011), fatal workplace injuries in the U.S.

are on the decline, but the rates of fatalities in the workplace are still alarmingly high. In

2011, 4,609 workers were killed on the job in the U.S. The private sector industry

accounted for 4,188 fatalities in the workplace while the government sector accounted for

421 fatalities. Within the private sector, 738 fatalities were in construction (Bureau of

Labor Statistics, 2011). In addition, non-fatal workplace injuries in the U.S. among

private industry, state government, and local government lead to 1, 181, 290 days away

from work due to injury. Twenty percent of the days away from work due to injury were

concentrated among laborers, nursing aids, janitors/cleaners, truck drivers, and police

officers (Bureau of Labor Statistics, 2011). The incidence rate of non-fatal accidents

among private sector employees was 190 cases per 10 000 workers compared to 105 for

non-private sector employees (Bureau of Labor Statistics, 2011).

The United States is not the only economically developed nation that is plagued

by high rates of occupational accidents. Fatal workplace injuries account for 590 deaths

in the United Kingdom annually. In contrast to the U.S, a smaller number of fatal

workplace injuries were concentrated among construction workers, causing 49 deaths, at

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an incidence rate of 2.3 per 100,000 workers (Health and Safety Executive, 2011).

Further, the agriculture sector in the United Kingdom accounted for the highest incidence

of fatalities (33), at a rate of 9.7 per 100,000 (Health and Safety Executive, 2011). In

addition, an estimated 591,000 employees in the United Kingdom had a non-fatal

accident at work, leading to 368,000 separate cases of absences lasting longer than 3

days. Service industries accounted for the largest number of non-fatal major injuries,

accounting for 18,466 injuries (Health and Safety Executive, 2011).

Occupational accident rates are commonly used as an indicator of how safe a

work environment is for employees across industries and countries (WHO, 2001);

however caution should be taken when interpreting accident rate statistics. For example,

the reporting of accidents may vary by country (Hӓmӓlӓinen, Saarela, & Takala, 2009),

making it difficult to distinguish whether a region has low accident rates due to safe work

environments or an underreporting of accidents. For example, Hӓmӓlӓinen, Saarela, and

Takala (2009) note that the accident rates in China and India appear to be too low.

Considering both China and India have a high concentration of workers in agriculture and

construction, which are industries known to have higher accident rates, it is very plausible

that accident reporting is low in both countries.

In addition to the problem of underreported accidents, accident rate statistics also

fail to describe the underlying factors in the workplace that create an unhealthy or unsafe

work environment. For example, a workplace may have a high accident rate, but this

statistic does not explain why accidents are occurring. In order to fully understand how to

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increase employee well-being and safety it is necessary to appreciate how workplace

hazards affect the work environment and impact employee health.

Although definitions and groupings can vary, workplace hazards can be broadly

grouped into two categories; psychosocial and environmental hazards (Cox, 1993).

Psychosocial hazards can include job content, work pace, interpersonal relationships, and

job control. Environmental hazards can include a lack of working space, poor lighting,

heat exposure, and excessive noise (Leka & Jain, 2010). Psychosocial and environmental

hazards have been be linked to increased accident rates and absences from work due to

injury (Clarke, 2006; Oliver, Cheyne, Tomás, & Cox, 2002); however the occupational

stress produced by workplace hazards can also have mental and physical consequences

beyond accidents. For example, psychosocial hazards have been found to be negatively

related to mental health (Standsfeld & Candy, 2006). Further, environmental hazards

such as excessive noise levels have been linked to increased stress levels among

employees (Topf, 2000). A growing body of research also suggests that high levels of

environmental workplace hazards are linked to negative physical health outcomes such as

obesity and poor physical health (Schulte, Wagner, Downs, & Miller, 2008).

Although occupational stress is related to negative health outcomes such as poor

mental health, obesity, and absences (Burton et al., 1999; Stansfeld & Candy, 2006;

Torres & Nowson, 2007), there is research to suggest that physical activity can buffer the

negative effects of stress on health outcomes (Sonnentag & Fritz, 2007). Importantly,

occupational stress may result in negative health outcomes due to a lack of recovering or

psychological detachment from work (Craig & Cooper, 1992). Virtanen et al. (2009)

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suggest that occupational stress can be amplified if there is a lack of recovery after work,

which could take the form of physical activity during leisure time. Sonnentag (2001)

explains that leisure activities can play a factor in a person’s psychological recovery after

work. Leisure activities can include recreational activities that involve physical activity,

or simply doing physically demanding household chores. It is believed that physical

activities provide a temporary relief from job related demands, and allow the body’s

functional systems to recover (Sonnentag, 2001). Additionally, Yeung (1996) explains

that physical activity provides a cognitive distraction from job related activities.

Importantly, physical activity may buffer the negative effects that stress has on employee

health.

Physical activity alone may not be as effective in buffering the negative effects of

stress if an individual has a poor diet. High stress levels have been linked to unhealthy

eating habits, such as eating foods high in saturated fat (Ng & Jeffery, 2003). Payne,

Jones, and Harris (2005) argue that stress has a direct effect on eating behaviors, even

more so than the link between stress and exercise behaviors. People who feel stressed are

more likely to eat unhealthy foods, which can contribute to poor health. However, people

who maintain a healthy diet can prevent weight gain and chronic disease (Harvard School

of Public Health, 2013). Individuals who eat a healthy diet low in saturated fats and high

in lean meats, fruits, and vegetables tend to maintain a healthy body weight compared to

people who eat unhealthy diets (Harvard School of Public Health, 2013). Research also

suggests that individuals who maintain a healthy diet have stronger immune systems,

allowing a person’s body to better cope with stress and illnesses associated with stress

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(Venkatraman & Pendergast, 2002). Further, Jackson, Knight, and Rafferty (2010)

believe that positive health behaviors, such as eating a healthy diet can buffer the

negative effects that stress has on developing mental health problems

It is evident that obesity, poor mental health, occupational accidents and illnesses

have both financial and health implications for the individual employee and the

organizations that employs them (Marmot, Friel, Bell, Houweling, & Taylor, 2008).

Clearly, several factors come into play, which can contribute to an employees’ health.

Personal health problems such as suffering from poor mental health or being overweight

can contribute to decreased health and well-being. In addition, hazards in the workplace

such as psychosocial and environmental hazards can have a negative impact on employee

health and well-being. Using the framework set out by NIOSH (2013) it is essential that

both health behaviors and health hazards that occur within and outside the workplace be

considered when promoting total worker health.

Based on the total worker health framework proposed by NIOSH the purpose of

the current study is twofold. The first goal of this study is to extend past literature on the

antecedents of occupational stress, obesity, poor mental health, and absences due to

sickness. Specifically, the current study will examine how workplace hazards impact

employee stress, obesity, mental health, and workplace absences due to illness. The

second goal of this study is to determine whether physical activity and a healthy diet can

buffer the negative effects of workplace hazards upon the health outcomes stated above.

Importantly, it is vital for organizations to understand how employee well-being can be

influenced by health behaviors both within and outside the workplace in order to

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recognize how health can be promoted. Finally, the current paper will consider the

changing demographics of the workforce, in order to properly promote total worker

health. Given the aging workforce it is essential that employers develop strategies to

promote the health of older workers, who are at a higher risk for health problems and

injury (Houx & Jolles, 1993).

Currently, there is a gap in the literature with regards to how environmental

hazards affect psychological health outcomes. Numerous studies have examined how

environmental hazards can affect physical health outcomes such as muscular skeletal

problems (Magnavita et al., 1999); however less attention has been paid to how

environmental hazards can impact psychological outcomes such as general mental health

or acute psychological distress. In addition, few studies have examined both psychosocial

and environmental hazards as an antecedent to health outcomes. Finally, few studies have

given proper attention to the aging workforce, and how to best promote the health of

older workers (University of Iowa, 2009).

Another gap in the literature is that many studies have examined how exercise can

buffer the negative effects of occupational stress without recognizing other important

health behaviors (Steptoe, Wardle, Pollard, Canaan, & Davies, 1996). For example, there

is a need for studies to examine how a healthy diet can potentially buffer the negative

effects of occupational stress. In addition, the combination of a healthy diet and exercise

is known to improve health (Center for Disease Control, 2011), however, few studies

have examined how the combination of a healthy diet and exercise can affect a more

distal outcome in the workplace, such as absences due to illness.

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Finally, it appears that there is an overabundance of studies that have used U.S.

samples of participants when examining health outcomes such as obesity, mental health,

and illnesses. It is also important to examine the health behaviors and health outcomes of

different country populations to ensure that the information uncovered by health

promotion research is generalizable worldwide.

The current study will bridge the gap in the literature in several important ways.

First, the current study will go beyond examining the effects of environmental hazards on

physical health outcomes, and include an investigation of how environmental and

psychosocial hazards affect both physical and psychological health outcomes. Secondly,

the current study will improve upon past research by investigating how both exercise and

diet can buffer the negative effects of stress. Additionally, the current study will bridge

the gap in the literature by examining the antecedents of physical and mental health

outcomes among a non U.S. sample. A study based on a non U.S. sample is much needed

in the health research field, allowing researchers to understand how health behaviors

differ across cultures. Finally, the current study will take into consideration the changing

needs of an aging workforce for health and safety promotion.

Obesity

Before the antecedents and influences on specific health outcomes are examined,

it is necessary to understand the physiological underpinnings of the human body. Since

obesity is a major health risk among the workforce, a discussion of what obesity is and

how obesity affects the body is important. In addition, a review of the human body’s

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physiological processes is essential in understanding how obesity affects the body, and

how the environment and individual factors can affect obesity.

Overweight and obesity are defined as excessive fat accumulation which can

impair health (WHO, 2013). Both the Center for Disease Control and the World Health

Organization define someone as being overweight if they have a body mass index (BMI)

greater than or equal to 25. Further, anyone with a BMI equal to or greater than 30 is

considered obese. BMI is a common index of weight to height that is used to classify

adults as obese or overweight (WHO, 2013). BMI is calculated by dividing a person’s

weight in kilograms by the square of his/her height in meters (CDC, 2012). The WHO

(2013) states that BMI is useful because it is a population-level measure of overweight

and obesity, as it is the same for both sexes and for all adult ages. However, BMI should

be used with caution because the BMI numbers do not always correspond to the same

level of body fat for different people.

According to the WHO (2013) the main cause of being overweight or obese is due

to an energy imbalance between calories consumed and calories expended. When an

individual consumes more calories than they expend weight gain will result. Body weight

will decrease when an individual consumes less calories, increases energy expenditure

through physical activity, or a combination of both. The Harvard School of Public Health

(2013) explains that the body will store excess calories as body fat, which can accumulate

over time, resulting in weight gain.

There are several factors that can cause or contribute to obesity. The WHO (2013)

cites increased consumption of energy-dense foods, which are high in fat as a major

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contributor to weight gain. In addition, decreased physical activity due to the sedentary

nature of many forms of work and changing modes of transportation have contributed to

obesity. Further, the negative health behaviors people practice can interact with their

genetics, increasing the likelihood of being overweight or obese (WHO, 2013). For

example, someone who has a genetic predisposition to fat accumulation and eats an

unhealthy diet is at a greater risk for developing obesity compared to a person who does

not have a genetic predisposition to store fat but also eats an unhealthy diet.

There are several health consequences to being overweight or obese. Obesity is

the second leading cause of death in the U.S. after tobacco use (Harvard School of Public

Health, 2013). Obesity is linked to a long list of health conditions such as heart disease,

stroke, diabetes, high blood pressure, unhealthy cholesterol, asthma, sleep apnea,

gallstones, kidney stones, infertility, and 11 types of cancer. Further, obesity is linked to

social and emotional problems such as discrimination, lower wages, lower quality of life

and depression. Additionally, obese individuals are at an increased risk for developing

mental health disorders (Harvard School of Public Health, 2013).

Mental Health

Although obesity is linked to mental health problems such as depression (Harvard

School of Public Health, 2013), poor mental health is a serious health risk on its own. In

order to fully understand the severity of poor mental health as a health risk for the

working population, it is critical to review what mental health is and how it affects the

human body.

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According to the Center for Disease Control (2011) mental health is defined as a

state of well-being in which individuals realize their own abilities, can cope with the

normal stresses of life, can work productively, and are able to make a contribution to their

communities. Surprisingly, it is estimated that only 17% of the working population are

considered to be in a state of optimal mental health (CDC, 2011). Since approximately

83% of the population is not considered to be in “optimal mental health” it is important

for researchers and organizations to determine how to increase mental health to promote

total worker health (NIOSH, 2013).

The CDC (2011) states that there are 3 main indicators of mental health. The first

indicator of mental health is emotional well-being. Emotional well-being includes

perceived life satisfaction, happiness, cheerfulness, and peacefulness. The second

indicator of mental health is psychological well-being. Psychological well-being includes

factors such as self-acceptance, personal growth, openness to new experiences, optimism,

hopefulness, purpose in life, control of one’s environment, spirituality, self-direction, and

positive relationships. The third indicator of mental health is social well-being, which

includes social acceptance, beliefs in the potential of people, personal self-worth and a

sense of community.

Closely related to mental health is mental illness. Although both terms are often

used interchangeably, mental health and mental illness represent different psychological

states (CDC, 2011). Mental illness is defined collectively as all diagnosable mental

disorders or health conditions that are characterized by alterations in thinking, mood,

behavior, or a combination of all three conditions, associated with distress, and/or

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impaired functioning (CDC, 2011). The most common type of mental illness is

depression, which affects 26% of the U.S adult population.

Mental health disorders can be triggered by many factors. The Mayo Clinic

(2013) states that genetics can play a role in the likelihood of developing a mental health

disorder. People with biological relatives who have had a mental disorder are at an

increased risk for developing a disorder themselves. Certain genes can also increase an

individuals’ risk of developing a disorder, and stressful life situations can trigger these

genes, leading to a disorder. In addition, environmental exposures during birth can cause

mental disorders. For example, exposure to viruses, toxins, and drugs while in the womb

have been linked to mental illness.

The Mayo Clinic (2013) also cites negative life experiences as a risk factor for

developing a mental health disorder. Stressful life events such as the death of a loved one,

financial problems, and high stress can trigger mental illnesses. In addition, a poor

childhood upbringing resulting in decreased self-esteem or a history of sexual and

physical abuse can contribute to the development of a mental disorder. Specifically,

negative life experiences can lead to unhealthy patterns of thinking related to mental

illness, such as pessimism.

Mental disorders have been shown to be related to many chronic health problems

such as immune suppression, diabetes, cancer, cardiovascular disease, asthma, and

obesity. In addition, mental health disorders are strongly related to risk behaviors of

chronic disease, including physical inactivity, smoking, excessive drinking, and

insufficient sleep (CDC, 2011). Importantly, mental health problems and stress resulting

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from feeling lonely or depressed can suppress an individual’s immune system, which can

increase susceptibility to other health problems (American Psychological Association,

2006).

Immune System

Before the antecedents and influences on specific health outcomes are examined it

is necessary to understand the physiological underpinnings of the human body. The main

system in the human body that fights infection and disease is the immune system. The

National Institute of Allergy and Infectious Disease (2011) describes the immune system

as a network of cells, tissues, and organs that work together to defend the body against

microbes, bacteria, parasites, fungi, and viruses. The immune systems seeks out these

“invaders” and destroys them. The immune system is comprised of millions of cells that

communicate information back and forth when an infection is detected. Specifically, the

immune system contains certain types of white blood cells named B lymphocytes and T

lymphocytes (National Institutes of Health, 2013). B lymphocytes become cells that

produce antibodies, making it easier for the immune system to destroy harmful antigens.

T lymphocytes attack antigens directly and help control the immune response. Once the

immune system is alerted of an infection, powerful chemicals are produced and released

to attack the “invaders”. The substances produced by the immune cells allow the cells to

regulate their own growth and behavior, enlist other immune cells, and direct the newly

recruited immune cells to the spot of the infection (National Institute of Allergy and

Infectious Disease, 2011).

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Importantly, one major factor that can harm the immune system is stress. It is

believed that the mediating factor between stress and immune system suppression is the

stress hormone cortisol (Randall, 2011). Cortisol’s main function is to restore

homeostasis to the body following exposure to stress. Unfortunately, long term cortisol

secretion can block T lymphocytes from multiplying by preventing some T cells from

recognizing signals from other immune cells and can reduce the bodies’ inflammation

response. Cortisol’s ability to suppress the immune response can leave individuals

suffering from chronic stress and very vulnerable to infection (Randall, 2011).

Notably, people who are older or are already sick are more prone to stress-related

immune changes (APA, 2006). For example, individuals who suffer from mild depression

show signs of a suppressed immune system such as a weaker T lymphocyte immune

response, which is the bodies’ major defence mechanism against viruses and bacteria

(Glaser, McGuire, & Glaser, 2002). In addition, immune response significantly decreases

as the age of the individual increases. It appears that both sickness and age contribute to a

suppressed immune system (APA, 2006). Further, people who are both sick and older

suffer the greatest health problems with a suppressed immune system (Segerstrom &

Miller, 2004).

Physiological Processes of Stress

In light of the fact that stress can lead to immune suppression and can negatively

impact health, it is important to understand the underlying mechanisms behind stress in

order to determine how stress can be reduced. O`Leary (1992) explains that acute stress

results when an immediate situation or event is perceived as a stressor. Acute stress

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causes the activation of the autonomic nervous system (ANS). The ANS consists of two

branches; the sympathetic nervous system and the parasympathetic nervous system. The

sympathetic nervous system is responsible for physiological processes which increase

arousal and attention, including increased heart rate, dilated pupils, respiration, and the

release of adrenalin. These physiological changes were adaptive in the past when humans

had to survive in the wilderness. These processes helped humans survive by preparing the

body to “fight or flee” a potential threat or predator. The parasympathetic system is

designed to bring back homeostasis to the ANS, by returning physiological processes

back to a normal rate; decreased heart rate, slower rates of respiration, and reduced levels

of adrenalin (O`Leary, 1992).

Long-term stress results when the amount or frequency of experienced stressors

are too great for the parasympathetic system to stabilize. Long-term stress can cause

excessive secretion of epinephrine, norepinephrine, and can suppress the immune system

(O`Leary, 1992). Stress activates the sympathetic nervous system and the HPA

activation-adrenomedullary system. Cortisol levels also take longer to return to normal

among stressed individuals (O`Leary, 1992). Long-term stress can lead to reduced well-

being, insomnia, nightmares, and disturbing dreams (The American Institute of Stress,

2011).

So far, I have discussed the effects of stress on various health outcomes, including

obesity, mental health, and illnesses. Although there are many different antecedents to

stress; the main precursor to experienced stress is the individuals’ perception that an

event or situation (whether acute or chronic) is perceived as a threat (O’leary, 1992).

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There are many different factors in a person’s environment that may be perceived as a

threat and lead to stress. Therefore, it is important to identify specific threats and stressors

in an individual’s environment in order to help reduce and/or remove a potential stressor

from the workplace.

It is necessary to go beyond a general framework for stress and investigate how

the environment that a person spends most of their waking life in (work) can affect health

and well-being. Specifically, the investigation of how hazards in the workplace affect

employee stress levels is important for several reasons. First, the ability for organizations

to identify and remove workplace hazards can have a large impact on employee health

and well-being. Further, healthy employees tend to incur less health care costs for

employers and healthy employees also tend to be more productive (CDC, 2011).

Workplace Hazards

Before presenting an in-depth analysis about how workplace hazards affect

individual and organizational outcomes it is important to clearly define the meaning of

workplace hazards. Researchers have given differing interpretations about what

workplace hazards are and how to group them. Cox (1993) explains that work hazards

can be broadly grouped into two categories; physical and psychosocial hazards. Physical

hazards can include biological, biochemical, chemical, and radiological hazards. Physical

hazards have also been labelled as environmental/ambient factors in some instances. For

example, the International Labor Office (ILO, 2001) defines a hazard as an ambient

factor that has the potential to cause harm, illness, or injury as a result of exposure.

Hazardous ambient factors include excessive noise, heat, or exposure to harmful

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chemicals or toxins (ILO, 2001). In addition, Taylor, Baldry, Bain, and Ellis (2003) group

physical hazards into proximate environment (work technology, work station design) and

ambient environment (work building, lighting, temperature, air quality, and acoustics).

Further, the World Health Organization (WHO, 2010) labels environment and equipment

hazards as inadequate equipment availability, suitability or maintenance, poor

environmental conditions such as lack of space, poor lighting, and excessive noise. It is

important to note that the WHO (2010) groups environment and equipment hazards into

the category of psychosocial hazards. Rugulies et al. (2007) advocate that physical

aspects of the work environment (e.g. noise, exposure to heat) do not constitute

psychosocial work hazards and should be treated separately, even if these exposures have

psychological effects. It is also important to point out that the WHO’s environmental

hazards are very similar to other researchers’ definition of physical and/or environmental

hazards, which are often labelled as distinct from psychosocial hazards.

In contrast psychosocial hazards may be considered a broader domain than

physical/environmental hazards. For example, the International Labor Office (ILO)

(1986) defines psychosocial work hazards as the interactions among job content, work

organization and management, environmental, organizational conditions, employee

competencies, and needs. Cox (1993) offers a slightly different definition of psychosocial

hazards, which are described as aspects of job content, work organizational management,

environmental, social, and organizational conditions which can cause psychological or

physical harm. The WHO argues that there is reasonable consensus in the literature about

the nature of psychosocial hazards. The WHO (2010) groups psychosocial hazards into

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10 categories; job content, workload and work pace, work schedule, control, environment

and equipment, organizational culture and function, interpersonal relationships at work,

role in organization, career development, and home-work interface.

Workplace Hazards and Stress

Workplace hazards can affect both psychological and physical health (WHO,

2001). Given that both psychosocial and physical workplace hazards can affect an

individual’s health it is important to point out that most studies examining the hazards-

stress-health relationship have omitted physical work hazards (Cox, 1993). Importantly,

the psychological effects of physical hazards can be as harmful as the direct physical

harm that a hazardous chemical can have on the human body. An employee may be

aware, suspect, or fear that they are being exposed to a harmful hazard, which can induce

psychological stress (Cox, 1993). For example, exposure to harmful fumes may have a

psychological effect on a worker through the direct effects on the brain, as a result of the

unpleasant smell of fumes and through the workers fear that such exposure may be

harmful (Cox, 1993). Therefore, occupational stress and health outcomes can be focused

on 2 main areas; stress and health outcomes associated with physical workplace hazards

and the stress and health outcomes associated with exposure to psychosocial workplace

hazards.

Physical Hazards and Health

According to the Fifth European Working Conditions survey, employees are

exposed to physical work hazards at a similar rate as 20 years prior. It is alarming that

there has not been a decrease in physical hazards exposure among workers over the past

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20 years, despite advances in technology and increased awareness of workplace hazards.

According to the Fifth European Working Conditions survey (Eurofound, 2010), 16% of

employees are subject to tiring and uncomfortable positions during their entire work day,

33% carry heavy loads at least a quarter of their working day, and 23% of employees are

exposed to vibrations. Although many employees still do work that involves physical

labor, physical workplace hazards are not limited to manual laborers and blue-collared

workers. Employees in the rapidly expanding service industry also carry out physical

work. For example, cashiers carry out repetitive movements during most of their working

time. Further, high work intensity in all work occupations can reduce the likelihood that

employees will use protective equipment as well as ergonomic devices that can alleviate

some physically demanding aspects of work (Eurofound, 2010).

Overall, research suggests that poor physical working conditions can affect both

an employee’s experience of stress and their psychological and physical health (WHO,

2010). Results from the Fourth and Fifth European Working Conditions survey

(Eurofound, 2007; Eurofound, 2010) indicate that employees with high levels of exposure

to physical hazards are more likely to report that their health is at risk as a result of their

work. An important study conducted by Lu (2008) discovered that the top five physical

hazards among employees were ergonomic hazards (72.2%), heat (66.6%), overwork

(66.6%), poor ventilation (54.8%), and chemical exposure (50.8%). The most common

illnesses reported were gastrointestinal problems (57.4%), backache (56%), headache

(53.2%), and fatigue/weakness (53.2%). Most physical work hazards can be objectively

measured, fairly reliably and validly and are therefore easily monitored in the workplace

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(Cox, 1993). Further, in some cases standards exist for a minimum level of exposure to

physical hazards such as noise levels in the workplace (Canadian Center for Occupational

Health and Safety, 2011).

Physical workplace hazards have been shown to affect rates of employee sickness

and injury. Lund et al. (2006) examined the effects of physical work environment on

sickness absences among employees in Denmark. For both male and female employees,

sickness absences were increased by bending or twisting of the neck and back, working

standing and/or squatting during most of the work day, lifting or carrying heavy loads,

and pushing or pulling heavy loads. Physically strenuous work environments can lead to

musculoskeletal disorders, especially among employees who work in the construction and

agriculture sector (Lee, Yeh, Chen, & Wang, 2005). Among the most prevalent

musculoskeletal injuries are neck, shoulder, hand, upper back and elbow injuries. Further,

physical working conditions are a strong predictor of upper extremity disorders in

manufacturing and service industries (Lee et al. 2005).

Taylor, Baldry, Bain, and Ellis (2003) also argue that ambient environmental

factors such as the work building, lighting, temperature, air quality, and acoustics can

have a negative impact on employee health (Stolwijk, 1991). Among employees surveyed

by Taylor et al. (2003), physical hazards thought to be responsible for increased sickness

absences included variable temperature conditions and poor ventilation. Ambient factors

were also stronger predictors of sickness absences compared to proximate factors, e.g.

cardiovascular health. Importantly, for some physical hazards it is the extremes of

physical work conditions which are associated with the experience of stress and negative

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health outcomes (Cox, 1993). For example, employees can often adapt to mid-range

conditions without effort or attention; however extremely cold or hot conditions may

induce discomfort and stress (Cox, 1993). In the case of other hazards, the simple

presence of the hazard or perceived threat of its presence can induce stress. For example,

doctors and nurses report anxiety when dealing with patients who might be affected with

HIV (Dworkin, Albrecht, & Cooksey, 1991).

The physical layout of a physical work environment can also affect employee

health. Croon, Sluiter, Kuijer, and Frings-Dresen (2005) conducted a literature review

about the effects of office layout on worker health and performance. Croon et al. (2005)

found that working in open-concept workplaces reduced privacy and job satisfaction. In

addition, Pejtersen et al. (2006) examined the effects of the indoor physical climate on

employee health. The sample consisted of office employees distributed among naturally

ventilated and mechanically ventilated office buildings. Five of the office buildings had

open-plan offices and eight of the office buildings had a mixture of cellular, multi-person,

and open plan offices. The results indicated that employees who worked in the open-

office environments were more likely to perceive thermal discomfort, poor air quality and

noise. Employees who worked in the multi-person offices and cellular offices reported

more complaints about central nervous system health issues and mucus membrane

symptoms.

Physical hazards are not the only kinds of workplace hazards that can impact

employee health. Not only can psychosocial hazards affect the level of response that an

individual may have to physical hazards (Stolwijk, 1991), psychosocial hazards can

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impact employee health directly (Eurofound, 2011). Subsequently, it is necessary to

review psychosocial hazards in the workplace in order for researchers and employers to

understand how to improve employee health and reduce employee exposure to workplace

hazards.

Psychosocial Hazards and Health

According to Eurofound (2011) psychosocial stressors in the workplace are on the

rise. The most prominent psychosocial workplace hazards are stress (62%), bullying and

harassment (37%) and overwork (29%). The rise in psychosocial hazards can be

attributed to the arrival of bullying and harassment as the second most common hazard.

According to Eurofound (2011) reports of bullying and harassment have almost doubled

since 2008, with a rise from 20% to 37% in 2010. Psychosocial work hazards are also

more common in larger workplaces. For example, stress and bullying/harassment have

shown dramatic increases among organizations with over 200 employees and even

steeper increases among organizations with over 1,000 employees. Stress and

bullying/harassment are also more common in the public sector compared to the private

sector. However, the incidence of overwork is similar for both sectors (Eurofound, 2011).

According to Leka and Jain (2010) there is reasonable consensus among

researchers that psychosocial hazards in the workplace which are experienced as stressful

have the potential to cause significant harm to employees. Leka and Jain (2010) argue

that several different forms of psychosocial workplace hazards can cause significant

damage to employee health and well-being. According to Leka and Jain (2010) there are

several psychosocial factors that can lead to poor health; job content, workload and work

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pace, work schedule, control, interpersonal relationships, organizational function and

culture, workplace harassment, lack of career development opportunities, and home-work

interface.

Psychosocial hazards may affect both physical and psychological health directly

or indirectly through the experience of stress. Most attention has been given to the

possible indirect, stress mediated effects of psychosocial hazards on health (Cox, 1993).

Work situations are experienced as stressful when they are perceived as involving

important work demands which are not well matched to the knowledge and skills of

employees (WHO, 2010). Research has shown a link between occupational stress and

increased risk of health problems such as heart disease, depression and musculoskeletal

disorders (WHO, 2010). Job strain has also been linked to negative outcomes such as

migraines, psychological distress and work injury (Wilkins & Beaudet, 1998).

Studies that have examined the relationship between physical hazards and health

tend to be grouped by the specific physical hazard. In contrast, research that focuses on

psychosocial hazards in the workplace appear to be grouped by the health outcome,

grouping several psychosocial hazards together. For example, some studies have

examined how psychosocial hazards can affect mental health and functioning (Stenfors et

al., 2013) while other studies have chosen to focus on sickness absences (Lund et al.,

2005) or somatic health complaints (Wilkins & Beaudet, 1998) as an outcome.

The current literature provides support that psychosocial hazards and occupational

stress resulting from exposure to workplace hazards can lead to poor health (WHO,

2010). Subsequently, it is not surprising that researchers have investigated the link

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between psychosocial hazards in the workplace and sickness absence among employees.

According to Lund et al. (2005) psychosocial factors in the workplace are predictive of

sickness absences among women and men. Nielsen et al. (2006) has also found a link

between psychosocial hazards in the workplace and sickness absence. In addition,

Niedhammer, Chastang, and David (2008) recently examined the contribution of

psychosocial work hazards to poor health using data from the National French SUMER

survey. The study revealed that low levels of decision latitude and social support and high

psychological demands were predictive of poor self-reported health and reported

sickness. Sickness absence has been used as an indicator of health in many studies

(Kivimäki et al., 2003); however sickness absences can be considered a distal indicator of

health. Health complaints and symptoms are a more proximal indicator of health since

health complaints stem directly from a health problem.

Psychosocial hazards have also been linked to various health complaints,

including migraines, sleeping problems, and stomach symptoms (Asa, Brulin, Angquist,

& Barnekow-Bergkvist, 2005). Further, psychosocial hazards such as low co-worker

support have been found to be related to general health complaints among men and work

injury and psychological distress among women (Wilkins & Beaudet, 1998). Workplace

bullying and harassment are also strong predictors of psychological and somatic health

complaints (Mikkelsen & Einarsen, 2002). Some studies have also found that

psychosocial hazards in the workplace can lead to health problems such as reported

musculoskeletal pain (Sembajwe et al., 2013). Lack of supervisor support was the

strongest predictor of musculoskeletal pain among the healthcare workers surveyed by

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Sembajwe et al. (2013). Psychosocial hazards, including low job control, high job

demands, and low work-related social support also predict quality of life (Cheung,

Kawachi, Coakley, Schwartz, & Colditz, 2000).

Psychosocial hazards in the workplace can also lead to mental and cognitive

health problems. Boschman et al. (2013) examined the relationship between psychosocial

work environment and mental health complaints among blue-collar workers.

Psychosocial hazards such as a lack of job control, few learning opportunities and low

future prospectives were related to a need for recovering after work, distress, depression,

and post-traumatic stress disorder. Stenfors et al. (2013) also found evidence that

psychosocial hazards in the workplace lead to higher rates of cognitive complaints such

as problems with concentration, memory, decision-making, psychiatric problems and

sleeping problems. It is evident from the current literature review that psychosocial

hazards in the workplace can affect both psychological and physical health.

The Current Study

The current study will examine the effects of both physical workplace hazards and

psychosocial workplace hazards on health outcomes among a sample of civil servant

workers from Northern Ireland. Specifically, the effects of physical and psychosocial

workplace hazards on absences due to sickness, BMI, and general mental health will be

examined. Refer to Figure 1, below for a visual representations of the study’s model.

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Figure 1

The Current Study

For the current study, workplace hazards were grouped into two distinct

categories based on the recommendations of Cox (1993); physical workplace hazards and

psychosocial workplace hazards. For the present study, the definition of physical hazards

will be based on descriptions set forth by Cox (1993); biological, biochemical, chemical,

and radiological hazards which can cause injury, illness, or death. In addition,

characteristics of office building design and layout, which can potentially impact

employee health will be included in the definition of physical work hazards, as described

by the WHO (2010). Examples of physical workplace hazards can include a lack of

space, poor lighting, poor ventilation and excessive noise. For the current study

psychosocial hazards will be based on the definition stated by Cox (1993); aspects of job

Physical Hazards

Psychosocial Hazards

BMI

Mental Health

Sickness Absences

Physical

Activity

Age

Diet

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content, work organization and management and of social and organizational conditions

which have the potential for physical and psychological harm. Specifically, psychosocial

hazards include organizational function and culture, role in organization, career

development, decision latitude/control, interpersonal relationships at work, home-work

interface, task design, workload/work pace and work schedule (Cox, 1993).

Much of the current discussions about the hazard-stress-health relationships have

primarily focused on psychosocial hazards in the workplace while giving less attention to

physical hazards in the workplace (Cox, 1993). Further, there is a need for researchers to

explore and compare the differential negative health consequences between physical and

psychosocial hazards in the workplace. For example, physical hazards may directly

impact the human body by exposure to harmful substances. In addition, physical hazards

may also indirectly affect the body by means of psychological stress induced by an

individual worrying about the potential health consequences of exposure to a harmful

substance (Cox, Griffiths, & Rial-Gonzalez, 2000). In contrast, psychosocial hazards may

not cause a direct physical health problem, but may lead to health problems due to

symptoms related to psychological distress. Since both physical and psychosocial hazards

can negatively affect health via multiple processes, it is necessary to examine whether

physical and psychosocial hazards are equally troublesome for employee health.

Identifying which types of workplace hazards are most detrimental to employee health is

necessary for organizations and policy makers to promote strategies to best protect and

promote total worker health.

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Further, there is a lack of research examining how physical hazards can impact

psychological outcomes such as mental health and how psychosocial hazards can impact

physical health outcomes. Importantly, the present study will advance the current

literature and investigate how physical hazards impact mental health, after controlling for

psychosocial hazards. Further, the relationship between psychosocial hazards and

physical health outcomes will be examined, while controlling for psychosocial hazards.

The strength of this study is that controlling for physical or psychological hazards allows

for incremental validity; examining whether psychosocial hazards predicts health

outcomes above and beyond physical hazards, and whether physical hazards predicts

health outcomes above and beyond psychosocial hazards.

There is also a need for occupational health psychology researchers to integrate

the use of self-report data and more objective measures of health, such as BMI or

employee sickness absence records. Some researchers argue that self-report measures,

which are a common measurement technique within the field of organizational

psychology are plagued by biases of the participant (Spector, 1994). To counteract the

potential effects of respondent bias many researchers prefer to use non-self-report

measures of health, arguing that many of the biases can be controlled (Spector, 2006).

However, in some instances self-report data is much less labor intensive, and can be a

valuable tool. For example, it could be argued that absences due to sickness or BMI are

more accurate measures of health because the information is based on objective

measures. It is important to note that if the data used to calculate BMI or absences is

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based on self-report data, it can still be a nice addition to a study that only uses

psychological measures of health such as self-reported well-being.

Spector (1994) explains that the benefit to using objective measures is that

“facts” can be checked for accuracy. Further, self-report data that is based on a physical

measurement can also be checked for accuracy. For example, an employee may report

what their height and weight are (indicators of BMI); therefore, measures of body fat can

be checked to confirm the accuracy of the statements if needed. Self-report measures are

also very useful when the intent of the research is to capture a construct which is

expected to be comprised of perceptions, such as employee perceptions of safety climate

(Cooper & Phillips, 2004). Further, relying on one form of measurement can lead to

method variance biases (Spector, 1994). For example, self-reports of physical health may

be subject to social desirability, with people not wanting to admit they are unhealthy

(Spector, 2006). If, however body fat percentage is measured, the effects of social

desirability are removed from the equation. The current study will incorporate both self-

report measures of psychological health and self-report measures of physical health.

Thus far, the current paper has reviewed the effects of physical and psychosocial

hazards on the general health of individuals. It is important to understand the many ways

in which physical and psychosocial hazards can impact the general health of the working

population. Knowledge about workplace hazards can allow employers to identify hazards

and improve employee health. To further help organizations and employees understand

how workplace hazards can impact employee health it is also necessary to narrow the

scope of investigation to specific health outcomes. Focusing on specific health outcomes

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can allow for an understanding about how both physical and psychosocial hazards

directly affect different aspects of health. In addition, understanding the specific health

consequences associated with workplace hazards aids organizations and policy makers in

the development of workplace hazard regulations to protect and promote total worker

health.

Workplace Hazards and Obesity

One area of research that needs to be expanded is the investigation of how

workplace hazards can affect employee body fat percentage and obesity. Little research

has specifically examined the effects of work conditions on BMI (Schulte et al., 2007).

Further, few studies have explored obesity and work hazards together, in order to develop

public health strategies targeted at reducing obesity. Schulte et al. (2008) argue that there

is a need for researchers to investigate the relationship between workplace hazards and

obesity; while seemingly independent, occupational hazards and obesity can be, and often

are interrelated, although most studies related to occupational hazards and obesity were

not designed to test the presence of effect modification. Schulte et al. (2008) also point

out that workplace exposures can lead to obesity and obesity can modify important

outcomes such as morbidity and mortality.

Within the literature, studies tend to examine the effects of physical workplace

hazards on the health of obese individuals, who are at an increased risk of injury. For

example, Maeda, Kaneko and Ohta (2006) found that obesity is a risk factor for heat

stress among employees, with obese individuals more likely to suffer from heat stress

compared to non-obese individuals. Exposure to respirable workplace contaminants such

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as dust, solvents and irritants may also increase the prevalence of obesity among men

with certain blood types or long-term occupational exposure (Suadicani, Hein, &

Gyntelberg, 2005). Individuals with high fat diets have also been found to accelerate the

development of plaque in the circulatory system following exposure to harmful

substances such as carbon nanotubes (Li et al., 2007). Vibration from work equipment is

another physical workplace hazard for obese employees. Wieslander, Norbäck, Göthe and

Juhlin (1989) reported that damage to the body resulting from obesity may compromise

muscular, neural and vascular tissues, making the body more susceptible to vibration-

induced injuries.

Studies that have examined the association between physical hazards and obesity

have mostly been limited to obese employees being at a greater risk for suffering injuries

due to physical workplace hazards. In contrast, studies that have examined the link

between psychosocial hazards and obesity have focused on how psychosocial hazards and

associated stress can lead to obesity. A recent study by Lallukka et al. (2008) revealed

that psychosocial hazards and job strain were positively related to BMI among a sample

of civil servant workers from the WhiteHall II study. In addition, Ostry, Radi, Louie and

LaMontagne (2006) found an association between psychosocial work conditions and

BMI among a sample of Australian blue-collar and white-collar workers. High exposure

to psychosocial demands were related to increased BMI across both men and women.

Moreover, Kivimaki et al. (2003) found that employees who face psychosocial hazards

such as workplace bullying have an average of 1 unit higher BMI compared to employees

who do not suffer from workplace bullying. A 1 unit increase in BMI is a serious health

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concern because even a small increase in BMI can lead to health complications such as

coronary heart disease (Kivimaki et al., 2006).

Many studies have also used the job-demand-control model as a framework for

examining the relationship among psychosocial hazards and obesity. Hellerstedt and

Jeffery (1997) showed a positive relationship among high work demands, low job control

and BMI. In addition, Martikainen and Marmot (1999) found that psychosocial hazards

based on the job-demand-control framework were related to increased incidence of

obesity among employees. Another study conducted by Netterstrom et al. (1991)

surveyed a sample of Danish employees and found that job strain resulting from

psychosocial hazards such as high demands and low control over work was related to

increased incidence of weight gain.

Yamada et al. (2007) hypothesized that work could facilitate weight gain and

obesity in 3 major ways. First, jobs stress could impact health behaviors such as alcohol

consumption and sedentary leisure activities that are related to weight gain. Secondly,

psychological strain could lead to modification of endocrine factors related to weight

gain. Thirdly, overwork could result in fatigue and inhibit behaviors that prevent weight

gain and fat accumulation. Importantly, all three risk factors for obesity described by

Yamada et al. (2007) involve some form of occupational stress, which then leads to poor

health habits or negative physiological changes to the body, which in turn can lead to

negative health outcomes such as weight gain.

Researchers argue that the occupational stress resulting from employee exposure

to physical and psychosocial hazards may help explain why workplace hazards can lead

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to increased obesity levels (Ostry, Radi, Louie, & LaMontagne, 2006). Cox (1993)

explains that workplace hazards can lead to occupational stress; the psychological effects

of physical hazards can be as harmful as the direct physical harm that a hazardous

chemical can have on the human body. An employee may be aware, suspicious, or fear

that they are being exposed to a harmful hazard, which can induce psychological stress.

In addition, it is well documented that physical workplace hazards increase occupational

stress (WHO, 2010).

The current study will investigate how physical and psychosocial hazards in the

workplace are related to BMI. There is research linking stress and eating habits to BMI

(Harvard School of Public Health, 2013), however few studies have specifically

examined the link between workplace hazards and BMI. Additionally, most studies that

have looked at physical hazards and obesity have focused on obese employees being at an

increased risk for injury. There is a need for more research on the link between physical

hazards and the development of obesity. Further, researchers need to examine how not

only stress, but psychosocial hazards can contribute to BMI. Examining the effects that

workplace hazards have on BMI is important because employees who are exposed to

workplace hazards will likely suffer from greater rates of obesity. For example,

employees who face many physical and psychosocial hazards in the workplace will

perceive greater levels of stress, likely resulting in unhealthy behaviors and increased

BMI. The current study will bridge the gap in the literature and examine the association

between workplace hazards and BMI. This leads into the first hypotheses of the present

study.

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Hypothesis 1a. Physical workplace hazards will be positively related to BMI.

Hypothesis 1b. Psychosocial workplace hazards will be positively related to

BMI.

As previously discussed, focusing on specific health outcomes is a powerful tool

for determining how both physical and psychosocial hazards affect different aspects of

health. Thus far the effects of physical and psychosocial hazards upon the general well-

being of employees have been examined. The specific effects of physical and

psychosocial hazards upon obesity have also been reviewed in this paper. Additionally,

another important health outcome to focus on is the mental health of employees. Mental

health can impact an individuals’ physical and mental well-being (WHO, 2010). Further,

workplace hazards can negatively impact employee mental health (Cox, Griffiths, &

Leka, 2005).

Psychosocial Hazards and Mental Health

Psychosocial hazards can greatly affect an individuals’ mental health and well-

being. Occupational stress, depression, and anxiety can be directly linked to the exposure

of psychosocial hazards in the workplace (Cox, Griffiths, & Leka, 2005). As previously

discussed in the current paper, researchers have defined and grouped psychosocial

hazards in different ways. Across research findings work characteristics such as lack of

job control, low decision latitude, low skill discretion, and job strain have been associated

with the risk of depression, poor health functioning, anxiety, distress, and fatigue (WHO,

2010).

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Stansfeld and Candy (2006) conducted a comprehensive meta-analysis on the

psychosocial work environment and mental health. Eleven articles met the inclusion

criteria. Stansfeld and Candy (2006) included several psychosocial categories in their

analyses; decision authority, decision latitude, psychological demands and work social

support, in addition to components of the job-strain model and effort-reward imbalance

model. The results of the meta-analysis revealed that job strain, low decision latitude, low

social support, high psychological demands, effort-reward imbalance and high job

insecurity predicted common mental disorders.

Further, a longitudinal study conducted by Stansfeld, Fuherer, Shipley, and

Marmot (1999) surveyed a sample of UK men which set out to understand the casual

relationship between work characteristics and psychiatric disorders. The results of the

study indicated that demands at work increased the risk of psychiatric disorders while

social support and high decision authority decreased the risk of psychiatric disorders. In

addition, high efforts and low rewards were associated with an increased risk of

developing psychiatric disorders.

Studies have also examined the effects of psychosocial hazards on general mental

health, using the well validated General Mental Health Questionnaire (GHQ12).

Recently, Laaksonen, Rahkonen, Martikainen, and Lahelma (2006) investigated the

effects of psychosocial work hazards on general mental health, using the GHQ12

questionnaire as a measure of mental health. Psychosocial work hazards such as job

demands, lack of job control, unfair organizational practices and workload were

examined. The results confirmed that all psychosocial hazards, excluding unfair

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organizational practices independently predicted general mental health. Niedhammer et

al. (2006) also examined the effects of psychosocial hazards upon general mental health

and depressive symptoms. Psychosocial hazards examined in the study included job

strain, low decision latitude, lack of social support, and over-commitment. The GHQ12

was used to assess general mental health. The results demonstrated that each psychosocial

hazard predicted general mental health and depressive symptoms.

Another important study by Rugulies, Bultmann, Aust, and Burr (2006) examined

the effects of psychosocial work characteristics on depressive symptoms among a sample

of the Danish workforce between 1995 and 2000. The result revealed that women with

low levels of influence at work and low social support were at the greatest risk for

experiencing depressive symptoms. Among men, job insecurity predicted severe

depression symptoms. It is important to point out that many of the studies reviewed thus

far use the term “psychosocial characteristics” or “psychological demands”, even though

the psychosocial factors examined would fall under the category of psychosocial hazards

according to Cox (1993).

Given the abundant amount of literature linking psychosocial hazards to mental

health, it is clear that investigating this relationship may not add much new and valuable

information to the scientific literature. Although it is important to review which kinds of

workplace hazards impact mental health, it is also important to focus on relationships that

have been investigated less often and can make the greatest contribution to the literature;

the link between physical hazards and mental health.

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Physical Hazards and Mental Health

Research suggests that physical hazards in the workplace can have a negative

impact on mental and cognitive outcomes such as anxiety, depression, distress, decision

making, and attention (Cox, Griffiths, & Rial-González, 2000). Although there is a

plethora of research concerning the effects of psychosocial hazards on mental health

(WHO, 2010), there is much less research investigating the effects of physical workplace

hazards upon mental health. Cox (1993) argues that physical workplace hazards can

directly and indirectly affect psychological health. For example, exposure to harmful

substances such as metals, pesticides and solvents can change brain chemistry, and

actually induce temporary or long-term mental health problems (Canadian Environmental

Law Association, 2011). In contrast, worrying about exposure to physical hazards can

induce stress (Cox, 1993).

Many studies that investigate the link between physical workplace hazards and

mental functioning have focused on the physiological effects of hazards on psychological

health. For example, physical hazards such as heat exposure have been linked to reduced

cognitive functioning as a result of dehydration directly affecting the brain (Cian et al.,

2000). As previously discussed, exposure to harmful chemicals and substances can

directly affect brain chemistry, resulting in symptoms such as anxiety, irritability and

depression (Canadian Environmental Law Association, 2011). Less attention has been

given to psychological effects that chemicals and toxins can have on behavior (Evans,

2003). For example, over-exposure to lead can impede self-regulatory behavior in

children, which is then related to behavioral conduct disorders such as fighting and

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aggression (Sciarillo, Alexander, & Farrell, 1992). Further, some behavioral reactions to

chemicals/toxins are caused by the psychological trauma associated with threats to

personal health (Evans, 2003). Individuals who have discovered that they have been

exposed to hazardous materials can suffer from psychological distress and post-traumatic

stress disorders in some instances (Edelstein, 2002).

There have been some important advances in the research area concerning the

effects of environmental characteristics upon mental health. Research has examined how

the built environment (e.g. housing, office buildings) can affect mental health. According

to Evans (2003) environmental characteristics that can directly affect mental health

include housing, crowding, noise, indoor air quality and lighting. The built environment

can also indirectly affect mental health by altering psychosocial conditions which are

known to alter mental health. For example, higher residential density can interfere with

the development of socially supportive relationships within a household, reducing social

support and increasing psychological distress (Evans, 2003).

Studies have examined how high-rise buildings and floor level can affect mental

health. Evans, Wells, and Moch (2003) found that high-rise housing can lead to elevated

psychological distress. It is believed that high-rise buildings can lead to social isolation if

the building does not have sufficient space to aid in the development of interaction with

neighbors, such as patios or backyards (Evans, 2003). Further building quality such as the

structural quality of the building, maintenance, upkeep and physical hazards have been

linked to mental health (Halpern, 1995). Although many of the studies mentioned above

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were conducted on the home environment, it is appropriate to predict that the building a

person works in everyday would also affect mental health.

Research has also investigated how noise can impact mental health, although most

studies concerning noise and mental health have been limited to airport noise exposure or

non-occupational settings (Evans, 2003). Early studies have supported that there is a

positive relationship between aircraft noise exposure and an increased incidence of

psychiatric admissions (Evans, 2003). Further, a study conducted by Lercher, Evans,

Meis, and Kofler (2002) revealed that noise pollution in neighborhoods can increase

psychological distress. Exposure to excessive noise has also been linked to an increase in

psychotropic drug use among adults (Knipschild & Oudshoorn, 1977).

Lack of sufficient light is a physical workplace hazard which can impact mental

health. Levels of illumination, specifically the amount of daylight an individual is

exposed to can impact psychological wellbeing (Evans, 2003). Importantly, seasonal

affective disorder (SAD) is a form of depression that is linked to the amount of daylight

exposure a person is exposed to (Mayo Clinic, 2013). Individuals who are chronically

exposed to shorter hours of daylight suffer more sadness, fatigue, and clinical depression

in some instances. Although there is less research investigating illumination levels in

occupational settings, studies examining the link between daylight exposure and mental

health among the general population have been fruitful. Research has shown that hospital

patients recover more quickly when in a brightly lit room (Beauchemin & Hayes, 1996)

and school children with insufficient exposure to daylight suffer from more behavioral

problems (McColl & Veitch, 2001). Importantly, employees who suffer from SAD can be

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aided by properly lit work areas. Mills, Tompkins, and Schlangen (2007) report that

employees suffering from SAD can increase mental health, productivity, and alertness by

working in a well-lit office area.

The research reviewed thus far has indicated that physical hazards both within and

outside of the workplace can negatively impact mental health and well-being (Evans,

2003). Physical hazards such as chemicals or fumes can physiologically affect the brain

and mental health (Canadian Environmental Law Association, 2011). Further, an

individuals’ appraisal that they have been exposed to a physical hazard can lead to stress

and changes in mental health (Edelstein, 2002).

The current study will specifically examine how physical workplace hazards are

related to general mental health. There is abundant literature on the relationship between

psychosocial hazards and mental health (Rugulies et al., 2006), however more research is

needed to explore the relationship between physical workplace hazards and mental

health. Despite the fact that certain chemicals and physical hazards can directly and

indirectly affect mental health (Cox, 1993), occupational health psychology has not given

appropriate attention to the link between physical hazards and mental health. Examining

the association between physical hazards and mental health is important because

employees who are exposed to many physical hazards will likely be at risk for mental

health problems. The current study will bridge the gap in the literature and test the

association between physical workplace hazards and mental health, while controlling for

psychosocial hazards. The literature suggests that psychosocial hazards are a strong

predictor of mental health; therefore controlling for psychosocial hazards will

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demonstrate whether physical hazards predict mental health beyond psychosocial

hazards. This leads into the second hypothesis of the current study.

Hypothesis 2. Physical workplace hazards will be negatively related to general

mental health.

The current paper has examined how physical and psychosocial factors in the

workplace can affect general health, obesity, and mental health. In addition, absences due

to sickness, illness, and injury can be used as an accurate proxy for employee health

(Kivimäki, Head, Ferrie, Shipley, Vahtera, & Marmot, 2003). Absences due to sickness,

illness, and injury are considered by many researchers to be an accurate measure of

health, especially if the data can be fact checked (Spector, 1994), allowing for a

comparison between self-report data and employee records. Therefore a review of the

effects of workplace hazards on health would not be complete without examining the

relationship between workplace hazards and employee absences. Examining workplace

absences due to sickness can allow researchers to compare absence rates with self-

reported health problems. If self-reported health problems are indeed serious or

accurately reported then employee health should also be accurately portrayed by means

of sickness absences.

Physical Workplace Hazards and Employee Absences

Physical workplace hazards are cause for concern for both employers and

employees. Based on a review of the GAZAL data (France’s national gas and electricity

company), Melchoir et al. (2005) state that physical work conditions account for 42% of

absences due to sickness and injury for men and 13% for women. The association

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between physical hazards and sickness absences applies to both white collar and blue

collar jobs. Niedhammer, Chastang, David, and Kellecher (2008) analyzed data on both

blue-collar and white-collar workers and found that the workplace physical hazards most

strongly associated with sickness absences were ergonomic hazards, physical and

chemical exposures, and biological exposures for women. Ergonomic hazards included

postural constraints and vibrations. Among physical exposures, noise and extreme

temperatures were most commonly reported while chemical and biological exposure were

defined by frequency of exposure. Haukenes, Mykletun, Knudsen, Hansen, and Maeland

(2011) also conclude that physical hazards are a real threat to employees’ health both

among skilled and unskilled occupations.

In most cases, studies that have investigated the link between physical hazards

and absences have examined ergonomic factors such as working posture, physical

demands such as carrying heavy loads, and ambient factors such as noise (Allebeck &

Mastekaasa, 2004). A well designed study by Boedekker (2001) examined several

physical hazards to determine the relationship between workplace physical hazards and

absences due to sickness. The physical hazards index included physical demands such as

heavy lifting, forced body postures, vibrations, noise, and unfavorable climate. The

Boedekker study revealed that vibrations and physical demands such as heavy lifting

were significantly related to sickness absences. Further, sick leave from work due to

accidents was strongly related to physical demands, vibrations, and work restraints,

including unadaptable workplace design and unhandy tools. Taking a broad approach,

D’Errico and Costa (2011) also investigated the association between several kinds of

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workplace hazards (both physical and organizational factors) and sickness absence.

Among the most serious physical workplace hazards, exposure to risk of injury was

significantly related to sickness absences among men and women. Noise and vibration

were also related to an increased risk of sickness absences among male employees.

In contrast, some studies have chosen to focus on specific workplace physical

hazards instead of examining a general index of workplace hazards. One area of

investigation that has received attention by researchers are the effects of physical

demands and physical loads on employee sickness absences. Hoogendoorn et al. (2002)

conducted a study examining how high physical workload and low job satisfaction can

increase the risk of sickness absences due to lower back pain. The results of the

Hoogendoorn study found that flexion and rotation of the trunk and lifting were risk

factors for sickness absences due to low back pain. More recently, Lund, Labriola,

Christensen, Bultmann, and Villadsen (2006) investigated the relationship between

physical workplace hazards such as uncomfortable work positions, physical workload and

sickness absences. A sample from the Danish workforce was used, with data from the

Danish work environment cohort study and a national register. For both males and

females, extreme bending or twisting of the neck or back, working mainly standing or

squatting, lifting or carrying heavy loads, and pushing or pulling loads were significant

risk factors for sickness absence. Labriola, Lund, and Burr (2006) also took a similar

approach by examining the effects of physical demands on absences due to sickness. The

Labriola et al. (2006) study confirmed the findings from previous studies that extreme

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bending and twisting of the body and repetitive monotonous work are associated with

increased sickness absences.

Excessive noise in the workplace is another physical hazard which has been

linked to increased sickness absences among employees. Evidence suggests that noise is

one of the most common stressors in the physical work environment among the

industrialized workforce in North America and Europe (Kjelberg, 1990; Tempest, 1985).

Fried (2002) specifically examined the effects of excessive noise levels on sickness

absences among a sample of industrial Israeli workers. Sound levels were measured using

a recording device, with samples taken twice per day. Upon analysis, the study found that

higher noise levels were correlated with increased sickness absence rates. Women also

reported more sickness absences compared to men. Employees may also develop health

problems and be less able to cope with stress resulting from excessive noise levels in the

workplace when they are not fairly compensated with an adequate salary (Ose, 2004).

Based on the comprehensive literature review in the current study, physical

hazards in the workplace are a grave risk factor for employee health, injury, and absences

due to sickness. Both clusters of physical hazards and individual physical hazards can

negatively impact employee health and lead to increased rates of sickness absences

(Allebeck & Mastekaasa, 2004). The association between physical hazards in the

workplace and employee absences due to sickness are also well documented across

different countries and occupations (Melchoir et al., 2005; Haukenes, Mykletun,

Knudsen, Hansen, and Maeland, 2011

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The current study will investigate the relationship between physical workplace

hazards and sickness absences. Within the literature there is a tendency for studies to

focus on the effects of psychosocial hazards or physical hazards upon sickness absences

separately. It is important to examine how workplace hazards can affect sickness

absences because sickness absence is an accurate predictor of employee health (Kivimaki

et al., 2006), the main focus of total worker health. The present study is unique because

the effects of physical hazards upon sickness absences will be examined, while

controlling for psychosocial hazards. Controlling for psychosocial hazards allows for an

examination of whether physical hazards impact sickness absences beyond the effects of

psychosocial hazards. It is likely that employees who face many physical hazards will

suffer from more illness resulting in missed work days compared to employees who face

few physical hazards. This leads into the third hypothesis.

Hypothesis 3a. Physical workplace hazards will be positively related to absences

due to sickness.

It is evident from the current literature review that physical hazards in the

workplace are a significant risk factor for sickness absences among employees (Melchoir

et al., 2005). When identifying hazards in the workplace it is necessary to conduct a

comprehensive analysis of which hazards exists to accurately evaluate the real health and

safety threats that employees face. Therefore, in order to fully identify the full array of

possible workplace hazards it is also necessary to consider psychosocial hazards, in

addition to physical hazards. Psychosocial hazards may be more difficult for

organizations to identify compared to physical hazards because psychosocial hazards may

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not stem from a tangible factor that an organization can visibly see. Nonetheless,

psychosocial hazards in the workplace have been proven to be detrimental to employee

health and linked to increased sickness absences (Stansfeld, Fuhrer, Shipley, & Marmot,

1999). Therefore, the current paper will review the association between psychosocial

workplace hazards and sickness absences to conduct a complete review of the literature.

Psychosocial Workplace Hazards and Employee Absences

Psychosocial hazards in the workplace have been linked to employee absences

due to sickness or injury in several studies. One of the most comprehensive reviews

concerning working conditions and workplace absences was conducted by Allebeck and

Mastekaasa (2004). Allebeck and Mastekaasa (2004) reviewed 20 studies that addressed

the association between psychosocial working environment and sickness absence. Most

studies appeared to be based on the demand-control-support model. The majority of the

studies supported the hypothesis that higher job demands lead to increased sickness

absences. Job control was found to be related to lower absences among employees in

almost every study examined by Allebeck and Matekaasa (2004). Mixed results were also

found for support, with job support from colleagues or supervisors being associated with

lower absences in a few instances (de Jong, Reuvers, Houtman, Bongers, & Komper,

2000). Workplace bullying was also found to be associated with higher absence rates, as

well as role uncertainty (Kivimäki, M., Elovainio, M., & Vahtera; Heaney & Clemans,

1995).

Studies have also used data from the Whitehall II study, which confirms the

findings from the Allebeck and Mastekaasa (2004) review that psychosocial hazards are

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related to increased absences due to sickness. The WhiteHall II study is a large public

data-set that was established in 1985 by a team of researchers to investigate the

importance of social class for health by following a cohort of 10, 308 employed civil

servant workers across the United Kingdom (Marmot et al., 2004). North, Syme, Feeney,

Shipley, and Marmot (1996) determined that both high demands and low control at work

are associated with higher rates of sickness absences among lower grade employees. Ari

Väänänen et al. (2003) report that lack of job autonomy and low social support are risk

factors for sickness absences among men and women. Sickness absences resulting from

psychiatric disorders have also been linked to job control and social support (Stansfeld,

Fuhrer, Shipley, & Marmot, 1999).

Since the Allebeck and Matekaasa (2004) review, which was based on studies

published up until 2002, several new studies have emerged which have examined the

association between psychosocial work hazards and sickness absences in the workplace.

Melchior, Niedhammer, Berkamn, and Goldberg (2003) used data from the French

GAZAL study (France’s National Gas and Electricity Company) to examine how

psychosocial hazards affect sickness absences. The results of the Melchior et al. (2003) 6

year prospective study revealed that low job control and low social support below the

median predicted increased absence rates among men (17%) and women (24%). In

addition, the Danish IPAW study (Intervention Project on Absence and Well-being)

conducted by Nielsen et al. (2004) was a 5 year project that investigated psychosocial

hazards as independent predictors of sickness absence days per year. The results of the

study indicated that low levels of job control predicted high absence rates among both

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men and women. Nielson et al. (2006) also showed that job control and job predictability

significantly predicted workplace absences. Another study conducted on a sample of

Danish employees also determined that sickness absence was predicted among men by

high emotional demands and high demands for hiding emotions. Among women, role

conflicts, low rewards, and poor management quality predicted sickness absences (Lund

et al., 2005).

Research also suggests that an examination of psychosocial hazards in the

workplace and absences due to sickness should not be limited to components of the

demand-control-support model. Instead a comprehensive approach to the measurement

and definition of psychosocial hazards and sickness absences is needed (Rugulies et al.,

2007). Voss, Floderus, and Diderichensen (2001) found that bullying in the workplace

was associated with a doubled risk of sickness absence for women. In contrast, for men,

the strongest predictor of sickness absence was anxiety about reorganization of the

workplace. Kivimaki, Elovainio, and Vahtera (2000) have also confirmed that workplace

bullying can lead to increased absences due to sickness in the workplace. Kivimaki et al.

(1997) found that income is a strong predictor of sickness absences, with lower income

employees at a 1.7 greater odds of reporting sickness absences. Further, D’Errico and

Costa (2010) report that psychosocial factors such as job insecurity are related to

increased absences due to sickness.

The current study will investigate the relationship between psychosocial

workplace hazards and sickness absences. As previously stated, within the literature there

is a tendency for studies to focus on the effects of psychosocial hazards or physical

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hazards upon sickness absences separately. It is important to examine how psychosocial

hazards can affect sickness absence because sickness absence is an accurate predictor of

employee health. The present study is unique because the effects of psychosocial hazards

upon sickness absences will be examined, while controlling for physical hazards.

Controlling for physical hazards allows for an examination of whether psychosocial

hazards impact sickness absences beyond the effects of physical hazards. It is reasonable

to predict that employees who face high levels of psychosocial hazards will miss more

work days due to sickness compared to employees who face few psychosocial hazards.

Further, the effects of psychosocial hazards on sickness absence will likely have a unique

impact on sickness absence, separate from physical hazards.This leads into the third

hypothesis.

Hypothesis 3b. Psychosocial workplace hazards will be positively related to

absences due to sickness.

The current paper has argued that workplace hazards can have a negative impact

on employee health, obesity, mental health, and absences due to sickness. It is important

to identify which workplace hazards are present in the workplace; however it is equally

important to understand factors that can ameliorate the negative impact that workplace

hazards can have on an individuals’ health. One area of research that is growing in

occupational health psychology is how physical activity and exercise can enhance worker

health and help employees’ cope with workplace stressors (Proper et al., 2003). Before

the relationships between workplace hazards, exercise and employee health are examined

it can be helpful to gain an understanding of what exercise is and the frequency with

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which the European and North American workforce engage in this positive health

behavior.

Physical Activity and Exercise

According the World Health Organization (2013) physical activity is defined as

any bodily movement produced by skeletal muscles that requires energy expenditure. In

contrast, exercise is defined as a subcategory of physical activity that is planned,

structured, and repetitive with the goal of maintaining or improving physical fitness.

Physical activity includes exercise as well as other activities that require some degree of

physical exertion such as manual labor, recreational activities, household chores, or

walking to work. Researchers often use the terms physical activity and exercise

interchangeably, which leads to an inaccurate understanding of both terms (Harvard

School of Public Health, 2012).

A recent study conducted by Hallal et al. (2012) compared physical activity levels

worldwide with data from adults 15 years or older, from 122 countries. Worldwide,

31.1% of adults are physically inactive, ranging from 17% in Southeast Asia to

approximately 43% in the Americas and the Mediterranean. Physical inactivity tends to

rise with increased age, and is higher among women than men and is greater in high-

income countries. Subsequently, it is not surprising that rates of physical inactivity are

extremely high in the U.S. and the U.K. According to the CDC (2011) physical inactivity

in the U.S. ranges from 10% to 43% depending on the region. The U.K., another high-

income country also struggles with physical inactivity, with approximately 40% to 49%

of adults considered physically inactive.

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Physical inactivity has been identified as the fourth leading risk factor for global

mortality, accounting for 6% of global deaths. The WHO (2013) estimates that physical

inactivity is accountable for 21-25% of breast and colon cancers, 27% of diabetes, and

30% of heart disease cases globally. Physical inactivity is also attributable to 9% of

premature mortality worldwide. Importantly, if inactivity could be decreased by 10% or

25% worldwide, more than 1-3 million lives could be saved each year (Lee et al., 2012).

The good news is that increasing physical activity can eliminate or reverse the

negative health consequences associated with years of sedentary living. Regular exercise

and physical activity can improve overall health and functioning of the body and reduce

the likelihood of developing heart disease, diabetes, and is a key element of weight loss

(Harvard School of Public Health, 2013). The U.S Department of Health and Human

Services (2008) state that being physically active improves longevity and quality of life,

helps protect people from developing heart disease, stroke, and high blood pressure.

Physical activity also protects people from developing certain cancers, type 2 diabetes,

osteoporosis, depression, anxiety, and improves sleep, heart and lung function, and a

healthy body weight.

Physical activity, specifically exercise can vary in intensity. Intensity refers to the

rate at which an activity is being performed or the amount of effort required to perform

an activity such as exercise (WHO, 2011). According to the CDC (2011), moderate

physical activity burns approximately 3.5 to 7 calories per minute, and can include

activities such as walking, hiking, or roller skating. On the other hand, vigorous activity

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burns more than 7 calories per minute, and can include activities such as jogging, circuit

training, or mountain climbing.

The Harvard School of Public Health (2013) recommends that healthy adults get a

minimum of 2 ½ hours of moderate-intensity aerobic activity or a minimum of 1 ¼ hours

per week of vigorous intensity aerobic activity. Increasing the amount of physical activity

to 5 hours of moderate or 2 ½ hours of vigorous-intensity aerobic activity provides even

more health benefits. Adults should also do muscle-training activities at least two days a

week to improve muscle tone and strength (Harvard School of Public Health, 2013). It is

well documented that increasing physical activity levels can improve health (WHO,

2011), however a combination of physical activity and a proper diet is most effective in

maintaining a healthy body weight and overall health (CDC, 2011). In order to effectively

promote health in the workplace researchers and employers need to consider how an

employees’ diet influences health and understand how eating habits develop.

Diet and Eating Habits

Eating a healthy and well-balanced diet is vital for improving and maintaining

good health. According to the WHO (2013) a healthy and well-balanced diet contains

essential vitamins and minerals which are necessary to boost the immune system. A

healthy diet also protects the human body against certain types of diseases, especially

diabetes, cardiovascular diseases, certain types of cancers, and skeletal conditions.

Importantly, a well-balanced diet also contributes to a healthy bodyweight (WHO, 2013).

The U.S. government recently revamped their dietary recommendations from a

food pyramid to a template called “MyPlate”, which is heavily based on the consumption

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of fruits and vegetables. (Harvard School of Public health, 2013). The Harvard School of

Public Health has developed a framework named the new healthy eating plate, which is

argued to fix the flaws in the USDA’s MyPlate framework. The new healthy eating plate

is based on the latest science about how food, drink, and activity choices affect health.

The new healthy eating plate uses an illustration of a plate, with 4 main sections inside

the plate; vegetables, whole grains, healthy protein, and fruits. There are also sections

outside of the plates for healthy oils and water. The rationale is that most people eat off a

plate, so a plate is an ideal blueprint for an eating plan. The Harvard School of Public

Health (2013) recommends that people fill half their plate with produce such as colorful

vegetables and fruits. A quarter of an individuals’ plate should be for whole grains such

as brown rice or multigrain bread. A healthy source of protein such as fish, poultry,

beans, or nuts can make up the rest of the plate. Additionally, it is recommended that

healthy oils are used for cooking such as olive oil. Finally, a meal can be completed with

a glass of water, tea, or coffee.

Many people know that eating a healthy, well-balanced diet is good for their

health; however, many people struggle to maintain a healthy diet and body weight. The

problem is that knowledge of a health behavior does not always lead to the practicing of a

health behavior (Ajzen, 1991). The U.S. government can set guidelines about healthy

eating, but that is only part of the complex puzzle about how to get people to eat healthy.

To truly understand the complex process about how people start and maintain healthy

behaviors it is necessary to examine how health behaviors develop into habits.

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Health Behaviors and Habit Formation

Although there is no universal definition, health behaviors can be described as

behaviors people carry out to enhance or maintain their health (Cohen, Bowness, & Felix,

1990). Positive health behaviors can include exercising and eating healthy. In contrast,

negative health behaviors include smoking or being physically inactive. Understanding

what kind of health behaviors people should be practicing in order to maintain and/or

improve health is the first step towards getting people to make informed choices about

their health. However, knowledge of a health behavior does not always lead to an

individual practicing a health behavior (Ajzen, 1991). Health behaviors can develop into

health habits, which are firmly established and performed behaviors which occur

automatically, without awareness (Cohen, Bowness, & Felix, 1990). For example, a

negative health behavior such as eating unhealthy snacks late at night can develop into an

ingrained behavior, which turns into a negative health habit.

The current paper has reviewed a wide array of health benefits that physical

activity and a healthy diet offer to people. One important aspect of physical activity that

needs to be discussed in further detail is that physical activity, especially exercise can

help reduce stress (Cotton, 1990). There is strong empirical evidence that physical

activity reduces stress; however, it is vital to understand how physical activity affects

stress at the physiological level. An understanding from the physiological level about

how physical activity affects stress has implications for how employees and organizations

deal with stress in the workplace.

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Physical Activity and Stress

According to the Mayo Clinic (2013), physical activity can directly reduce stress

in several ways. First, physical activity increases the production of neurotransmitters and

endorphins, which are known to increase a positive mood. The Harvard School of Public

Health (2013) also states that physical activity reduces the body’s levels of stress

hormones, including adrenaline and cortisol. Behavioral factors also contribute to the

positive effects of physical activity (Harvard School of Public Health, 2013). For

example, a person’s self-image will improve as their strength and stamina increase.

Further, regular physical activity can lower symptoms associated with depression and

anxiety. Physical activity can also improve sleep quality, which can in turn help an

individual cope with stress. The Mayo Clinic (2013) also explains that physical activity

can act as a cognitive distraction from daily stressors, allowing a person to relax and

forget about their problems.

In order to minimize the negative effects that occupational stress has on

employees and organizations many employers have attempted to tackle the problem by

promoting physical activity in the workplace (Con et al., 2009). Several factors determine

how beneficial physical activity will be for an individual; the type of physical activity,

frequency of participation, compliance to exercise programs, and employee interest in

exercise (Stein, 2001). The association between physical activity and the reduction of

stress has been investigated by a number of researchers (Cotton, 1990). There is strong

evidence to support that individuals who are aerobically fit are more resistant to the

negative physiological and psychological effects of stress compared to non-aerobically fit

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individuals. People who are more aerobically fit may also recover more quickly from

stress (Bonita, 1991).

Researchers from different fields have attempted to explore how physical activity

and exercise help people cope with stress. Researchers have employed different

experimental frameworks when examining the stress buffering effects of exercise

(Gerber, Kellman, Hartman, & Puhse, 2009). For example, some studies have explored

how acute exercise can lead to reduced reactivity to stressors and increase an individual’s

recovery when exposed to an experimentally induced stressor (Boutcher, Hopp, &

Boutcher, 2010). In line with research conducted by Boutcher et al. (2010), a meta-

analyses found that acute bouts of exercise have a significant impact on blood pressure

response to a psychosocial stressor (Hammer, Taylor, & Steptoe, 2006). In contrast, some

researchers have chosen to explore how chronic exercise (via increased fitness levels) can

suppress an individuals’ stress reactions and boost recovery from experimentally induced

stress (Jackson & Dishman, 2006). Salmon (2001) states that several studies have

confirmed that habitual exercise habits protect individuals from the harmful effects of

stress on physical and mental health, although causality is not clear.

There is a substantial amount of research that has investigated how physical

activity and exercise can affect the relationship between stress and stress induced

illness/health complaints. One of the first studies to investigate whether exercise can act

as a stress buffer was conducted by Kobasa, Maddi, and Puccetti (1982). The Kobasa

study revealed that exercise interacted with stressful events, reducing the incidence of

illness among business executives. Research by Brown (1991) confirm the early findings

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found in the Kobasa et al. (1982) study. Both exercise and aerobic fitness were found to

moderate the stress-illness relationship. Physically active and physically fit individuals

were healthier and had fewer visits to healthcare facilities when exposed to life stressors

as well (Brown, 1991). Carmack et al. (1999) also found evidence that exercise inhibits

the development of physical symptoms and anxiety associated with stressors among

college students. Based on a similar sample, Louchbaum, Lutz, Sells, Ready, and Carson

(2004) found that strenuous exercise was related to lower levels of psychosomatic

complaints among individuals subject to increased stress. Ensel and Lin (2004) also

found evidence that exercise reduced psychosomatic complaints among people who

encounter high levels of stress in their lives.

Recently, Gurber and Puhse (2009) conducted a comprehensive narrative review

of studies testing exercise as a stress buffer, reviewing articles from 1982 up until 2008.

The main goal of the Gurber and Puhse review was to determine if exercise and fitness

protect against stress-induced health complaints. Over half of the studies reviewed

supported the premise that people with high exercise levels exhibit less health problems

when they encounter stress. Further analyses revealed that the stress-moderation effects

were consistently found across different samples and varying methodological approaches.

Gurber and Puhse (2009) argue that more studies are needed to provide an understanding

about how much exercise is needed to trigger stress-buffer effects.

A research area that has also received substantial attention is how leisure activities

outside of work help people recover from occupational stress. Surprisingly, the research

area of leisure time activities outside of work has remained segmented from research

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investigating exercise as a stress buffer. A key differentiation is that studies that examine

leisure time activities include physical activities of varying intensities, ranging from

structured, intense exercise to light physical activity such as walking to work (Saftlas et

al., 2004). Additionally, studies that have examined leisure time activities include both

physical activities such as playing sports and less physical activities such as volunteering

(Sonnentag, 2001). In contrast, studies that have looked at exercise as a stress buffer

generally focus on structured, moderate-intense exercise (Gerber & Puhse, 2009).

Importantly, studies that have investigated the effects of leisure time physical activities

on occupational stress have noted that physical activity has psychological as well as

physiological implications for recovery (Geurts & Sonnentag, 2006).

Recovery can be described as a process which reverses the negative consequences

of job demands and reduces stress levels back to normal, allowing a person to function at

the same level prior to induced stress (Craig & Cooper, 1992). Recovery experiences

during leisure time physical activity play a critical role in a person’s health and mood.

Sonnentag, Binnewies, and Mojza (2008) found that low psychological detachment from

work during the evening predicted fatigue and negative affect among employees the

following morning at work. Further, mastery experiences during the evening predicted

relaxation and positive affect the following morning at work. Additionally, Winwood,

Bakker, and Winefield (2007) report that leisure time activities such as social activity,

participating in hobbies, and exercise affect the relationship between work strains and

sleep quality. The results from the Winwood et al. (2007) study also revealed that

participants who engaged in higher levels of leisure time activity reported increased

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recovery between work periods and lower chronic fatigue. Importantly, people who

consistently recover from work strain between work periods may be able to avoid

negative health outcomes related to work strain.

It is important for employers and researchers to understand that leisure activities

that include physical activity and exercise can help lower employee stress levels because

it can be a tool used to promote total worker health. It is also equally important that

researchers demonstrate how physical activity can buffer the negative effects of stress on

important health outcomes in the workplace. Research that can demonstrate how physical

activity promotion can improve employee health, and in turn lead to increased profits for

an organization are vital. From an organizational perspective, unhealthy employees will

likely accumulate more sickness absences compared to healthy employees. Subsequently,

if researchers can make a strong argument that physical activity promotion can reduce

employee absences and increase profits and productivity, organizations will be more

motivated to promote employee health. Further, employees who are of a healthy body

weight and in good mental health will be more productive, which translates into more

money for organizations (Burton et al., 2005).

Physical Activity and Obesity

The positive health benefits of physical activity are not limited to stress reduction.

Much evidence supports the claim that physical activity is a vital component to

maintaining a healthy bodyweight and body composition (Harvard School of Public

Health, 2013). Despite the fact that diet and physical activity have been linked to obesity

for many years, the connection has recently experienced a renewal of interest. There is

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mounting evidence that the current prevalence of obesity is more closely related to a

decrease in energy expenditure than the traditionally accepted imbalance between energy

consumption and energy expenditure (Struber, 2004). A number of factors influence how

many calories a person burns each day, including age, body size, and genes. However, the

most variable factor and most modifiable factor that influences obesity is the amount of

physical activity people get each day (Harvard School of Public Health, 2013).

Researchers argue that physical activity prevents obesity in several ways (Hu,

2008). According to the Harvard School of Public Health (2013) physical activity

increases people’s total energy expenditure, which helps people maintain an energy

balance. Physical activity also decreases total body fat and fat around the waist.

Resistance exercises such as weightlifting and strength training activities aimed at

building muscle mass increases the amount of energy that the body burns throughout the

day. Increased muscle mass also results in more calories burned while at rest. Finally,

physical activity has been proven to reduce depression and anxiety (U.S. Department of

Health and Human Services, 2008). The resulting mood boost from physical activity may

motivate people to stick to their exercise regimens over time.

The intensity of physical activity plays a major role in how much fat a person can

burn. A study conducted by Slentz, Aiken, and Houmard (2005) randomly assigned

overweight and physically inactive participants to 1 of 4 groups; a control group that did

not exercise, low intensity, moderate intensity, and high intensity physical activity group.

After six months participants from the high intensity group lost a significant amount of

abdominal fat, whereas the other three groups had no change in abdominal fat. Studies

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conducted by several researchers have also confirmed that vigorous activities are more

effective for weight control compared to light physical activities such as slow walking

(Mekary et al., 2009).

Evidence also suggests that physical activity may also reduce obesity and body fat

storage by interacting with stress. A study by Yin, Davis, Moore, Treiber (2005)

examined whether physical activity buffers the effects of chronic stress on body fat

composition among a sample of adolescents. Body fat composition was measured by

using skin fold measurements, BMI, and waist circumference. Yin et al. (2005) found that

physical activity interacted with stress, predicting all three measures of body fat

composition. It appears that greater levels of physical activity buffer the effects of

chronic stress on adiposity measures.

It is important to note that Yin et al (2005) reported that there were no prior

studies investigating physical activity as a moderator of the effect of stress on obesity. A

thorough literature review was conducted for the current study. To the best of this

authors’ knowledge there have been few if any studies since Yin et al. (2005) to directly

test the hypothesis that physical activity moderates the relationship between stress and

body fat/body composition. Interestingly, there have been many studies that have

confirmed that physical activity can reduce stress (Gerber & Puhse, 2009) and that

physical activity can buffer the effects of stress-induced health complaints (Carmack et

al., 1999); however there is a large gap in the literature with regards to physical activity

buffering the effects of stress on body fat composition. Further, the only apparent study

that did examine the stress buffering effects of physical activity on adiposity was based

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on a sample of adolescents, which is not a representative sample of the working

population. There is a need for researchers to examine how physical activity can buffer

the effects of stress on body fat composition among a representative and generalizable

sample.

The current study has reviewed literature which suggests that both physical and

psychosocial hazards in the workplace increase employee stress levels. Research has

confirmed that physical and psychosocial hazards in the workplace can lead to an

unhealthy body weight and body fat composition. Additionally, there is a plethora of

research which has proven that physical activity can buffer the negative effects of stress

(Ensel & Lin, 2004), including body fat composition (Yin et al., 2005). The current paper

has already hypothesized physical and psychosocial hazards in the workplace will be

associated with increased BMI. Expanding on the previous prediction of this paper, it

would be appropriate to predict that physical activity levels will play a role in how stress

associated with workplace hazards affects employee body fat composition. Previous

research has linked stress to obesity, and has also discussed the stress buffering effects of

exercise; however few studies have specifically examined how physical activity can

buffer the negative effects of workplace hazards on BMI. For example, an employee who

faces many workplace hazards but frequently exercises will likely have a lower BMI

compared to a person who is faced with many workplace hazards but does not exercise.

Exercise can both directly reduce body fat and increase a person’s ability to cope with

stress, indirectly improving body composition. The current study will bridge the gap in

the literature, and hypothesize the following:

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Hypothesis 4a. Physical activity will moderate the relationship between physical

workplace hazards and BMI, meaning that employees who have high levels of physical

activity will be better able to cope with exposure to physical hazards, and have a lower

BMI compared to employees who have low levels of physical activity.

Hypothesis4b. Physical activity will moderate the relationship between

psychosocial workplace hazards and BMI, meaning that employees who have high levels

of physical activity will be better able to cope with exposure to psychosocial hazards, and

have a lower BMI compared to employees who have low levels of physical activity.

I have argued that physical activity will buffer the relationship between stress

resulting from occupational hazards and BMI. It is important to point out that BMI is

only one indicator of a person’s health. It is also important to consider the mental health

of a person to truly encompass a complete picture of health. Mental health is an important

indicator of a person’s overall health because mental health problems can have a

psychological as well as a physiological impact on the human body (CDC, 2011). Mental

health is not completely controlled by genetics and can be influenced by the environment

and an individual’s health habits (CDC, 2011).

Physical Activity and Mental Health

One of the most modifiable behaviors that affects mental health is physical

activity (Harvard School of Public Health, 2013). Physical activity plays an important

role in the management of mild to moderate mental health problems, especially

depression and anxiety (Paluska & Schwenk, 2000). People with mental health problems

tend to be less physically active; however increased physical activity, including both

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aerobic and resistance training have been shown to reduce depressive symptoms. In

addition, anxiety symptoms and panic disorders improve with regular exercise, similar to

the effects of meditation or relaxation (Pauluska & Schwenk, 2000). Exercise can also

enhance mental health by improving self-esteem, cognitive function, and can alleviate

symptoms of social withdrawal (Sharma, Madaan, & Petty, 2006).

According to the Mental Health Foundation (2013) physical activity and exercise

release chemicals in the brain that are associated with mood enhancing effects.

Specifically, exercise releases endorphins which have been shown to reduce pain and

create a euphoric state (Paluska & Schwenk, 2000). Improvements in mood by means of

exercise can partly be explained by exercise-induced increases in blood circulation to the

brain and by an influence on the hypothalamic-pituary-adrenal (HPA) axis (Sharma,

Madaan, & Petty, 2006). The Mental Health Foundation (2013) lists several benefits of

physical activity which can impact mental health; improved sleep, increased interest in

sex, better endurance, stress relief, improved mood, increased energy and stamina, and

increased mental alertness.

Physical activity may also buffer stress and reduce stress-induced mental health

problems by creating a temporary distraction from one’s problems and increase social

interactions (Peluso & Andrade, 2005). Physical activity can offer a diversion from

unpleasant stimuli, leading to improved affect and strengthen mental health (Pauluska &

Schwenk, 2000). Further, social interactions gained from group exercise routines or

meeting new people when being physically active can reduce feelings of loneliness and

buffer stress (Pauluska & Schwenk, 2000). Further, many of the studies reviewed have

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shown that physical activity can distract people from their problems and increase social

interactions, both of which increase resilience to stress and promote a healthy mental

state (Fox, 1999).

One of the main ways that physical activity and exercise are thought to improve

or maintain mental health is by reducing a persons’ physiological reactivity to stress

(Guszkowska, 2003). In essence, it is believed that physical activity acts as a stress buffer

between a stressor and mental illness. In line with the stress buffering hypothesis, Norris,

Carroll, and Cockrane (1992) examined the effects of physical activity and exercise

training on psychological stress and well-being in an adolescent population. Adolescents

were assigned to either high or moderate intensity aerobic training, flexibility training, or

a control group. Analyses revealed that participants in the high intensity exercise group

reported significantly less stress compared to the other experimental groups. The

relationship between stress and anxiety/depression/hostility was considerably weakened

for the high intensity exercise group. An experiment conducted by Rejeski, Thompson,

Brubaker, and Miller (1992) further confirmed the positive effects of physical activity on

stress and mental health by examining how acute exercise buffers psychosocial stress

responses. Rejeski et al. (1992) revealed that exercise reduced blood-pressure reactivity

when participants were exposed to mental and interpersonal threat. Additionally, aerobic

exercise reduced both the frequency and intensity of anxiety-related thoughts that occur

in anticipation of a threat.

More recently, Salmon (2001) conducted a literature review on the effects of

physical exercise on anxiety, depression and sensitivity to stress. Salmon (2001) notes

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that there is strong evidence to support the theory that exercise leads to enduring

resilience to stress. Salmon also cites that results of cross-sectional and longitudinal

studies are consistent in indicating that aerobic exercise has antidepressant and anxiolytic

effects which protects against the harmful consequences of stress. More recent research

also confirms that physical activity can increase resilience to stress and reduce stress-

induced mental health problems, especially depression (Southwick, Vythilingam, &

Charney, 2005).

The beneficial effects of physical activity on stress and mental health are not

limited to aerobic exercise. Recently, Hartfield, Havenhand, Khalsa, and Krayer (2011)

tested the effectiveness of yoga for the improvement of well-being and resilience to stress

among a sample of British employees. Compared to a control group, participants who

practiced yoga for 6 weeks reported improvements in energy, confidence, increased life

satisfaction, and self-confidence during stressful situations. A study by Streeter, Gerbarg,

Saper, Ciraulo, and Brown, (2012) also confirms that low-impact physical activity such

as yoga or stretching can be effective for increasing resilience to stressful situations. It is

believed that yoga practices reduce allostatic load in stress response systems, returning

the body to optimal homeostasis. Both depression and PTSD can increase the allostatic

load on the body, exacerbating stress. Steeter et al. (2012) state that yoga can reduce the

allostatic load on the body and promote mental health via buffering the negative effects

of stress. Field (2011) conducted a review concerning the health benefits of yoga. In line

with the research discussed thus far an overwhelming amount of research suggests that

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yoga both decreases stress and reduces the prevalence of psychological conditions such

as anxiety and depression.

The current paper has reviewed how stress resulting from workplace hazards can

negatively impact mental health (Leka, 2010). Further, there is strong evidence that

physical activity can buffer the negative effects of stress and reduce the incidences of

stress-induced mental health problems (Guszkowska, 2003). Importantly, varying forms

of physical activity can be beneficial for mental health, ranging from yoga to aerobic

exercise (Streeter et al., 2012). Despite strong evidence that physical activity can buffer

stress-induced mental health problems, few if any studies have specifically examined

how physical activity can moderate the relationship between workplace hazards and

mental health. It is likely that physical hazards are perceived as stressful, and

participating in physical activity releases neurotransmitters which are known to increase

mood and mental health. Further, employees who are physically fit may be more resilient

to the effects of stress caused by both physical and psychosocial hazards, reducing the

risk of developing mental health problems. Additionally, there is a need for researchers to

develop strategies for employees to deal with occupational stress stemming from

workplace hazards and the resulting impact stress can have on mental health. This leads

into the fifth set of hypotheses.

Hypothesis 5a. Physical activity will moderate the relationship between physical

workplace hazards and mental health, meaning that employees who have high levels of

physical activity will be better able to cope with exposure to physical hazards, and have

better mental health compared to employees who have low levels of physical activity.

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Hypothesis 5b. Physical activity will moderate the relationship between

psychosocial workplace hazards and mental health, meaning that employees who have

high levels of physical activity will be better able to cope with exposure to psychosocial

hazards, and have better mental health compared to employees who have low levels of

physical activity.

As previously discussed, BMI and mental health can both be considered proximal

indicators of health. Proximal indicators of health (signs and symptoms of health,

disease-specific outcomes) are ideal for determining an individuals’ health (Brenner,

Curbow, & Legrow, 1995). However, it is also important to measure distal outcomes of

health to determine how the health of an employee can impact an organization. Distal

indicators are not a direct result of health, but can be indirectly related to health. For

example, employees may report poor health but still make it to work every day. Therefore

measuring a more distal outcome of health such as absences due to sickness can help

demonstrate how an employees’ health impacts both the organization (e.g. productivity)

and the employee (frequency of sicknesses). Further, examining how modifiable

behaviors such as physical activity can affect distal health outcomes such as employee

absences may motivate employers to pay closer attention to their employees’ health since

it affects organizational profitability.

Physical Activity and Absences Due to Sickness

Research clearly indicates that physical activity and exercise are beneficial for

maintaining and improving physical and mental health (Harvard School of Public Health,

2013). Notably, the link between physical activity levels and employee absences due to

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sickness is not always straightforward. For example, physical activity can directly

improve health and reduce the likelihood of developing chronic illnesses such as type 2

diabetes, metabolic syndrome, some forms of cancers and cardiovascular disease (CDC,

2011). Chronic illnesses negatively impact quality of life and health (CDC, 2011), which

in turn could result in employees missing work due to health problems. Studies that have

examined the association between physical activity/fitness and employee absences can be

grouped into 2 main categories; the effect of fitness program participation on employee-

related absences and the association between fitness/physical activity and absenteeism

(Aldana & Pronk, 2001).

Studies have confirmed that physical activity programs in the workplace can

buffer stress and reduce the prevalence of employee absences. An early study by Cox,

Shephard, and Corey (1981) used a control trial to determine the effects of a fitness

program on physiological fitness and work absences. Analyses revealed that employees

who participated in the fitness program increased their cardiovascular fitness and

decreased their body fat, resulting in a 22% drop in workplace absences. Further,

employees with high program participation showed the greatest decreases in workplace

absences. Nine years after the Cox et al (1981) study Lynch, Golaszewski, Clearie, Snow,

and Vickery (1990) conducted a longitudinal study comparing absenteeism rates between

fitness program participants and non-participants before and after 1 and 2 years of

participation. The Lynch study found that employees who participated in the fitness

program demonstrated a 1.2 day decrease in yearly absences compared to employees who

did not participate in a fitness program. Overall, greater participation in the fitness

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program was associated with greater decreases in workplace absences. Similarly,

Lechner, deVries, Adriaansen and Drabbels (1997) examined the effectiveness of

different levels of participation in an employee fitness program on absenteeism over the

course of 2 years. Results revealed that high program participation was associated with a

significant decline in absences due to sickness. The high participation group showed an

average of 4.8 days of reduced sickness absence compared to the low participation group

and the control group.

More recently, many high quality studies have also examined physical fitness

programs in the workplace and employee absences due to sickness. Rongen et al. (2013)

conducted a meta-analysis, evaluating the effectiveness of workplace health promotion

programs. Many of the studies examined by Rongen et al. (2013) used employee sickness

absences as an outcome variable. For example Tveito and Erikson (2009) found that

physical exercise programs in the workplace were related to decreased sickness absence

rates. In addition, Zavanela et al. (2012) evaluated the effectiveness of resistance training

on employee absences. Analyses revealed that employee sickness absences dropped over

a 24 week period. Further, Reijonsaaire et al. (2012) looked at the effectiveness of a web-

based physical activity promotion program for employees. Reijonsaaire et al. (2012)

found that sickness absences were significantly reduced among employees with the

highest participation rates in the physical activity program.

A few studies have also examined the association between

physical/cardiovascular fitness and employee absences due to sickness. Early studies such

as Baun, Bemacki, and Tsai (1986) was one of the first studies to find an association

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between exercise and decreased absences due to sickness. Sickness absences were also

found to be higher among people who did not exercise. In line with earlier research, both

Tucker, Aldana, and Friedman (1990) and Steinhart, Greenhow, and Stewart (1991)

found a strong association between high cardiorespiratory fitness and physical activity

levels with decreased sickness absences.

Building upon previous research, Jacobson and Aldana (2001) examined the

relationship between frequency of aerobic activity and illness related absenteeism among

a large sample of U.S. employees. After controlling for confounding variables (gender,

SES) a significant relationship was found between absenteeism and exercising.

Differences in absenteeism were found between people who didn’t exercise and people

who exercised for 20 minutes, once or twice per week. The results revealed that even

small amounts of physical activity can have an impact on employee absences.

Additionally, Kyröläinen et al. (2008) examined the relationship between physical fitness,

BMI, and sickness absence among military personnel. Results showed that individuals

with the greatest number of sickness absences exhibited lower muscle fitness and shorter

running distance compared to individuals with shorter sickness absences. In addition,

high BMI and poor aerobic endurance were related to a higher incidence of absenteeism.

Van den Heuvel et al. (2005) took a unique approach to investigating the link between

physical activity and absenteeism rates by focusing on sporting activity and work

absences. Analyses showed that employees who practiced sports over a four year time

period missed work 20 days less than employees who did not practice any sporting

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activities. Van den Heuvel et al. (2005) concluded that employees practicing sports take

sick leave less often than people who do not participate in sports.

The current paper has reviewed that both workplace physical activity programs

(Rongen et al., 2013) and physical fitness/physical activity (Steinhart, Greenhow, &

Stewart, 1991) are related to decreased sickness absences across diverse samples of

employees. Research also cites that physical activity can buffer the negative effects of

occupational stress (Salmon, 2001), which can also lead to increased absenteeism. Most

studies that have investigated the link between physical activity and sickness absences

have not given proper attention to the root causes of employee absences e.g., stress-

induced health problems.

There is a need for studies to identify which kinds of stressors impact employee

absences in order to specify which workplace factors are most harmful to employees.

Further, many studies have investigated the link between physical activity and sickness

absence without investigating how physical activity can interact with stress and predict

employee absences due to sickness (Rongen et al., 2013). The current study will bridge

the gap in the literature by looking beyond the casual link between physical activity and

sickness absences. The present paper has argued that physical activity can both reduce

stress and make people more resilient to stress. It has also been argued that employees

who face many workplace hazards will suffer from increased stress levels and sickness

absences. In contrast, employees who exercise the most will likely have lower levels of

stress and sickness absences compared to employee who do not exercise often. This leads

into the sixth set of hypotheses.

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Hypothesis 6a. Physical activity will moderate the relationship between physical

workplace hazards and absences due to sickness, meaning that employees who practice

high levels of physical activity will be better able to cope with exposure to physical

hazards, and have less sickness absences compared to employees who practice low levels

of physical activity.

Hypothesis 6b. Physical activity will moderate the relationship between

psychosocial workplace hazards and absences due to sickness, meaning that employees

who practice high levels of physical activity will be better able to cope with exposure to

psychosocial hazards, and have less sickness absences compared to employees who

practice low levels of physical activity.

Despite the clear stress buffering effects and health benefits of physical activity

(Harvard School of Public Health, 2013) the current study is one of few studies to

specifically examine how physical activity can buffer stress resulting from occupational

hazards and the resulting impact on obesity, mental health, and sickness absences. It is

important to note that physical activity is not the only positive health behavior that can

help employees deal with stress and impact individual and organizational outcomes.

Studies have researched how stress may impact the eating habits of people (Kim & Kim,

2009), however there is a need for researchers to also examine how a person’s diet can

affect stress and health. Although studies have confirmed that people under stress eat less

healthy foods (Ng & Jeffery, 2003), it may also be the case that people who eat a healthy

diet are better equipped to deal with stress. The current study will take an unconventional

approach and examine how a healthy diet may help employees deal with stress.

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Diet and Stress

A person’s diet and stress are interrelated, however it is often difficult to

determine whether stress affects a person’s diet or whether a person’s diet affects stress.

The most plausible answer is that the cause and effect relationship can go both ways.

Stress can affect what an individual eats (Ng & Jeffery, 2003), and what an individual

eats may affect stress levels. According to Dallman (2003) people under chronic stress

have high levels of a chemical called glucocorticoid in their bloodstream. Glucocorticoids

initiate the release of stress hormones, which increase cravings for sugary foods. Dallman

(2003) argues that people eat comfort foods in an attempt to reduce the chronic stress

response in the body. Chronic stimulation of neurotransmitter sites can lead to a depletion

in neurotransmitters over time. One key modifiable behavior that can affect

neurotransmitter receptor cite is a person’s diet (Dallman, 2003).

High glycemic carbohydrates such as potatoes or white rice can cause spikes in

blood sugar, dopamine, serotonin, as well as opioids, which can desensitize

neurotransmitter receptor sites. A decrease of neurotransmitters released into the

bloodstream can lead to mental as well as behavioral problems over time, making people

more susceptible to stress (National Institute of Mental Health, 2013). A diet low in

protein can also cause a depletion in neurotransmitters. A high protein meal can actually

raise levels of certain neurotransmitters such as epinephrine (Dallman, 2003). Omega-3

fatty acids, which are routinely found in fish such as salmon can also affect brain

biochemistry, physiology, functioning, and play a role in some neuropsychiatric diseases

and cognitive decline (Bourre, 2005). Importantly, dietary omega-3 fatty acids appear to

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be effective in the prevention of stress and disorders such as depression (Bourre, 2005).

Researchers also recommend that certain nutrients are helpful for the body when people

are under stress. Helpful nutrients include antioxidants such as vitamin C, vitamin E, Beta

carotene, phytochemicals, B-vitamins, and water (Purdue University, 2013).

Roberts, Vaziri, and Barnard (2002) also note that diet can affect blood pressure,

which is a key indicator of stress as well as a good determinant about how someone will

respond to a stressful situation. A diet high in fat and refined carbohydrates has been

shown to increase blood pressure and increase stress on the body. In contrast, a low-fat

diet, with fruits, vegetables, and low fat dairy can reduce blood pressure (Appel et al.,

2005). High-fat diets can lead to increased blood pressure when cholesterol accumulates

in the arteries, making it more difficult for blood to flow freely (Harvard School of Public

Health, 2013). Further, diets high in sodium can also increase blood pressure. When

sodium is released into the bloodstream, sodium causes blood to expand in the arteries,

which increases internal pressure within the arteries (CDC, 2013). Reducing sodium

intake can dramatically decrease blood pressure and reduce the risk of hypertension

(Sacks et al., 2001). Research supports the association between stress and blood pressure;

however research tends to focus on stress as a predictor of blood pressure (Vrijkotte,

Doornen, & de Geus, 2000). It is important to note that people who suffer from high

blood pressure are also less capable of coping with stress compared to individuals with

lower blood pressure (Hixson, Gruchow, & Morgan, 1998). Therefore, a well-balanced

diet that promotes healthy blood pressure should help buffer effects on physical health. A

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well-balanced diet is also vital for maintaining a healthy body weight, which can further

improve health and reduce the risk of hypertension (CDC, 2011).

Diet and Obesity

The relationship between diet and obesity is surprisingly straight forward;

consume the same number of calories that the body burns over time and weight will stay

stable. Consume more calories than the body burns, weight will go up (Harvard School of

Public Health, 2013). Importantly, not all foods are created equal, and some types of

foods are more useful for controlling weight and preventing chronic diseases. For

example, foods such as whole grains, vegetables, fruits, and nuts can help control weight

and prevent diseases, including heart disease, stroke, and diabetes (Dansinger et al.,

2005). Importantly, the type of fat people eat is more important than the amount of fat

consumed. A study which examined the health behaviors of 42,000 nurses found a link

between weight gain and consumption of unhealthy fats (trans fats, saturated fats) but

consumption of healthy fats (monounsaturated fats, polyunsaturated fats) were not linked

to weight gain (Field, Willett, Lissner, & Colditz, 2007). There is also evidence that diets

high in protein are beneficial for weight loss because people tend to feel fuller on fewer

calories after high protein meals compared to high carbohydrate meals (Halton & Hue,

2004). The current paper has reviewed the stress buffering effects of a healthy diet as

well as the significant role that a healthy diet plays in weight management and obesity.

There is a need for studies to examine how stress resulting from occupational hazards can

impact employee obesity. Further, to the best of this author’s knowledge, few studies

have investigated how diet can buffer the negative effects of occupational hazards upon

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employee obesity rates. Many studies have reviewed how occupational stress can lead to

poor eating habits (Dallman, 2003), however there is a need for occupational health

psychologists to recognize that eating habits can also affect an employee’s ability to cope

with stress, which can also impact body composition (Hixson, Gruchow, & Morgan,

1998).

The current study will bridge the gap in the literature and examine how diet

interacts with stress resulting from occupational hazards and impact obesity in the

workplace. Based on research reviewed in this paper, employees who face more

occupational hazards will be more stressed, which can lead to increased fat accumulation.

Further, employees who eat a well-balanced diet will be healthier and better able to cope

with stress and regulate calorie consumption, reducing the risk of becoming overweight.

Therefore, it is likely that employees who eat a healthy diet will have a lower BMI

compared to employees who eat an unhealthy diet. This leads into the seventh set of

hypotheses for the current study.

Hypothesis 7a. Diet will moderate the relationship between physical workplace

hazards and BMI, meaning that employees who eat a healthy diet will be better able to

cope with exposure to physical hazards, and have a lower BMI compared to employees

who eat an unhealthy diet.

Hypothesis 7b. Diet will moderate the relationship between psychosocial

workplace hazards and BMI, meaning that employees who eat a healthy diet will be

better able to cope with exposure to psychosocial hazards, and have a lower BMI

compared to employees who eat an unhealthy diet.

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It is evident that a person’s diet has a large impact on overall health. For example,

there is strong evidence to suggest that a healthy, well-balanced diet is vital for

maintaining physical health, a healthy weight and preventing the onset of chronic

diseases (Dansinger et al., 2005). In order to promote total worker health employers and

researchers need to recognize that the eating habits of employees can affect both the

health of the employee and the organization. Importantly, diet can also affect more than

physical health. The food people eat can play a large role in maintaining good mental

health and preventing the development of mental illnesses (Mental Health Foundation,

2013). Thus far the effects of diet on physical health have been reviewed; however a full

review of the health benefits of a healthy diet would not be complete without examining

how diet also affects mental health.

Diet and Mental Health

According to the Mental Health Foundation (2013) good nutrition is essential for

maintaining good mental health and a number of mental health conditions may be

influenced by dietary factors. An individual’s diet can impact both short-term and long-

term mental health. Evidence also suggests that foods play an important role in the

development, management, and prevention of specific mental health problems such as

depression, schizophrenia, attention deficit disorder, and Alzheimer’s disease. Mental

health can be maintained and improved by ensuring that an individuals’ diet provides

adequate amounts of complex carbohydrates, essential fats, amino acids, vitamins,

minerals, and water (Mental Health Foundation, 2013).

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A number of studies have linked the intake of certain nutrients with mental health

disorders, including several different types of depression. For example, countries with

lower levels of fish in their diet suffer from higher rates of depression (Hibbeln,

Nieminen, Blasbalg, Riggs, & Lands, 2006). Fish contain healthy omega-3 fatty acids and

B vitamins, which have been linked to improved mental functioning and decreased rates

of depression (Frasure-Smith, Lesperance, & Julien, 2004). In contrast, countries with

higher consumption rates of fish, such as Japan have significantly lower rates of

depression (Hibbeln et al., 2006). Complex carbohydrates and food components such as

folic acid, selenium, and tryptophan have also been linked to a decrease in depressive

symptoms (Leung & Kaplan, 2009). Studies have found that individuals with low levels

of folate or folic acid in their diet are significantly more likely to be diagnosed with

mental health problems compared to people with higher intake levels (Morris, Fava,

Jacques, Selhub, & Rosenberg, 2003). Similarly, mental health disorders have been

linked to diets low in zinc, and vitamins B1, B2, and C (Bourre, 2006).

Fluid intake can also affect mood and mental health. Mild dehydration can impair

cognitive performance, decrease mood, and affect behavior (Ganio et al., 2011). An

average adult loses approximately 2.5 litres of water daily through the lungs as water

vapour, through skin perspiration, and through the kidneys as urine (Naghii, 2000). If an

individual does not adequately replenish lost fluids, symptoms of dehydration can occur,

including irritability, loss of concentration, and reduced mental functioning (Mental

Health Foundation, 2013). Another contributing factor to dehydration and mental health

problems is the overconsumption of caffeinated beverages. Consuming too much caffeine

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can increase blood pressure, anxiety, depressive symptoms, disrupt sleep, and

dehydration (Rogers, Smith, Heatherley, & Pleydell-Pearce, 2008).

The current study has reviewed the wide array of health benefits that a healthy,

well-balanced diet can offer. A healthy diet can buffer stress and improve both physical

and mental health (Mental Health Institute, 2013). Interestingly, research from the

biological and health sciences has remained segmented from organizational and

occupational health psychology. For example, there are studies linking diet to mental

health (Hibbeln et al., 2006); however the link between diet and mental health have rarely

been examined in an organizational context. There is a need for occupational and

organizational psychologists to investigate how improving employee eating habits and

mental health can be applied to a workplace setting. Additionally, there is a need for

studies to examine how a healthy diet can interact with stress resulting from occupational

hazards and impact employee mental health.

The present study will examine how a healthy diet can affect the relationship

between workplace hazards and mental health. The current study has argued that

employees who eat a well-balanced diet are at a reduced risk for developing mental

health problems and are also better equipped to cope with stress. In line with the current

argument, employees who eat a healthy diet are better able to cope with stress and less

likely to develop mental health problems compared to people who eat an unhealthy diet.

This leads into the eighth set of hypotheses.

Hypothesis 8a. Diet will moderate the relationship between physical workplace

hazards and mental health, meaning that employees who eat a healthy diet will be better

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able to cope with exposure to physical hazards, and have better mental health compared

to employees who have an unhealthy diet.

Hypothesis 8b. Diet will moderate the relationship between psychosocial

workplace hazards and mental health, meaning that employees who eat a healthy diet will

be better able to cope with exposure to psychosocial hazards, and have better mental

health compared to employees who have an unhealthy diet.

Research strongly supports that an individual’s diet can impact many aspects of

health, ranging from mental health to obesity (Hassan et al., 2003). The role of an

occupational health psychologist is to promote worker health (Fox & Spector, 2013). In

order to promote worker health, occupational health psychologists need to be able to

demonstrate to employers that the health of their employees can have financial

implications for the organization. Research shows that absences due to sickness are a

huge financial burden for organizations (Mercer, 2010). There is also strong evidence that

a healthy diet can improve health and buffer stress (Bourre, 2005), which could

potentially reduce the amount of sickness absences employees take. Linking employee

health practices to organizational outcomes such as absenteeism is a method

psychologists can use to push employers to invest in the health of their employees.

Diet and Sickness Absences

There is clear evidence that obesity and an unhealthy diet are related to poor

health (CDC, 2013). Further, several studies have linked obesity and general health to

sickness absences in the workplace (Bertera, 1990). Despite clear evidence that diet has a

profound effect on health (Hibbeln et al., 2006) and can affect an individual’s ability to

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cope with stress (Dallman, 2003), there is a lack of research specifically examining how

an individual’s diet can impact sickness absences in the workplace. Many studies that

have examined health and sickness absences have focused on obesity and chronic health

problems (Ferrie et al., 2007). Previous research may have given less attention to the link

between diet and sickness absences because diet may be considered a more distal

predictor of health status and sickness absences. For instance, a poor diet may not directly

lead to increased sickness absences, nonetheless diet can impact a person’s ability to cope

with stress (Bourre, 2005), which in turn can affect sickness absence. It may be the case

that researchers choose to focus on obesity and sickness absences because the association

is more direct, possibly resulting in a stronger association.

Although there are few studies that have specifically examined the link between

diet and sickness absences, there are studies that have focused on workplace health

promotion programs and absenteeism (Bertera, 1990). The main weakness in the

literature is that workplace health promotion programs provide information about how to

practice healthy behaviors without evaluating whether there were any changes in the

actual practice of health behaviors (Arneson & Ekberg, 2005). Further, many health

promotion studies in the workplace focus on whether a program brought about changes in

the outcome variable (e.g. absences or employee stress), without giving any attention to

health behavior changes (Harden, Peersmen, Oliver, Mauthner, & Oakley, 1999). Harden

et al. (1999) also point out that health promotion programs need more rigorous evaluation

for their effectiveness. Studies have tended to give more attention to the link between

work characteristics and sickness absences (Head et al., 2006), without acknowledging

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that health behaviors such as diet can buffer occupational stress (Bourre, 2005) and

potentially reduce sickness absences.

In addition there are studies that have investigated how to promote healthy eating

in the workplace without examining the link between diet and organizational outcomes

such as absenteeism or productivity. For example, Plotnikoff et al. (2005) examined the

use of email in promoting nutrition behavior in a work context. Employees who received

the email information showed increases in healthy eating behaviors and balancing food

intake with physical activity levels. Studies have also examined factors that can predict

employee participation in healthy eating programs in the workplace, ranging from

company size to the presence of a cafeteria (Lassen et al., 2007). Although it is critical to

understand how to promote health behaviors in the workplace, it is also important to

evaluate whether health promotion in the workplace has an impact on organizations such

as employee absences.

The current study has reviewed how a healthy diet can significantly improve the

health of an employee, and help employees deal with stress (Hixson, Gruchow, &

Morgan, 1998). Further, the current study has argued that there is a need for researchers

to examine how the health behaviors employees practice can affect both individual health

and organizational outcomes such as absenteeism. Given the strong link between diet and

health, and health and sickness absences it is surprising that previous researchers have not

investigated the potential stress buffering effects of diet upon employee sickness

absences. It is likely that employees who eat a well-balanced diet are healthier than

employees who eat an unhealthy diet, allowing their body’s to better cope with stress

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from occupational hazards. Further, employees who are healthier and better able to cope

with stress would likely suffer from less health problems and less sickness absences. This

leads into the ninth set of hypotheses for the present study.

Hypothesis 9a. Diet will moderate the relationship between physical workplace

hazards and sickness absences, meaning that employees who eat a healthy diet will be

better able to cope with exposure to physical hazards, and have less sickness absences

compared to employees who have an unhealthy diet.

Hypothesis 9b. Diet will moderate the relationship between psychosocial

workplace hazards and sickness absences, meaning that employees who eat a healthy diet

will be better able to cope with exposure to psychosocial hazards, and have less sickness

absences compared to employees who have an unhealthy diet.

Aging Workforce

Promoting total worker health involves more than promoting modifiable health

behaviors among the working population. In some instances, factors beyond an

employees’ control can affect health and safety both within and outside the workplace.

Specifically, an employee’s age can affect their health and safety. For example, older

workers are more likely to sustain severe injuries in the workplace compared to younger

workers and miss more workdays due to poor health (Wegman & McGee, 2004).

According to the Healthy Aging for a Sustainable Workforce Report (University of Iowa,

2009), current knowledge about keeping older workers safe and healthy is insufficient.

Organizations and researchers must explore new ways to promote healthy aging in the

workplace.

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Longer life expectancies, low birth rates, and the aging of the “baby boomer”

generation all impact the demographics of the current and future workforce (Harrington

& Heidkamp, 2013). The aging workforce creates significant economic, health, and social

challenges in the U.S. and internationally (United Nations, 2007). The U.S. census

predicts that by 2050, 19.6 million American workers will be 65 or older, making up 19%

of the total U.S. workforce. Further, the number of people in the workforce who are 65

years or older is expected to grow by 75% while the number of people in the workforce

who are 25 to 54 is only expected to grow by 2% (Harrington & Heidkamp, 2013).

Given the growing proportion of older individuals in the workplace it is necessary

to promote working conditions that allow aging workers to continue to work productively

and safely. Organizations and researchers can use the total worker health framework set

forth by NIOSH (2013) to enhance the health of older employees. Specifically, the CDC

(2013) states that emerging factors beyond the workplace jointly contribute to many

health and safety problems that confront workers. One important factor that can affect

health and safety beyond workplace factors is a person’s age. According to the 2004

National Academic Panel report on “Health and Safety Needs of Older Workers” more

research is needed to understand how to prevent work-related injury, illness, and promote

health among older workers. Further, knowledge gaps need to be filled to better

understand the biological, physiochemical, and psychosocial factors that affect aging

workers.

Despite negative stereotypes towards aging individuals, older individuals or

employees are just as cognitively capable of completing work compared to their younger

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counterpart s (National Institute of Mental Health, 2013). Further, older employees are no

more likely to suffer from mental health problems than younger employees (National

Institute of Mental Health, 2013). Distribution of onset of mental health problems can

differ by age, however older individuals appear to be just as mentally healthy, or healthier

than their younger peers. For example, younger adults (18-29 years old) suffer from the

highest rates of depression of any age group. In addition, the age group of 18-25 is most

likely to suffer from serious mental illness (National Institute of Mental Health, 2013). In

contrast, older adults are at an increased risk for mental health problems such as post-

traumatic stress disorder and panic disorder (Kessler et al., 2007). Despite the fact that the

current paper has reviewed many predictors of mental health, the research on the

association between age and mental health is not clear cut. Based on the current literature

review it appears that age is not an accurate predictor of overall, general mental health.

Rather, age may be better suited for predicting which kinds of mental health problems an

individual is at risk for developing. Importantly, one area of health where age can play a

significant factor is in the body composition (e.g., body fat vs. lean muscle tissue) of an

individual.

Age and BMI

One major health concern about the aging workforce is the increase in obesity

rates. Adults age 60 and over are more likely to be obese compared to younger adults

(Ogden et al., 2012). Further, BMI appears to linearly increase with age among women,

and increases with age among men in two stages; a dramatic increase between 20-40

years and then a slower increase between 40-60 years of age (Welon, Szklarska, Bielicki,

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& Malina, 2002). Research also cites that BMI is age dependent when used as an

indicator of body fatness, meaning a person’s age is a strong predictor of their BMI

(Gallagher et al., 1996).

To further complicate matters, increases in BMI are related to several chronic

diseases and a compromised immune system (Harvard School of Public Health, 2013).

Excess body fat can lead to an impaired immune system, further leading to a greater

incidence and severity of infectious diseases (Marti, Marcos, & Martinez, 2001). Further,

older individuals who have an impaired immune system or suffer from chronic health

problems have increased difficulty coping with occupational stress (Harvard School of

Public Health, 2013). Older employees are at a high risk for being obese (Ogden et al.,

2012), and a lack of physical or psychological resources to cope with stress can lead to

weight gain through two main mechanisms; weight gain related to an increased release in

stress hormones and weight gain associated with emotional eating (Torres & Nowson,

2007). To summarize, age is a risk factor for obesity, which negatively impacts the

immune system, which in turn can reduce a person’s ability to cope with stress, which

can eventually lead to more weight gain.

In order to promote total worker health, researchers and organizations need to

recognize that age-related increases in body fat can negatively impact employee health

and can be a financial burden for organizations (Finkelstein, Fiebelkorn, & Wang, 2005).

Organizations should actively promote physical activity and positive health behaviors in

the workplace; however employers need to target older employees, who are at the

greatest risk for obesity (Ogden et al., 2012). Many studies have examined health

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promotion/interventions in the workplace (Franche et al., 2005); however research has

failed to discuss the implications of workplace health promotion for older employees.

Based on the information reviewed in the current paper, employees accumulate

more body fat as they age. Additionally, high rates of occupational hazards and stress

also predict weight gain. It is likely that 2 major risk factors for obesity (stress and age)

will interact to amplify the probability of weight gain. For example, an employee who is

in their fifties and encounters a high rate of occupational hazards will most likely be at a

greater risk for being obese compared to an employee in their twenties, who encounters

many workplace hazards. This leads into the tenth set of hypotheses for the present study.

Hypothesis 10a. Age will moderate the relationship between physical workplace

hazards and BMI, meaning that younger employees will be better able to cope with

exposure to physical hazards, and have lower BMI compared to older employees.

Hypothesis 10b. Age will moderate the relationship between psychosocial

workplace hazards and BMI, meaning that younger employees will be better able to cope

with exposure to psychosocial hazards, and have lower BMI compared to older

employees.

The current paper has discussed the implications that age can have for employee

health. Specifically, age is a significant predictor of obesity (Welon et al., 2002), and

obesity is predictive of many chronic diseases, such as Type 2 diabetes, heart disease, and

certain types of cancers (CDC, 2011). In light of the fact that age is a significant predictor

of health status, including obesity and chronic health diseases, it is likely that age will

also impact a more distal outcome such as sickness absence. For example, age can play a

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role in obesity related chronic diseases, which in turn may affect how many sick days

employees take. Finally, Kivimäki et al. (2003) have argued that sickness absence can be

used as a global measure of health.

Age and Sickness Absence

Studies that have examined employee sickness absence need to give proper

attention to the effects that age can have on sickness absence. Data on age are often

presented as confounding variables, without any attempt to explain how age can impact

sickness absence (Allebeck et al., 2004). Further, some studies have controlled for age

when examining sickness absences without giving a proper explanation as to why age

was controlled for (Nielson et al., 1996). For example, Niedhammer et al. (1998) cites

age as a potential confounder variable when examining sickness absence, without giving

any explanation why age could be a confounding variable.

The limited number of studies that have examined the effects of age on sickness

absence have revealed important information. Brage, Nygard, and Tellnes (1998) found

evidence that long-term sickness absence due to musculoskeletal health problems was

strongly associated with age. Older employees are also at a greater risk for suffering

lower back injuries (Guo et al., 1995) and long-term work-related disability (Cheadle et

al., 1994). Further, Voss et al. (2008) compared self-reported sickness absence with

registered records of sickness absence. The results indicated that employees 50 years and

older had both the highest self-reported sickness absence and the most registered sickness

absences compared to younger age groups.

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The likely explanation for increased rates of sickness absence among older

employees is that sickness absence is a distal predictor of health. Specifically, sickness

absence rates are likely a result of a decline in physical health, which can come with older

age (Houx & Jolles, 1993). Further, age-related health decline may make it more likely

that stress resulting from occupational hazards will have a greater impact on health and

sickness absences among older employees. For example, older employees who are

exposed to occupational hazards will likely incur more sickness absences compared to

younger employees who are exposed to the same workplace hazards. The current study

will bridge the gap in the literature by being one of few studies to examine the

relationship between age and sickness absence without using age as a control variable.

The current study will propose the following:

Hypothesis 11a. Age will moderate the relationship between physical workplace

hazards and sickness absences, meaning that younger employees will be better able to

cope with exposure to physical hazards, and have less sickness absences compared to

older employees

Hypothesis 11b. Age will moderate the relationship between psychosocial

workplace hazards and sickness absences, meaning that younger employees will be better

able to cope with exposure to psychosocial hazards, and have less sickness absences

compared to older employees.

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CHAPTER 2

METHOD

Participants

Participants from the Northern Ireland Civil Service Workforce Health Survey

(Addley, Douglas, Mallon, & Mathewson, 2000), commissioned by the Northern Ireland

Civil Service (NICS) Workplace Health Committee was used for the current study. Refer

to Figure A1 in Appendices for an overview of the NICS research model. The study was

part of the NICS Workplace Health Improvement Programme. The Programme was

developed based on the idea that workplace health promotion has the potential to address

many of the antecedents of individual as well as public health while also making an

important contribution to organizational performance. The goal of the NICS Workplace

Health Improvement Programme was to complement pre-existing structures already in

place to support staff and promote their health. Health promotion areas of interest include

health and safety, welfare and occupational health services, work-life balance and health

promotion activities that take place across all departments and agencies on a regular

basis.

The entire data set consisted of 16,651 civil servant workers from Northern

Ireland, collected over the course of three months. Participants were 50.4% female and

49.6% male. The average age of participants was 38, ranging from 16 to 70 years of age.

Approximately 26% of the sample reported being single, 63% married, 5% cohabiting

and 6% separated or divorced. Approximately 94% of participants were permanent

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employees, 2% were on a fixed term/contract, and 4% were temporary workers. No data

was collected on ethnicity. Participant response rates varied by job grade. Response rates

were highest among Senior civil service workers (88.2%) and lowest among Industrial

staff (29.1%). For a complete description of all the job grades refer to Table B1 in

Appendices.

Measures

Physical Workplace Hazards. The following question was asked to participants

to measure physical hazards in the workplace: “Which of the following, in your view, are

causing your workplace to be unsafe or unhealthy?” Participants were then presented

with 13 potential hazards to choose from and select. Example options include the

lighting, noise levels, and unsafe floor surfaces. All items were summed, and participants

received a score ranging from 0 to 13, with higher scores indicating higher exposure

levels to physical hazards. Refer to Appendix A for the full list of options.

Based on previous research, physical hazards such as noise, lighting, and

ventilation are not always related to one another, although each factor is considered a

potential physical hazard in the workplace (Cox, 1993). For example, a workplace may

be excessively noisy but have adequate lighting. Both ventilation and lighting can affect

employees, however the absence or presence of one physical hazard is not related to the

absence or presence of another physical hazards. Each potential hazard can uniquely

contribute to and define the construct of workplace physical hazards. In line with the

current argument, the current paper argues that the construct of physical workplace

hazards is a formative model. The direction of causality is from the items to the construct

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in a formative model, whereas the direction of causality is from the construct to the items

in a reflective model (Edwards & Bagozzi, 2000). Therefore, a measure of internal

consistency would not be appropriate for a measure of physical hazards because items are

not expected to be correlated in a formative model.

Psychosocial Workplace Hazards. An adapted version of the U.K. Health and

Safety Executive’s Management Standards Work-Related Stress Indicator Tool (HSE

MS) was used to measure psychosocial hazards in the workplace. A general question was

first presented to participants: “Which if any of the following are causing you unwanted

stress in your job?” Example options include having too much work to do, poor

communication, and sexual harassment. All items were summed, and participants

received a score ranging from 0 to 45, with higher scores indicating higher exposure

levels to psychosocial hazards. Refer to Appendix B for a full list of questions.

The U.K. Health and Safety Executive’s Management Standards Work-Related

Stress indicator tool (HSE MS) is a widely used tool to measure stress in the U.K.

(Edwards et al., 2008). Recently, Edwards et al. (2008) conducted an analysis of the

psychometric properties of HSE MS using data from 39 organizations. Although the

original HSE MS has subscales for job demands, support, and control, analyses by

Edwards et al. (2008) suggest that deriving a single measure of work-related stress by

combining the subscales is appropriate.

The current study expands on the work of Edwards et al. (2008) and not only

derive a single score from the HSE MS, but also argue that the scale is based on a

formative model in contrast to a reflective model. The direction of causality is from the

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items to the construct in a formative model, whereas the direction of causality is from the

construct to the items in a reflective model. For the HSE MS scale, it would not be

expected that individual items will be highly correlated because each indicator uniquely

contributes to the meaning of the construct (MacKenzie, Podsakoff, & Jarvis, 2005). For

example, an employee may have high work demands such as unreasonable deadlines, but

also have boring or repetitive work. Both unreasonable deadlines and boring work can

independently contribute to stress, and both need not be related to contribute to stress.

Therefore, a measure of internal consistency would not be appropriate because it would

not be expected that indicators will be correlated or covary in a formative model

(Edwards & Bagozzi, 2000).

Physical Activity. The current study used self-report measures of physical

activity. The main advantage to the use of self-report measures of physical activity is that

these measures are practical, low cost, have a low participation burden and are well

accepted among researchers (Prince et al., 2008). In contrast, direct measures of exercise

and physical activity are often time and cost intensive, intrusive for participants, and

require specialized training and physical proximity of the participant for data collection

(Prince et al., 2008). In addition, self-report measures of physical activity have been

shown to be extremely reliable and demonstrate acceptable validity (Baranowski, 1988).

A main strength of the current study is that both exercise frequency and intensity are

measured. Sallis (1991) points out that researchers have not consistently measured

frequency and intensity of exercise despite the fact that both factors are necessary to

understand physical activity and fitness (Harvard School of Public Health, 2013).

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Three questions were used to measure the frequency and intensity of exercise.

Each item begins with the following question: “How many times a week on average do

you do the following kinds of exercise for more than 20 minutes?” Next, participants

were presented with three options. For example, “Strenuous exercise (Heart beats rapidly)

(e.g., running jogging, football, squash, basketball, vigorous swimming)”. Participants

then list the times per week they participated in a certain activity. The same format

follows for moderate exercise and mild exercise. Refer to Appendix C for the full list of

questions.

Diet. Dietary habits were measured using one question, with seven different

options for a response. For example, a question asks “Which of the following, if any, do

you consciously try to limit or avoid? (Tick all that apply). Participants are then given the

option to indicate which foods they try and avoid, e.g., sugar, fast foods, salt, fat/fatty

foods, caffeine, and red meat. Refer to Appendix D to view the full list of questions. For

the current study, a principal components analysis revealed one factor past the point of

inflection on a scree plot test. An exploratory factor analysis revealed that one factor was

present, with factor loadings ranging from .45 to .60. Overall, the internal consistency

was acceptable (α= .70).

The current measure of dietary intake is appropriate for the current study because

all six options used in the current study are predictive of health and well-being. For

example, consumption of fast foods and foods high in fat have been linked to coronary

heart disease (Ascherio, Katan, Zock, Stampfer, & Willett, 1999). High consumption of

sugary foods have been linked to obesity (Gibson, 2006) and high intake of sodium is

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associated with higher blood pressure (Vollmer et al., 2001). Consumption of red meat

has been linked to an increased risk of mortality (Sinha et al., 2009) and overconsumption

of caffeine can cause health problems such as anxiety and heart palpitations (National

Institute of Health, 2013). Overall, the items used in the current study to measure dietary

intake are predictive of an individual’s diet and health.

BMI. Two questions were used to determine participants’ BMI. The first question

asked “What is your weight without clothes?” Participants then list their weight in stones,

pounds, of kilograms. The second question used to determine BMI asked participants

“What is your height in bare feet?” Participants then listed their height in feet, inches, or

centimeters. All weight measurements were converted to kilograms and all height

measurements were converted to meters. Next the metric system formula was used to

calculate BMI. Based on the recommendations of the CDC (2013) BMI was calculated by

using the following formula: weight in kilograms divided by height in meters squared.

BMI scores < 18.5 are considered underweight. A normal weight score would be

18.5-24.9, overweight would be 25-29.9, and obesity would be a BMI of 30 or greater.

BMI is an accurate predictor for certain diseases such as heart disease, high blood

pressure, Type 2 diabetes, gallstones, breathing problems, and certain cancers for both

men and women (National Heart, Ling, and Blood Institute, 2013). Despite the strengths

of using BMI there are some limitations. BMI may overestimate body fat in athletes and

others who have a muscular build and BMI may underestimate body fat in older

individuals and others who have lost muscle (National Heart, Lung, and Blood Institute,

2013). Nonetheless, BMI is an efficient tool for measuring body composition because it is

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less time intensive and more cost effective than using other methods such as bioelectrical

impedance or hydrostatic underwater weighing.

General Mental Health-12 Questionnaire (GHQ-12). The GHQ-12 (Goldberg

& Williams, 1988) was used to measure the general mental health of participants. The

GHQ-12 is used to detect mental health problems in the general population. The

questionnaire assesses participants’ current state and asks if that differs from his or her

usual state. An example question from the questionnaire is “Have you recently been able

to concentrate on whatever you are doing?” Participants then choose between 4 options;

better than usual, same as usual, less than usual, or much less than usual. The first two

options on the questionnaire are scored as zero, and the third and fourth options are

scored as 1. Thus, each participant will produce an overall score out of a maximum

possible score of 12. A score of 4 or more is indicative of mild psychiatric symptoms.

Refer to Appendix E to view a copy of the GHQ-12. For the current study a principal

components analysis revealed one factor past the point of inflection on a scree plot test.

An exploratory factor analysis revealed that one factor was present, with factor loadings

ranging from .64 to .82. The reliability of the questionnaire was also found to be very

high in the current study (α = .92).

The validity of the GHQ-12 as a tool for detecting mental health problems has

been established by comparing the GHQ-12 scores with the Composite International

Diagnostic Interview, which can generate diagnoses using the DSM-IV systems

(Goldberg et al., 1997). Further, the GHQ-12 has been shown to produce similar

diagnoses of psychiatric problems as compared to clinical interviews. For example,

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Hardy et al. (1999) validated the GHQ-12 with a sample of the U.K. workforce and found

a strong correlation (.70) with an independent standardized clinical interview. Both

Goldberg et al. (1997) and Hardy et al. (1999) report that people scoring four or more

points on the GHQ-12 (GHQ-12 scoring method) are identified as likely cases for minor

psychiatric disorders while individuals scoring three or less points are classified as

psychologically healthy. Further, the GHQ-12 scoring method has been found to be a

more reliable scoring method compared to the likert scoring method (Hankins, 2008).

According to Houdmont et al. (in press) the GHQ-12 scoring method was designed to

reduce measurement errors that might be introduced by an individuals’ tendency to

endorse extreme responses or to over-use the scale mid-points. The current paper used the

GHQ-12 scoring method in order to reduce measurement errors and accurately measure

psychiatric problems among employees.

Absences Due to Sickness. Workplace absences were measured with the

following question: “In the last year, how many working days were you absent from work

because you were sick, injured, or disabled?” Participants were then instructed to write

the approximate number of days. Sickness absences are an important variable to measure

because it can be used as both a global measure of health (Kivimäki et al., 2003) and be

used as an organizational outcome which affects an employer’s profits. There is some

debate as to whether self-reported sickness absences is as reliable as certified sickness

absence records (Marmot et al., 1995); however research suggests that employees are

fairly accurate at reporting absences due to sickness (Voss, Stark, Alfredsson, Vingard, &

Josephson, 2008). Ferrie et al. (2005) also confirms that there is a strong agreement

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between self-reported sickness absences and number of annual recorded sickness absence

days among both sexes. Self-reported sickness absences are also strongly related to

overall health. Finally, data on absences are often collected routinely by employers,

which reduces the potential recall and response set biases attributable to self-reported

indicators of health (Folger & Belew, 1985).

Control Variables. Gender was controlled for in the current study. Research has

shown that there are gender differences in body mass index between men and women.

For example, Kuan, Ho, Shuhaili, and Gudum (2011) found that men are more likely to

be obese compared to women and women are more likely to be underweight. Males and

females differ in their “ideal” body image, which likely contributes to differences in BMI.

Kuan et al. (2011) report that females preferred their ideal figure to be underweight

whereas more males chose an overweight figure as an ideal body type. Kuan et al. (2011)

also found that women were more likely to diet, use self-induced vomiting to lose weight

and use laxatives and exercise as a weight-loss strategy. Therefore, the effects of gender

on BMI were controlled for in the current study.

Gender is also a critical determinant of mental health and mental illness (WHO,

2013). Eaton et al. (2012) report that women are more likely to be diagnosed with anxiety

or depression, while men are at a higher risk for substance abuse or antisocial disorders.

Eaton et al. (2012) explain that women are more likely to internalize emotions, which can

lead to withdrawal, loneliness, and depression. In contrast, men are more likely to

externalize emotions, which can lead to aggression and impulsive behavior. Therefore,

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the current study controlled for any effects that gender may have on self-reported mental

health.

There are also gender differences in sickness absences among employees.

Laaksonen, Martikainen, Rahkonen, and Lahelma (2008) found that women had a 46%

higher risk for sickness absences compared to men. Importantly, the overall gender

differences in sickness absence are due to women being more likely to suffer from short

absences spells and men more likely to have longer-term sickness spells. Laaksonen et al.

(2010) report that gender differences in sickness absence are also attributable to the

differences in mental and behavioral disorders and musculoskeletal disease. North et al.

(1993) also found gender differences in sickness absence. North et al. (1993) concluded

that gender differences in sickness absence were due to socioeconomic differences

between working men and women. Based on the discussed research above the current

study controlled for the effects of gender on sickness absence.

Procedures

Refer to Figure A1 in Appendices for an overview of the NICS Workplace Health

Improvement Programme model. First, a pilot test of the questionnaire was conducted

with a random sample of employees from NICS to ensure the questionnaire was easy to

complete. The pilot test was also used to predict what the response rates would look like

for the full survey. The results from the pilot test proved that the survey worked well.

Nonetheless, some minor changes were made to the wording and format of the survey as

a result of the pilot test.

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Next, self-completion postal questionnaires were sent out to all 28,937 NICS

staff. The questionnaire was designed to specifically meet the needs of NICS and

incorporate key benchmark variables regarding employee health and organizational

needs. The survey was sent out on Monday, March the 27th, 2000. Reminder letters were

sent out to all participants 3 weeks after the initial survey was sent out and the survey was

closed on May the 30th, 2000. At the closing date of the survey a total of 16,651

employees completed the questionnaire. The response rate was approximately 57.5%.

Splitting the Data by Pay Grade. The data in the current study were split into 2

groups; employees in the senior level pay grade and employees in the lower level pay

grade. Employees in the senior level pay grade included senior civil service workers and

senior principal workers. The lower level pay grade group consisted of deputy principals,

staff officers, industrial staff, administrative assistants and officers, and executive

officers. The rationale for testing the hypotheses separately was that occupational pay

grade has been shown to be an important predictor of health, especially among British

civil servant workers (Marmot et al., 1991). Further, Emslie, Hunt, and Macintyre (1999)

found that employees in the lower pay grades were more likely to be exposed to poor

working conditions and suffer an increased incidence of health problems. Employees in

lower pay grades are also more likely to miss work due to sickness or injury (Hemingway

et al., 1997), and be less physically and mentally healthy (North et al., 1993) compared to

higher pay grade employees.

Although studies have found health differences between pay grades (Marmot et

al., 1996), more studies need to compare the highest pay grade employees to those from

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the lower pay grades. For example, it is likely that the differences in exposure to

workplace hazards are greater between the senior pay grade and the remaining pay grades

compared to the differences between each individual pay grade. Further, differences in

health status between lower pay grades have been shown to be less extreme than the

differences between the health status of the highest pay grade employees and the

remaining lower pay grades (Marmot, Rose, Shipley, & Hamilton, 1978; Marmot et al.,

1995).

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CHAPTER 3

RESULTS

Data Screening

All statistical analyses were conducted using SPSS 16.0. The data consisted of 16,

636 participants. Before beginning the analyses, the data were standardized and

examined for outliers with unusually large z-scores. Case ID 12644 and 6952 had

unusually high z-scores of 8.9 for the exercise variable, and were therefore deleted. Case

3313 had a high z-score of 6.8 for the psychosocial hazard variable, and was deleted.

Case 12690 had a z-score of 8.0 for the BMI variable and was deleted. Cases 12029,

2747, 11487, 7723, 5055, 3516, 11543, 11459, 14459, 14937 and 5966 had unusually

high z-scores of 11.65 for the sickness absence variable, and were therefore deleted.

Next, multivariate outliers were examined for Hypotheses 1a through 11b, examining

Standardized residuals, Cook’s Distance, and Standardized DfBetas. Case 6065 was

deleted because the standardized residual was 12.18, which is unusually high. Due to

missing data, pairwise deletion was used in all subsequent analyses for both data sets.

Descriptive Statistics of the Measured Variables

The descriptive statistics for the entire sample were computed. Means and

standard deviations for all of the scales and questions are presented in Table B2. The

mean score for physical hazards was 1.33, with a standard deviation of 1.98. The mean

score for psychosocial hazards was 5.98, with a standard deviation of 5.43. In addition,

the mean score for BMI was 25.53, with a standard deviation of 4.23. The mean score for

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the GHQ-12 measure of general mental health was 2.44, with a standard deviation of

3.48. The mean days of sickness absence were 11.44, with a standard deviation of 28.84.

Further, the mean days of exercise were 7.69, with a standard deviation of 3.94. The

mean score for diet was 2.61, with a standard deviation of 1.61. Finally, the mean age

was 38.87, with a standard deviation of 10.13.

The descriptive statistics for the senior level pay grade (N = 1025) and lower level

pay grade employees (N = 14, 873) were computed and compared. Means, standard

deviations and t-test results for all of the scales and questions are presented in Table B3.

Senior level pay grade employees (M = 0.74, SD = 1.57) were exposed to significantly

less physical hazards compared to lower level pay grade (M = 1.38, SD = 2.00), t(15,896)

= -10.05, p < .001. In contrast, senior level pay grade (M = 5.83, SD = 4.66) did not

significantly differ in exposure to psychosocial hazards compared to lower level pay

grade (M = 6.05, SD = 5.49), t(15,896) = -1.27, p > .05. Further, senior level pay grade

(M = 25.31, SD = 3.21) did not significantly differ from lower level pay grade in BMI (M

= 25.52, SD = 4.29), t(14,909) = -1.49, p > .05. In addition, senior level pay grade did

report better mental health scores, via lower scores (M = 2.10, SD = 3.06) compared to

lower level pay grade (M = 2.48, SD = 3.47), t(15,896) = -3.36, p < .05). Furthermore,

senior level pay grade reported significantly less sickness absences (M = 5.57, SD =

18.44) compared to lower level pay grade (M = 11.81, SD = 29.03), t(14,736) = -6.53, p <

.001. Additionally, senior level pay grade (M = 7.67, SD = 3.64) did not significantly

differ in exercise frequency compared to lower level pay grades, (M = 7.71, SD = 3.96),

t(12,757) = -0.24, p > .05; however senior level pay grade did report a healthier diet (M =

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3.05, SD = 1.57) compared to lower level pay grade (M = 2.59, SD = 1.60), t(15,896) =

8.81, p <.001. Finally, senior level pay grade (M = 47.59, SD = 7.85) had a significantly

higher mean age compared to lower level pay grade (M = 38.18, SD = 9.95), t(15,735) =

29.40.

Finally, Skewness and Kurtosis were calculated for each variable, for both senior

pay grade and the lower pay grade group. For the senior pay grade, the Skewness and

Kurtosis of the measured variables are as follows: BMI (0.66, 1.76); GHQ-12 score (1.60,

1.70); sickness absence (2.02; 3.84); physical hazards (2.58, 7.38); psychosocial hazards

(1.22, 1.93); physical activity (2.18, 8.85); diet (-.14, -.52); and age (-.64, .09).

For the lower pay grade, the Skewness and Kurtosis of the measured variables are as

follows: BMI (1.08, 2.77); GHQ-12 score (1.39, .78); sickness absence (1.13, .18);

physical hazards (1.44, 1.52); psychosocial hazards (1.12, 1.20); physical activity (1.75,

5.65); diet (.20, -.65); and age (.19, -.57). The Kurtosis for physical activity was high for

both senior and lower pay grades (8.85 & 5.65). Additionally, physical hazards had a

high kurtosis for the senior pay grade (7.38), indicating a Leptokurtic distribution. The

distributions suggests that values are concentrated around the mean, with thicker tails,

meaning there is a high probability for extreme values.

Correlation Analyses

Hypotheses 1-11 were examined for the entire sample (N = 16,636), looking at the

relationships between the independent variables, dependent variables and moderator

variables. Table B2 presents the correlations among all the variables. In support of

hypothesis 1a, physical hazards were positively related to BMI, (r = .02, p < .05). In

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support of hypothesis 1b, psychosocial hazards were positively related to BMI, (r = .07, p

< .05). In support of hypothesis 2, physical hazards were positively related to a higher

score on the GHQ-12 mental health questionnaire, with higher scores signifying worse

mental health, (r = .16, p < .001). Although not a hypothesis, psychosocial hazards were

significantly related to a higher score on the GHQ-12, (r = .46, p < .001). In support of

hypothesis 3a, physical hazards were significantly related to sickness absence, (r = .07, p

< .001). In support of hypothesis 3b, psychosocial hazards were significantly related to

sickness absence, (r = .10, p < .001). In partial support of hypotheses 4a, 4b, 5a, 5b, 6a,

and 6b, exercise was significantly related to BMI (r = -.07, p < .001), GHQ-12 (r = -.09,

p < .001) and sickness absence (r = -.040, p < .001). In partial support of hypothesis 7a

and 7b, diet was negatively related to BMI, (r = -.03, p < .001), however diet was not

significantly related to GHQ-12 (r = -.002, p > .05) or sickness absence (r = .02, p > .05).

In partial support of hypotheses 10a and 10b, age was positively related to BMI (r = .17,

p < .001), however age was not significantly related to sickness absence, (r = .003, p >

.05).

Hypotheses 1-11 were also examined for the senior level pay grade sample,

looking at the relationship between the independent variables, dependent variables and

moderator variables. Table B4 presents the correlations among all the variables. Failing

to support hypothesis 1a, physical hazards were negatively related to BMI, (r = -.08, p <

.05). In support of hypothesis 1b, psychosocial hazards were positively related to BMI, (r

= .08, p < .05). In support of hypothesis 2, physical hazards were positively related to a

higher score on the GHQ-12 mental health questionnaire, with higher scores signifying

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worse mental health, (r = .12, p < .001). Although not a hypothesis, psychosocial hazards

were significantly related to a higher score on the GHQ-12, (r = .48, p < .001). Failing to

support hypothesis 3a, physical hazards were not significantly related to sickness

absence, (r = .05, p > .05). Failing to support hypothesis 3b, psychosocial hazards were

not significantly related to sickness absence, (r = .05, p > .05). In partial support of

hypotheses 4a and 4b, exercise was significantly related to BMI (r = -.14, p < .001);

however exercise was not related to GHQ-12 (r = -.01, p > .05) and sickness absence (r =

-.003, p > .05). In partial support of hypothesis 7a and 7b, diet was negatively related to

BMI, (r = -.12, p < .001), however diet was not significantly related to GHQ-12 (r = .04,

p > .05) or sickness absence (r = .02, p > .05). In partial support of hypotheses 10a and

10b, age was positively related to BMI (r = .15, p < .001), however age was not

significantly related to sickness absence, (r = -.01, p > .05).

Hypotheses 1-11 were also examined for the lower level pay grade sample,

looking at the relationship between the independent variables, dependent variables and

moderator variables. Table B5 presents the correlations among all the variables.

Supporting hypothesis 1a, physical hazards were positively related to BMI, (r = .03, p <

.05). In support of hypothesis 1b, psychosocial hazards were positively related to BMI, (r

= .07, p < .01). In support of hypothesis 2, physical hazards were positively related to a

higher score on the GHQ-12 mental health questionnaire, with higher scores signifying

worse mental health, (r = .16, p < .001). Although not a hypothesis, psychosocial hazards

were significantly related to a higher score on the GHQ-12, (r = .46, p < .001). In support

of hypothesis 3a, physical hazards were significantly related to sickness absence, (r = .07,

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p < .001). Additionally, in support of hypothesis 3b, psychosocial hazards were

significantly related to sickness absence, (r = .11, p < .001). In partial support of

hypotheses 4a, 4b, 5a, 5b, 6a and 6b exercise was significantly related to BMI (r = -.06, p

< .001), GHQ-12 (r = -.10, p < .001) and sickness absence (r = -.04, p < .001). In partial

support of hypothesis 7a, 7b, 9a and 9b diet was negatively related to BMI, (r = -.02, p <

.05) and sickness absence (r = .02, p < .05); however diet was not significantly related to

GHQ-12 (r = -.004, p > .05). In partial support of hypotheses 10a, 10b, 11a and 11b age

was positively related to BMI (r = .18, p < .001) and sickness absence, (r = .02, p < .05).

Regression Analyses

Regression analyses were conducted to test hypotheses 1-3. The results for the

linear regression analyses for hypotheses 1-3 are presented in Tables B6 to B8. The linear

regression analyses failed to support Hypothesis 1a for the senior level pay grade

employees. Although significant, the effect was in the opposite direction, with physical

workplace hazards being negatively related BMI, controlling for gender and psychosocial

hazards, B = -.17, t(891) = -2.50, p < .05. The results for Hypothesis 1a indicate that

physical hazards are associated with a reduced BMI for the senior level pay grade.

Physical hazards and the control variables also explained a significant proportion of

variance in BMI, R2 = .10, F(3, 892) = 31.30, p < .01. The linear regression analyses

supported Hypothesis 1a for the lower level pay grade employees, in which physical

workplace hazards significantly predicted BMI, controlling for gender and psychosocial

hazards, B = .04, t(14,000) = 2.16, p < .05. The results for Hypothesis 1a indicate that

physical hazards are associated with an increased BMI for the lower level pay grade.

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Physical hazards and the control variables also explained a significant proportion of

variance in BMI, R2 = .03, F(3, 14,001) = 165.18, p < .05. Refer to Table B6 for results.

The linear regression analyses supported Hypothesis 1b for the senior level pay

grade employees, in which psychosocial workplace hazards significantly predicted BMI,

controlling for gender and physical hazards, B = .08, t(891) = 3.64, p < .01. The results

for Hypothesis 1b indicate that psychosocial hazards are associated with an increased

BMI for the senior level pay grade employees. Psychosocial hazards and the control

variables also explained a significant proportion of variance in BMI, R2 = .10, F(3, 892) =

31.27, p < .01. The linear regression analyses also supported Hypothesis 1b for the lower

level pay grade employees, in which psychosocial workplace hazards significantly

predicted BMI, controlling for gender and physical hazards, B = .05, t(14,000) = 7.09, p <

.01. The results for Hypothesis 1b indicate that psychosocial hazards are associated with

an increased BMI for the lower level pay grade. Psychosocial hazards and the control

variables also explained a significant proportion of variance in BMI, R2 = .03, F(3,

14,001) = 165.18, p < .01. Refer to Table B6 for results.

The linear regression analyses failed to support Hypothesis 2 for the senior level

pay grade employees, in which physical workplace hazards did not significantly predict

GHQ-12 scores, while controlling for gender and psychosocial hazards, B = -.04, t(1015)

= -.76, p > .05. The results for Hypothesis 2 indicate that physical hazards are not

associated with improved mental health for the senior level pay grade. However, physical

hazards and the control variables explained a significant proportion of variance in GHQ-

12 scores, R2 = .21, F(3, 1016) = 31.27, p < .05. The linear regression analyses also

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failed to support Hypothesis 2 for the lower level pay grade employees, in which physical

workplace hazards did not significantly predict GHQ-12 scores, while controlling for

gender and psychosocial hazards, B = -.03, t(14,852) = -1.89, p > .05. The results for

Hypothesis 2 indicate that physical hazards are not associated with improved mental

health for the lower level pay grade. However, physical hazards and the control variables

explained a significant proportion of variance in GHQ-12 scores, R2 = .21, F(3, 14,853) =

1.33, p < .05. Refer to Table B7 for results.

The linear regression analyses failed to support Hypothesis 3a for the senior level

pay grade employees, in which physical workplace hazards did not significantly predict

sickness absences, controlling for gender and psychosocial hazards, B = .28, t(940) = .70,

p > .05. The results for Hypothesis 3a indicate that physical hazards are not associated

with increased sickness absences for the senior level pay grade. However, physical

hazards and the control variables explained a significant proportion of variance in

sickness absences, R2 = .01, F(3, 941) = 4.48, p < .01. The linear regression analyses

supported Hypothesis 3a for the lower level pay grade employees, in which physical

workplace hazards significantly predicted sickness absences, controlling for gender and

psychosocial hazards, B = .43, t(13,770) = 3.22, p < .01. The results for Hypothesis 3a

indicate that physical hazards are associated with increased sickness absences for lower

level pay grade. Physical hazards and the control variables also explained a significant

proportion of variance in sickness absences, R2 = .02, F(3, 13771) = 97.03, p < .01. Refer

to Table B8 for results.

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The linear regression analyses failed to support Hypothesis 3b for the senior level

pay grade employees, in which psychosocial workplace hazards did not significantly

predict sickness absences, controlling for gender and physical hazards, B = .14, t(940) =

1.06, p > .05. The results for Hypothesis 3b indicate that psychosocial hazards are not

associated with increased sickness absences for the senior level pay grade. However,

psychosocial hazards and the control variables explained a significant proportion of

variance in sickness absences, R2 = .01, F(3, 941) = 4.48, p < .01. The linear regression

analyses did support Hypothesis 3b for the lower level pay grade employees, in which

psychosocial workplace hazards significantly predicted sickness absences, controlling for

gender and physical hazards, B = .53, t(13,770) = 10.93, p < .01. The results for

Hypothesis 3b indicate that psychosocial hazards are associated with increased sickness

absences for the lower level pay grade. Psychosocial hazards and the control variables

also explained a significant proportion of variance in sickness absences, R2 = .02, F(3,

13,771) = 97.03, p < .01. Refer to Table B8 for results.

Supported Interactions

Refer to Table B27 for a results summary of supported interactions. For all

moderation analyses workplace hazards and the moderator variable were mean centered

to reduce multicollinearity between the standard errors of the IV and moderator. Next, the

mean centered IV and moderator were multiplied together to create an interaction

variable.

Age was examined as a moderator between physical hazards and sickness

absence. The results for the moderation analyses are presented in Table B23. First,

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physical hazards and age, along with the control variables gender and psychosocial

hazards were entered as predictors of the dependent variable, sickness absence. The

results for the lower level pay grade indicated that there was a direct effect for physical

hazards on sickness absence, B = .42, t(13,657) = 3.18, p < .05. The addition of the

interaction term between physical hazards and age significantly added to the prediction of

sickness absence, F Change = 2.92, p < .05, supporting Hypothesis 11a. Further, the

simple slopes were tested for significance using an interaction effects spreadsheet

(Dawson, 2011).The simple slopes were found to be significant, B = .09, t(13,657) = .04,

p < .05 and B = 1.55, t(13,657) = .03, p < .05. (Age values set to 20 & 50). (See Figure

A16 for simple slopes plot).

Finally, age was examined as a moderator between psychosocial hazards and

sickness absence. The results for the moderation analyses are presented in Table B24.

First, psychosocial hazards and age, along with the control variables gender and physical

hazards were entered as predictors of the dependent variable, sickness absence. The

results for the lower level pay grade indicated that there was a direct effect for

psychosocial hazards on sickness absence, B = .54, t(13,657) = 11.11, p < .05. The

addition of the interaction term between psychosocial hazards and age significantly added

to the prediction of sickness absence, F Change = 3.58, p < .05, supporting Hypothesis

11b. Further, the simple slopes were tested for significance using an interaction effects

spreadsheet (Dawson, 2011). The simple slopes were found to be significant, B = .77,

t(13,657) = .02, p < .05 and B = 1.07, t(13,657) = .01, p < .05. (Age values set to 20 &

50). (See Figure A17 for simple slopes plot).

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Non-Supported Interactions

Physical Activity. Refer to Table B9 for results. Physical activity did not

moderate the relationship between physical hazards and BMI for the senior level pay

grade, F Change = .37, p > .05, and the lower level pay grade, F Change = .47, p > .05,

failing to support hypothesis 4a. See Figure A2 for simple slopes. Additionally, physical

activity did not moderate the relationship between psychosocial hazards and BMI for the

senior level pay grade, F Change = .003, p > .05, and the lower level pay grade, F

Change = .08, p > .05, failing to support Hypothesis 4b. Refer to Table B10 for results

and Figure A3 for simple slopes.

Further, physical activity did not moderate the relationship between physical

hazards and mental health for the senior level pay grade, F Change = .19, p > .05, and the

lower level pay grade, F Change = .48, p > .05, failing to support Hypothesis 5a. Refer to

Table B11 for results and Figure A4 for simple slopes. In addition, physical activity did

not moderate the relationship between psychosocial hazards and mental health for the

senior level pay grade, F Change = .04, p > .05, and the lower level pay grade, F Change

= 3.24, p > .05, failing to support Hypothesis 5b. Refer to Table B12 for results and

Figure A5 for simple slopes.

Next, physical activity did not moderate the relationship between physical hazards

and sickness absence for the senior level pay grade, F Change = .10, p > .05, and the

lower level pay grade, F Change = 1.29, p > .05, failing to support Hypothesis 6a. Refer

to Table B13 for results and Figure A6 for simple slopes. Finally, physical activity did

not moderate the relationship between psychosocial hazards and sickness absence for the

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senior level pay grade, F Change = 2.41, p > .05, and the lower level pay grade, F

Change = .01, p > .05, failing to support Hypothesis 6b. See Table B14 for results and

Figure A7 for simple slopes.

Diet. Diet did not moderate the relationship between physical hazards and BMI

for the senior level pay grade, F Change = .91, p > .05, and the lower level pay grade, F

Change = 1.85, p > .05, failing to support Hypothesis 7a. Refer to Table B15 for results

and Figure A8 for simple slopes. Additionally, diet did not moderate the relationship

between psychosocial hazards and BMI for the senior level pay grade, F Change = 2.46,

p > .05, and the lower level pay grade, F Change = .52, p > .05, failing to support

Hypothesis 7b. Refer to Table B16 for results and Figure A9 for simple slopes.

Next, diet did not moderate the relationship between physical hazards and mental

health for the senior level pay grade, F Change = 1.75, p > .05, and the lower level pay

grade, F Change = .72, p > .05, failing to support Hypothesis 8a. Refer to Table B17 for

results and Figure A10 for simple slopes. In addition, diet did not moderate the

relationship between psychosocial hazards and mental health for the senior level pay

grade, F Change = 1.76, p > .05, and the lower level pay grade, F Change = .06, p > .05,

failing to support Hypothesis 8b. Refer to Table B18 for results and Figure A11 for

simple slopes.

Additionally, diet did not moderate the relationship between physical hazards and

sickness absence for the senior level pay grade, F Change = .52, p > .05, and the lower

level pay grade, F Change = .00, p > .05, failing to support Hypothesis 9a. Refer to Table

B19 for results and Figure A12 for simple slopes. Finally, diet did not moderate the

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relationship between psychosocial hazards and sickness absence for the senior level pay

grade, F Change = .11, p > .05, and the lower level pay grade, F Change = 1.64, p > .05,

failing to support Hypothesis 9b. Refer to Table B20 for results and Figure A13 for

simple slopes.

Age. Age also did not moderate the relationship between physical hazards and

BMI for the senior level pay grade, F Change = 1.67, p > .05, and the lower level pay

grade, F Change = .26, p > .05, failing to support Hypothesis 10a. Refer to Table B21 for

results and Figure A14 for simple slopes. Further, age did not moderate the relationship

between psychosocial hazards and BMI for the senior pay grade, F Change = 3.43, p >

.05, and the lower level pay grade, F Change = .06, p > .05, failing to support Hypothesis

10b. Refer to Table B22 for results and Figure A15 for simple slopes.

Moreover, age did not moderate the relationship between physical hazards and

sickness absence for the senior level pay grade, F Change = 1.18, p > .05, failing to

support Hypothesis 11a. Refer to Table 23 for results and Figure A16 for simple slopes.

Finally, age did not moderate the relationship between psychosocial hazards and sickness

absence for the senior level pay grade, F Change = .02, p > .05, failing to support

Hypothesis 11b. Refer to Table B24 for results and Figure A17 for simple slopes.

Alternative Analyses

Literature discussed throughout the current paper has suggested that both BMI

and sickness absences can be affected by a person’s age. Therefore, hypotheses 1-3 were

also tested by splitting employees into groups based on age; under 30, 30-40, 40-50, and

above 50. The rationale is that employees in the younger age groups will have a lower

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BMI and less sickness absences. The current paper examined age as a moderator between

hazards and health outcomes; however it is also important to examine whether health can

differ within particular age groups. Identifying the health needs of specific age groups

can aid policy makers in the creation of age-specific health interventions. The results

from the regression analyses indicated small, but no major differences in health outcomes

by grouping employees into age groups. Refer to Table B25 for Results.

Research reviewed in the current paper has also suggested that an employees’

salary/pay grade can influence health outcomes. Specifically, employees in lower level

pay grades may face more occupational stress and have less resources to cope with stress

and health problems compared to senior level pay grade employees. In order to examine

whether there were differences in health outcomes among lower pay grade employees the

lower level pay grade employees were divided into 4 groups; pay grade B, pay grade C,

pay grade D and industrial staff. Regression analyses were conducted to re-test

hypotheses 1-3, using the new grouping scheme. The results indicated that there were

small differences in health outcomes based on splitting the lower pay grade into 4 smaller

groups; however, there was no clear trend demonstrating stronger effects of hazards on

health as you get to lower pay grades. The results confirmed that the initial grouping of

employees into senior level and lower level pay grade may be optimal for examining

health differences between pay grades. The differences in health outcomes appear to be

strongest between senior level pay grade and lower level pay grade. Refer to Table B26

for results.

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CHAPTER 4

DISCUSSION

The purpose of this dissertation was to investigate the link between workplace

hazards and employee health. More specifically, I examined how physical and

psychosocial hazards in the workplace can impact body mass index (BMI), mental health,

and sickness absence. Further, the purpose of this dissertation was to examine how

modifiable health behaviors and personal factors can buffer the negative effects of

workplace hazards on health. Specifically, I examined how physical activity, diet, and

age affect how physical and psychosocial hazards impact BMI, mental health, and

sickness absence.

Although the link between occupational stress and employee health has been well-

studied, the link between specific workplace hazards and employee physical and mental

health has not. Additionally, there is a need for studies to operationally define what a

workplace hazard is. Moreover, there is a need for researchers to determine which

factors can buffer the negative effects that workplace hazards can have on employee

health. This dissertation has bridged the gap in the literature by proposing a unifying

definition of workplace hazards and by examining how both physical and psychosocial

workplace hazards can impact different health outcomes.

Finally, this dissertation expanded upon previous research by investigating how

modifiable health behaviors and age can interact with workplace hazards to predict

employee health. It was predicted that both physical and psychosocial workplace hazards

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would negatively impact employees’ physical and mental health. Overall, the results

confirmed the prediction that physical and psychosocial hazards would be related to a

higher BMI among employees; however there was one instance where physical hazards

were related to a lower BMI for the senior level pay grade. Contrary to what I predicted,

physical hazards were not related to mental health; however psychosocial hazards were

related to worse mental health. Further, I predicted that physical and psychosocial

workplace hazards would be positively related to sickness absences. Among the lower

level pay grade employees, both physical and psychosocial hazards predicted sickness

absences. Interestingly, physical and psychosocial hazards did not predict sickness

absences among senior level pay grade employees. It was also hypothesized that physical

activity, diet, and age would all moderate the relationship between hazards and health

outcomes; where greater physical activity, a healthier diet, and a lower age would help

buffer the negative effects of hazards on health. The results did confirm that age

moderates the relationship between hazards and sickness absence among the lower pay

grade employees; however, physical activity and diet did not affect the relationship

between hazards and health for both pay grades.

Theoretical Implications

Workplace Hazards and BMI. In order to discuss the theoretical implications of

the findings in the current study, the relationship between workplace hazards and BMI

will be reviewed. Research has found a consistent link between occupational stress and

weight gain among employees (Martikainen & Marmot, 1999). Further, some studies

have confirmed that both physical and psychosocial hazards in the workplace can lead to

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increased stress levels and weight gain (Schulte et al., 2007). In line with previous

research, the first hypothesis tested in this study was that physical hazards in the

workplace would be positively related to BMI, meaning that increased exposure to

physical hazards would lead to an increase in BMI. In support of hypothesis 1a, physical

hazards were positively related to BMI among the lower level pay grade employees;

however, for the senior level pay grade, physical hazards were negatively related to BMI,

contradicting previous research. At first, the findings may seem strange that physical

hazards are related to a lower BMI for senior level employees. One possible explanation

is that employees in the lower pay range may have more physically demanding jobs,

whereas senior level employees are more likely to be managing people, and not actually

completing physically demanding tasks. Senior level employees who face more physical

hazards may be getting more physical activity compared to other senior level employees.

In contrast, senior level employees who are being exposed to less physical hazards may

be completing less physically demanding tasks which involve some level of physical

activity. Consequently, senior level employees who are more physically active

throughout the workday may in turn have a lower BMI due to increased energy

expenditure. This relationship may not hold true for lower level employees because these

employees may all be completing equally physical tasks. Therefore, higher exposure to

physical hazards may indicate a more physically active job for senior level employees,

but not for lower level employees.

Hypothesis 1b predicted that psychosocial workplace hazards would be positively

related to BMI, meaning that higher exposure to psychosocial hazards would lead to an

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increased BMI. Much research has confirmed that occupational stress can lead to weight

gain (Stansfeld & Candy, 2006) and that psychosocial hazards in the workplace are one

of the leading causes of stress in the workplace (Eurofound, 2011). The results of this

study confirmed hypothesis 1b, where greater exposure to psychosocial hazards were

related to an increased BMI for both senior level and lower level pay employees. The

results of this dissertation highlight the fact that not only are psychosocial hazards a

common stressor for employees, but psychosocial hazards also have a significant impact

on the physical health of employees. Additionally, this study demonstrates that

psychosocial hazards impact employee health across varying pay grades, underlining the

strength of the relationship between psychosocial hazards and body composition.

Workplace Hazards and Mental Health. A plethora of research has

demonstrated that psychosocial hazards in the workplace can negatively impact mental

health (Rugulie et al., 2006). The negative impact that psychosocial hazards can have on

mental health range from psychiatric disorders (Stansfeld et al., 1999) to poor general

mental health (Niedhammer et al., 2006). Given the abundant literature linking

psychosocial hazards to mental health, future examination of this relationship may not

add much new information to the scientific literature. Therefore, the current study did not

examine the association between psychosocial hazards and mental health. Although it is

important to review which kinds of workplace hazards impact mental health, it is also

important to focus on relationships that have been investigated less often and can make

the greatest contribution to the literature; the link between physical hazards and mental

health.

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Studies of the link between physical hazards and health have tended to focus on

how physical hazards can result in bodily injury, e.g., musculoskeletal injuries (WHO,

2001) or physiological damage to the brain, e.g., brain exposure to harmful substances

(Canadian Environmental Law Association, 2011). I chose to take a unique perspective

and examine how exposure to physical hazards can negatively impact general mental

health. Research has revealed that occupational stress can have a negative impact on

mental health (Cox et al., 2000) and that physical hazards are a common stressor in the

workplace for many employees (Eurofound, 2011). This suggests that occupational stress

resulting from physical hazards can contribute to the worsening of mental health. I

predicted that physical hazards would be related to worse mental health, meaning higher

exposure to physical hazards would be related to a higher score on the GHQ-12 scale.

The results of this study failed to support hypothesis 2, in that exposure to physical

hazards was not related to mental health among either the senior level or lower level pay

grade employees. The failure to find a significant relationship between physical hazards

and mental health may be due to the fact that certain types/severities of physical hazards

may cause more psychological stress than others, e.g., toxic fumes vs. a hot working

environment. The complex relationship between physical hazards and mental health may

make it difficult to find an effect without an improved measurement of workplace

hazards.

Workplace Hazards and Sickness Absence. Physical working conditions may

be attributable to upwards of 42% of sickness absences annually (Melchoir et al., 2005).

Physical workplace hazards have also been found to be strongly associated with absences

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due to physical injury (Allebeck & Mastekaasa, 2004) and illness (Haukenes et al., 2011).

In line with previous research findings, hypothesis 3a of the present study predicted that

physical hazards in the workplace would be positively related to sickness absence. In

support of hypothesis 3a, increased exposure to physical hazards were related to

increased sickness absence for the lower level pay grade; however, for the senior level

pay grade, physical hazards were not related to sickness absence, failing to support

hypothesis 3a. One explanation for why physical hazards impact sickness absences for

lower level pay grade employees and not senior level pay employees could be differences

in exposure to physical hazards. As previously mentioned, employees in the lower pay

range may have more physically demanding jobs and have a greater exposure to physical

hazards. In contrast, senior level employees are more likely to be managing people, and

not actually completing physically demanding tasks, lowering their exposure to physical

hazards. Studies have also reported that employees in lower pay grades work in less safe

environments, with more workplace hazards compared to higher paid employees

(Melchoir et al., 2005), possibly accounting for increased sickness absences. Finally,

socioeconomic differences in morbidity and mortality have been found between pay

grades (North et al., 1993). For example, lower paid employees might have less

healthcare benefits to prevent or treat illnesses compared to higher paid employees,

leading to more days away from work due to sickness.

In addition to physical hazards, psychosocial workplace hazards are one of the

leading causes of stress in the workplace (Eurofound, 2011). Research has established

that psychosocial hazards in the workplace are related to increased sickness absences

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(Lund et al., 2005). Many studies have been limited to linking components of the job-

demand-control model to absences (Berkamn & Goldberg, 2003); however, the current

study has expanded on previous research by examining how several different kinds of

psychosocial hazards can impact sickness absence. I hypothesized that psychosocial

hazards in the workplace would be positively related to sickness absence. In support of

this hypothesis, exposure to psychosocial hazards predicted sickness absences for the

lower pay grade employees; however, psychosocial hazards were not significantly related

to sickness absence among the senior pay grade employees. One explanation for the

findings of this study could be that lower pay grade employees are exposed more to

certain psychosocial hazards compared to senior pay grade employees. For example,

lower pay grade employees have lower job control and higher job demands (North et al.,

2006), which likely contributes to stress levels and sickness absences. Also, as stated

earlier in the discussion section, socioeconomic differences in morbidity and mortality

have been found between pay grades (North et al., 1993). Specifically, employees in

lower pay grades tend to have lower perceived health (Marmot et al., 1995), and may

have less resources to cope with injury and illness compared to employees in higher pay

grades.

Physical Activity. Given the negative impact that workplace hazards can have on

employee health, it is important to investigate ways to help employees cope with these

hazards. Physical activity has been shown to have a wide array of health benefits,

including reducing the risk of chronic diseases, improving quality of life, and stress

reduction (Harvard School of Public Health, 2013). People who are more physically

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active also tend to maintain a healthy body weight (Harvard School of Public Health,

2013), have better mental health and functioning (CDC, 2011), and tend to miss fewer

days of work due to sicknesses and injury (Rogen et al., 2013). Physical activity can also

help buffer stress people experience on a daily basis (Mayo Clinic, 2013). Despite these

health benefits, few studies have investigated whether physical activity can buffer

occupational stress experienced from exposure to physical and psychosocial hazards in

the workplace. Further, to the best of this authors’ knowledge, few studies have examined

how physical activity can buffer the negative effects of physical and psychosocial hazards

upon BMI, mental health, and sickness absence. Given the fact that physical activity can

improve mental and physical health (Mayo Clinic, 2013), while also acting as a stress

buffer (Harvard School of Public Health, 2013), it is probable that physical activity would

buffer the stress associated with exposure to physical and psychosocial hazards upon

physical and mental health.

Based on the scientific literature, and the reasoning explained above, I

hypothesized that physical activity would moderate the effects of physical and

psychosocial hazards upon BMI, general mental health, and sickness absence.

Specifically, hypotheses 4a to 6b predicted that employees who are more physically

active would be better able to cope with stress from exposure to physical and

psychosocial hazards. The increased ability to cope with stress would therefore result in a

lower BMI, better mental health, and less sickness absences compared to employees who

were not very physical active. The results of this study failed to support hypotheses 4a to

6b; physical activity did not act as a moderator between workplace hazards and BMI,

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mental health, or sickness absence. An explanation for the inability of the current study to

find an effect for physical activity may be explained by the differing intensity levels of

physical activity and exercise. First, physical activity and exercise have different

physiological effects on the human body because the intensity of physical exertion is

dissimilar. Exercise results in a higher, sustained increase in heart rate, as well as a

greater amount of calories consumed by the human body compared to general physical

activity (Caspersen, Powell, & Christenson, 1985). General physical activity may not be

at a high enough intensity to elicit a physiological response sufficient to have an impact

on bodyweight, mental health, or sickness absence. This differentiation between physical

activity and exercise intensity can aid in the explanation of why exercise shows more

consistent effects on improving health compared to general physical activity (Driver &

Taylor, 2000).

In addition, health status and health behaviors beyond physical activity likely play

an important role in the health of employees (CDC, 2011). For example, an employee

may be physically active, may smoke or consume large amounts of alcohol, which could

negate any positive effects of physical activity on health. An employee may also suffer

from chronic health conditions, such as diabetes, heart problems, or high blood pressure,

which have all been linked to obesity, poor mental health and sickness absences (CDC,

2011; Rogen et al., 2013). Physically activity alone may not be enough for an individual

who suffers from a chronic health condition to reduce sickness absence. Further, some

employees may already suffer from psychiatric or mental health disorders, where

physical activity alone may not be effective for treating a mental disorder. It is likely that

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the effects of overall health status have a stronger impact on BMI, mental health, and

sickness absence than physical activity alone would have.

Another possible reason why the current study did not find support that physical

activity acts as a stress buffer between workplace hazards and health outcomes may be

that employees choose alternative methods to cope with stress. Physical activity can help

individuals cope with stress (Mayo Clinic, 2013); however, some employees may choose

to cope with stress using alternative methods, such as seeking support from co-workers

and friends, while other employees may practice relaxation techniques, such as

meditation. Physical activity should not be viewed as the only viable method for stress

reduction, rather physical activity should be viewed as one of many possible alternatives

for dealing with stress.

Diet. In addition to exercise, eating a healthy diet can play a significant role in

employee health. A healthy, well-balanced diet can help people maintain a healthy body

weight, reduce obesity (Harvard School of Public Health, 2013) and help prevent or treat

the onset of mental health problems and chronic diseases (Mental Health Foundation,

2013). Further, a healthy diet can help people cope with stress by means of lowering

blood pressure and increasing immune system functioning (Roberts et al., 2002). Despite

the clear health benefits that consuming a healthy diet boast, few studies have specifically

investigated whether diet can buffer occupational stress experienced from exposure to

physical and psychosocial hazards in the workplace. Further, to the best of this authors’

knowledge, few studies have examined how a healthy diet can buffer the negative effects

of physical and psychosocial hazards upon BMI, mental health, and sickness absence.

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Given the fact that consuming a healthy diet can improve mental and physical health

(Mayo Clinic, 2013), while also acting as a stress buffer (Harvard School of Public

Health, 2013), it is probable that eating a healthy diet would buffer the stress associated

with exposure to physical and psychosocial hazards upon physical and mental health.

I hypothesized that diet would moderate the effects of physical and psychosocial

hazards upon BMI, general mental health, and sickness absence. Specifically, hypotheses

7a to 9b predicted that employees who eat a healthier diet would be better able to cope

with stress from exposure to physical and psychosocial hazards. The increased ability to

cope with stress would therefore result in a lower BMI, better mental health, and less

sickness absences compared to employees who do not have a healthy diet. The results of

this study failed to support hypotheses 7a to 9b; diet did not act as a moderator between

workplace hazards and BMI, mental health, or sickness absence. An explanation for the

inability of the current study to find an effect for diet may be explained by the

differentiation between chronic versus short term health benefits of a healthy diet. The

health benefits of a healthy diet are largely based on how long a person has been eating

healthy (Robert et al., 2000). For example, someone who has been eating healthy for

several years will likely be in better health compared to someone who has just recently

made healthy changes to their diet. In addition, an employee who has been eating healthy

for several years will likely be in better overall health, and show healthier responses to

stress via blood pressure compared to an employee who has been eating healthy for a

couple of months.

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Another possible explanation for why diet did not act as a moderator in the

current study is that eating healthy foods are only one aspect of a healthy, well-balanced

diet. For example, the quantity of food a person consumes and the timing of meals can

affect how a diet will impact health (Harvard School of Public Health, 2013). For

instance, an individual may eat healthy foods, but skip breakfast or not eat for hours,

which cause erratic spikes in blood glucose levels (Boyne et al., 2003). In addition,

someone could be eating nutritious foods, but be consuming too many calories per day,

leading to weight gain and health problems (Harvard School of Public Health, 2013).

Finally, a person could be eating healthy foods, but not consuming enough calories per

day, leading to nutritional deficiencies (Holloszy & Fontana, 2007). Nutritional

deficiencies are a major health risk because they can lead to mental and physical health

problems (Wachs, 2009).

Finally, the overall health status of an individual is one of the strongest predictors

of health outcomes (Mayo Clinic, 2013), including BMI, mental health, and sickness

absences. In addition, health behaviors beyond eating a healthy diet can play an important

role on employee health (CDC, 2011). For example, an employee may have a healthy diet

but does not exercise and smokes often, both of which could negate the benefits of a

healthy diet. An employee may also suffer from chronic health conditions, such as

diabetes, heart problems, or high blood pressure, which have all been linked to obesity,

poor mental health and sickness absences (CDC, 2011; Rogen et al., 2013). Additionally,

some employees may already suffer from psychiatric or mental health disorders, where

diet alone may not be effective for treating a mental disorder. It is likely that the effects

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of overall health status have a stronger impact on BMI, mental health, and sickness

absence than diet alone would have.

Age. In addition to modifiable health behaviors such as physical activity and

consuming a healthy diet, factors that an individual has no control over, such as age, can

impact health. For instance, it has been shown that BMI increases with age (Welon et al.,

2002) and older employees also miss more days of work due to sickness and injury (Voss

et al., 2008). To date, few studies have tested whether older employees have a more

difficult time coping with occupational stress. Given that older employees tend to have

more health problems such as chronic disease (CDC, 2011), it is probable that the lower

health status of older employees would make them more susceptible to the negative

effects of occupational stress on health.

I hypothesized that age would moderate the effects of physical and psychosocial

hazards upon BMI and sickness absence. Specifically, hypotheses 10a to 11b predicted

that older employees would be less capable of coping with stress resulting from physical

and psychosocial hazards in the workplace, resulting in a higher BMI and more sickness

absences compared to younger employees. The results of the present study failed to

support hypotheses 10a and 10b, meaning age did not have an impact on whether

physical or psychosocial workplace hazards affected BMI. The present study’s inability

to find any significant effects for age on BMI could be related to the different health

consequences of BMI for older versus younger individuals. For example, a higher BMI

has been found to be inversely related to mortality among older people, after adjusting for

waist circumference (Janssen & Katzmarzyk, 2005). Thin older people are also at a

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higher risk for death (Grabowski & Ellis, 2001), indicating that there are some health

benefits for older people to maintain some body fat (Chapman, 2008). Further, BMI does

not measure fat distribution. For example, older people tend to accumulate more visceral

fat (abdominal fat) compared to younger people (Zamboni et al., 1997), and visceral fat

poses the most health threats to people (Harvard School of Public Health, 2013).

Subsequently, age can impact fat distribution; however, age may not dramatically impact

measures of BMI, which do not measure fat distribution.

In addition, the results of the current study supported hypothesis 11a, where age

moderated the relationship between physical hazards and sickness absence for the lower

level pay grade employees; however age did not moderate the relationship between

physical hazards and sickness absence for the senior level pay grade. Further, the results

supported hypothesis 11b, where age significantly moderated the relationship between

psychosocial hazards and sickness absence for the lower level pay grade employees;

however age did not act as a moderator for the senior level pay grade employees. Age

likely did not play a factor among the senior level pay grade employees because earlier

findings from the current study found that workplace hazards did not impact sickness

absences among this pay grade; therefore if no initial relationship exists between hazards

and sickness absence, it is likely that the age of the employee would not alter this

relationship. In contrast, workplace hazards did have an impact on sickness absence for

lower level pay grade employees and age played a role in how often employees exposed

to workplace hazards were absent from work due to sickness or injury.

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Healthcare Differences between Europe and the United States. It is also

important to note the differences in healthcare systems between Europe, specifically

Northern Ireland and the United States. The sample used for this study consisted of

Northern Ireland civil servant workers, who have access to universal healthcare, provided

by National Health Service (NHS). In the U.K., universal healthcare is governed by the

government and funded from taxes (Grosios, Gahan, & Burbidge, 2010). In contrast, the

U.S. healthcare system is funded by public and private insurance, with large fees for

many patients. In the U.S., many individuals who are below the poverty line can’t afford

expensive insurance premiums and medical expenses that are not covered by insurance

(Schoen, Osborn, Squires, & Doty, 2013). The differences in access to healthcare may

explain why this study did not find that physical activity or diet buffered stress.

Employees in the U.K. may have higher baseline line health status compared to American

employees, who don’t have equal access to healthcare. A study examining the effects of

workplace hazards on health in the U.S. may find that physical activity and diet buffer

stress among a less healthy U.S. sample.

Organizational Implications

The current study has several important implications for organizations. First, this

study used the Total Worker Health framework set forth by NIOSH (2013) to highlight

the importance of recognizing that both work-related factors and health factors beyond

the workplace jointly contribute to the health and safety of employees. NIOSH (2013)

describes total worker health as a strategy integrating occupational safety and health

protection with health promotion to prevent worker injury and illness and to advance

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health and well-being. The present study has stressed the importance of promoting

worker health and well-being while also protecting worker safety and health. This study

found that exposure to workplace hazards negatively impacts employee health and that

the promotion of health behaviors can improve employee health.

An important factor for organizations to consider when they are deciding how to

promote worker health is which intervention level they want to focus on (primary,

secondary, or tertiary). According to LaMontagne et al. (2007), the goal of primary

prevention is to reduce the potential risk factors or alter the nature of the stressor before

workers experience stress-related symptoms or disease. Examples of primary prevention

include job redesign or workload reduction. Secondary prevention aims to help equip

workers with knowledge, skills, and resources to cope with stressful conditions.

Secondary prevention targets the employees’ response to stressors, and can include

cognitive behavioral therapy or stress coping classes. Finally, according to LaMontagne

et al. (2007), the goal of tertiary prevention is to treat, compensate, and rehabilitate

workers with enduring stress-related symptoms or disease. Examples of tertiary

prevention include occupational therapy or return-to-work programs.

Another important factor for organizations to consider when deciding how to go

about promoting employee health is whether to promote health behaviors within the

workplace, outside of the workplace, or both. Many researchers and practitioners now

acknowledge that it is necessary to promote employee health both within and outside the

organization, while at the same time protecting the privacy and rights of employees

(NIOSH, 2013). For example, employers could promote healthy eating in the workplace

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by offering healthy food choices in the cafeteria. Organizations could also go a step

further, and promote physical activity outside of the workplace by offering subsidies for

employee gym memberships. In order to promote total worker health employers should

aim to promote worker health both inside and outside of the workplace, because both

factors impact employee health. Further, in order to promote total worker health,

organizations should aim for implementing preventative measures of stress and ill health,

before negative health issues arise.

The current study found that both physical hazards and psychosocial hazards in

the workplace can negatively impact health, as demonstrated by reliable indicators of

health; specifically BMI, general mental health, and sickness absences. One of the most

important ways organizations can improve the health and well-being of their employees

is by reducing/removing exposure to physical and psychosocial hazards. By focusing on

primary prevention, organizations can remove a stressor/hazard from the workplace, and

help prevent the onset of stress and illness among employees. For example, organizations

could reduce employee exposure to physical hazards by developing strict safety

procedures for employees to follow when working in hazardous conditions. Further,

employers could investigate whether there are safer ways for employees to complete job

tasks, while minimizing exposure to potential physical hazards. With regards to

psychosocial hazards, organizations can aim to improve communication among

employees with conflict management skills development (LaMontagne et al., 2007).

Teaching employees how to effectively communicate with each other can help reduce

potential conflict in the workplace and minimize employee exposure to psychosocial

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hazards (Brew & David, 2004). Although ideal, it is often difficult and time-consuming

for organizations to implement primary level interventions for health promotion. For

example, the nature of some jobs may make it difficult or impossible for job redesign,

where exposure to certain physical or psychosocial hazards are inherent for a job.

Therefore, some organizations may find it more helpful to target secondary level

interventions, to help employees deal with hazards that can’t be removed from the

workplace.

Organizational Implications of Physical Activity. When primary prevention is

not possible, organizations can consider using secondary level interventions to improve

employee health, such as promoting physical activity. Although the current study did not

find that physical activity affected the relationship between workplace hazards and health

outcomes, physical activity did have a direct effect on BMI for both pay grades. The

results of this study confirm that physical activity is a viable method for reducing BMI,

confirming previous research (Sallis et al., 2003). Further, physical activity had a direct

effect on mental health and sickness absence for the lower pay grades, suggesting that

physical activity is a viable method for improving the mental health of employees while

also reducing sickness absences. Physical activity promotion can be considered a

secondary level intervention in most cases, unless physical activity is used as a

rehabilitation method for treating employees who are already sick or ill. Physical activity

promotion can be used as a secondary level intervention because physical activity can be

used to help employees’ cope with stress and alter their responses to stressors.

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Importantly, health interventions in the workplace that target physical activity have been

proven to be effective in improving employee health (Groeneveld et al., 2010).

Organizations can promote physical activity in several ways. First, employers can

promote physical activity within the workplace by offering opportunities to be more

physically active at work. For example, Marshal (2004) found that less organized

approaches, which promote incidental physical activity with and around the workplace

were most effective. For instance, initiatives set in place to promote regular walk/stress

breaks at work or promoting the use of stairs versus taking the elevator have been shown

to be effective for weight reduction (Yancey et al., 2008). Further, Marsh (2004)

established that individually based programs, where materials were tailored to individual

needs were most successful. For instance, programs should be tailored based on an

individuals’ readiness for change; ranging from whether a person has no desire to practice

physical activity to an individual who wants to start being physically active, but does not

know how to begin (Daley & Duda, 2006). It may be optimal to give information about

the health risks of being physically inactive to people who do not want to change their

activity levels; however, information about what kinds of exercises to practice may be

best for someone who wants to become more active, but lacks sufficient knowledge about

how to incorporate physical activity into their daily routine.

Importantly, social support and workplace initiatives to increase social support for

physical activity can aid in adopting a healthier lifestyle (Stokes, Henley, & Herget,

2006). For example, unstructured support from co-workers can help motivate people to

get more physical activity; however, to increase the effectiveness of social support,

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organizations can create a healthy culture in the workplace. For example, employers can

organize friendly competitions between co-workers, where employees can track how

many steps they have taken in a day using a pedometer. Notably, organizations that create

a culture where physical activity is the norm may have the best chance for sustaining

positive health behavior changes (Marshal, 2004).

Although this study did not find effects for physical activity on mental health and

sickness absence for the senior level pay grade employees, physical activity promotion is

still important. For example, it is not wise for an individual who is inactive to jump right

into a demanding exercise program because this could lead to injury. Physical activity

promotion can be used as an intermediate step between being inactive and participating in

an exercise program, in order to prepare a person’s body for more demanding exercise.

Promoting physical activity as an intermediate step to exercise for workplace initiatives is

important because an employer does not want to deal with lawsuits from injured

employees. For example, a corporate exercise promotion program could lead to injury

among unhealthy employees who are not ready to perform vigorous exercise. Physical

activity is a solution to slowly introducing employees to an exercise program, reducing

the likelihood of an injury.

Organizational Implications of Diet. In addition to promoting physical activity

in the workplace, complete health promotion programs should aim to target multiple

health behaviors in order to bring about the greatest possible change in employee health

(Groveneveld et al., 2010). Although the current study did not find that diet affected the

relationship between workplace hazards and health, the results indicated that diet had a

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direct effect on BMI for the senior pay grade and mental health for the lower pay grade.

Diet can be considered a secondary level intervention because diet would be used to help

employees’ cope with stress and alter their responses to stressors. Importantly, health

interventions in the workplace that target eating habits have been proven to be effective

in improving employee health (Groeneveld et al., 2010).

Organizations can promote a healthy diet for their employees in several ways.

Research suggests that educational training in the workplace can improve an employees’

diet and health (Maes et al., 2012). For example, providing employees with information

about healthy eating, especially by means of multimedia methods, have been shown to

increase positive attitudes towards healthy foods and the adoption of healthier eating

behaviors (Beaudoin et al., 2007). For instance, providing employees with information

about healthy eating through a website, and personalizing information so employees can

keep track of their diet may aid in promoting effective behavior change. Similar to

physical activity interventions, promoting healthy eating by personalizing a program to

an individuals’ readiness for change (Daley & Duda, 2006) may be the best option for

creating lasting changes in an employees’ diet. An employees’ readiness to adopt healthy

eating habits can range from whether a person has no desire to eat healthy to an

individual who wants to start eating healthy, but does not know how to begin. It may be

optimal to give information about health risks about an unhealthy diet to people who do

not want to change their eating behaviors; however, material about how to shop for

healthy foods may be best for someone who wants to eat healthy, but doesn’t possess

enough knowledge about what foods to eat.

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The work environment can also affect an employees’ eating habits and diet.

Engbers et al. (2005) argue that an important addition to a health promotion program are

environmental modifications, which can influence dietary intake. For example,

environmental changes in the workplace that have been shown to increase healthy eating

include the following: increasing the availability and variety of healthful food options,

reducing the price of healthy foods in worksite cafeterias and vending machines, and

making healthy food options more visible to employees (Story et al., 2008). In addition to

the physical aspect of the work environment, social support and workplace initiatives to

increase social support for healthy eating can aid in dietary changes (Kelsey, Earp, &

Kirkley, 1997). For instance, unstructured support from co-workers can help motivate

people to maintain a healthy diet; however, to increase the effectiveness of social support,

organizations can create a healthy culture in the workplace. Further, employers can

organize friendly competitions between co-workers, where employees can track the

healthy foods they eat. Importantly, organizations that create a culture where eating

healthy is the norm may have the best chance for sustaining positive health behavior

changes (Kristal, Glanz, Tilley, & Li, 2000).

Although the current study did not find effects of diet on BMI for lower level

employees, mental health for the senior level employees, or sickness absence for both pay

grades, promoting a healthy diet is crucial for optimal health (Harvard School of Public

Health, 2013). It is important to note that the benefits of a healthy diet are largely based

on how long a person has been eating healthy (Robert et al., 2000). For example,

someone who has been eating healthy for several years will likely be in better health

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compared to someone who has just recently made healthy changes to their diet.

Therefore, thinking of health promotion in terms of long-term health benefits may help

organizations and individuals realize that just because there may not be any immediate

tangible rewards for a healthy diet, the long-term rewards may still be as valuable. For

instance, healthier employees in the future can save organizations lost revenues by means

of less sickness absences and lower insurance premiums (Danna & Griffin, 1999).

Further, the long-term health benefits of eating a healthy diet can equate to increased

quality of life and optimal health for the individual (Mayo Clinic, 2013).

Organizational Implications of the Aging Workforce. Promoting total worker

health involves more than promoting modifiable health behaviors among the working

population. In some instances, factors beyond an employees’ control can affect health and

safety both within and outside the workplace. The current study found that age moderated

the relationship between physical hazards and sickness absence and psychosocial hazards

and sickness absence among the lower level pay grade employees. Additionally, although

age did not moderate the relationship between workplace hazards and BMI, age did show

a direct effect on BMI for both pay grades. When organizations develop health

interventions for their workers, the age of their employees is an often overlooked aspect

of a health intervention. Age does not fit the normal framework for health interventions

because age is not a modifiable behavior. Despite age being an often overlooked variable

in health intervention research, the rapidly aging workforce is forcing researcher and

organizations to pay attention to the unique health needs of these individuals.

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Importantly, although age is a factor that an individual has no control over, older

employees can practice positive health behaviors, to slow the decline in age-related

functional fitness and health. According to Kenny, Yardley, Martineau, and Jay (2008),

an average decline of 20% in physical work capacity has been reported between the ages

of 40 and 60, due to decreases in aerobic and musculoskeletal capacity. These declines

can contribute to decreased work capacity and increase the likelihood of work-related

injuries and illness. Kenny et al. (2008) argue that well-organized, management

supported worksite health interventions encouraging physical activity during work hours

could potentially decrease the incidence of age-related injury and illness.

Promoting both a healthy diet and physical activity among older workers may

have a greater impact on health compared to younger employees (University of Iowa,

2009). In the context of older workers, the promotion of physical activity and diet may be

used as a secondary or tertiary level intervention. From a secondary intervention level,

increased physical activity and a healthier diet may help older employees cope with

stressors in the workplace. From a tertiary intervention level, physical activity and dietary

changes may be used to help rehabilitate older employees suffering from health problems.

Older employees are at the greatest risk of sustaining sever injuries in the workplace and

are more likely to miss more days of work per year due to sickness and injury (Wegman

& McGee, 2004). Therefore, health interventions targeted towards improving the health

of the highest risk group for occupational injury and sickness, e.g., older workers, should

be viewed as a promising area for improving employee health and organizational

productivity.

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Due to a lack of attention paid to the health needs of the aging workforce, there

are currently few guidelines or standard practices that organizations can use as a

framework for tailoring health interventions to their older employees. Luckily, many of

the same principles that are used for health interventions of younger employees can be

used for older employees. For instance, Marsh (2004) established that individually based

programs, where materials were tailored to individual needs were most successful.

Programs should be tailored based on an individuals’ readiness for changing a health

behavior. Further, less organized approaches which promote incidental physical activity

or healthy eating during work hours may be most effective. For instance, initiatives set in

place to promote regular walk/stress breaks at work or creating “healthy snack days” for

employees may bring about behavior change. However, some aspects of health promotion

should be tailored to an employee’s age. For example, it is essential that lower impact

forms of physical activity are promoted for older employees. Strenuous physical activity

can lead to injury among older employees, who are at an increased risk of joint and bone

injuries (Cummings et al., 1993).

Strengths of Current Study

The current study has made several important contributions to Occupational

Health Psychology. First, the present study has used total worker health as a framework

for investigating occupational stress and health behaviors. Total Worker Health is a

concept based on taking all aspects of a person’s health into account when promoting

health (NIOSH, 2013). One strength of the current study is that the health of an employee

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is examined from both within and outside the workplace, allowing for a comprehensive

evaluation about what factors best predict a person’s health.

Researchers have differed in their definition of what physical and psychosocial

hazards in the workplace encompass. The current study has attempted to organize a

framework for what constitutes workplace hazards, and attempted to create an all-

inclusive definition of physical and psychosocial hazards, which can be used as a

framework for future researchers.

Thirdly, the present study is one of few to examine the effects of both physical

and psychosocial workplace hazards upon employee health. For example, studies have

examined how psychosocial hazards impact mental health or how physical hazards affect

musculoskeletal disorders; however few studies have examined the effects of both

physical and psychosocial hazards on health outcomes. Further, the current study

investigated the effects of physical hazards on health outcomes, while controlling for

psychosocial hazards. Additionally, the effects of psychosocial hazards on health

outcomes were examined, while controlling for physical hazards. Controlling for one type

of hazard allows for an examination about whether one hazard (e.g. physical) predicts a

health outcome beyond and above the effects of another hazard (e.g. psychosocial).

Specifically, the current study found that physical hazards can negatively impact BMI

and sickness absences, above and beyond psychosocial hazards. Additionally, it was

found that psychosocial hazards can negatively impact BMI, mental health, and sickness

absence, above and beyond the effects of physical hazards.

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Additionally, the current study noted that there are potential differences in both

health outcomes and health behaviors based on an employees’ pay grade level. A strength

of the present study is that it recognizes that lower paid employees have differing health

needs compared to employees in the highest salary bracket. Lower paid employees may

lack financial and social resources to enable them to cope with exposure to workplace

hazards. The current study highlights the fact that organisations should take the divergent

health needs of employees in different pay grades when designing and implementing

health interventions in the workplace.

A final strength of the current study is that it recognizes new challenges that the

aging workforce may face in the future. For example, aging employees are at an

increased risk for chronic diseases (NIOSH, 2013) and workplace accidents compared to

younger employees (Voss et al., 2008). The demographics of the workforce is changing,

and aging workers are at an increased risk for health problems such as obesity and

chronic diseases. The present study highlights the fact that researchers and organizations

may need to tailor health promotion programs to this section of the workforce.

Specifically, older employees are at an increased risk for obesity compared to younger

employees. Increased obesity rates can lead to health problems for individual employees

and can be a financial burden for employers, e.g., increased sickness absences and

healthcare costs. The present study highlights the point that different approaches may

need to be taken when promoting the health of older employees.

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Weaknesses of Current Study

There are some potential problems with the current study. First, the present study

does not have direct measures of the mechanisms of occupational stress, rather measures

of physical and psychosocial hazards are used as proxies for occupational stress. For

example, participants list what physical or psychosocial hazards are causing them

unwanted stress at work; however the responses are in a yes/no format. The yes/no format

may not capture whether certain workplace hazards are more severe, causing more stress

than other hazards. A direct measure of the severity and frequency of hazards or a direct

measure of perceived occupational stress may best capture how hazards in the workplace

are creating stress.

Another weakness of the current study is that a cross-sectional design was used,

where all data was collected at the same time-point. Therefore, it is difficult to determine

whether the outcome is affected by predictor or the predictor is affected by the outcome.

Cross-sectional studies make it difficult to interpret the sequence of events. For example,

since workplace hazards, health outcomes, and physical activity were measured at the

same time it is not possible to completely confirm whether physical activity can affect the

relationship between workplace hazards and health.

There are also limitations to using BMI as an indicator of health and obesity.

First, the current study used self-report data on height and weight to calculate

participants’ BMI. Self-report measures of weight may not be reliable measures because

individuals may underreport their weight, due to being self-conscious about their

bodyweight. In addition, BMI does not measure fat distribution across the body. Fat

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152

accumulation around the waist and abdomen can put people at a greater risk for chronic

disease and poor health compared to fat distributed in other parts of the body (Harvard

School of Public Health, 2013). Finally, BMI may not be an accurate predictor of health

for individuals that are muscular, e.g., athletes, because BMI takes weight and height into

account, without accounting for differences in bodyweight due to muscle. For example,

the BMI of a muscular athlete may list them as obese because they are heavy for their

height, not taking into account that muscle is contributing to a heavier weight (CDC,

2013). Nonetheless, BMI is a useful indicator of health, because it not only predicts

chronic disease, but it is also a cost effective and easy tool to use.

Another possible limitation for the present study is the confounding factor of

physically demanding work and physical activity levels. For example, employees who

have more physically demanding job tasks may be getting more physical activity than

employees who work primarily in an office setting. Further, employees who have

physically demanding jobs may also be exposed to more physical workplace hazards. It

may be the case that exposure to physical hazards is also related to greater levels of

physical activity. For instance, office employees who are exposed to less physical hazards

may also have less physically demanding jobs. Taking into account the nature of an

employees’ job, and how it can impact health and health behaviors is a potential factor to

consider for future research.

Future Research

There are many different avenues for future research on the topic of workplace

hazards and employee health outcomes. First, it is important to consider the role that

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resources have on occupational stress and employee health. Future research could

examine how workplace hazards impact employee health by framing a study using the

Job-Demand-Resource Model (JD-R) by Demerouti, Bakker, Nachreiner, and Schaufeli

(2001). The JD-R Model by Demerouti, et al. (2001) categorizes employee working

conditions into demands and resources. In the context of the current study, job demands

can be characterized by physical and psychosocial hazards in the workplace. Resources

can be described as factors that help employees cope with workplace stressors, and help

employees attain job goals and personal growth. Resources can include job conditions

which help employees deal with stress or personal characteristics e.g., coping skills

(Schaufeli & Bakker, 2004). Future research could examine how healthy lifestyle factors

act as resources to help buffer the negative effects that workplace hazards have on health.

The current study examined how exercise and diet can act as stress buffers; however

future researchers may want to examine how multiple healthy lifestyle factors grouped

together can buffer stress. Healthy lifestyle factors could include exercise, diet, drinking

and smoking behaviors. It is likely that a group of healthy lifestyle factors would be more

likely to buffer stress compared to examining individual health behaviors. It is probable

that employees who have more healthy lifestyle resources will be better able to cope with

occupational stress, and have more favorable health outcomes.

Additionally, future research could incorporate ideas from positive psychology to

help determine the best ways to promote total worker health. For example, positive

psychology emphasises prevention across all areas of psychology, focusing on primary

prevention of health problems rather than just treating a health problem (Seligman &

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154

Csikszentmihalyi, 2000). Organizations and researchers should aim to incorporate

positive psychology into worksite health interventions. For instance, future health

interventions in the workplace should place a greater emphasis on primary prevention,

which has been shown to be more cost-effective compared to secondary or tertiary

interventions (Cecchini et al., 2010). Instead of research focusing on how to treat

employees who are already suffering from health problems, researchers should

investigate how organizations can do a better job of removing hazards from the

workplace.

Importantly, positive psychology research indicates that positive states are more

than the absence of negative states (Peters & Czapinski, 1990). Future research should

examine the conceptual differences between positive and negative states, and incorporate

this differentiation into total worker health promotion. For instance, “happy” is not the

same as “not sad”, and each state has differing biologically based mechanisms (Russell,

2003). Applying positive psychology to the current study, the absence of physical and

psychosocial hazards in the workplace does not always equate to a happier and healthier

worker. Researchers should investigate the effectiveness of using both primary and

secondary interventions in the workplace. Primary interventions can be the first line of

defence against occupational stressors; however, using health promotion strategies as a

secondary intervention level may help improve employee health above and beyond the

initial removal of a stressor.

In addition, future research should incorporate direct measures of the mechanisms

of occupational stress. The current study used workplace hazards as indicators of

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155

occupational stress; however having direct measures of occupational stress may make the

link between workplace hazards, stress, and health outcomes more clear. For example, in

most cases, increased exposure to workplace hazards was related to more negative health

outcomes; however it is plausible that not all of the workplace hazards equally contribute

to occupational stress. In order to determine which kinds of workplace hazards are most

stressful for employees, future researchers could group hazards by severity. It is likely

that certain hazards, such as exposure to toxic chemicals will induce more stress

compared to working in a hot climate. Therefore, some sort of grouping or weighing

technique may be effective in making the link between workplace hazards, occupational

stress, and health more clear.

Future research could also improve upon the current study by implementing a

longitudinal study instead of using a cross-sectional design. A longitudinal study allows

for the collection of data at different time-points, making it easier to establish whether

one variable affects another variable over time. A longitudinal design can be used to

establish the sequence of events among predictor and outcome variables. For example,

future research could measure workplace hazards at one time-point, and measure health

outcomes six months later, in order to establish if workplace hazards can impact future

health.

Furthermore, other indicators of obesity and health could be used in addition to

using BMI. Although BMI is easy to measure and inexpensive, it is an indirect measure

of body fat, which can be prone to error. BMI is also a less accurate predictor of body fat

for the elderly (Harvard School of Public Health, 2014). Alternative methods for

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156

measuring body fat include measuring waist circumference, skinfold thickness,

bioelectrical impedance, and hydrostatic weighing (Harvard School of Public Health,

2014). The most accurate method for measuring body fat is hydrostatic weighing, where

individuals are weighed in air, while submerged in a tank of water. However, this method

is time consuming and expensive in some instances. Measuring skin thickness with

calipers or measuring waist circumference can be a more convenient and inexpensive

method for measuring body fat, although these methods can be prone to measurement

error. Finally, biometrical impedance can be a safe and relatively inexpensive method for

measuring body fat; however an individual’s’ hydration levels can affect the accuracy of

readings. Future researchers should weigh the pros and cons of each method for

measuring body fat, and determine if cost, accuracy, or convenience is a priority. Ideally,

research should incorporate multiple methods for measuring body fat, in order to get the

most accurate and reliable measures.

One interesting factor to consider for future research is physical activity related to

work tasks or pay grade. Lower pay grade employees tend to have more physically

demanding jobs (Hemingway et al., 1997). For example, lower pay grade jobs may

include more bending, stretching, standing, and lifting objects (Sekine et al., 2006). The

main issue is whether occupational physical activity should be considered when

examining the physical activity levels of employees. The distinction between

occupational physical activity and leisure physical activity could be made if the goal of a

future study is to examine both forms of physical activity separately. The other option

would be to include both occupational and leisure physical activity into one measure of

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157

general physical activity. Finally, if the goal was to examine the individual effects of

leisure physical activity on health, occupational physical activity could be controlled for

to account for varying levels of occupational physical activity among employees.

Additionally, future research could investigate how physical and psychosocial

hazards in the workplace affect sleep quality. Given that the current study found that

workplace hazards can negatively impact health outcomes such as BMI, mental health

and sickness absence, it is also likely that workplace hazards would impact sleep quality.

Previous research has linked work demands and occupational stress to reduced sleep

quality (Caruso, Hitchcock, Dick, Russo, & Schmit, 2004); however few studies have

directly linked exposure to both physical and psychosocial hazards to sleep quality. An

individual’s ability to attain a healthy and consistent sleep schedule is vital to overall

health and well-being (The National Sleep Foundation, 2009). Therefore, future research

should investigate whether workplace hazards impact sleep quality, and devise ways to

buffer the negative effects of workplace hazards on sleep quality.

Finally, it is often difficult to convince organizations to implement health

interventions in the workplace because organizations are not convinced that these

interventions will have any benefit. Studies linking or demonstrating the potential

benefits to the employees and organization in terms of financial gain and improved

organizational and individual health are critical for gaining organizational support for a

worksite health intervention.

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Conclusions

In conclusion, some novel relationships were established in this study. First,

psychosocial hazards were found to predict BMI among both pay grades. Further,

physical hazards predicted BMI for both pay grades; however, contrary to prediction,

physical hazards were inversely related to BMI among the senior pay grade employees.

Moreover, it was found that physical hazards were not related to mental health. In

addition, physical and psychosocial hazards only predicted sickness absence among lower

pay grade employees. The current study also found that physical activity and diet did not

buffer the negative effects of workplace hazards on BMI, mental health, and sickness

absence. Nevertheless, physical activity was significantly related to BMI for both pay

grades and mental health and sickness absence for the lower pay grades.

Additionally, diet was significantly related to BMI for the senior pay grade and mental

health for the lower pay grade. Finally, age moderated the relationship between physical

hazards and sickness absence and psychosocial hazards and sickness absence among the

lower level pay grade employees. The results of this study show that physical and

psychosocial hazards in the workplace can be detrimental to employee health.

Importantly, physical activity was related to a lower BMI, improved mental health, and

less sickness absences, while a healthy diet was related to a lower BMI for both pay

grades. Promoting physical activity and a healthy diet are viable methods for improving

employee health. Further, when organizations are designing worksite health

interventions, physical activity, diet, and employee age should be factored into a plan

designed to promote total worker health.

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APPENDICES

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APPENDIX A

Physical Workplace Hazards Which of the following, in your view are causing your workplace to be unsafe or unhealthy? (Tick all that apply)

1) The lighting 2) The heating 3) The air quality 4) Noise levels 5) Unsafe work area (e.g. cluttered or badly 6) Set up office area) 7) Hazardous chemicals/microbiological agents 8) Radiological hazards 9) Wiring/cabling 10) Unsafe floor surfaces 11) Water leaks 12) Inadequate seating or desk space 13) Inadequate access to staff facilities (tea/coffee, rest areas, toilets) 14) Other (please specify)___________________________________

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APPENDIX B

HSE MS Psychosocial Hazards Scale Which, if any, of the following are causing you unwanted stress in your job? (Tick all that apply)

1) Long working hours 2) New technology 3) Having too much work to do 4) Having too little work to do 5) Changes to your job 6) Boring or repetitive work 7) Shift work 8) People you manage 9) Too much responsibility 10) Dealing with the public 11) Excessive travel 12) Balancing family and work

commitments 13) Working beyond your level of

ability 14) Unrealistic targets 15) Unreasonable deadlines 16) Constant interruptions 17) Keeping up with

emails/voicemails 18) Performing tasks outside your job

specifications 19) Lack of consultation about

decisions that affect you 20) Lack of freedom to plan your

work 21) Disliking the work you do 22) The lack of flexibility in your

work pattern

23) How you are treated by your manager

24) Poor morale where you work 25) Poor relationships with colleagues 26) Feeling undervalued 27) Being bullied in the last year 28) Sexual harassment in the last year 29) Sectarian harassment in the last

year 30) Lack of support from

management/colleagues 31) Poor communication 32) Being ignored or excluded 33) Being criticized 34) Lack of recognition at work 35) Office politics 36) Lack of equal opportunities 37) Poor working conditions 38) Lack of career progress 39) Inadequate training 40) Lack of job security 41) Being understaffed 42) The way work is shared 43) Being unclear about what you are

supposed to do 44) None of these, I am not stressed at

work 45) Other (Please

specify)____________

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APPENDIX C

Exercise How many times a week, on average do you do the following kinds of exercise for more than 20 minutes? (Please write the appropriate number on each line. If NONE, please write ‘0’).

a) Strenuous Exercise (Heart beats rapidly) (e.g. Running, jogging, football, squash, basketball, vigorous swimming, vigorous long distance cycling, high impact aerobics and other exercises of a similar intensity)………………………………………...times a week

b) Moderate Exercise (Not exhausting) (e.g. Fast walking, badmington, easy

swimming, easy cycling, volleyball, popular dancing, heavy gardening, low impact aerobics and other exercises of a similar intensity)………………………………………...times a week

c) Mild Exercise (Minimal effort) (e.g. Yoga, golf, easy walking, bowls, light

gardening and other exercises of a similar intensity)………………………………………...times a week

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APPENDIX D

Diet Which of the following, if any, do you consciously try to limit or avoid? (Tick all that apply) Salt Fat/fatty foods Caffeine Sugar Red meat Fast foods Other (Please specify) _______________

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APPENDIX E

General Mental Health-12 Questionnaire (GHQ-12) Please read this before you start this section. We would like to know how your health has been in general, over the past few weeks. Please answer ALL the questions by putting a tick in the box under the answer which you think most applies to you. Have you recently…

a) Been able to concentrate on whatever you are doing?

Better than usual

Same as usual

Less than usual

Much less than usual

b) Lost much sleep over worry? Not at all

No more than usual

Rather more than usual

Much more than usual

c) Felt that you are playing a useful part in things?

More so than usual

Same as usual

Less useful than usual

Much more than usual

d) Felt capable of making decisions about things?

More so than usual

Same as usual

Less so than usual

Much less than usual

e) Felt constantly under strain? Not at all

No more than usual

Rather more than usual

Much more than usual

f) Felt you couldn’t overcome your difficulties?

Not at all

No more than usual

Rather more than usual

Much more than usual

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g) Been able to enjoy your normal day-to-day activities

More so than usual

Same as usual

Less so than usual

Much less than usual

h) Been able to face up to your problems?

More so than usual

Same as usual

Less so than usual

Much less able

i) Been feeling unhappy and depressed?

Not at all

No more than usual

Rather more than usual

Much more than usual

j) Been losing confidence in yourself?

Not at all

No more than usual

Rather more than usual

Much more than usual

k) Been thinking of yourself as a worthless person?

Not at all

No more than usual

Rather more than usual

Much more than usual

l) Been feeling reasonably happy, all things considered?

More so than usual

Same as usual

Less so than usual

Much less than usual

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FIGURE A1

NICS Research Overview

Securing Commitment

Needs Analysis

Action

Evaluation

NICS Workforce Health Survey

Reports and Recommendations

NICS-Wide Improvement Plans

drawn up by OHS/WHC

Department/Agency Improvement Plans drawn up by local

implementation teams

Delivery of service wide improvement

plan

Delivery of department/agency improvement plans

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FIGURE A2

Physical Activity as a Moderator between Physical Hazards & BMI

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FIGURE A3

Physical Activity as a Moderator between Psychosocial Hazards & BMI

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FIGURE A4

Physical Activity as a Moderator between Physical Hazards and GHQ-12

0

2

4

6

8

10

12

Low Physical Hazards High Physical Hazards

GH

Q-1

2

Lower Pay Grade

Low PhysicalActivity

High PhysicalActivity

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FIGURE A5

Physical Activity as a Moderator between Psychosocial Hazards & GHQ-12

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FIGURE A6

Physical Activity as a Moderator between Physical Hazards & Sickness Absence

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FIGURE A7

Physical Activity as a Moderator between Psychosocial Hazards & Sickness Absence

0

1

2

3

4

5

6

7

8

Low PsychosocialHazards

High PsychosocialHazards

Sic

kn

ess

Ab

sen

ce D

ays

Senior Pay Grade

Low PhysicalActivity

High PhysicalActivity

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221

FIGURE A8

Diet as a Moderator between Physical Hazards & BMI

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FIGURE A9

Diet as a Moderator between Psychosocial Hazards & BMI

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FIGURE A10

Diet as a moderator between Physical Hazards & GHQ-12

0

2

4

6

8

10

12

Low Physical Hazards High Physical Hazards

GH

Q-1

2

Senior Pay Grade

Low Health Diet

High HealthDiet

0

2

4

6

8

10

12

Low Physical Hazards High Physical Hazards

GH

Q-1

2

Lower Pay Grade

Low Health Diet

High HealthDiet

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FIGURE A11

Diet as a Moderator between Psychosocial Hazards & GHQ-12

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FIGURE A12

Diet as a Moderator between Physical Hazards & Sickness Absence

0

1

2

3

4

5

6

7

8

Low Physical Hazards High Physical Hazards

Sic

kn

ess

Ab

sen

ce D

ays

Senior Pay Grade

Low Health Diet

High HealthDiet

0

1

2

3

4

5

6

7

8

Low Physical Hazards High Physical Hazards

Sic

kn

ess

Ab

sen

ce D

ays

Lower Pay Grade

Low Health Diet

High HealthDiet

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FIGURE A13

Diet as a Moderator between Psychosocial Hazards & Sickness Absence

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FIGURE A14

Age as a Moderator between Physical Hazards & BMI

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FIGURE A15

Age as a Moderator between Psychosocial Hazards & BMI

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FIGURE A16

Age as a moderator between Physical Hazards & Sickness Absence

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FIGURE A17

Age as a Moderator between Psychosocial Hazards & Sickness Absence

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TABLE B1

Glossary Of Job Grades

Industrials Industrial Grade Employees AA Administrative Assistants (NICS), (Northern Ireland Office-NIO) AO Administrative Officer (NICS) EO11/EO1 Executive Officer Grades 2 & 1 (NICS), Grade C (NIO) SO Staff Officer (NICS), Grade B2 (NIO) DP Deputy Principal (NICS), Grade B1 (NIO) Grades 6&7 Senior Principal & Principal (NICS), Grade A (NIO) Grades 5+ Senior Civil Service (Assistant Secretary, Under Secretary & Permanent

Secretary)

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TABLE B2

Means, Standard Deviations, and Correlations of Measured Variables for Entire Sample

Variable Mean 1 2 3 4 5 6 7 8

1 Physical Hazards

1.33

(1.98)

1

2 Psychosocial Hazards

5.98

(5.43)

.04 1

3 BMI 25.53

(4.23)

.02* .07* 1

4 GHQ-12 2.44

(3.48)

.16** .46** .04** 1

5 Sickness Absence

11.44

(28.84)

.07** .10** .03* .17** 1

6 Exercise 7.69

(3.94)

.02* -.02* -.07** -.09** -.04** 1

7 Diet 2.61

(1.61)

.06* .05** -.03** -.002 .02 .11** 1

8 Age 38.87

(10.13)

.05 -.06* .17** -.02* .003 -.09** .18** 1

* = p < .05; ** = p < .01 (two-tailed) Standard deviations appear in parentheses below means.

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TABLE B3

Comparison of Means for Measured Variables between Senior Pay Grade and Lower Pay Grade

Variables Senior Pay Grade Sample Means

Lower Pay Grade Sample Means

df t value

Physical Hazards 0.74

(1.57)

1.38

(2.00)

15, 896 -10.05***

Psychosocial Hazards

5.83

(4.66)

6.05

(5.49)

15, 896 -1.27

BMI 25.31

(3.21)

25.52

(4.29)

14, 909 -1.49

GHQ-12 2.10

(3.06)

2.48

(3.47)

15, 896 -3.36*

Sickness Absence 5.57

(18.44)

11.81

(29.03)

14, 736 -6.53**

Exercise 7.67

(3.64)

7.71

(3.96)

12, 757 -0.24

Diet 3.05

(1.57)

2.59

(1.60)

15, 896 8.81***

Age 47.59

(7.85)

38.18

(9.95)

15, 735 29.40***

* = p < .05; ** = p < .01; *** = p < .001. Standard deviations appear in parentheses below means.

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TABLE B4

Correlations of Measured Variables for Senior Pay Grade

Variable 1 2 3 4 5 6 7 8 9

1 Physical Hazards

1

2 Psychosocial Hazards

.03* 1

3 BMI -.08* .08* 1

4 GHQ-12 .12** .48** -.05 1

5 Sickness Absence

.05 .05 -.02 .17** 1

6 Exercise .02 -.06 -.14** -.01 -.003 1

7 Diet .05 .08** -.12** .04 .02 .16** 1

8 Age -.16** -.10** .15** .01 -.01 -.04 .11** 1

9 Gender .12* .07* -.28* .02* .11** -.01 .14** __ 1

* = p < .05; ** = p < .01 (two-tailed); Gender: Males coded as 1, females coded as 2, in all Tables.

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TABLE B5

Correlations of Measured Variables for Lower Pay Grade

Variable 1 2 3 4 5 6 7 8 9

1 Physical Hazards

1

2 Psychosocial Hazards

.03* 1

3 BMI .03** .07** 1

4 GHQ-12 .16** .46** .05** 1

5 Sickness Absence

.07** .11** .03** .17** 1

6 Exercise -.02* -.02* -.06** -.10** -.04** 1

7 Diet .06** .05** -.02* -.004 .02* .10** 1

8 Age -.03** -.05** .18** -.01 .02* -.09** .17** 1

9 Gender .09** -.02* -.17** .04* .09** -.14** .13** __ 1

* = p < .05; ** = p < .01 (two-tailed)

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TABLE B6

Regression Analyses: Physical & Psychosocial Hazards Predicting BMI

Senior Pay Grade Lower Pay Grade

Variable B SE B β B SE B β

Gender -2.14 .25 -.28** -1.47 .07 -.17** Psychosocial Hazards .08 .02 .12** .05 .01 .06**

Physical Hazards -.17 .07 -.08* .04 .02 .02*

R2 .10

31.27**

.03

165.18** F

* = p < .05; ** = p < .01 (two-tailed)

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TABLE B7

Regression Analyses: Physical Hazards Predicting GHQ-12

Senior Pay Grade Lower Pay Grade

Variable B SE B β B SE B β

Gender -.07 .20 -.01 .36 .05 .05** Psychosocial Hazards .32 .02 .49** .30 .01 .46**

Physical Hazards -.04 .06 -.02 -.03 .01 -.02

R2 .23

31.27**

.21

1.33** F

* = p < .05; ** = p < .01 (two-tailed)

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TABLE B8

Regression Analyses: Physical & Psychosocial Hazards Predicting Sickness Absences

Senior Pay Grade Lower Pay Grade

Variable B SE B β B SE B β

Gender 4.58 1.44 .10** 5.11 .49 .09** Psychosocial Hazards .14 .13 .04 .53 .05 .10**

Physical Hazards .28 .40 .02 .43 .13

R2 .01

4.48**

.02

97.03** F

* = p < .05; ** = p < .01 (two-tailed)

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TABLE B9

Physical Activity as a Moderator between Physical Hazards & BMI

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender -2.20

(.27)

-.28** -2.15

(.27)

-.28 -1.57

(.08)

-.18** -1.57

(.08)

-.18**

Psych Hazards .08

(.03)

.11** .08

(.03)

.11** .05

(.01)

.06** .05

(.01)

06**

Physical Hazards -.16

(.07)

-.08* -.16

(.07)

-.08* .04

(.02)

.02 .04

(.02)

.02*

Physical Activity -.12

(.03)

-.14** -.12

(.03)

-.13* -.10

(.01)

-.09** -.10

(.01)

-.09**

Physical Hazards x Physical Activity

.01

(.02)

.02 -.00

(.01)

-.01

R2 .11**

23.97**

.11**

.37

.04**

122.56**

.04**

.47 F Change

Note: Physical Hazards and Physical Activity were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas Psych is abbreviation for Psychosocial Hazards in all Tables

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TABLE B10

Physical Activity as a Moderator between Psychosocial Hazards & BMI

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender -.22

(.27)

-.28** -2.20

(.27)

-.28** -1.57

(.08)

-.18** -1.57

(.08)

.04**

Physical Hazards -.16

(.07)

-.08* -.16

(.07)

-.08* .04

(.02)

.02* .04

(.02)

.46**

Psych Hazards .08

(.03)

.11** .08

(.03)

.11** .05

(.01)

.06** .05

(.01)

-.02

Physical Activity -.12

(.03)

-.14** -.12

(.03)

-.14** -.10

(.01)

-.09** -.10

(.01)

-.08**

Psych Hazards x Physical Activity

.00

(.01)

.002 .00

(.002)

-.28

R2 .11**

23.97**

.11**

.003

.04**

122.56**

.04**

.08 F Change Note: Psychosocial Hazards and Physical Activity were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B11

Physical Activity as a Moderator between Physical Hazards & GHQ-12

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender -.06

(.22)

-.01 -.06

(.22)

-.01 .29

(.06)

.04** .29

(.06)

.04**

Psych Hazards .32

(.02)

.49** .32

(.02)

.49** .29

(.01)

.46** .29

(.01)

.46**

Physical Hazards -.05

(.06)

-.02 -.05

(.06)

-.02 -.03

(.02)

-.02 -.03

(.02)

-.02

Physical Activity .02

(.03)

.02 .02

(.03)

.03 -.07

(.01)

-.08** -.07

(.01)

-.08**

Physical Hazards x Physical Activity

.01

(.02)

.02 -.002

(.00)

-.01

R2 .23**

64.63**

.23**

.19

.22**

826.15**

.22**

.48

F Change Note: Physical Hazards and Physical Activity were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B12

Physical Activity as a Moderator between Psychosocial Hazards & GHQ-12

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender -.06

(.22)

-.01 -.06

(.22)

-.01 .29

(.06)

.04** .29

(.06)

.04**

Physical Hazards -.05

(.06)

-.02 -.05

(.06)

-.02 -.03

(.02)

-.02 -.03

(.02)

-.02

Psych Hazards .32

(.02)

.49** .32

(.02)

.49** .29

(.01)

.46** .29

.01)

.46**

Physical Activity .02

(.03)

.02 .02

(.03)

.02 -.07

(.01)

-.08** -.07

(.01)

-.08**

Psych Hazards x Physical Activity

-.001

(.01)

-.01 -.002

(.001)

-.02

R2 .23**

64.63**

.23**

.04

.22*

826.15**

.22*

3.24 F Change Note: Psychosocial Hazards and Physical Activity were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B13

Physical Activity as a Moderator between Physical Hazards & Sickness Absence

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender 4.55

(1.53)

.10** 4.55

(1.53)

.10** 4.89

(.54)

.08** 4.90

(.54)

.08**

Psych Hazards

.14

(.14)

.04 .14

(.14)

.04 .52

(.05)

.10** .52

(.05)

.10**

Physical Hazards .28

(.42)

.02 .27

(.43)

.02 .43

(.14)

.03** .43

(.14)

.03**

Physical Activity -.003

(.17)

.00 .02

(.19)

.01 -.19

(.07)

-.03** -.19

(.07)

-.03**

Physical Hazards x Physical Activity

.04

(.12)

.01 -.04

(.03)

-.01

R2 .01*

2.99*

.23*

.10

.02*

63.72**

.02*

1.29 F Change Note: Physical Hazards and Physical Activity were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B14

Physical Activity as a Moderator between Psychosocial Hazards & Sickness Absence

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender 4.55

(1.53)

.10** 4.55

(1.53)

.10** 4.89

(.54)

.08** 4.90

(.54)

.08**

Physical Hazards .28

(.42)

.02 .27

(.43)

.02 .42

(.14)

.03** .43

(.14)

.03**

Psych Hazards .14

(.14)

.04 .14

(.14)

.04 .52

(.05)

.10** .52

(.05)

.10**

Physical Activity -.003

(.17)

.00 .02

(.19)

.01 -.19

(.07)

-.03** -.19

(.07)

-.03**

Physical Hazards x Physical Activity

.04

(.12)

.01 -.04

(.03)

-.01

R2 .01*

2.99*

.01*

2.41*

.02*

63.72**

.02*

.01 F Change Note: Psychosocial Hazards and Physical Activity were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B15

Diet as a Moderator between Physical Hazards & BMI

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender -2.06

(.37)

-.27** -2.07

(.25)

-.27** -1.46

(.13)

-.17** -1.46

(.13)

-.17**

Psych Hazards .09

(.02)

.13** .09

(.02)

.013** .05

(.01)

.06** .05

(.01)

.06**

Physical Hazards -.17

(.07)

-.08* -.14

(.08)

-.07 .04

(.02)

.02* .04

(.02)

.02*

Diet -.17

(.07)

-.09** -.19

(.07)

-.10** -.01

(.02)

-.004 -.01

(.02)

-.004

Physical Hazards x Diet

-.04

(.04)

-.04 -.02

(.01)

-.01

R2 .10**

25.35**

.10**

.91

.03*

123.95**

.03*

1.85 F Change Note: Physical Hazards and Diet were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B16

Diet as a Moderator between Psychosocial Hazards & BMI

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender -2.06

(.25)

-.27** -2.07

(.25)

-.27** -1.46

(.13)

-.17** -1.46

(.07)

-.17**

Physical Hazards -.17

(.07)

-.08* -.16

(.07)

-.08* .04

(.02)

.02* .04

(.02)

.02*

Psych Hazards .09

(.02)

.13** .10

(.02)

.14** .05

(.01)

.06** .05

(.01)

.06**

Diet -.17

(.07)

-.09** -.18

(.07)

-.09** -.01

(.02)

-.004 -.01

(.02)

-.004

Psychos Hazards x Diet

-.02

(.01)

-.05 -.003

(.004)

-.01

R2 .10**

25.35**

.10**

2.46

.03*

123.95**

.03*

.52 F Change Note: Psychosocial Hazards and Diet were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B17

Diet as a Moderator between Physical Hazards & GHQ-12

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender -.07

(.21)

-.01 -.08

(.21)

-.01 .39

(.05)

.06** .39

(.05)

.06

Psych Hazards .32

(.02)

.49** .32

(.02)

.49** .29

(.01)

.46** .29

(.01)

.46**

Physical Hazards -.04

(.06)

-.02 -.01

(.06)

-.004 -.02

(.01)

-.01 -.02

(.01)

-.01

Diet .00

(.05)

.00 -.02

(.06)

-.01 -.07

(.02)

-.03** -.07

(.02)

-.03**

Physical Hazards x Diet

-.04

(.03)

-.04 -.01

(.01)

-.01

R2 .23**

77.27**

.23**

1.75

.21**

1001.10**

.21**

.72 F Change Note: Physical Hazards and Diet were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B18

Diet as a Moderator between Psychosocial Hazards & GHQ-12

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender -.07

(.21)

-.01 -.07

(.21)

-.01 .39

(.05)

.06** .39

(.05)

.06**

Physical Hazards -.04

(.06)

-.02 -.04

(.06)

-.02 -.02

(.01)

-.01 -.02

(.01)

-.01

Psych Hazards .32

(.02)

.49** .33

(.02)

.50** .29

(.01)

.46** .29

(.01)

.46**

Diet .00

(.05)

.00 -.01

(.05)

-.003 -.07

(.02)

-.03** -.07

(.02)

-.03**

Psych Hazards x Diet

-.01

(.01)

-.04 .00

(.003)

-.002

R2 .23**

77.27**

.23**

1.76

.21**

1001.10**

.21**

.06 F Change Note: Psychosocial Hazards and Diet were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B19

Diet as a Moderator between Physical Hazards & Sickness Absence

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender 4.56

(1.45)

.10** 4.56

(1.45)

.10** 5.08

(.50)

.09** 5.08

(.50)

.09**

Psych Hazards .14

(.13)

.04 .14

(.13)

.04 .52

(.05)

.10** .52

(.05)

.10**

Physical Hazards .28

(.40)

.02 .41

(.44)

.04 .42

(.13)

.03 .42

(.13)

.03

Diet -.03

(.38)

-.002 -.12

(.41)

-.01 .07

(.15)

.004 -.07

(.15)

.004

Physical Hazards x Diet

-.16

(-.23)

-.03 -.003

(.08)

.00

R2 .01*

3.36**

.01*

.52

.02*

72.82**

.02*

.002 F Change Note: Physical Hazards and Diet were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B20

Diet as a Moderator between Psychosocial Hazards & Sickness Absence

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender 4.56

(1.45)

.10** 4.56

(1.45)

.10* 5.08

(50)

.09** 5.08

(.50)

.09**

Physical Hazards .28

(.40)

.02 .29

(40)

.02 .42

(.13)

.03** .42

(.13)

.03**

Psych Hazards .14

(.13)

.04 .16

(.14)

.04 .52

(.05)

.10** .52

(.05)

.10**

Diet -.03

(.38)

-.002 -.04

(.39)

-.003 .07

(.15)

.004 .06

(.15)

.004

Psych Hazards x Diet

-.03

(.08)

-.01 .04

(.03)

.01

R2 .01*

3.36*

.01*

.11

.02**

72.82**

.02**

1.64 F Change Note: Psychosocial Hazards and Diet were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B21

Age as a Moderator between Physical Hazards & BMI

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender -2.00

(.26)

-.26** -2.03

(.26)

-.27 -1.25

(.07)

-.15** -1.26

(.07)

-.15**

Psych Hazards .09

(.02)

.12** .09

(.02)

.13** .06

(.01)

.07** .06

(.01)

.07**

Physical Hazards -.16

(.07)

-.08* -.09

(.09)

-.05 .04

(.02)

.02** .04

(.02)

.02*

Age .03

(.01)

.06* .02

(.02)

.05 .07

(.004)

.16** .07

(.004)

.16**

Physical Hazards x Age

-.01

(.01)

.05 .001

(.002)

.004

R2 .10**

24.29**

.10**

1.67

.06**

215.46**

.06**

.26 F Change Note: Physical Hazards and Age were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B22

Age as a Moderator between Psychosocial Hazards & BMI

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender -2.00

(.26)

-.26** -2.03

(.26)

-.27** -1.25

(.07)

-.15** -1.26

(.07)

-.15**

Physical Hazards -.16

(.07)

-.08* -.16

(.07)

-.08* .04

(.02)

.02* .04

(.02)

.02**

Psych Hazards .09

(.02)

.12** .13

(.03)

.18** .06

(.01)

.07** .06

(.01)

.07**

Age .03

(.01)

.06* .02

(.01)

.06* .07

(.004)

.16** .07

(.004)

.16**

Psych Hazards x Age

-.01

(.003)

-.08 .00

(.001)

.002

R2 .10**

24.29**

.10**

3.43*

.06**

215.46**

.06**

.06 F Change Note: Psychosocial Hazards and Age were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B23

Age as a Moderator between Physical Hazards & Sickness Absence

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender 5.01

(1.52)

.14** 5.15

(1.52)

.12** 5.45

(.50)

.09** 5.43

(.50)

.09**

Psych Hazards .15

(.14)

.04 .14

(.14)

.04 .54

(.05)

.10** .54

(.05)

.10**

Physical Hazards .33

(.41)

.03 .01

(.50)

.00 .42

(.13)

.03** .45

(.13)

.03**

Age .09

(.08)

.04 .11

(.09)

.05 .11

(.03)

.04** .12

(.03)

.04**

Physical Hazards x Age

.05

(.05)

.05 .02

(.01)

.02*

R2 .02**

3.58**

.02**

1.18

.02**

77.21**

.02**

2.92* F Change Note: Physical Hazards and Age were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B24

Age as a Moderator between Psychosocial Hazards & Sickness Absence

Model 1

(Senior Pay Grade) Model 2

(Senior Pay Grade) Model 1

(Lower Pay Grade) Model 2

(Lower Pay Grade)

Variable B β B β B β B β

Gender 5.01

(1.52)

.14** 5.03

(1.52)

.11** 5.45

(.50)

.09** 5.47

(.50)

.09**

Physical Hazards .33

(.41)

.03 .33

(.41)

.03 .42

(.13)

.03** .42

(.13)

.03**

Psych Hazards .15

(.14)

.04 .13

(.19)

.03 .54

(.05)

.10** .55

(.05)

.10**

Age .09

(.08)

.04 .09

(.08)

.04 .11

(.03)

.04** .12

(.03)

.04**

Psych Hazards x Age

.002

(.02)

.01 .01

(.01)

.02*

R2 .02

3.58**

.02

.02

.02**

77.21**

.02**

3.58* F for change in R2 Note: Psychosocial Hazards and Age were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas

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TABLE B25

Regression Analyses Split by Age

Age Group IV’s DV DF Model R square < 30 Physical hazards,

Psychosocial hazards, Gender

BMI (3, 2900) .03**

GHQ-12 (3, 2900) .20** Sickness Absence (3, 2935) .02**

30-40 BMI (3, 6073) .04** GHQ-12 (3, 6471) .20** Sickness Absence (3, 6036) .02**

40-50 BMI (3, 3996) .03** GHQ-12 (3, 4216) .22** Sickness Absence (3, 3878) .03**

50+ BMI (3, 2368) .01** GHQ-12 (3, 2511) .26** Sickness Absence (3, 2299) .01**

*p < .05. **p < .01.

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TABLE B26

Regression Analyses Split within Lower Pay Grade

Pay Grade IV’s DV DF Model R square B Physical hazards,

Psychosocial hazards, Gender

BMI (3, 2772) .03**

GHQ-12 (3, 2932) .24** Sickness Absence (3, 2715) .02**

C BMI (3, 4221) .04** GHQ-12 (3, 4460) .19** Sickness Absence (3, 4160) .02**

D BMI (3, 6140) .02** GHQ-12 (3, 6549) .12** Sickness Absence (3, 6078) .02**

Industrial BMI (3, 856) .01** GHQ-12 (3, 900) .28** Sickness Absence (3, 806) .04**

*p < .05. **p < .01.

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TABLE B27

Supported Moderation Analyses: Sickness Absence as DV

Model 1

(Low Grade) Model 2

(Low Grade) Model 1

(Low Grade) Model 2

(Low Grade)

Var B β B β R2 B β B β R2

Gender 5.45

(.50)

.09** 5.43

(.50)

.09** .020 5.45

(.50)

.09** 5.47

(.50)

.09** .020

Phys Hazard .42

(.13)

.03** .45

(.13)

.03** .021 .42

(.13)

.03** .42

(.13)

.03** .021

Psych Hazard .54

(.05)

.10** .54

(.05)

.10** .021 .54

(.05)

.10** .55

(.05)

.10** .021

Age .11

(.03)

.04** .12

(.03)

.04** .022 .11

(.03)

.04** .12

(.03)

.04** .022

Psych Hazard x Age

.01

(.01)

.02** .023

Phys Hazard x Age

.02

(.01)

.02* .023

R2 .02**

77.21**

.02**

.2.92*

.02**

77.21**

.02**

3.58* F Change Note: Psychosocial Hazards, Physical Hazards, and Age were centered at their means. *p < .05. **p < .01. Standard errors appear in parentheses under unstandardized betas Phys is abbreviation for Physical Hazards, Psych is abbreviation for Psychosocial Hazards