Immediate Care of Newbor1

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    Thigh muscles VASTUS LATERALIS (best site)

    RECTUS FEMORIS (alternate site)

    6. Weighing and taking of Other Anthropometric

    Measurements

    Weight: 3000g 3,400g (7 7.5 lbs.) Internationalstandard

    : 6 6.5 lbs. average birth weight of a Filipino

    newborn

    > lower limit normal : 2,500g

    Height : 19 21 inches (ave. 50 cm)

    HC : 33 35 cm/13 14 inches

    CC : 31 33 cm/12 13 inches

    AC : 31 33 cm/12 13 inches

    7. Vital signs

    Checking when infants is asleep/quietGentle, minimal handling and watchful eyes

    PHYSIOLOGIC CHANGES IN THE NEWBORN

    1. Weight Loss : 7 10 days

    5% to 10% of BW (6 10 oz)

    After day 10, weight gain of 1 lb per month from 1 6

    months

    BW is doubled at 6 months and tripled at 1 year

    2. Jaundice : 2 7 days for full terms and 3 10 days forpreterm

    Because of liver immaturity and fetal increase in RBCs

    (polycythemia)

    Expose to morning sunlight

    3. Fever : 2 4 days

    Primarily because of dehydration

    4. Anemia : 4 6 months

    NEWBORN SKIN MARKS

    1. LANUGO - fine, downy hair, more in preterms2. VERNIX CASEOSA - whitist, cheesy, odorless usually on

    folds of the skin; more in full term

    protects skin and prevents heat loss

    3. MILIA - white, pinpoint papules on the

    nose/chin/cheecks

    disappears as early as 2 weeks or 3 4 weeks

    4. MONGOLIAN SPOT - grayish blue patch at the lower

    back from accumulation of the pigment cells

    melanocytes

    disappears by school age

    5. NEVI(STORK BITES) - red spots found at the back of

    the neck and above eyelids

    disappears spontaneously before 1 year

    6. ERYTHEMIA TOXICUM NEONATORUM - newborn rash

    pink papular rash appearing on the body within 24 48

    hours after birth; harmless

    disappears within a few days

    SIGNIFICANT NEWBORN REFLEXES

    A. FEEDING REFLEXES

    Rooting if the cheeck or the corner of the mouth is

    touched, hr turns to that side; for food location

    Diasappears at 3 4 months when he can follow moving

    objectsLast period of disappearance : 7 months

    2. Sucking anything that touches the lips is sucked;

    present even before birth

    disappears at 6 months

    3. Extrusion or spitting up anything that touches the

    anterior tongue is extruded, protects infant from

    swallowing inedible substances

    - disappears at 4 6 months

    Swallowing swallows anything that touches the

    posterior tongue

    B. PROTECTIVE REFLEXES

    Sneezing and coughing protect and clear the air

    passages

    Yawning protects cells from depleted oxygen

    Blinking protects eyes from objects coming near it

    C. MORO or STARTLE embracing motion of the arms in

    response to loud noise, jarring of the crib and falling

    sensation

    Best index of CNS integrity; absence indicates BRAIN

    DAMAGEDisappears by the end of 4th or 5th month

    D. TONIC NECK REFLEX/FENCING when head is turned

    to one side, the arm and leg on that side extend and

    opposite arm and leg flex

    disappears at 3 4 months

    E. BABINSKI fanning or hyperextension of the toes

    when the sole is stroked from the heel upwards

    F. DARWIN dancing reflex; few quick alternating stepswhen the newborn is held upright and his feet touch a

    hard surface

    Disappears at 4 weeks

    G. MAGNET If pressure is applied on the soles of the

    feet while infant lies supine, he pushes back against the

    pressure

    A test of spinal cord integrity

    H. CROSSED EXTENSION if one leg of a newborn lying

    supine is extended and the sole is irritated by rubbing it

    with a sharp object, he will raise the other leg and

    extend it as if trying to push away the hand irritating the

    first leg.

    A test of spinal cord integrity

    SKIN DISORDERS IN INFANT

    1. DIAPER DERMATITIS

    1. Diaper rash/contact dermatitis : inflammation of the

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    skin caused by irritants moisture, heat and chemical

    substances

    Erythemia in the genital signals the beginning of the

    rash

    Progresses from macules and papules to eroded, moist

    or crushed lesions

    2. Ammonia dermatitis: diffuse erythema in the perianaland gluteal areas caused by breakdown of urea in the

    urine to ammonia by bacteria in the feces.

    Progresses to shiny, red and excoriated skin.

    3. INTERTRIGO: maceration of any two skin surfaces in

    close opposition/chafing of the skin.

    Common in obese infants in gluteal and neck folds due

    to poor ventilation, high humidity and poor hygiene.

    4. Nursing responsibilities: provide health teachings

    Meticulous skin/care hygiene particularly along skin

    creases

    Keeping areas involved well ventilated and free ofirritating substances, use loose diaper/clothing

    Quickly changing diapers after soiling after washing the

    area with water or bland soap and water if needed. Pat

    dry areas with soft cloth or towel; expose to air for few

    minutes before dipering

    II. MILARIA/HEAT RASH/PRICKLY HEAT

    A. Fine; erythematous papular rash over shoulders neck

    and skin folds due to warm weather or overdressing.

    B. Intervention frequent bathing with cool plain water

    avoiding soap; light dressing. Keeping environment cool,

    application of bland dusting powder and calamine lotionIII. SEBORRHREA DERMATITIS

    A. Common recurrent skin disease called CRADLE CAP in

    neonates, dermatitis of the scalp in infants and dandruff

    in other children.

    Cause accumulation of sweat, sebum and dirt causing

    flat, adherent and greasy scales with pruritus, crushing

    usually indicates a secondary infection.

    C. Prevention Keeping involved areas clean, dry and

    cool and free of irritants.

    Treatment mineral oil, ointment or lotion to soften the

    scales before shampooing.IV. IMPETIGO

    Bacteria infection of the superficial layers of the skin

    invaded by streptococci, staphylococci, or pneumococci,

    commonly found on the face

    Causes: poor skin care, overcrowding, malnutrition

    Characteristics: macules, papules, pustules, crusts

    Treatment: meticulous hygiene of skin,

    hexachlorophene scrubbing of lesions to prevent

    nephritis and rheumatic fever.

    V. BOILS/FURUNCLES

    Bacterial infection of hair follicles common face, neck,

    axila, buttocks

    Progresses from papules to pustules than hard tender,

    hot nodless which form a pus point.

    Treatment personal hygiene, no squeezing, topical

    neomycin cream and diet high in protein, low in fats and

    carbohydrates.

    VI. ORAL MONILIASIS

    Also called oral thrush

    Fungi infection of the mouth

    Cause Candida Albicans

    Seen and white patches on the tongue

    Prevented by oral hygiene, care of maternal nipples,

    proper sterilization of feeding bottles/nipples

    Treatment Mycostatin or Nystatin oral paint

    SELECTED NEWBORN CONDITIONS

    I. HYPOGLYCEMIA low blood sugar

    less than 30 mg % in the first 72 of the full term and less

    than 45 mg % after 72

    less than 20 mg % in the preterm

    A. Etiologic factors prematurity, postmaturity, SGA,

    birth injuries, congenital defects, low APGAR,

    inadequate intake, stresses (cold stress, CS)

    B. Danger Signs jitteriness, apnea, tachypnea, irregular

    breathing plus signs of increased intracranial pressure:

    tense, bulging fontanel

    lethargy

    high-pitch shrill cry

    projectile vomiting

    absent MORO reflex

    tremors/convulsion

    C. Nursing implementation

    Give oral glucose

    Administer ordered 10 % - 25 % IV glucose, monitor rate

    of flow strictly to prevent hyperglycemia

    Keep warm

    Prevent infection: handwashing best measure

    Prevent convulsion: decrease environmental stimuli

    Monitor VS, behavior, serum glucose

    Handle gently

    II. HYPOTHERMIA low body temperature less than 36.5C

    A. Etiologic Factor prematurity, postmaturity, SGA,

    malnourished newborn

    absence of adequate brown fat to burn

    B. Danger Signs low body temperature, mottling,

    cyanosis, crying, increased activity, tachypnea

    C. Nursing Implementation

    * Keep warm: maintain in incubator (best place for

    maintaining body warmth)

    Prevent heat loss

    Oxygenate PRN

    Monitor temperature per axila

    III. HYPERBILIRUBINEMIA Increased serum bilirubin

    more than 12 13 mg %

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    - Normal serum bilirubin in newborn: 2 6 mg %

    A. Etiologic factors Rh and ABO incompatibility,

    infection, prematurity, drugs, breastfeeding (because of

    pregnanedial), polycythemia

    B. Assessment/Findings -

    pathologic jaundice (present in first 24)dark, concentrated urine

    lethargy, poor feeding

    pallor

    signs of increased urine

    C. Treatment: phototherapy and exchange transfusion

    D. Nursing responsibility: Detect and report early

    pathologic jaundice

    HEMOLYTIC DISEASE OF THE NEWBORN

    ERYTHROBLASTOSIS FETALIS

    A. Blood incompatibility characterized by:

    hemolytic anemia

    hyperbilirubinemia

    B. Types: Rh incompatibility and ABO incompatibility

    Rh incompatibility is more severe: does not usually

    affect the first child.

    C. Danger Signs severe paleness at birth and

    pathologic jaundice (appears in the first 24)

    Newborn is not jaundiced at birth because there isplacental excretion of excess bilirubin.

    D. Diagnosis: COOMBS TEST

    - Direct Coombs test uses newborn blood mix with

    Coombs reagent whereas indirect Coombs test uses

    maternal blood mixed with Rh (+) blood.

    Positive result: with RBC agglutination, mother has

    produced antibodies (+ isoimmunization)

    Negative result: without RBC agglutination; mother has

    not produced antibodies yet (- isoimmunizatiion)

    E. Prevention: RhoGAM

    RhoGAM is given to an Rh (-) mother, with Rh (+) fetus,

    abortus or ectopic pregnancy, with (_) COOMBS test.

    Action destroys fetal antigens (fetal RBCs) before

    mother produces antibodies.

    Given intramuscularly in the first 72 after delivery of a

    fullterm, abortus or ectopic pregnancy.

    F. Treatment: phototherapy and exchange transfusion

    PHOTOTHERAPY decreases serum bilirubin

    Nursing responsibilities:

    Undress infant leaving diapers

    Cover eyes with eye shield

    Have light 16 inches away from infant

    Turn gently every 2 hours

    Give sterile water in between regular milk feedings

    Monitor temperature, I & O, serum bilirubin, jaundice

    and side effects: rise in temperature, dehydration,

    priapism (painful penile rection), bronze skin, dark and

    concentrated urine, loose and green stools.

    Retinal damage if eyes are not shielded, sterility if

    genitalia is not covered .

    EXCHANGE TRANSFUSION decreases serum bilirubin

    and maternal antibodies, and elevates hemoglobin

    Nursing responsibilities:

    Have appropriate blood ready: Rh (-) and type (O), fresh,

    at room temperature with hematocrit 50 % + and pH 7.1

    or as specified by the physician, heparinized

    Check VS before and after 15 minutes during specially

    CR.

    NPO 3 4 hours before or aspirate stomach to preventvomiting and aspiration

    Have resuscitation equipment ready

    Place infant on his back with arms and legs restrained

    and under radiant warmer

    Albumin (1 gm/kg) maybe given 1 2 hours before to

    allow more binding sites for bilirubin making exchange

    more effective.

    Note and record the time of exchange more effective

    Note and record time of exchange, monitor exchanges

    10 % calcium gluconate maybe given after each 100 ml

    of blood exchanged to prevent hypocalcemia.Protamine sulfate maybe given after the exchange

    transfusion to prevent bleeding.

    After transfusion, leave umbilical catheter with IV plug

    for a repeat exchange or remove catheter, small

    pressure dressing applied and site observed for

    bleeding.

    V. RESPIRATORY DISTRESS SYNDROME/HYALINE

    MEMBRANE DISEASE

    Pulmonary condition common in preterms andcharacterized by hyaline membrane formed in the

    alveoli causing atelectasis.

    Etiologic Factors: prematurity, hypothermia, acidosis,

    hypoxia.

    Main pathologic finding: inadequate surfactant

    Major Assessment Findings:

    *Expiratory grunting -Tachypnea (more than72 minutes)

    * Flaring

    *See-saw breathing - Chest retractions and Lower chest

    THE PREMATURE AND POSTMATURE INFANT

    PREMATURE INFANT POSTMATURE INFANT

    a. Born at 36 weeks or less a. Born at 43 weeks or over

    b. Low birth-weight, poorly b. low birth-weight with

    placental

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    developed muscles and insufficiency from aging process

    fatty tissues

    c. Weak, lethargic, with poor c. wide awake, mentally

    alert

    muscle tone and reflexes

    d. Skin: red, wrinkled, transparent d. Skin: greenish

    (meconiumto translucent with visible stained, lethery

    desquamating

    capillaries, less subcutaneous parchment-like, absent or

    slight

    fats, MORE LANUGO, LESS lanugo and vernix caseosa

    VERNIX CASEOSA

    PREMATURE INFANT POSTMATURE INFANT

    e. Associated Problems e. Associated Problems

    1. Respiratory Distress Syndrome 1. Meconium

    aspiration

    2. Hypothermia 2. Hypothermia

    3. Hypoglycemia 3. Hypoglycemia

    4. Hyperbilirubinemia 4. Polycythemia =

    Hyperbilirubinemia

    5. Infection 5. Infection

    6. Rickets and anemia

    7. Mental retardation from kenicterus

    8. Retrolental Fibroplasia

    NURSING RESPONSIBILITIES FOR LOW BIRTH-WEIGHT

    INFANTS

    A. Establish and maintain airway.

    Resuscitation mostly necessary at birth because of poor

    APGAR.

    Suction using a sterile catheter and brief suctioning LESSTHAN 5 SECONDS per suctioning time as necessary.

    Safe use of oxygen to prevent oxygen toxicity.

    B. Keep warm inside ISOLETTE/INCUBATOR = the best

    place to keep him warm

    Monitor temperature per axilla

    Maintain heat and humidity

    C. Prevent infection

    Hand washing is the BEST way to prevent and its spread.

    Masking is the LEAST.(NOSOCOMIAL NURSERY INFECTIONS are hospital

    acquired infection and the MOST COMMON CAUSE is

    staphylococcus aureus).

    D. Monitor respiration, color, VS, I & O and weight

    E. Maintain hydration and nutrition to promote rapid

    growth.

    Usually led by NGT or GAVAGE because sucking and

    swallowing are poor; adhere to safety rules in gavage

    feeding

    F. Gentle and minimal handling

    G. Support parents; encourage verbalization, allowparenteral care as much as possible AFTER

    APPROPRIATE TEACHING.

    H. PREVENT maturity or low birth-weight conditions

    early and regular PRENATAL CARE is the best prevention

    to complications of pregnancy, labor and puerperium

    are also the best way to prevent high-risk newborn.