© 2008 LWW Chapter 5. Immediate Care of Acute Orthopedic Injuries.
Immediate Care of Newbor1
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Thigh muscles VASTUS LATERALIS (best site)
RECTUS FEMORIS (alternate site)
6. Weighing and taking of Other Anthropometric
Measurements
Weight: 3000g 3,400g (7 7.5 lbs.) Internationalstandard
: 6 6.5 lbs. average birth weight of a Filipino
newborn
> lower limit normal : 2,500g
Height : 19 21 inches (ave. 50 cm)
HC : 33 35 cm/13 14 inches
CC : 31 33 cm/12 13 inches
AC : 31 33 cm/12 13 inches
7. Vital signs
Checking when infants is asleep/quietGentle, minimal handling and watchful eyes
PHYSIOLOGIC CHANGES IN THE NEWBORN
1. Weight Loss : 7 10 days
5% to 10% of BW (6 10 oz)
After day 10, weight gain of 1 lb per month from 1 6
months
BW is doubled at 6 months and tripled at 1 year
2. Jaundice : 2 7 days for full terms and 3 10 days forpreterm
Because of liver immaturity and fetal increase in RBCs
(polycythemia)
Expose to morning sunlight
3. Fever : 2 4 days
Primarily because of dehydration
4. Anemia : 4 6 months
NEWBORN SKIN MARKS
1. LANUGO - fine, downy hair, more in preterms2. VERNIX CASEOSA - whitist, cheesy, odorless usually on
folds of the skin; more in full term
protects skin and prevents heat loss
3. MILIA - white, pinpoint papules on the
nose/chin/cheecks
disappears as early as 2 weeks or 3 4 weeks
4. MONGOLIAN SPOT - grayish blue patch at the lower
back from accumulation of the pigment cells
melanocytes
disappears by school age
5. NEVI(STORK BITES) - red spots found at the back of
the neck and above eyelids
disappears spontaneously before 1 year
6. ERYTHEMIA TOXICUM NEONATORUM - newborn rash
pink papular rash appearing on the body within 24 48
hours after birth; harmless
disappears within a few days
SIGNIFICANT NEWBORN REFLEXES
A. FEEDING REFLEXES
Rooting if the cheeck or the corner of the mouth is
touched, hr turns to that side; for food location
Diasappears at 3 4 months when he can follow moving
objectsLast period of disappearance : 7 months
2. Sucking anything that touches the lips is sucked;
present even before birth
disappears at 6 months
3. Extrusion or spitting up anything that touches the
anterior tongue is extruded, protects infant from
swallowing inedible substances
- disappears at 4 6 months
Swallowing swallows anything that touches the
posterior tongue
B. PROTECTIVE REFLEXES
Sneezing and coughing protect and clear the air
passages
Yawning protects cells from depleted oxygen
Blinking protects eyes from objects coming near it
C. MORO or STARTLE embracing motion of the arms in
response to loud noise, jarring of the crib and falling
sensation
Best index of CNS integrity; absence indicates BRAIN
DAMAGEDisappears by the end of 4th or 5th month
D. TONIC NECK REFLEX/FENCING when head is turned
to one side, the arm and leg on that side extend and
opposite arm and leg flex
disappears at 3 4 months
E. BABINSKI fanning or hyperextension of the toes
when the sole is stroked from the heel upwards
F. DARWIN dancing reflex; few quick alternating stepswhen the newborn is held upright and his feet touch a
hard surface
Disappears at 4 weeks
G. MAGNET If pressure is applied on the soles of the
feet while infant lies supine, he pushes back against the
pressure
A test of spinal cord integrity
H. CROSSED EXTENSION if one leg of a newborn lying
supine is extended and the sole is irritated by rubbing it
with a sharp object, he will raise the other leg and
extend it as if trying to push away the hand irritating the
first leg.
A test of spinal cord integrity
SKIN DISORDERS IN INFANT
1. DIAPER DERMATITIS
1. Diaper rash/contact dermatitis : inflammation of the
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skin caused by irritants moisture, heat and chemical
substances
Erythemia in the genital signals the beginning of the
rash
Progresses from macules and papules to eroded, moist
or crushed lesions
2. Ammonia dermatitis: diffuse erythema in the perianaland gluteal areas caused by breakdown of urea in the
urine to ammonia by bacteria in the feces.
Progresses to shiny, red and excoriated skin.
3. INTERTRIGO: maceration of any two skin surfaces in
close opposition/chafing of the skin.
Common in obese infants in gluteal and neck folds due
to poor ventilation, high humidity and poor hygiene.
4. Nursing responsibilities: provide health teachings
Meticulous skin/care hygiene particularly along skin
creases
Keeping areas involved well ventilated and free ofirritating substances, use loose diaper/clothing
Quickly changing diapers after soiling after washing the
area with water or bland soap and water if needed. Pat
dry areas with soft cloth or towel; expose to air for few
minutes before dipering
II. MILARIA/HEAT RASH/PRICKLY HEAT
A. Fine; erythematous papular rash over shoulders neck
and skin folds due to warm weather or overdressing.
B. Intervention frequent bathing with cool plain water
avoiding soap; light dressing. Keeping environment cool,
application of bland dusting powder and calamine lotionIII. SEBORRHREA DERMATITIS
A. Common recurrent skin disease called CRADLE CAP in
neonates, dermatitis of the scalp in infants and dandruff
in other children.
Cause accumulation of sweat, sebum and dirt causing
flat, adherent and greasy scales with pruritus, crushing
usually indicates a secondary infection.
C. Prevention Keeping involved areas clean, dry and
cool and free of irritants.
Treatment mineral oil, ointment or lotion to soften the
scales before shampooing.IV. IMPETIGO
Bacteria infection of the superficial layers of the skin
invaded by streptococci, staphylococci, or pneumococci,
commonly found on the face
Causes: poor skin care, overcrowding, malnutrition
Characteristics: macules, papules, pustules, crusts
Treatment: meticulous hygiene of skin,
hexachlorophene scrubbing of lesions to prevent
nephritis and rheumatic fever.
V. BOILS/FURUNCLES
Bacterial infection of hair follicles common face, neck,
axila, buttocks
Progresses from papules to pustules than hard tender,
hot nodless which form a pus point.
Treatment personal hygiene, no squeezing, topical
neomycin cream and diet high in protein, low in fats and
carbohydrates.
VI. ORAL MONILIASIS
Also called oral thrush
Fungi infection of the mouth
Cause Candida Albicans
Seen and white patches on the tongue
Prevented by oral hygiene, care of maternal nipples,
proper sterilization of feeding bottles/nipples
Treatment Mycostatin or Nystatin oral paint
SELECTED NEWBORN CONDITIONS
I. HYPOGLYCEMIA low blood sugar
less than 30 mg % in the first 72 of the full term and less
than 45 mg % after 72
less than 20 mg % in the preterm
A. Etiologic factors prematurity, postmaturity, SGA,
birth injuries, congenital defects, low APGAR,
inadequate intake, stresses (cold stress, CS)
B. Danger Signs jitteriness, apnea, tachypnea, irregular
breathing plus signs of increased intracranial pressure:
tense, bulging fontanel
lethargy
high-pitch shrill cry
projectile vomiting
absent MORO reflex
tremors/convulsion
C. Nursing implementation
Give oral glucose
Administer ordered 10 % - 25 % IV glucose, monitor rate
of flow strictly to prevent hyperglycemia
Keep warm
Prevent infection: handwashing best measure
Prevent convulsion: decrease environmental stimuli
Monitor VS, behavior, serum glucose
Handle gently
II. HYPOTHERMIA low body temperature less than 36.5C
A. Etiologic Factor prematurity, postmaturity, SGA,
malnourished newborn
absence of adequate brown fat to burn
B. Danger Signs low body temperature, mottling,
cyanosis, crying, increased activity, tachypnea
C. Nursing Implementation
* Keep warm: maintain in incubator (best place for
maintaining body warmth)
Prevent heat loss
Oxygenate PRN
Monitor temperature per axila
III. HYPERBILIRUBINEMIA Increased serum bilirubin
more than 12 13 mg %
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- Normal serum bilirubin in newborn: 2 6 mg %
A. Etiologic factors Rh and ABO incompatibility,
infection, prematurity, drugs, breastfeeding (because of
pregnanedial), polycythemia
B. Assessment/Findings -
pathologic jaundice (present in first 24)dark, concentrated urine
lethargy, poor feeding
pallor
signs of increased urine
C. Treatment: phototherapy and exchange transfusion
D. Nursing responsibility: Detect and report early
pathologic jaundice
HEMOLYTIC DISEASE OF THE NEWBORN
ERYTHROBLASTOSIS FETALIS
A. Blood incompatibility characterized by:
hemolytic anemia
hyperbilirubinemia
B. Types: Rh incompatibility and ABO incompatibility
Rh incompatibility is more severe: does not usually
affect the first child.
C. Danger Signs severe paleness at birth and
pathologic jaundice (appears in the first 24)
Newborn is not jaundiced at birth because there isplacental excretion of excess bilirubin.
D. Diagnosis: COOMBS TEST
- Direct Coombs test uses newborn blood mix with
Coombs reagent whereas indirect Coombs test uses
maternal blood mixed with Rh (+) blood.
Positive result: with RBC agglutination, mother has
produced antibodies (+ isoimmunization)
Negative result: without RBC agglutination; mother has
not produced antibodies yet (- isoimmunizatiion)
E. Prevention: RhoGAM
RhoGAM is given to an Rh (-) mother, with Rh (+) fetus,
abortus or ectopic pregnancy, with (_) COOMBS test.
Action destroys fetal antigens (fetal RBCs) before
mother produces antibodies.
Given intramuscularly in the first 72 after delivery of a
fullterm, abortus or ectopic pregnancy.
F. Treatment: phototherapy and exchange transfusion
PHOTOTHERAPY decreases serum bilirubin
Nursing responsibilities:
Undress infant leaving diapers
Cover eyes with eye shield
Have light 16 inches away from infant
Turn gently every 2 hours
Give sterile water in between regular milk feedings
Monitor temperature, I & O, serum bilirubin, jaundice
and side effects: rise in temperature, dehydration,
priapism (painful penile rection), bronze skin, dark and
concentrated urine, loose and green stools.
Retinal damage if eyes are not shielded, sterility if
genitalia is not covered .
EXCHANGE TRANSFUSION decreases serum bilirubin
and maternal antibodies, and elevates hemoglobin
Nursing responsibilities:
Have appropriate blood ready: Rh (-) and type (O), fresh,
at room temperature with hematocrit 50 % + and pH 7.1
or as specified by the physician, heparinized
Check VS before and after 15 minutes during specially
CR.
NPO 3 4 hours before or aspirate stomach to preventvomiting and aspiration
Have resuscitation equipment ready
Place infant on his back with arms and legs restrained
and under radiant warmer
Albumin (1 gm/kg) maybe given 1 2 hours before to
allow more binding sites for bilirubin making exchange
more effective.
Note and record the time of exchange more effective
Note and record time of exchange, monitor exchanges
10 % calcium gluconate maybe given after each 100 ml
of blood exchanged to prevent hypocalcemia.Protamine sulfate maybe given after the exchange
transfusion to prevent bleeding.
After transfusion, leave umbilical catheter with IV plug
for a repeat exchange or remove catheter, small
pressure dressing applied and site observed for
bleeding.
V. RESPIRATORY DISTRESS SYNDROME/HYALINE
MEMBRANE DISEASE
Pulmonary condition common in preterms andcharacterized by hyaline membrane formed in the
alveoli causing atelectasis.
Etiologic Factors: prematurity, hypothermia, acidosis,
hypoxia.
Main pathologic finding: inadequate surfactant
Major Assessment Findings:
*Expiratory grunting -Tachypnea (more than72 minutes)
* Flaring
*See-saw breathing - Chest retractions and Lower chest
THE PREMATURE AND POSTMATURE INFANT
PREMATURE INFANT POSTMATURE INFANT
a. Born at 36 weeks or less a. Born at 43 weeks or over
b. Low birth-weight, poorly b. low birth-weight with
placental
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developed muscles and insufficiency from aging process
fatty tissues
c. Weak, lethargic, with poor c. wide awake, mentally
alert
muscle tone and reflexes
d. Skin: red, wrinkled, transparent d. Skin: greenish
(meconiumto translucent with visible stained, lethery
desquamating
capillaries, less subcutaneous parchment-like, absent or
slight
fats, MORE LANUGO, LESS lanugo and vernix caseosa
VERNIX CASEOSA
PREMATURE INFANT POSTMATURE INFANT
e. Associated Problems e. Associated Problems
1. Respiratory Distress Syndrome 1. Meconium
aspiration
2. Hypothermia 2. Hypothermia
3. Hypoglycemia 3. Hypoglycemia
4. Hyperbilirubinemia 4. Polycythemia =
Hyperbilirubinemia
5. Infection 5. Infection
6. Rickets and anemia
7. Mental retardation from kenicterus
8. Retrolental Fibroplasia
NURSING RESPONSIBILITIES FOR LOW BIRTH-WEIGHT
INFANTS
A. Establish and maintain airway.
Resuscitation mostly necessary at birth because of poor
APGAR.
Suction using a sterile catheter and brief suctioning LESSTHAN 5 SECONDS per suctioning time as necessary.
Safe use of oxygen to prevent oxygen toxicity.
B. Keep warm inside ISOLETTE/INCUBATOR = the best
place to keep him warm
Monitor temperature per axilla
Maintain heat and humidity
C. Prevent infection
Hand washing is the BEST way to prevent and its spread.
Masking is the LEAST.(NOSOCOMIAL NURSERY INFECTIONS are hospital
acquired infection and the MOST COMMON CAUSE is
staphylococcus aureus).
D. Monitor respiration, color, VS, I & O and weight
E. Maintain hydration and nutrition to promote rapid
growth.
Usually led by NGT or GAVAGE because sucking and
swallowing are poor; adhere to safety rules in gavage
feeding
F. Gentle and minimal handling
G. Support parents; encourage verbalization, allowparenteral care as much as possible AFTER
APPROPRIATE TEACHING.
H. PREVENT maturity or low birth-weight conditions
early and regular PRENATAL CARE is the best prevention
to complications of pregnancy, labor and puerperium
are also the best way to prevent high-risk newborn.