imbalances stimuli Hyperemesis gravidarum

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Hyperemesis gravidarum What is it? Excessive vomiting that leads to dehydration, starvation, and even death among pregnant populations Related to increased estrogen levels Assessment Hypotension Elevated H&H Decreased urine output Hypokalemia Weight loss Ketonuria Interventions 48 hours of NPO status IV fluids Antiemetics as ordered Vitamins Decrease environmental stimuli Clear liquids and small dry feedings as tolerated. Give either cold or hot food, nothing room temp. Critical labs Potassium Have the patient on tele Monitor for symptoms of shock and fluid volume deficit. Complications Dehydration Wernicke's encephalopathy from vitamin B1 deficiency Mallory-Weiss tears (esophageal tears and bleeding) Esophageal bleeding Pneumothorax Acute tubular necrosis Electrolyte and acidbase imbalances Diagnostic studies Potassium, sodium, chloride, and protein levels are decreased due to losses from vomiting. Blood urea nitrogen, non protein nitrogen, and uric acid levels are increased due to renal compromise and hemoconcentration. Hemoglobin (Hb) level and hematocrit (HCT) are increased due to hemoconcentration. Urinalysis reveals ketones and, possibly, protein; urine specific gravity increases. Vitamin B1 and B6 levels are decreased due to impaired intake. Thyroid-stimulating hormone, thyroxine, and triiodothyronine levels may be mildly increased.

Transcript of imbalances stimuli Hyperemesis gravidarum

Hyperemesis gravidarum What is it?

Excessive vomiting that leads to dehydration, starvation, and even death among pregnant populationsRelated to increased estrogen levels

Assessment ❖ Hypotension ❖ Elevated H&H❖ Decreased urine

output ❖ Hypokalemia ❖ Weight loss❖ Ketonuria

Interventions ❖ 48 hours of NPO status ❖ IV fluids❖ Antiemetics as ordered❖ Vitamins ❖ Decrease environmental

stimuli❖ Clear liquids and small dry

feedings as tolerated. ❖ Give either cold or hot food,

nothing room temp.

Critical labs ❖ Potassium❖ Have the patient on tele❖ Monitor for symptoms of

shock and fluid volume deficit.

Complications ❖ Dehydration ❖ Wernicke's encephalopathy from

vitamin B1 deficiency ❖ Mallory-Weiss tears (esophageal

tears and bleeding) ❖ Esophageal bleeding ❖ Pneumothorax ❖ Acute tubular necrosis ❖ Electrolyte and acid‑base

imbalances

Diagnostic studies ❖ Potassium, sodium, chloride, and protein levels

are decreased due to losses from vomiting. ❖ Blood urea nitrogen, non protein nitrogen, and

uric acid levels are increased due to renal compromise and hemoconcentration. Hemoglobin (Hb) level and hematocrit (HCT) are increased due to hemoconcentration.

❖ Urinalysis reveals ketones and, possibly, protein; urine specific gravity increases.

❖ Vitamin B1 and B6 levels are decreased due to impaired intake.

❖ Thyroid-stimulating hormone, thyroxine, and triiodothyronine levels may be mildly increased.