Electus Servisol Contact Clean & Lube · If breathing is shallow or has stopped, ensure clear...

Electus Distribution Pty Ltd Chemwatch Hazard Alert Code: 4

Electus Servisol Contact Clean & Lube

Chemwatch: 36-4825Version No: 3.1.1.1Safety Data Sheet according to WHS and ADG requirements

Issue Date: 13/07/2015Print Date: 18/12/2017

L.GHS.AUS.EN

SECTION 1 IDENTIFICATION OF THE SUBSTANCE / MIXTURE AND OF THE COMPANY / UNDERTAKING

Product Identifier

Product name Electus Servisol Contact Clean & Lube

Synonyms Not Available

Proper shipping name AEROSOLS

Other means of identification Not Available

Relevant identified uses of the substance or mixture and uses advised against

Relevant identified usesApplication is by spray atomisation from a hand held aerosol packApplication is by spray atomisation from a hand held aerosol pack, cleaner and lubricant.

Details of the supplier of the safety data sheet

Registered company name Electus Distribution Pty Ltd

Address 320 Victoria Road Rydalmere NSW 2116 Australia

Telephone 1300 738 555

Fax 1300 758 500

Website https://www.fischerconnectors.com

Email [email protected]

Emergency telephone number

Association / Organisation Not Available

Emergency telephone numbers +61 2 45774866 (George Jones)

Other emergency telephonenumbers

Not Available

SECTION 2 HAZARDS IDENTIFICATION

Classification of the substance or mixture

HAZARDOUS CHEMICAL. DANGEROUS GOODS. According to the WHS Regulations and the ADG Code.

Poisons Schedule S6

Classification [1]Aerosols Category 1, Gas under Pressure (Compressed gas), Skin Corrosion/Irritation Category 2, Carcinogenicity Category 2, Reproductive ToxicityCategory 2, Specific target organ toxicity - single exposure Category 3 (narcotic effects), Specific target organ toxicity - repeated exposure Category 2,Acute Aquatic Hazard Category 2, Chronic Aquatic Hazard Category 2

Legend: 1. Classified by Chemwatch; 2. Classification drawn from HSIS ; 3. Classification drawn from EC Directive 1272/2008 - Annex VI

Label elements

Hazard pictogram(s)

SIGNAL WORD DANGER

Hazard statement(s)

H222 Extremely flammable aerosol.

H280 Contains gas under pressure; may explode if heated.

H315 Causes skin irritation.

H351 Suspected of causing cancer.

H361 Suspected of damaging fertility or the unborn child.

H336 May cause drowsiness or dizziness.

H373 May cause damage to organs through prolonged or repeated exposure.

H411 Toxic to aquatic life with long lasting effects.

AUH044 Risk of explosion if heated under confinement.

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Precautionary statement(s) Prevention

P201 Obtain special instructions before use.

P210 Keep away from heat/sparks/open flames/hot surfaces. - No smoking.

P211 Do not spray on an open flame or other ignition source.

P251 Pressurized container: Do not pierce or burn, even after use.

P260 Do not breathe dust/fume/gas/mist/vapours/spray.

P271 Use only outdoors or in a well-ventilated area.

P281 Use personal protective equipment as required.

P273 Avoid release to the environment.

P280 Wear protective gloves/protective clothing/eye protection/face protection.

Precautionary statement(s) Response

P308+P313 IF exposed or concerned: Get medical advice/attention.

P362 Take off contaminated clothing and wash before reuse.

P312 Call a POISON CENTER or doctor/physician if you feel unwell.

P391 Collect spillage.

P302+P352 IF ON SKIN: Wash with plenty of soap and water.

P304+P340 IF INHALED: Remove victim to fresh air and keep at rest in a position comfortable for breathing.

P332+P313 If skin irritation occurs: Get medical advice/attention.

Precautionary statement(s) Storage

P405 Store locked up.

P410+P403 Protect from sunlight. Store in a well-ventilated place.

P410+P412 Protect from sunlight. Do not expose to temperatures exceeding 50 °C/122 °F.

P403+P233 Store in a well-ventilated place. Keep container tightly closed.

Precautionary statement(s) Disposal

P501 Dispose of contents/container in accordance with local regulations.

SECTION 3 COMPOSITION / INFORMATION ON INGREDIENTS

Substances

See section below for composition of Mixtures

Mixtures

CAS No %[weight] Name

110-54-3 30-60

127-18-4 10-30

Not avail. <10

68476-85-7. 10-30

SECTION 4 FIRST AID MEASURES

Description of first aid measures

Eye Contact

If aerosols come in contact with the eyes: Immediately hold the eyelids apart and flush the eye with fresh running water. Ensure complete irrigation of the eye by keeping eyelids apart and away from eye and moving the eyelids by occasionally lifting the upper and lower lids. Seek medical attention without delay; if pain persists or recurs seek medical attention. Removal of contact lenses after an eye injury should only be undertaken by skilled personnel.

Skin Contact

If solids or aerosol mists are deposited upon the skin:Flush skin and hair with running water (and soap if available). Remove any adhering solids with industrial skin cleansing cream. DO NOT use solvents. Seek medical attention in the event of irritation.

Inhalation

If aerosols, fumes or combustion products are inhaled:Remove to fresh air. Lay patient down. Keep warm and rested. Prostheses such as false teeth, which may block airway, should be removed, where possible, prior to initiating first aid procedures. If breathing is shallow or has stopped, ensure clear airway and apply resuscitation, preferably with a demand valve resuscitator, bag-valve mask device, orpocket mask as trained. Perform CPR if necessary. Transport to hospital, or doctor.

Ingestion

Not considered a normal route of entry.For advice, contact a Poisons Information Centre or a doctor.Avoid giving milk or oils. Avoid giving alcohol. If spontaneous vomiting appears imminent or occurs, hold patient's head down, lower than their hips to help avoid possible aspiration of vomitus.

n-hexane

perchloroethylene

mineral oil

hydrocarbon propellant

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Indication of any immediate medical attention and special treatment needed

for intoxication due to Freons/ Halons; A: Emergency and Supportive Measures

Maintain an open airway and assist ventilation if necessary Treat coma and arrhythmias if they occur. Avoid (adrenaline) epinephrine or other sympathomimetic amines that may precipitate ventricular arrhythmias. Tachyarrhythmias caused by increasedmyocardial sensitisation may be treated with propranolol, 1-2 mg IV or esmolol 25-100 microgm/kg/min IV. Monitor the ECG for 4-6 hours

B: Specific drugs and antidotes:There is no specific antidote

C: Decontamination Inhalation; remove victim from exposure, and give supplemental oxygen if available. Ingestion; (a) Prehospital: Administer activated charcoal, if available. DO NOT induce vomiting because of rapid absorption and the risk of abrupt onset CNS depression. (b) Hospital:Administer activated charcoal, although the efficacy of charcoal is unknown. Perform gastric lavage only if the ingestion was very large and recent (less than 30 minutes)

D: Enhanced elimination:There is no documented efficacy for diuresis, haemodialysis, haemoperfusion, or repeat-dose charcoal.

POISONING and DRUG OVERDOSE, Californian Poison Control System Ed. Kent R Olson; 3rd EditionDo not administer sympathomimetic drugs unless absolutely necessary as material may increase myocardial irritability. No specific antidote. Because rapid absorption may occur through lungs if aspirated and cause systematic effects, the decision of whether to induce vomiting or not should be made by an attending physician. If lavage is performed, suggest endotracheal and/or esophageal control. Danger from lung aspiration must be weighed against toxicity when considering emptying the stomach. Treatment based on judgment of the physician in response to reactions of the patient

Treat symptomatically. Following acute or short term repeated exposures to n-hexane:

Large quantities of n-hexane are expired by the lungs after vapour exposure (50-60%). Humans exposed to 100 ppm demonstrate an n-hexane biological half life of 2 hours. Initial attention should be directed towards evaluation and support of respiration. Cardiac dysrhythmias are a potential complication.

INGESTION:Ipecac syrup should be considered for ingestion of pure hexane exceeding 2-3ml/kg. Extreme caution must be taken to avoid aspiration since small amounts of n-hexane intratracheally, producea severe chemical pneumonitis.

[Ellenhorn and Barceloux: Medical Toxicology]BIOLOGICAL EXPOSURE INDEX - BEI

BEIs represent the levels of determinants which are most likely to be observed in specimens collected in a healthy worker who has been exposed to chemicals to the same extent as a worker withinhalation exposure to the Exposure Standard (ES or TLV).Determinant Index Sampling Time Comments1. 2,5-hexanedione in urine 5 mg/gm creatinine End of shift NS2. n-Hexane in end-exhaled air SQ NS: Non-specific determinant; Metabolite observed following exposure to other materials.SQ: Semi-quantitative determinant; Interpretation may be ambiguous - should be used as a screening test or confirmatory test.For acute or short term repeated exposures to perchloroethylene:

Tetrachloroethylene / perchloroethylene is well absorbed through the lungs with peak levels more important than duration in determining blood concentration. Lungs excrete most of the absorbedtetrachloroethylene in an unchanged state; about 3% is converted by the liver to form trichloracetic acid and subsequently excreted by the kidney. Exhaled material has a biological half-life of 65hours.

INHALATION:The treatment of acute inhalation exposures is supportive with initial attention directed to evaluation / support of ventilation and circulation. As with all hydrocarbons care must be taken to reducethe risk of aspiration by proper positioning and medical observation.

INGESTION:The ingestion level at which emesis should be induced is difficult to predict in the absence of extensive human studies. The role of charcoal and cathartics remains uncertain.

[Ellenhorn and Barceloux: Medical Toxicology]BIOLOGICAL EXPOSURE INDEX - BEI

These represent the determinants observed in specimens collected from a healthy worker who has been exposed at the Exposure Standard (ES or TLV):Determinant Index Sampling Time Comments1. Perchloroethylene in end-exhaled air 10 ppm Prior to last shift of work-week2. Perchloroethylene in blood 1 mg/L Prior to last shift of work-week3. Trichloroacetic acid in urine 7 mg/L End of work-week NS, SQ NS: Non-specific determinant; also seen after exposure to other materialsSQ: Semi-quantitative determinant - Interpretation may be ambiguous; should be used as a screening test or confirmatory test.

SECTION 5 FIREFIGHTING MEASURES

Extinguishing media

SMALL FIRE:Water spray, dry chemical or CO2

LARGE FIRE:Water spray or fog.

Special hazards arising from the substrate or mixture

Fire Incompatibility Avoid contamination with oxidising agents i.e. nitrates, oxidising acids, chlorine bleaches, pool chlorine etc. as ignition may result

Advice for firefighters

Fire Fighting

FOR FIRES INVOLVING MANY GAS CYLINDERS: To stop the flow of gas, specifically trained personnel may inert the atmosphere to reduce oxygen levels thus allowing the capping of leakingcontainer(s). Reduce the rate of flow and inject an inert gas, if possible, before completely stopping the flow to prevent flashback. DO NOT extinguish the fire until the supply is shut off otherwise an explosive re-ignition may occur. If the fire is extinguished and the flow of gas continues, used increased ventilation to prevent build-up, of explosive atmosphere. Use non-sparking tools to close container valves. Be CAUTIOUS of a Boiling Liquid Evaporating Vapour Explosion, BLEVE, if fire is impinging on surrounding containers. Direct 2500 litre/min (500 gpm) water stream onto containers above liquid level with the assistance remote monitors. Alert Fire Brigade and tell them location and nature of hazard. May be violently or explosively reactive. Wear breathing apparatus plus protective gloves. Prevent, by any means available, spillage from entering drains or water course. If safe, switch off electrical equipment until vapour fire hazard removed.

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Use water delivered as a fine spray to control fire and cool adjacent area. DO NOT approach containers suspected to be hot. Cool fire exposed containers with water spray from a protected location. If safe to do so, remove containers from path of fire. Equipment should be thoroughly decontaminated after use.

--------------------------------------------------------------GENERAL--------------------------------------------------------------

Alert Fire Brigade and tell them location and nature of hazard. May be violently or explosively reactive. Wear breathing apparatus plus protective gloves. Consider evacuation Fight fire from a safe distance, with adequate cover. If safe, switch off electrical equipment until vapour fire hazard removed. Use water delivered as a fine spray to control fire and cool adjacent area. DO NOT approach cylinders suspected to be hot. Cool fire-exposed cylinders with water spray from a protected location. If safe to do so, remove containers from path of fire.

--------------------------------------------------------------FIRE FIGHTING PROCEDURES:--------------------------------------------------------------

The only safe way to extinguish a flammable gas fire is to stop the flow of gas. If the flow cannot be stopped, allow the entire contents of the cylinder to burn while cooling the cylinder and surroundings with water from a suitabledistance. Extinguishing the fire without stopping the gas flow may permit the formation of ignitable or explosive mixtures with air. These mixtures may propagate toa source of ignition.

--------------------------------------------------------------SPECIAL HAZARDS--------------------------------------------------------------

Excessive pressures may develop in a gas cylinder exposed in a fire; this may result in explosion. Cylinders with pressure relief devices may release their contents as a result of fire and the released gas may constitute a further source of hazard for thefire-fighter. Cylinders without pressure-relief valves have no provision for controlled release and are therefore more likely to explode if exposed to fire.

--------------------------------------------------------------FIRE FIGHTING REQUIREMENTS:--------------------------------------------------------------

The need for proximity, entry and flash-over protection and special protective clothing should be determined for each incident, by a competent fire-fightingsafety professional.

Fire/Explosion Hazard

Liquid and vapour are highly flammable. Severe fire hazard when exposed to heat or flame. Vapour forms an explosive mixture with air. Severe explosion hazard, in the form of vapour, when exposed to flame or spark. Vapour may travel a considerable distance to source of ignition. Heating may cause expansion or decomposition with violent container rupture. Aerosol cans may explode on exposure to naked flames. Rupturing containers may rocket and scatter burning materials. Hazards may not be restricted to pressure effects. May emit acrid, poisonous or corrosive fumes. On combustion, may emit toxic fumes of carbon monoxide (CO).

Combustion products include:,carbon monoxide (CO),carbon dioxide (CO2),hydrogen chloride,phosgene,other pyrolysis products typical of burning organic material.Contains low boiling substance: Closed containers may rupture due to pressure buildup under fire conditions.May emit clouds of acrid smoke

HAZCHEM 2Y

SECTION 6 ACCIDENTAL RELEASE MEASURES

Personal precautions, protective equipment and emergency procedures

See section 8

Environmental precautions

See section 12

Methods and material for containment and cleaning up

Minor Spills

Clean up all spills immediately. Avoid breathing vapours and contact with skin and eyes. Wear protective clothing, impervious gloves and safety glasses. Shut off all possible sources of ignition and increase ventilation. Wipe up. If safe, damaged cans should be placed in a container outdoors, away from all ignition sources, until pressure has dissipated. Undamaged cans should be gathered and stowed safely.

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Major Spills

Remove leaking cylinders to a safe place. Fit vent pipes. Release pressure under safe, controlled conditions Burn issuing gas at vent pipes. DO NOT exert excessive pressure on valve; DO NOT attempt to operate damaged valve.Clear area of personnel and move upwind. Alert Fire Brigade and tell them location and nature of hazard. May be violently or explosively reactive. Wear breathing apparatus plus protective gloves. Prevent, by any means available, spillage from entering drains or water courses No smoking, naked lights or ignition sources. Increase ventilation. Stop leak if safe to do so. Water spray or fog may be used to disperse / absorb vapour. Absorb or cover spill with sand, earth, inert materials or vermiculite. If safe, damaged cans should be placed in a container outdoors, away from ignition sources, until pressure has dissipated. Undamaged cans should be gathered and stowed safely. Collect residues and seal in labelled drums for disposal. Clear area of all unprotected personnel and move upwind. Alert Emergency Authority and advise them of the location and nature of hazard. May be violently or explosively reactive. Wear full body clothing with breathing apparatus. Prevent by any means available, spillage from entering drains and water-courses. Consider evacuation. Shut off all possible sources of ignition and increase ventilation. No smoking or naked lights within area. Use extreme caution to prevent violent reaction. Stop leak only if safe to so do. Water spray or fog may be used to disperse vapour. DO NOT enter confined space where gas may have collected. Keep area clear until gas has dispersed.

Personal Protective Equipment advice is contained in Section 8 of the SDS.

SECTION 7 HANDLING AND STORAGE

Precautions for safe handling

Safe handling

The conductivity of this material may make it a static accumulator., A liquid is typically considered nonconductive if its conductivity is below 100 pS/m and isconsidered semi-conductive if its conductivity is below 10 000 pS/m., Whether a liquid is nonconductive or semi-conductive, the precautions are the same.,A number of factors, for example liquid temperature, presence of contaminants, and anti-static additives can greatly influence the conductivity of a liquid.

Natural gases contain a contaminant, radon-222, a naturally occurring radioactive gas. During subsequent processing, radon tends to concentrate inliquefied petroleum streams and in product streams having similar boiling points. Industry experience indicates that the commercial product may containsmall amounts of radon-222 and its radioactive decay products (radon daughters). The actual concentration of radon-222 and radioactive daughters inprocess equipment (IE lines, filters, pumps and reactor units) may reach significant levels and produce potentially damaging levels of gamma radiation. Apotential external radiation hazard exists at or near any pipe, valve or vessel containing a radon enriched stream or containing internal deposits ofradioactive material. Field studies, however, have not shown that conditions exist that expose the worker to cumulative exposures in excess of generalpopulation limits. Equipment containing gamma-emitting decay products should be presumed to be internally contaminated with alpha-emitting decayproducts which may be hazardous if inhaled or ingested. During maintenance operations that require the opening of contaminated process equipment, theflow of gas should be stopped and a four hour delay enforced to allow gamma-radiation to drop to background levels. Protective equipment (including highefficiency particulate respirators (P3) suitable for radionucleotides or supplied air) should be worn by personnel entering a vessel or working oncontaminated process equipment to prevent skin contamination or inhalation of any residue containing alpha-radiation. Airborne contamination may beminimised by handling scale and/or contaminated materials in a wet state. [TEXACO]

Avoid all personal contact, including inhalation. Wear protective clothing when risk of exposure occurs. Use in a well-ventilated area. Prevent concentration in hollows and sumps. DO NOT enter confined spaces until atmosphere has been checked. Avoid smoking, naked lights or ignition sources. Avoid contact with incompatible materials. When handling, DO NOT eat, drink or smoke. DO NOT incinerate or puncture aerosol cans. DO NOT spray directly on humans, exposed food or food utensils. Avoid physical damage to containers. Always wash hands with soap and water after handling. Work clothes should be laundered separately. Use good occupational work practice. Observe manufacturer's storage and handling recommendations contained within this SDS.Atmosphere should be regularly checked against established exposure standards to ensure safe working conditions are maintained.

Other information

Store below 38 deg. C. Keep dry to avoid corrosion of cans. Corrosion may result in container perforation and internal pressure may eject contents of can Store in original containers in approved flammable liquid storage area. DO NOT store in pits, depressions, basements or areas where vapours may be trapped. No smoking, naked lights, heat or ignition sources. Keep containers securely sealed. Contents under pressure. Store away from incompatible materials. Store in a cool, dry, well ventilated area. Avoid storage at temperatures higher than 40 deg C. Store in an upright position. Protect containers against physical damage. Check regularly for spills and leaks. Observe manufacturer's storage and handling recommendations contained within this SDS.

Conditions for safe storage, including any incompatibilities

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Suitable containerDO NOT use aluminium or galvanised containersAerosol dispenser. Check that containers are clearly labelled.

Storage incompatibility

Butane/ isobutanereacts violently with strong oxidisers reacts with acetylene, halogens and nitrous oxides is incompatible with chlorine dioxide, conc. nitric acid and some plastics may generate electrostatic charges, due to low conductivity, in flow or when agitated - these may ignite the vapour.

Segregate from nickel carbonyl in the presence of oxygen, heat (20-40 C)Haloalkenes are highly reactive. Some of the more lightly substituted lower members are highly flammable; many members of the group are peroxidisable and polymerisable. Avoid reaction or contact with potassium or its alloys - although apparently stable on contact with a wide rage of halocarbons, reaction products may beshock-sensitive and may explode with great violence on light impact. Severity generally increases with the degree of halocarbon substitution andpotassium-sodium alloys give extremely sensitive mixtures.

BRETHERICK L.: Handbook of Reactive Chemical HazardsAvoid reaction with metal halides and active metals, eg. sodium (Na), potassium (K), calcium (Ca), zinc (Zn), powdered aluminium (Al), magnesium(Mg) and magnesium alloys. Avoid contact with rubber, and plastics such as methacrylate polymers, polyethylene and polystyrene

Tetrachloroethylene:decomposes slowly in contact with water or moisture forming trichloroacetic acid and hydrochloric acid reacts violently with concentrated nitric acid (producing carbon dioxide), strong oxidisers, strong alkalis, powdered chemically active metals such asaluminium, barium, beryllium, lithium, zinc is incompatible with nitrogen tetroxide, finely divided metals decomposes in UV light, on contact with red-hot metals, and at temperatures above 150 C, releasing hydrogen chloride, carbon monoxide and phosgene corrodes metals in the presence of moisture can oxidise in presence of air and light.

The presence of 0.5% trichloroethylene as an impurity caused generation of dichloroacetylene during unheated drying over solid sodium hydroxide.Subsequent fractional distillation produced an explosion.Propane:

reacts violently with strong oxidisers, barium peroxide, chlorine dioxide, dichlorine oxide, fluorine etc. liquid attacks some plastics, rubber and coatings may accumulate static charges which may ignite its vapours Compressed gases may contain a large amount of kinetic energy over and above that potentially available from the energy of reaction produced by thegas in chemical reaction with other substances

SECTION 8 EXPOSURE CONTROLS / PERSONAL PROTECTION

Control parameters

OCCUPATIONAL EXPOSURE LIMITS (OEL)

INGREDIENT DATA

Source Ingredient Material name TWA STEL Peak Notes

Australia Exposure Standards n-hexane Hexane (n-Hexane) 72 mg/m3 / 20 ppm Not Available Not Available Not Available

Australia Exposure Standards perchloroethylene Perchloroethylene 340 mg/m3 / 50 ppm 1020 mg/m3 / 150 ppm Not Available Not Available

Australia Exposure Standards mineral oil Oil mist, refined mineral 5 mg/m3 Not Available Not Available Not Available

Australia Exposure Standards hydrocarbon propellant LPG (liquified petroleum gas) 1800 mg/m3 / 1000 ppm Not Available Not Available Not Available

EMERGENCY LIMITS

Ingredient Material name TEEL-1 TEEL-2 TEEL-3

n-hexane Hexane 260 ppm Not Available Not Available

perchloroethylene Perchloroethylene; (Tetrachloroethylene) Not Available Not Available Not Available

hydrocarbon propellant Liquified petroleum gas; (L.P.G.) 65,000 ppm 2.30E+05 ppm 4.00E+05 ppm

Ingredient Original IDLH Revised IDLH

n-hexane 1,100 [LEL] ppm Not Available

perchloroethylene 150 ppm Not Available

mineral oil 2500 mg/m3 Not Available

hydrocarbon propellant 2,000 [LEL] ppm Not Available

MATERIAL DATA

For liquefied petroleum gases (LPG):TLV TWA: 1000 ppm, 1800 mg/m3 (as LPG)ES TWA: 1000 ppm, 1800 mg/m3 (as LPG)OES TWA: 1000 ppm, 1750 mg/m3; STEL: 1250 ppm, 2180 mg/m3 (as LPG)IDLH Level: 2000 ppm (lower explosive limit)No chronic systemic effects have been reported from occupational exposure to LPG. The TLV-TWA is based on good hygiene practices and is thought to minimise the risk of fire or explosion.Odour Safety Factor(OSF)OSF=0.16 (hydrocarbon propellant)

For butane:Odour Threshold Value: 2591 ppm (recognition)Butane in common with other homologues in the straight chain saturated aliphatic hydrocarbon series is not characterised by its toxicity but by its narcosis-inducing effects at high concentrations.The TLV is based on analogy with pentane by comparing their lower explosive limits in air. It is concluded that this limit will protect workers against the significant risk of drowsiness and othernarcotic effects.Odour Safety Factor(OSF)OSF=0.22 (n-BUTANE)

For n-hexane:

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Odour Threshold Value: 65 ppmNOTE: Detector tubes for n-hexane, measuring in excess of 100 ppm, are available commercially.Occupational polyneuropathy may result from exposures as low as 500 ppm (as hexane), whilst nearly continuous exposures of 250 ppm have caused neurotoxic effects in animals. Many literaturereports have failed to distinguish hexane from n-hexane and on the assumption that the commercial hexane contains 30% n-hexane, a worst case recommendation for TLV is assumed to reduce therisk of peripheral neuropathies (due to the metabolites 2,5-heptanedione and 3,6-octanedione) and other adverse neuropathic effects.Concurrent exposure to chemicals (including MEK) and drugs which induce hepatic liver oxidative metabolism can reduce the time for neuropathy to appear.Odour Safety Factor(OSF)OSF=0.15 (n-HEXANE)

Odour Threshold Value for tetrachloroethylene (perchloroethylene): 47 ppm (detection), 71 ppm (recognition)NOTE: Detector tubes for perchloroethylene, measuring in excess of 10 ppm, are commercially available.Exposure at or below the TLV-TWA is thought to prevent discomfort and subjective complaints arising during controlled human studies and prolonged industrial exposure to perchloroethylene at100 ppm and 200 ppm.The STEL is recommended to minimise the potential of anaesthetic effects.Possible liver injury is unlikely as these values are thought to provide a wide margin of safety.On the basis of pharmacokinetic parameters and metabolic fate of perchloroethylene and in the absence of exposures which induce overt liver injury there is no expectation, by ACGIH, of adetectable increased risk of human liver cancerNOTE K: The classification as a carcinogen need not apply if it can be shown that the substance contains less than 0.1%w/w 1,3-butadiene (EINECS No 203-450-8). - European Union (EU)List of harmonised classification and labelling hazardous substances, Table 3.1, Annex VI, Regulation (EC) No 1272/2008 (CLP) - up to the latest ATP

Exposure controls

Appropriate engineeringcontrols

Engineering controls are used to remove a hazard or place a barrier between the worker and the hazard. Well-designed engineering controls can behighly effective in protecting workers and will typically be independent of worker interactions to provide this high level of protection.The basic types of engineering controls are:Process controls which involve changing the way a job activity or process is done to reduce the risk.Enclosure and/or isolation of emission source which keeps a selected hazard "physically" away from the worker and ventilation that strategically "adds" and"removes" air in the work environment. Ventilation can remove or dilute an air contaminant if designed properly. The design of a ventilation system mustmatch the particular process and chemical or contaminant in use.Employers may need to use multiple types of controls to prevent employee overexposure.

General exhaust is adequate under normal conditions. If risk of overexposure exists, wear SAA approved respirator. Correct fit is essential to obtainadequate protection.Provide adequate ventilation in warehouse or closed storage areas.Air contaminants generated in the workplace possess varying "escape" velocities which, in turn, determine the "capture velocities" of fresh circulating airrequired to effectively remove the contaminant.

Type of Contaminant: Speed:

aerosols, (released at low velocity into zone of active generation) 0.5-1 m/s

direct spray, spray painting in shallow booths, gas discharge (active generation into zone of rapid air motion) 1-2.5 m/s (200-500 f/min.)

Within each range the appropriate value depends on:

Lower end of the range Upper end of the range

1: Room air currents minimal or favourable to capture 1: Disturbing room air currents

2: Contaminants of low toxicity or of nuisance value only. 2: Contaminants of high toxicity

3: Intermittent, low production. 3: High production, heavy use

4: Large hood or large air mass in motion 4: Small hood-local control only

Simple theory shows that air velocity falls rapidly with distance away from the opening of a simple extraction pipe. Velocity generally decreases with thesquare of distance from the extraction point (in simple cases). Therefore the air speed at the extraction point should be adjusted, accordingly, afterreference to distance from the contaminating source. The air velocity at the extraction fan, for example, should be a minimum of 1-2 m/s (200-400 f/min.) forextraction of solvents generated in a tank 2 meters distant from the extraction point. Other mechanical considerations, producing performance deficits withinthe extraction apparatus, make it essential that theoretical air velocities are multiplied by factors of 10 or more when extraction systems are installed orused.

Personal protection

Eye and face protection

No special equipment for minor exposure i.e. when handling small quantities.OTHERWISE: For potentially moderate or heavy exposures:

Safety glasses with side shields. NOTE: Contact lenses pose a special hazard; soft lenses may absorb irritants and ALL lenses concentrate them. Close fitting gas tight goggles Safety glasses with side shields.Chemical goggles.Contact lenses may pose a special hazard; soft contact lenses may absorb and concentrate irritants. A written policy document, describing the wearingof lenses or restrictions on use, should be created for each workplace or task. This should include a review of lens absorption and adsorption for theclass of chemicals in use and an account of injury experience. Medical and first-aid personnel should be trained in their removal and suitable equipmentshould be readily available. In the event of chemical exposure, begin eye irrigation immediately and remove contact lens as soon as practicable. Lensshould be removed at the first signs of eye redness or irritation - lens should be removed in a clean environment only after workers have washed handsthoroughly. [CDC NIOSH Current Intelligence Bulletin 59], [AS/NZS 1336 or national equivalent]

Skin protection See Hand protection below

Hands/feet protection

Wear general protective gloves, eg. light weight rubber gloves.No special equipment needed when handling small quantities. OTHERWISE: For potentially moderate exposures: Wear general protective gloves, eg. light weight rubber gloves. For potentially heavy exposures: Wear chemical protective gloves, eg. PVC. and safety footwear.

Body protection See Other protection below

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Other protection

No special equipment needed when handling small quantities.OTHERWISE:

Overalls. Skin cleansing cream. Eyewash unit. Do not spray on hot surfaces. The clothing worn by process operators insulated from earth may develop static charges far higher (up to 100 times) than the minimum ignition energiesfor various flammable gas-air mixtures. This holds true for a wide range of clothing materials including cotton. Avoid dangerous levels of charge by ensuring a low resistivity of the surface material worn outermost.

BRETHERICK: Handbook of Reactive Chemical Hazards.

Thermal hazards Not Available

Recommended material(s)

GLOVE SELECTION INDEX

Glove selection is based on a modified presentation of the: "Forsberg Clothing Performance Index". The effect(s) of the following substance(s) are taken into account in the computer-generated selection: Electus Servisol Contact Clean & Lube

Material CPI

BUTYL C

CPE C

NEOPRENE C

NEOPRENE/NATURAL C

NITRILE C

NITRILE+PVC C

PE/EVAL/PE C

PVA C

PVC C

SARANEX-23 C

SARANEX-23 2-PLY C

TEFLON C

VITON C

VITON/CHLOROBUTYL C

VITON/NITRILE C

* CPI - Chemwatch Performance IndexA: Best SelectionB: Satisfactory; may degrade after 4 hours continuous immersionC: Poor to Dangerous Choice for other than short term immersionNOTE: As a series of factors will influence the actual performance of the glove, a finalselection must be based on detailed observation. -* Where the glove is to be used on a short term, casual or infrequent basis, factors such as"feel" or convenience (e.g. disposability), may dictate a choice of gloves which might otherwisebe unsuitable following long-term or frequent use. A qualified practitioner should be consulted.

Respiratory protection

Type AX Filter of sufficient capacity. (AS/NZS 1716 & 1715, EN 143:2000 & 149:2001, ANSIZ88 or national equivalent)

Where the concentration of gas/particulates in the breathing zone, approaches or exceeds the"Exposure Standard" (or ES), respiratory protection is required.Degree of protection varies with both face-piece and Class of filter; the nature of protectionvaries with Type of filter.

Required MinimumProtection Factor

Half-FaceRespirator

Full-FaceRespirator

Powered AirRespirator

up to 10 x ES AX-AUS -AX-PAPR-AUS /Class 1

up to 50 x ES -AX-AUS / Class1

-

up to 100 x ES - AX-2 AX-PAPR-2 ^

^ - Full-faceA(All classes) = Organic vapours, B AUS or B1 = Acid gasses, B2 = Acid gas or hydrogencyanide(HCN), B3 = Acid gas or hydrogen cyanide(HCN), E = Sulfur dioxide(SO2), G =Agricultural chemicals, K = Ammonia(NH3), Hg = Mercury, NO = Oxides of nitrogen, MB =Methyl bromide, AX = Low boiling point organic compounds(below 65 degC)

Cartridge respirators should never be used for emergency ingress or in areas of unknownvapour concentrations or oxygen content. The wearer must be warned to leave the contaminatedarea immediately on detecting any odours through the respirator. The odour may indicate thatthe mask is not functioning properly, that the vapour concentration is too high, or that the maskis not properly fitted. Because of these limitations, only restricted use of cartridge respirators isconsidered appropriate.

Generally not applicable.Aerosols, in common with most vapours/ mists, should never be used in confined spaceswithout adequate ventilation. Aerosols, containing agents designed to enhance or mask smell,have triggered allergic reactions in predisposed individuals.

SECTION 9 PHYSICAL AND CHEMICAL PROPERTIES

Information on basic physical and chemical properties

Appearance Clear extremely flammable liquid with solvent odour; does not mix with water.

Physical state Compressed Gas Relative density (Water = 1) Not Available

Odour Not AvailablePartition coefficient n-octanol /

waterNot Available

Odour threshold Not Available Auto-ignition temperature (°C) Not Available

pH (as supplied) Not Available Decomposition temperature Not Available

Melting point / freezing point(°C)

Not Available Viscosity (cSt) Not Available

Initial boiling point and boilingrange (°C)

Not Available Molecular weight (g/mol) Not Applicable

Flash point (°C) *-81 (propellant) Taste Not Available

Evaporation rate Not Available Explosive properties Not Available

Flammability HIGHLY FLAMMABLE. Oxidising properties Not Available

Upper Explosive Limit (%) Not AvailableSurface Tension (dyn/cm or

mN/m)Not Available

Lower Explosive Limit (%) Not Available Volatile Component (%vol) 100 (VOC)

Vapour pressure (kPa) Not Available Gas group Not Available

Solubility in water (g/L) Immiscible pH as a solution (1%) Not Available

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Vapour density (Air = 1) >1 VOC g/L Not Available

SECTION 10 STABILITY AND REACTIVITY

Reactivity See section 7

Chemical stability

Elevated temperatures. Presence of open flame. Product is considered stable. Hazardous polymerisation will not occur.

Possibility of hazardousreactions

See section 7

Conditions to avoid See section 7

Incompatible materials See section 7

Hazardous decompositionproducts

See section 5

SECTION 11 TOXICOLOGICAL INFORMATION

Information on toxicological effects

Inhaled

Inhalation of vapours may cause drowsiness and dizziness. This may be accompanied by narcosis, reduced alertness, loss of reflexes, lack of coordinationand vertigo.No health effects were seen in humans exposed at 1,000 ppm isobutane for up to 8 hours or 500 ppm for 8 hours/day for 10 days. Isobutane can haveanaesthetic and asphyxiant effects at high concentrations, well above the lower explosion limit of 1.8% (18,000 ppm).Butane is a simple asphyxiant and is mildly anaesthetic at high concentrations (20-25%). 10000 ppm for 10 minutes causes drowsiness.Narcotic effects may be accompanied by exhilaration, dizziness, headache, nausea, confusion, incoordination and unconsciousness in severe casesExposure to high concentrations of fluorocarbons may produce cardiac arrhythmias or cardiac arrest due sensitisation of the heart to adrenalin ornoradrenalin. Deaths associated with exposures to fluorocarbons (specifically halogenated aliphatics) have occurred in occupational settings and ininhalation of bronchodilator drugs.Bronchospasm consistently occurs in human subjects inhaling fluorocarbons. At a measured concentration of 1700 ppm of one of the commerciallyavailable aerosols there is a biphasic change in ventilatory capacity, the first reduction occurring within a few minutes and the second delayed up to 30minutes. Most subjects developed bradycardia (reduced pulse rate).Bradycardia is encountered in dogs when administration is limited to upper respiratory tract (oropharyngeal and nasal areas). Cardiac arrhythmias can beexperimentally induced in animals (species dependency is pronounced with dogs and monkeys requiring lesser amounts of fluorocarbon FC-11 than rats ormice). Sensitivity is increased by injection of adrenalin or cardiac ischaemia/necrosis or pulmonary thrombosis/bronchitis. The cardiotoxic effects of thefluorocarbons originate from irritation of the respiratory tract which in turn reflexively influences the heart rate (even prior to absorption of the fluorocarbon)followed by direct depression of the heart after absorption.Exposure to fluorocarbon thermal decomposition products may produce flu-like symptoms including chills, fever, weakness, muscular aches, headache,chest discomfort, sore throat and dry cough. Complete recovery usually occurs within 24 hours of exposure.The paraffin gases C1-4 are practically nontoxic below the lower flammability limit, 18,000 to 50,000 ppm; above this, low to moderate incidental effects suchas CNS depression and irritation occur, but are completely reversible upon cessation of the exposure.The vapour is discomfortingWARNING:Intentional misuse by concentrating/inhaling contents may be lethal.Inhalation hazard is increased at higher temperatures.Acute effects from inhalation of high concentrations of vapour are pulmonary irritation, including coughing, with nausea; central nervous system depression- characterised by headache and dizziness, increased reaction time, fatigue and loss of co-ordinationCentral nervous system (CNS) depression may include nonspecific discomfort, symptoms of giddiness, headache, dizziness, nausea, anaesthetic effects,slowed reaction time, slurred speech and may progress to unconsciousness. Serious poisonings may result in respiratory depression and may be fatal.Some aliphatic hydrocarbons produce axonal neuropathies. Isoparaffinic hydrocarbons produce injury to the kidneys of male rats. When albino rats wereexposed to isoparaffins at 21.4 mg/l for 4 hours, all animals experienced weakness, tremors, salivation, mild to moderate convulsions, chromodacryorrhoeaand ataxia within the first 24 hours. Symptoms disappeared after 24 hours.Several studies have evaluated sensory irritation in laboratory animals or odor or sensory response in humans. When evaluated by a standard procedure toassess upper airway irritation, isoparaffins did not produce sensory irritation in mice exposed to up to 400 ppm isoparaffin in air. Human volunteers wereexposed for six hours to 100 ppm isoparaffin. The subjects were given a self-administered questionnaire to evaluate symptoms, which included dryness ofthe mucous membranes, loss of appetite, nausea, vomiting, diarrhea, fatigue, headache, dizziness, feeling of inebriation, visual disturbances, tremor,muscular weakness, impairment of coordination or paresthesia. No symptoms associated with solvent exposure were observed. With a human expert panel,odour from liquid imaging copier emissions became weakly discernible at approximately 50 ppm. Numerous long-term exposures have been conducted in animals with only one major finding observed. Renal tubular damage has been found in kidneys ofmale rats upon repeated exposures to isoparaffins. It does not occur in mice or in female rats. This male rat nephropathy has been observed with a numberof hydrocarbons, including wholly vaporized unleaded gasoline. The phenomenon has been attributed to reversible binding of hydrocarbon to alpha2-globulin. Since humans do not synthesize alpha2-globulin or a similar protein, the finding is not considered to be of biological significance to man. Noclinically significant renal abnormalities have been found in refinery workers exposed to hydrocarbons.When evaluated for developmental toxicity in rats, isoparaffins were neither embryotoxic nor teratogenic. Isoparaffins were consistently negative on standardbacterial genotoxicity assays. They were also non-genotoxic in in vivo mammalian testing for somatic or germ cell mutations (mouse micronucleus test andrat dominant lethal assay, respectively).Mullin et al: Jnl Applied Toxicology 10, pp 136-142, 2006Acute intoxication by halogenated aliphatic hydrocarbons appears to take place over two stages. Signs of a reversible narcosis are evident in the first stageand in the second stage signs of injury to organs may become evident, a single organ alone is (almost) never involved.

Depression of the central nervous system is the most outstanding effect of most halogenated aliphatic hydrocarbons. Inebriation and excitation, passing intonarcosis, is a typical reaction. In severe acute exposures there is always a danger of death from respiratory failure or cardiac arrest due to a tendency tomake the heart more susceptible to catecholamines (adrenalin)

Material is highly volatile and may quickly form a concentrated atmosphere in confined or unventilated areas. The vapour may displace and replace air inbreathing zone, acting as a simple asphyxiant. This may happen with little warning of overexposure.Symptoms of asphyxia (suffocation) may include headache, dizziness, shortness of breath, muscular weakness, drowsiness and ringing in the ears. If theasphyxia is allowed to progress, there may be nausea and vomiting, further physical weakness and unconsciousness and, finally, convulsions, coma anddeath. Significant concentrations of the non-toxic gas reduce the oxygen level in the air. As the amount of oxygen is reduced from 21 to 14 volume %, thepulse rate accelerates and the rate and volume of breathing increase. The ability to maintain attention and think clearly is diminished and muscularcoordination is somewhat disturbed. As oxygen decreases from 14-10% judgement becomes faulty; severe injuries may cause no pain. Muscular exertionleads to rapid fatigue. Further reduction to 6% may produce nausea and vomiting and the ability to move may be lost. Permanent brain damage may resulteven after resuscitation at exposures to this lower oxygen level. Below 6% breathing is in gasps and convulsions may occur. Inhalation of a mixturecontaining no oxygen may result in unconsciousness from the first breath and death will follow in a few minutes.Accidental high level exposure to tetrachloroethylene (perchloroethylene) has produced lightheadedness, unconsciousness and liver and kidney damage in

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workers. In at least two cases such exposures were fatal. Sudden death resulting from acute exposure to anesthetic vaporconcentrations is presumed to result from either excessive depression of the respiratory center or the onset of a fatal cardiac arrhythmia induced byepinephrine sensitisation.The nervous system is a major target organ in humans exposed to tetrachloroethylene by inhalation.Acute exposure, depending on concentration, can result in reversible mood and behavioral changes,impairment of coordination, or anesthetic effects. The anesthetic and preanesthetic central nervous system effects of tetrachloroethylene were documentedin four volunteer subjects (one male, sex not specified for the other three) exposed acutely to concentrations ranging from 500 to 2,000 ppm. Mood changes,slight ataxia, faintness, and dizziness occurred with exposure to concentrations of l,000-1,500 ppm for <2 hours. At exposure to 2,000 ppm for 5-7 minutes,subjects experienced a sensation of impending collapse. Slight lightheadedness was reported by six male volunteers exposed to tetrachloroethylene at aconcentration of 210-240 ppm for over 30 minutes. Dizziness has been reported after brief accidental exposure to high concentrations of tetrachloroethylenefumes while longer exposures resulted in collapse, coma, and seizures. There have been several experimental studies of acute neurological effects in adulthumans exposed to tetrachloroethylene. In one study, symptoms of neurological impairment were not reported after exposure to 106 ppm for 1 hour.Neurological symptoms of dizziness and drowsiness occurred at exposure to 216 ppm for 45 minutes to 2 hours; loss of motor coordination occurred atexposure to 280 ppm for 2 hours or 600 ppm for 10 minutes. Volunteers in this study were allowed to leave the exposure chamber when they experienceddiscomfort. Consequently, exposure times varied and ranged from 3 minutes to 2 hours. In another study, exposure to 100 ppm for 7 hours producedsymptoms such as headache, dizziness, difficulty in speaking, and sleepiness. Of five objective tests of central nervous systemperformance in humans exposed for 7 hours, none showed any abnormality except the Romberg test of balance, which was abnormal for three of the fivesubjects exposed for 7 hours a day for 5 consecutive days. Only one exposure level (100 ppm) was used in this study.Subjects exposed to 106 ppm in laboratory studies experienced slight eye irritation; dizziness and sleepiness were reported at 216 ppm; at exposures of 280ppm or 600 ppm for 10 minutes there was a loss of motor coordination. In another study subjects exposed for 7 hours at 101 ppm complained of eye irritationand subjective symptoms such headache, drowsiness and sleepiness.Intense irritation of the upper respiratory tract was reported in volunteers exposed to high concentrations (>l,000 ppm) of tetrachloroethylene.The liver is a target organ in humans accidentally exposed to high concentrations of tetrachloroethylene. Hepatocellular damage was documented by biopsyin a case study of a womanexposed occupationally to tetrachloroethylene fumes for 2.5 months. Liver damage also has been diagnosed by the presence of hepatomegaly, icterus, andelevations of serum glutamic oxaloacetic transaminase (SGOT), bilirubin, and urinary urobilinogen. These effects were generally observed several daysafter an acute exposure resulting in nervous system effects.Hepatic lesions are also induced in experimental animals during inhalation exposure to tetrachloroethylene. Mice are the species most sensitive to thiseffect. Hepatocellular vacuolizationoccurred after a single 4-hour exposure of mice to 200 ppm or greater concentrations of tetrachloroethylene.

Inhalation of vapours or aerosols (mists, fumes), generated by the material during the course of normal handling, may be damaging to the health of theindividual.

Ingestion

Not normally a hazard due to physical form of product.Considered an unlikely route of entry in commercial/industrial environments Swallowing of the liquid may cause aspiration of vomit into the lungs with the risk of haemorrhaging, pulmonary oedema, progressing to chemicalpneumonitis; serious consequences may result.Signs and symptoms of chemical (aspiration) pneumonitis may include coughing, gasping, choking, burning of the mouth, difficult breathing, and bluishcoloured skin (cyanosis).Many aliphatic hydrocarbons create a burning sensation because they are irritating to the GI mucosa. Vomiting has been reported in up to one third of allhydrocarbon exposures. While most aliphatic hydrocarbons have little GI absorption, aspiration frequently occurs, either initially or in a semi-delayedfashion as the patient coughs or vomits, thereby resulting in pulmonary effects. Once aspirated, the hydrocarbons can create a severe pneumonitis.Rats given isoparaffinic hydrocarbons (after 18-24 hours fasting) showed lethargy and/or general weakness, ataxia and diarrhoea. Symptoms disappearedwithin 24-28 hours.When tetrachloroethylene (perchloroethylene) is used in the treatment of hookworm (4.5 to 6.5 gm orally) the only adverse effect is inebriation. Transienthepatotoxicity in patients given single oral doses of up to 5 ml have been recorded.

Skin Contact

The material may accentuate any pre-existing dermatitis conditionDermally, isoparaffins have produced slight to moderate irritation in animals and humans under occluded patch conditions where evaporation cannot freelyoccur. However, they are not irritating in non-occluded tests, which are a more realistic simulation of human exposure. They have not been found to besensitisers in guinea pig or human patch testing. However, occasional rare idiosyncratic sensitisation reactions in humans have been reported.Spray mist may produce discomfort

In common with other halogenated aliphatics, fluorocarbons may cause dermal problems due to a tendency to remove natural oils from the skin causingirritation and the development of dry, sensitive skin. They do not appear to be appreciably absorbed.Open cuts, abraded or irritated skin should not be exposed to this materialToxic effects may result from skin absorptionEntry into the blood-stream through, for example, cuts, abrasions, puncture wounds or lesions, may produce systemic injury with harmful effects. Examinethe skin prior to the use of the material and ensure that any external damage is suitably protected.Industrial experience shows exposure to tetrachloroethylene (perchloroethylene) produces localised skin irritation. Prolonged dermal contact can causechemical burns and blistering.

The material produces severe skin irritation; evidence exists, or practical experience predicts, that the material either: produces severe inflammation of the skin in a substantial number of individuals following direct contact, and/or produces significant and severe inflammation when applied to the healthy intact skin of animals (for up to four hours), such inflammation being presenttwenty-four hours or more after the end of the exposure period. Skin irritation may also be present after prolonged or repeated exposure; this may result in a form of contact dermatitis (nonallergic). The dermatitis isoften characterised by skin redness (erythema) and swelling (oedema) which may progress to blistering (vesiculation), scaling and thickening of theepidermis. At the microscopic level there may be intercellular oedema of the spongy layer of the skin (spongiosis) and intracellular oedema of theepidermis.

NOTE: Prolonged contact is unlikely, given the severity of response, but repeated exposures may produce severe ulceration.

Eye

Although the material is not thought to be an irritant (as classified by EC Directives), direct contact with the eye may produce transient discomfortcharacterised by tearing or conjunctival redness (as with windburn).Instillation of isoparaffins into rabbit eyes produces only slight irritation.Direct contact with the eye may not cause irritation because of the extreme volatility of the gas; however concentrated atmospheres may produce irritationafter brief exposures..Intense irritation of the eyes of humans was noted following acute exposure to highconcentrations (930 ppm) of tetrachloroethylene vapors. Burning or stinging sensations in the eyes occurred after exposure to 600 or 280 ppm; very mildirritation was reported by four subjects at exposure to 216 or 106 ppm; and transient eye irritation was noted in six subjects during the first few minutes ofexposure at 75-80 ppm.Although no loss of color vision was noted in workers exposed to tetrachloroethylene at averageconcentrations of 15.3 and 10.7 ppm for men and women, respectively , loss of color vision was reported in dry cleaners exposed to an averageconcentration of 7.3 ppm for an average of 106 months. This effect may be a neurological effect rather than a direct effect on the eyes,

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Chronic

On the basis, primarily, of animal experiments, concern has been expressed that the material may produce carcinogenic or mutagenic effects; in respect ofthe available information, however, there presently exists inadequate data for making a satisfactory assessment.Harmful: danger of serious damage to health by prolonged exposure through inhalation. Serious damage (clear functional disturbance or morphological change which may have toxicological significance) is likely to be caused by repeated orprolonged exposure. As a rule the material produces, or contains a substance which produces severe lesions. Such damage may become apparentfollowing direct application in subchronic (90 day) toxicity studies or following sub-acute (28 day) or chronic (two-year) toxicity tests.Exposure to the material may cause concerns for human fertility, generally on the basis that results in animal studies provide sufficient evidence to cause astrong suspicion of impaired fertility in the absence of toxic effects, or evidence of impaired fertility occurring at around the same dose levels as other toxiceffects, but which are not a secondary non-specific consequence of other toxic effects.Halogenated oxiranes may arise following epoxidation of haloalkenes.The metabolism of haloethylenes by microsomal oxidation leading to epoxide formation across the double bond has been proposed. The resulting oxiranesare highly reactive and may covalently bind to nucleic acids leading to mutations and possible cancers A measure of such potential carcinogenicity is thedevelopment of significant preneoplastic foci in livers of treated rats.The carcinogenicity of halogenated oxiranes may lie in the reactivity of an epoxide intermediate. It is reported that 1,1-dichloroethylene, vinyl chloride,trichloroethylene, tetrachloroethylene and chloroprene, for example, are carcinogens in vivo - this may be a consequence of oxirane formation.Symmetrically substituted oxiranes such as 1,2-dichloroethylene and 1,1,2-2-tetrachloroethylene are more stable and less mutagenic than unsymmetricalchlorinated oxiranes such as 1,1-dichloroethylene, 1,1,2-trichloroethylene and monochloroethylene (vinyl chloride).The carcinogenicity of 1,1-dichloroethylene has primarily been associated with inhalation exposure while that of vinyl chloride, trichloroethylene andtetrachloroethylene occurs following exposure by both inhalation and oral routes. National Toxicology Program Toxicity Report Series Number 55; April2002 Various studies report an association between cancer and industrial exposure to tetrachloroethylene; IARC concluded that this evidence is sufficient toassign appropriate warnings. Similar warnings have been issued by IARC for vinyl fluoride. Similarly vinyl bromide exhibited neoplastic and tumourigenic activity in rats exposed by inhalation and is classified by various bodies as potentially carcinogenic.Substances such as chloroprene (2-chloro-1,3-butadiene), are reported to produce an increased frequency of chromosomal aberrations in the lymphocytesof Russian workers. Russian epidemiological studies also suggest an increased incidence of skin and lung cancer following exposure to chloroprene, aresult which is not supported by other studies.Generally speaking, the monohalogenated substances exhibit higher carcinogenic potential than their dihalogenated counterparts. Whether additionalsubstitution lessens such hazard is conjectural. Tetrafluoroethylene, for example, produced clear evidence of carcinogenic activity in a two-year inhalationstudy in rats and mice. National Toxicology Program Technical Report Series 450, April 1997Principal route of occupational exposure to the gas is by inhalation.

Chronic inhalation or skin exposure to n-hexane may cause peripheral neuropathy, which is damage to nerve ends in extremities, e.g. fingers, with loss ofsensation and characteristic thickening. Nerve damage has been documented with chronic exposures of greater than 500 ppm. Improvement in conditiondoes not immediately follow removal from exposure and symptoms may progress for two or three months. Recovery may take a year or more depending onseverity of exposure, and may not always be complete. Exposure to n-hexane with methyl ethyl ketone (MEK) will accelerate the appearance of damage, butMEK alone will not cause the nerve damage. Other isomers of hexane do not cause nerve damage. [Source: Shell Co.]Workers inhaling 232 to 385 ppm tetrachloroethylene (perchloroethylene) for 8 hours/day, 5 days/week for 2 to 6 years have shown abnormal hepaticfunction, including cirrhosis, with lightheadedness, headache, malaise and dizziness.Some adverse reproductive effects in women have been reported to be associated with occupational exposure to tetrachloroethylene in dry cleaningoperations. These effects include menstrual disorders and spontaneous abortion.Epidemiological studies of dry cleaning and laundry workers have shown significant excesses in mortality resulting from cancers of the lung, cervix,esophagus, kidney, skin, lymphatic/hematopoietic system, and/or colon .A small excess risk of liver cancer was found among female (7 observed, 2.1expected) but not male laundry and dry cleaning workers. A nested-case-control study of this population, which distinguished between laundry and drycleaning workers, indicated that all of the liver cancers were found among laundry workers. Renal cell carcinoma was also not increased among drycleaners relative to laundry workers. The investigators concluded that the excess risk of liver cancer among women working in laundries and dry cleaningshops was not explained by exposure to dry cleaning solvents.Results relevant to assessing the relationship between exposure to tetrachloroethylene and cancer risk are available from five cohort studies. Although dataon different levels or duration of exposure were available in some studies the number of observed cases of various cancers were generally too small to allowadequate statistical power for testing a dose-response relationship. Elevated relative risk for non-Hodgkins lymphoma were reported in three cohortstudies. IARC concluded that thereis evidence for consistently positive associations between exposure to tetrachloroethylene and the risks for oesophageal and cervical cancer andnon-Hodgkin's lymphoma. These associations appear unlikely to be due to chance, although confounding factors cannot be excluded and the total numbersin the cohort study are relatively small

Electus Servisol Contact Clean& Lube

TOXICITY IRRITATION

Not Available Not Available

n-hexane

TOXICITY IRRITATION

Dermal (rabbit) LD50: >2000 mg/kg[1] Eye(rabbit): 10 mg - mild

Inhalation (rat) LC50: 47945.232 mg/l/4H[2]

Oral (rat) LD50: 28710 mg/kg[2]

perchloroethylene

TOXICITY IRRITATION

Dermal (rabbit) LD50: >10000 mg/kg[1] Eye (rabbit): 162 mg -mild

Inhalation (mouse) LC50: 30.3 mg/l/6H[2] Skin (rabbit): 810 mg/24h -SEVERE

Oral (rat) LD50: 2629 mg/kg[2]

mineral oilTOXICITY IRRITATION

Not Available Not Available


TOXICITY IRRITATION

Inhalation (rat) LC50: 84.684 mg/l15 min[1] Not Available

Inhalation (rat) LC50: 90.171125 mg/l15 min[1]

Legend: 1. Value obtained from Europe ECHA Registered Substances - Acute toxicity 2.* Value obtained from manufacturer's SDS. Unless otherwise specifieddata extracted from RTECS - Register of Toxic Effect of chemical Substances

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Legend: – Data available but does not fill the criteria for classification – Data available to make classification

– Data Not Available to make classification

PERCHLOROETHYLENE

Disinfection by products (DBPs) re formed when disinfectants such as chlorine, chloramine, and ozone react with organic and inorganic matter in water.The observations that some DBPs such as trihalomethanes (THMs), di-/trichloroacetic acids, and 3-chloro-4-(dichloromethyl)-5-hydroxy-2(5H)-furanone(MX) are carcinogenic in animal studies have raised public concern over the possible adverse health effects of DBPs. To date, several hundred DBPs havebeen identified.Numerous haloalkanes and haloalkenes have been tested for carcinogenic and mutagenic activities. n general, the genotoxic potential is dependent on thenature, number, and position of halogen(s) and the molecular size of the compound. Short-chain monohalogenated (excluding fluorine) alkanes and alkenesare potential direct-acting alkylating agents, particularly if the halogen is at the terminal end of the carbon chain or at an allylic position. Dihalogenatedalkanes are also potential alkylating or cross-linking agents (either directly or after GSH conjugation),particularly if they are vicinally substituted (e.g.,1,2-dihaloalkane) or substituted at the two terminal ends of a short to medium-size (e.g., 2-7) alkyl moiety (i.e., alpha, omega-dihaloalkane). Fullyhalogenated haloalkanes tend to act by free radical or nongenotoxic mechanisms (such as generating peroxisome-proliferative intermediates) or undergoreductive dehalogenation to yield haloalkenes that in turn could be activated to epoxides.Haloalkenes are of concern because of potential to generate genotoxic intermediates after epoxidation. The concern for haloalkenes may be diminished ifthe double bond is internal or sterically hindered.The cancer concern levels of the 14 haloalkanes and haloalkenes, have been rated based on available screening cancer bioassay (pulmonary adenomaassay) and genotoxicity data. Five brominated and iodinated methane and ethane derivatives are given a moderate rating. Beyond the fact that bromine andiodine are better leaving groups than chlorine, there is also evidence that brominated THMs may be preferentially activated by a theta-class glutathioneS-transferase (GSTT1-1) to mutagens in Salmonella even at low substrate concentrations Furthermore, there are human carcinogenicity implicationsbecause of polymorphism in GSTT1-1. Human subpopulations with expressed GSTT1-1 may be at a greater risk to brominate THMs than humans who lackthe gene.Six, two, and one haloalkanes/ haloalkene(s) are given low-moderate, marginal, and low concern, respectively.The material may be irritating to the eye, with prolonged contact causing inflammation. Repeated or prolonged exposure to irritants may produceconjunctivitis.The material may produce severe skin irritation after prolonged or repeated exposure, and may produce a contact dermatitis (nonallergic). This form ofdermatitis is often characterised by skin redness (erythema) thickening of the epidermis. Histologically there may be intercellular oedema of the spongy layer (spongiosis) and intracellular oedema of the epidermis. Prolonged contact is unlikely,given the severity of response, but repeated exposures may produce severe ulceration.

WARNING: This substance has been classified by the IARC as Group 2A: Probably Carcinogenic to Humans.

MINERAL OIL

Toxicity and Irritation data for petroleum-based mineral oils are related to chemical components and vary as does the composition and source of the originalcrude.A small but definite risk of occupational skin cancer occurs in workers exposed to persistent skin contamination by oils over a period of years. This risk hasbeen attributed to the presence of certain polycyclic aromatic hydrocarbons (PAH) (typified by benz[a]pyrene).Petroleum oils which are solvent refined/extracted or severely hydrotreated, contain very low concentrations of both.

HYDROCARBONPROPELLANT

No significant acute toxicological data identified in literature search. for Petroleum Hydrocarbon Gases:In many cases, there is more than one potentially toxic constituent in a refinery gas. In those cases, the constituent that is most toxic for a particular endpointin an individual refinery stream is used to characterize the endpoint hazard for that stream. The hazard potential for each mammalian endpoint for each ofthe petroleum hydrocarbon gases is dependent upon each petroleum hydrocarbon gas constituent endpoint toxicity values (LC50, LOAEL, etc.) and therelative concentration of the constituent present in that gas. It should also be noted that for an individual petroleum hydrocarbon gas, the constituentcharacterizing toxicity may be different for different mammalian endpoints, again, being dependent upon the concentration of the different constituents ineach, distinct petroleum hydrocarbon gas.All Hydrocarbon Gases Category members contain primarily hydrocarbons (i.e., alkanes and alkenes) and occasionally asphyxiant gases like hydrogen.The inorganic components of the petroleum hydrocarbon gases are less toxic than the C1 - C4 and C5 - C6 hydrocarbon components to both mammalianand aquatic organisms. Unlike other petroleum product categories (e.g. gasoline, diesel fuel, lubricating oils, etc.), the inorganic and hydrocarbonconstituents of hydrocarbon gases can be evaluated for hazard individually to then predict the screening level hazard of the Category membersAcute toxicity: No acute toxicity LC50 values have been derived for the C1 -C4 and C5- C6 hydrocarbon (HC) fractions because no mortality wasobserved at the highest exposure levels tested (~ 5 mg/l) for these petroleum hydrocarbon gas constituents. The order of acute toxicity of petroleumhydrocarbon gas constituents from most to least toxic is:C5-C6 HCs (LC50 > 1063 ppm) > C1-C4 HCs (LC50 > 10,000 ppm) > benzene (LC50 = 13,700 ppm) > butadiene (LC50 = 129,000 ppm) > asphyxiantgases (hydrogen, carbon dioxide, nitrogen).Repeat dose toxicity: With the exception of the asphyxiant gases, repeated dose toxicity has been observed in individual selected petroleum hydrocarbongas constituents. Based upon LOAEL values, the order of order of repeated-dose toxicity of these constituents from most toxic to the least toxic is:Benzene (LOAEL .>=10 ppm) >C1-C4 HCs (LOAEL = 5,000 ppm; assumed to be 100% 2-butene) > C5-C6 HCs (LOAEL = 6,625 ppm) > butadiene(LOAEL = 8,000 ppm) > asphyxiant gases (hydrogen, carbon dioxide, nitrogen).Genotoxicity:In vitro: The majority of the Petroleum Hydrocarbon Gases Category components are negative for in vitro genotoxicity. The exceptions are: benzene and1,3-butadiene, which are genotoxic in bacterial and mammalian in vitro test systems.In vivo: The majority of the Petroleum Hydrocarbon Gases Category components are negative for in vivo genotoxicity. Theexceptions are benzene and 1,3-butadiene, which are genotoxic in in vivo test systemsDevelopmental toxicity: Developmental effects were induced by two of the petroleum hydrocarbon gas constituents, benzene and the C5 -C6 hydrocarbonfraction. No developmental toxicity was observed at the highest exposure levels tested for the other petroleum hydrocarbon gas constituents tested for thiseffect. The asphyxiant gases have not been tested for developmental toxicity. Based on LOAEL and NOAEL values, the order of acute toxicity of theseconstituents from most to least toxic is:Benzene (LOAEL = 20 ppm) > butadiene (NOAEL .>=1,000 ppm) > C5-C6 HCs (LOAEL = 3,463 ppm) > C1-C4 HCs (NOAEL >=5,000 ppm; assumed tobe 100% 2-butene) > asphyxiant gases (hydrogen, carbon dioxide, nitrogen).Reproductive toxicity: Reproductive effects were induced by only two petroleum hydrocarbon gas constituents, benzene and isobutane (a constituent ofthe the C1-C4 hydrocarbon fraction). No reproductive toxicity was observed at the highest exposure levels tested for the other petroleum hydrocarbon gasconstituents tested for this effect. The asphyxiant gases have not been tested for reproductive toxicity. Based on LOAEL and NOAEL values, the order ofreproductive toxicity of these constituents from most to least toxic is:Benzene (LOAEL = 300 ppm) > butadiene (NOAEL .>=6,000 ppm) > C5-C6 HCs (NOAEL .>=6,521 ppm) > C1-C4 HCs (LOAEL = 9,000 ppm; assumedto be 100% isobutane) > asphyxiant gases (hydrogen, carbon dioxide, nitrogen)

Acute Toxicity Carcinogenicity

Skin Irritation/Corrosion Reproductivity

Serious Eye Damage/Irritation STOT - Single Exposure

Respiratory or Skinsensitisation

STOT - Repeated Exposure

Mutagenicity Aspiration Hazard

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SECTION 12 ECOLOGICAL INFORMATION

Toxicity

Electus Servisol Contact Clean& Lube

ENDPOINT TEST DURATION (HR) SPECIES VALUE SOURCE

NotAvailable

Not Available Not AvailableNotAvailable

NotAvailable

n-hexane


LC50 96 Fish 2.5mg/L 4

EC50 48 Crustacea 3877.65mg/L 4

perchloroethylene


LC50 96 Fish 4mg/L 4

EC50 48 Crustacea 2.49mg/L 5

EC50 72 Algae or other aquatic plants ~0.2mg/L 4

BCF 240 Fish 350mg/L 4

NOEC 168 Crustacea 0.33mg/L 5

mineral oil


NotAvailable


NotAvailable



NotAvailable


NotAvailable

Legend: Extracted from 1. IUCLID Toxicity Data 2. Europe ECHA Registered Substances - Ecotoxicological Information - Aquatic Toxicity 3. EPIWIN Suite V3.12(QSAR) - Aquatic Toxicity Data (Estimated) 4. US EPA, Ecotox database - Aquatic Toxicity Data 5. ECETOC Aquatic Hazard Assessment Data 6. NITE(Japan) - Bioconcentration Data 7. METI (Japan) - Bioconcentration Data 8. Vendor Data

Toxic to aquatic organisms. Do NOT allow product to come in contact with surface waters or to intertidal areas below the mean high water mark. Do not contaminate water when cleaning equipment or disposing of equipmentwash-waters. Wastes resulting from use of the product must be disposed of on site or at approved waste sites.May cause long-term adverse effects in the aquatic environment. DO NOT discharge into sewer or waterways.

Persistence and degradability

Ingredient Persistence: Water/Soil Persistence: Air

n-hexane LOW LOW

perchloroethylene HIGH (Half-life = 720 days) MEDIUM (Half-life = 160.13 days)

Bioaccumulative potential

Ingredient Bioaccumulation

n-hexane MEDIUM (LogKOW = 3.9)

perchloroethylene LOW (BCF = 77.1)

Mobility in soil

Ingredient Mobility

n-hexane LOW (KOC = 149)

perchloroethylene LOW (KOC = 106.8)

SECTION 13 DISPOSAL CONSIDERATIONS

Waste treatment methods

Product / Packaging disposal

DO NOT allow wash water from cleaning or process equipment to enter drains. It may be necessary to collect all wash water for treatment before disposal. In all cases disposal to sewer may be subject to local laws and regulations and these should be considered first. Where in doubt contact the responsible authority. Consult State Land Waste Management Authority for disposal. Discharge contents of damaged aerosol cans at an approved site. Allow small quantities to evaporate. DO NOT incinerate or puncture aerosol cans. Bury residues and emptied aerosol cans at an approved site.

SECTION 14 TRANSPORT INFORMATION

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Labels Required

Marine Pollutant

HAZCHEM 2Y

Land transport (ADG)

UN number 1950

UN proper shipping name AEROSOLS

Transport hazard class(es)Class 2.1

Subrisk Not Applicable

Packing group Not Applicable

Environmental hazard Environmentally hazardous

Special precautions for userSpecial provisions 63 190 277 327 344

Limited quantity 1000ml

Air transport (ICAO-IATA / DGR)

UN number 1950

UN proper shipping name Aerosols, flammable; Aerosols, flammable (engine starting fluid)

Transport hazard class(es)

ICAO/IATA Class 2.1

ICAO / IATA Subrisk Not Applicable

ERG Code 10L


Environmental hazard Environmentally hazardous

Special precautions for user

Special provisions A145 A167 A802; A1 A145 A167 A802

Cargo Only Packing Instructions 203

Cargo Only Maximum Qty / Pack 150 kg

Passenger and Cargo Packing Instructions 203; Forbidden

Passenger and Cargo Maximum Qty / Pack 75 kg; Forbidden

Passenger and Cargo Limited Quantity Packing Instructions Y203; Forbidden

Passenger and Cargo Limited Maximum Qty / Pack 30 kg G; Forbidden

Sea transport (IMDG-Code / GGVSee)

UN number 1950

UN proper shipping name AEROSOLS

Transport hazard class(es)IMDG Class 2.1

IMDG Subrisk Not Applicable


Environmental hazard Marine Pollutant

Special precautions for user

EMS Number F-D, S-U

Special provisions 63 190 277 327 344 381 959

Limited Quantities 1000ml

Transport in bulk according to Annex II of MARPOL and the IBC code

Not Applicable

SECTION 15 REGULATORY INFORMATION

Safety, health and environmental regulations / legislation specific for the substance or mixture

N-HEXANE(110-54-3) IS FOUND ON THE FOLLOWING REGULATORY LISTS

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Australia Exposure Standards

Australia Hazardous Substances Information System - Consolidated Lists

Australia Inventory of Chemical Substances (AICS)

PERCHLOROETHYLENE(127-18-4) IS FOUND ON THE FOLLOWING REGULATORY LISTS




International Agency for Research on Cancer (IARC) - Agents Classified by the IARCMonographs

MINERAL OIL(NOT AVAIL.) IS FOUND ON THE FOLLOWING REGULATORY LISTS



International Agency for Research on Cancer (IARC) - Agents Classified by the IARCMonographs

HYDROCARBON PROPELLANT(68476-85-7.) IS FOUND ON THE FOLLOWING REGULATORY LISTS




International Air Transport Association (IATA) Dangerous Goods Regulations - Prohibited ListPassenger and Cargo Aircraft

National Inventory Status

Australia - AICS N (mineral oil)

Canada - DSL N (mineral oil)

Canada - NDSL N (perchloroethylene; hydrocarbon propellant; n-hexane; mineral oil)

China - IECSC N (mineral oil)

Europe - EINEC / ELINCS / NLP N (mineral oil)

Japan - ENCS N (hydrocarbon propellant; mineral oil)

Korea - KECI N (mineral oil)

New Zealand - NZIoC N (mineral oil)

Philippines - PICCS N (mineral oil)

USA - TSCA N (mineral oil)

Legend:Y = All ingredients are on the inventoryN = Not determined or one or more ingredients are not on the inventory and are not exempt from listing(see specific ingredients in brackets)

SECTION 16 OTHER INFORMATION

Other information

Ingredients with multiple cas numbers

Name CAS No

hydrocarbon propellant 68476-85-7., 68476-86-8.

Classification of the preparation and its individual components has drawn on official and authoritative sources as well as independent review by the Chemwatch Classification committee usingavailable literature references.

The SDS is a Hazard Communication tool and should be used to assist in the Risk Assessment. Many factors determine whether the reported Hazards are Risks in the workplace or othersettings. Risks may be determined by reference to Exposures Scenarios. Scale of use, frequency of use and current or available engineering controls must be considered.

Definitions and abbreviations

PC－TWA: Permissible Concentration-Time Weighted AveragePC－STEL: Permissible Concentration-Short Term Exposure LimitIARC: International Agency for Research on CancerACGIH: American Conference of Governmental Industrial HygienistsSTEL: Short Term Exposure LimitTEEL: Temporary Emergency Exposure Limit。IDLH: Immediately Dangerous to Life or Health ConcentrationsOSF: Odour Safety FactorNOAEL :No Observed Adverse Effect LevelLOAEL: Lowest Observed Adverse Effect LevelTLV: Threshold Limit ValueLOD: Limit Of DetectionOTV: Odour Threshold ValueBCF: BioConcentration FactorsBEI: Biological Exposure Index

This document is copyright.Apart from any fair dealing for the purposes of private study, research, review or criticism, as permitted under the Copyright Act, no part may be reproduced by any process without writtenpermission from CHEMWATCH.TEL (+61 3) 9572 4700.

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end of SDS

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