EKG Findings and Arrhythmias

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EKG findings and Arrhythmias Heart Blocks: First-degree AV block – normal sinus rhythm with PR interval ³ 0.2ms Second-degree, type 1 (Weckenbach) block – PR interval elongates from beat to beat until a PR is dropped Second-degree, type 2 (Mobitz) block – PR interval fixed but there are regular non-conducted P-waves leading to dropped beats Third-degree block – no relationship between P waves and QRS complexes. Presents with junctional escape rhythms or ventricular escape rhythm  Atrial Fibrillation The most common chronic arrhythmia From ischemia, atrial dilatation, surgical history, pulmonary diseases, toxic syndromes Classically, the pulse is irregularly irregular  Signs and Symptoms: Chest discomfort Palpitations Tachycardia, Hypotension + syncope

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EKG findings and Arrhythmias

Heart Blocks:

First-degree AV block – normal sinus rhythm with PR interval ³ 0.2ms

Second-degree, type 1 (Weckenbach) block – PR interval elongates from beat to beat until a PR is dropped

Second-degree, type 2 (Mobitz) block – PR interval fixed but there are regular non-conducted P-waves leading

to dropped beats

Third-degree block – no relationship between P waves and QRS complexes. Presents with junctional escape

rhythms or ventricular escape rhythm

 

Atrial Fibrillation

• The most common chronic arrhythmia

• From ischemia, atrial dilatation, surgical history, pulmonary diseases, toxic syndromes

• Classically, the pulse is irregularly irregular

 

Signs and Symptoms:

Chest discomfort• Palpitations

• Tachycardia,

• Hypotension + syncope

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Treatment :

• Control rate with b-blockers, CCB’s, and digoxin (not acutely)

• If fibrillations last >24hr then should anticoagulate with warfarin for at least 3 weeks before

cardioversion (prevents embolisms)

• If you cannot convert to normal sinus rhythm, the patient will require long-term anticoagulation.1st line is warfarin, 2nd line is aspirin

Cardioversion to convert to normal rhythm:

1st line – IV procainamide, sotalol, amiodarone

Electrical à shock of 100-200J followed by 360J

 

Atrial Flutter

• Less stable than Afib • The rate is slower than that of atrial fibrillation (approximately 250-350bpm) 

• Ventricular rate in atrial flutter is at risk of going too fast, thus atrial flutter is considered to be moredangerous (medically slowing this rate can cause a paradoxical increase in ventricular rates) 

• Classic rhythm is an atrial flutter rate of 300bpm with a 2:1 block resulting in a ventricular rate of 150bpm 

• Signs and symptoms similar to those of atrial fibrillation 

• Complications include syncope, embolization, ischemia, heart failure 

Classic EKG finding is a “sawtooth” pattern:

 

Treatment :

• If patient is stable, slow the ventricular rate with CCB’s or b-blockers (avoid procainamide because itcan result in increased ventricular rate as the atrial rate slows down)

• If cardioversion is going to take place be sure to anticoagulate for 3 weeks

• If patient is unstable must cardiovert à start at only 50J because is easier to convert to normal sinusrhythm than atrial fibrillation

 

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Multifocal Atrial Tachycardia (MFAT)

• An irregularly irregular rhythm where there are multiple concurrent pacemakers in the atria.

• Commonly found in pts with COPD

EKG shows tachycardia with ³ 3 distinct P waves

 

Treatment :

• Verapamil

• Treat any underlying condition

 

Supraventricular Tachycardia

• Many tachyarrhythmias originating above the ventricle

• Pacemaker may be in atrium or AV junction, having multiple pacemakers active at any one time

• Differentiating from ventricular arrhythmia may be difficult if there is also the presence of a bundlebranch block

Treatment :

• Very dependent on etiology

• May need to correct electrolyte imbalance

• May need to correct ventricular rate [digoxin, CCB, b-blockers, adenosine (breaks 90% of SVT)]

• If unstable requires cardioversion

• Carotid massage if patient has paroxysmal SVT

 

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Ventricular Tachycardia

• VTach is defined as ³ 3 consecutive premature ventricular contractions

• If sustained, the tachycardic periods last a minimum of 30s.

• Sustained tachycardia requires immediate cardioversion due to risk of going into ventricular fibrillation

 

Treatment :

• If hypotensive or no pulse existent do emergency defibrillation (200, then 300, then 360J)

• If patient is asymptomatic and not hypotensive, the first line treatment is amiodarone or lidocainebecause it can convert rhythm back to normal

 

Ventricular Fibrillation

• Erratic ventricular rhythm is a fatal condition.

• Has no rhyme or rhythm

 

Signs and Symptoms:

• Syncope

• Severe hypotension

• Sudden death

Treatment :• 1st line – Emergent cardioversion is the primary therapy (200-300-360J), which converts to normalrhythm almost 95% of the time

• Chest compressions rarely work

• 2nd line – Amiodarone or lidocaine

If treatment isn’t given in a timely matter, patient experiences failure of cardiac output and this progresses to

death.