DY-MARK ZINC-GAL GALVANISING ZINC RICH COATING ……Issue Date: 3-Oct-2011 CHEMWATCH 4736-52...

DY-MARK ZINC-GAL GALVANISING ZINC RICH COATINGChemwatch Independent Material Safety Data SheetIssue Date: 3-Oct-2011 CHEMWATCH 4736-52NC317ECP Version No:2.0

CD 2011/3 Page 1 of 26

Section 1 - CHEMICAL PRODUCT AND COMPANY IDENTIFICATION

PRODUCT NAMEDY-MARK ZINC-GAL GALVANISING ZINC RICH COATING

SYNONYMS"Pack Sizes: 250 ml, 500 ml, 1 litre, 4 litre"

PROPER SHIPPING NAMEPAINT

PRODUCT USE■ Used according to manufacturer's directions.

SUPPLIERCompany: Dy- Mark Pty LtdAddress:89 Formation StreetWacolQLD, 4076AustraliaTelephone: +61 7 3271 2222Emergency Tel:0403 186 708Fax: +61 7 3271 2751Email: [email protected]

Section 2 - HAZARDS IDENTIFICATION

STATEMENT OF HAZARDOUS NATUREHAZARDOUS SUBSTANCE. DANGEROUS GOODS. According to NOHSC Criteria, and ADG Code.

CHEMWATCH HAZARD RATINGS

Flammability Toxicity

Body Contact Reactivity

Chronic

SCALE: Min/Nil=0 Low=1 Moderate=2 High=3 Extreme=4

RISKRisk Codes Risk PhrasesR11 • Highly flammable.R20/21/22 • Harmful by inhalation, in contact with skin and if

swallowed.R36/37/38 • Irritating to eyes, respiratory system and skin.R48/20 • Harmful: danger of serious damage to health by prolonged

exposure through inhalation.R50/53 • Very toxic to aquatic organisms, may cause long- term

adverse effects in the aquatic environment.R62(3) • Possible risk of impaired fertility.R63(3) • Possible risk of harm to the unborn child.

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CD 2011/3 Page 2 of 26Section 2 - HAZARDS IDENTIFICATION

R65 • HARMFUL - May cause lung damage if swallowed.R67 • Vapours may cause drowsiness and dizziness.

SAFETYSafety Codes Safety PhrasesS16 • Keep away from sources of ignition. No smoking.S23 • Do not breathe gas/ fumes/ vapour/ spray.S24 • Avoid contact with skin.S25 • Avoid contact with eyes.S36 • Wear suitable protective clothing.S37 • Wear suitable gloves.S39 • Wear eye/ face protection.S51 • Use only in well ventilated areas.S09 • Keep container in a well ventilated place.S53 • Avoid exposure - obtain special instructions before use.S29 • Do not empty into drains.S401 • To clean the floor and all objects contaminated by this material, use water

and detergent.S07 • Keep container tightly closed.S35 • This material and its container must be disposed of in a safe way.S13 • Keep away from food, drink and animal feeding stuffs.S26 • In case of contact with eyes, rinse with plenty of water and contact Doctor or

Poisons Information Centre.S46 • If swallowed, IMMEDIATELY contact Doctor or Poisons Information Centre (show

this container or label).S57 • Use appropriate container to avoid environment contamination.S61 • Avoid release to the environment. Refer to special instructions/ safety data

sheets.S60 • This material and its container must be disposed of as hazardous waste.

Section 3 - COMPOSITION / INFORMATION ON INGREDIENTS

NAME CAS RN %zinc 7440-66-6 60-90xylene 1330-20-7 10-30naphtha petroleum, light aromatic solvent 64742-95-6. 10-30toluene 108-88-3 10-30n- hexane 110-54-3


CD 2011/3 Page 3 of 26Section 4 - FIRST AID MEASURES

EYE■ If this product comes in contact with the eyes:- Wash out immediately with fresh running water.- Ensure complete irrigation of the eye by keeping eyelids apart and away from eye and moving the eyelids by occasionally lifting the upper and lower lids.- Seek medical attention without delay; if pain persists or recurs seek medical attention.- Removal of contact lenses after an eye injury should only be undertaken by skilled personnel.

SKIN■ If skin contact occurs:- Immediately remove all contaminated clothing, including footwear.- Flush skin and hair with running water (and soap if available).- Seek medical attention in event of irritation.

INHALED■ - If fumes or combustion products are inhaled remove from contaminated area.- Lay patient down. Keep warm and rested.- Prostheses such as false teeth, which may block airway, should be removed, where possible, prior to initiating first aid procedures.- Apply artificial respiration if not breathing, preferably with a demand valve resuscitator, bag-valve mask device, or pocket mask as trained. Perform CPR if necessary.- Transport to hospital, or doctor, without delay.

NOTES TO PHYSICIAN■ Any material aspirated during vomiting may produce lung injury. Therefore emesis should not be inducedmechanically or pharmacologically. Mechanical means should be used if it is considered necessary to evacuatethe stomach contents; these include gastric lavage after endotracheal intubation. If spontaneous vomiting hasoccurred after ingestion, the patient should be monitored for difficult breathing, as adverse effects ofaspiration into the lungs may be delayed up to 48 hours.- Absorption of zinc compounds occurs in the small intestine.- The metal is heavily protein bound.- Elimination results primarily from faecal excretion.- The usual measures for decontamination (Ipecac Syrup, lavage, charcoal or cathartics) may be administered,although patients usually have sufficient vomiting not to require them.- CaNa2EDTA has been used successfully to normalise zinc levels and is the agent of choice.[Ellenhorn and Barceloux: Medical Toxicology].Following acute or short term repeated exposures to toluene:- Toluene is absorbed across the alveolar barrier, the blood/air mixture being 11.2/15.6 (at 37 degrees C.)The concentration of toluene, in expired breath, is of the order of 18 ppm following sustained exposure to100 ppm. The tissue/blood proportion is 1/3 except in adipose where the proportion is 8/10.- Metabolism by microsomal mono-oxygenation, results in the production of hippuric acid. This may be detectedin the urine in amounts between 0.5 and 2.5 g/24 hr which represents, on average 0.8 gm/gm of creatinine. Thebiological half-life of hippuric acid is in the order of 1-2 hours.- Primary threat to life from ingestion and/or inhalation is respiratory failure.- Patients should be quickly evaluated for signs of respiratory distress (eg cyanosis, tachypnoea,intercostal retraction, obtundation) and given oxygen. Patients with inadequate tidal volumes or poorarterial blood gases (pO2 50 mm Hg) should be intubated.- Arrhythmias complicate some hydrocarbon ingestion and/or inhalation and electrocardiographic evidence ofmyocardial damage has been reported; intravenous lines and cardiac monitors should be established inobviously symptomatic patients. The lungs excrete inhaled solvents, so that hyperventilation improvesclearance.- A chest x-ray should be taken immediately after stabilisation of breathing and circulation to documentaspiration and detect the presence of pneumothorax.- Epinephrine (adrenaline) is not recommended for treatment of bronchospasm because of potential myocardialsensitisation to catecholamines. Inhaled cardioselective bronchodilators (e.g. Alupent, Salbutamol) are thepreferred agents, with aminophylline a second choice.- Lavage is indicated in patients who require decontamination; ensure use.

BIOLOGICAL EXPOSURE INDEX - BEIThese represent the determinants observed in specimens collected from a healthy worker exposed at the

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CD 2011/3 Page 4 of 26Section 4 - FIRST AID MEASURES

Exposure Standard (ES or TLV):Determinant Index Sampling Time Commentso- Cresol in urine 0.5 mg/L End of shift BHippuric acid in urine 1.6 g/g creatinine End of shift B, NSToluene in blood 0.05 mg/L Prior to last shift of

workweekNS: Non-specific determinant; also observed after exposure to other materialB: Background levels occur in specimens collected from subjects NOT exposed.For acute or short term repeated exposures to xylene:- Gastro-intestinal absorption is significant with ingestions. For ingestions exceeding 1-2 ml (xylene)/kg,intubation and lavage with cuffed endotracheal tube is recommended. The use of charcoal and cathartics isequivocal.- Pulmonary absorption is rapid with about 60-65% retained at rest.- Primary threat to life from ingestion and/or inhalation, is respiratory failure.- Patients should be quickly evaluated for signs of respiratory distress (e.g. cyanosis, tachypnoea,intercostal retraction, obtundation) and given oxygen. Patients with inadequate tidal volumes or poorarterial blood gases (pO2 < 50 mm Hg or pCO2 > 50 mm Hg) should be intubated.- Arrhythmias complicate some hydrocarbon ingestion and/or inhalation and electrocardiographic evidence ofmyocardial injury has been reported; intravenous lines and cardiac monitors should be established inobviously symptomatic patients. The lungs excrete inhaled solvents, so that hyperventilation improvesclearance.- A chest x-ray should be taken immediately after stabilisation of breathing and circulation to documentaspiration and detect the presence of pneumothorax.- Epinephrine (adrenalin) is not recommended for treatment of bronchospasm because of potential myocardialsensitisation to catecholamines. Inhaled cardioselective bronchodilators (e.g. Alupent, Salbutamol) are thepreferred agents, with aminophylline a second choice. BIOLOGICAL EXPOSURE INDEX - BEIThese represent the determinants observed in specimens collected from a healthy worker exposed at theExposure Standard (ES or TLV):Determinant Index Sampling Time CommentsMethylhippu- ric acids 1.5 gm/gm creatinine End of shiftin urine

2 mg/min Last 4 hrs of shift.

Section 5 - FIRE FIGHTING MEASURES

EXTINGUISHING MEDIA■ Do not use a water jet to fight fire.

FIRE FIGHTING■ - Alert Fire Brigade and tell them location and nature of hazard.- May be violently or explosively reactive.- Wear breathing apparatus plus protective gloves.- Prevent, by any means available, spillage from entering drains or water course.- Consider evacuation (or protect in place).- Fight fire from a safe distance, with adequate cover.- If safe, switch off electrical equipment until vapour fire hazard removed.- Use water delivered as a fine spray to control the fire and cool adjacent area.- Avoid spraying water onto liquid pools.- Do not approach containers suspected to be hot.- Cool fire exposed containers with water spray from a protected location.- If safe to do so, remove containers from path of fire.When any large container (including road and rail tankers) is involved in a fire,consider evacuation by 500 metres in all directions.

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CD 2011/3 Page 5 of 26Section 5 - FIRE FIGHTING MEASURES

FIRE/EXPLOSION HAZARD■ - Liquid and vapour are highly flammable.- Severe fire hazard when exposed to heat, flame and/or oxidisers.- Vapour may travel a considerable distance to source of ignition.- Heating may cause expansion or decomposition leading to violent rupture of containers.- On combustion, may emit toxic fumes of carbon monoxide (CO).Combustion products include: carbon dioxide (CO2), other pyrolysis products typical of burning organicmaterial.Contains low boiling substance: Closed containers may rupture due to pressure buildup under fire conditions.May emit clouds of acrid smoke.

FIRE INCOMPATIBILITY■ - Reacts with acids producing flammable / explosive hydrogen (H2) gas.- Avoid contamination with oxidising agents i.e. nitrates, oxidising acids, chlorine bleaches, pool chlorineetc. as ignition may result.

HAZCHEM•3YE

Personal Protective EquipmentBreathing apparatus.Chemical splash suit.

Section 6 - ACCIDENTAL RELEASE MEASURES

MINOR SPILLS■ - Remove all ignition sources.- Clean up all spills immediately.- Avoid breathing vapours and contact with skin and eyes.- Control personal contact by using protective equipment.- Contain and absorb small quantities with vermiculite or other absorbent material.- Wipe up.- Collect residues in a flammable waste container.

MAJOR SPILLS■ - Clear area of personnel and move upwind.- Alert Fire Brigade and tell them location and nature of hazard.- May be violently or explosively reactive.- Wear breathing apparatus plus protective gloves.- Prevent, by any means available, spillage from entering drains or water course.- Consider evacuation (or protect in place).- No smoking, naked lights or ignition sources.- Increase ventilation.- Stop leak if safe to do so.- Water spray or fog may be used to disperse /absorb vapour.- Contain spill with sand, earth or vermiculite.- Use only spark-free shovels and explosion proof equipment.- Collect recoverable product into labelled containers for recycling.- Absorb remaining product with sand, earth or vermiculite.- Collect solid residues and seal in labelled drums for disposal.- Wash area and prevent runoff into drains.- If contamination of drains or waterways occurs, advise emergency services.

EMERGENCY RESPONSE PLANNING GUIDELINES (ERPG)The maximum airborne concentration below which it is believed that nearly all individuals could be exposedfor up to one hour WITHOUT experiencing or developing

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CD 2011/3 Page 6 of 26Section 6 - ACCIDENTAL RELEASE MEASURES

life-threatening health effects is:toluene 1000ppm

irreversible or other serious effects or symptoms which could impair an individual's ability to takeprotective action is:

toluene 300ppm

other than mild, transient adverse effects without perceiving a clearly defined odour is:toluene 50ppm

American Industrial Hygiene Association (AIHA)

Ingredients considered according to the following cutoffsVery Toxic (T+) >= 0.1% Toxic (T) >= 3.0%R50 >= 0.25% Corrosive (C) >= 5.0%R51 >= 2.5%

else >= 10%where percentage is percentage of ingredient found in the mixture

Personal Protective Equipment advice is contained in Section 8 of the MSDS.

Section 7 - HANDLING AND STORAGE

PROCEDURE FOR HANDLING■ - Containers, even those that have been emptied, may contain explosive vapours.- Do NOT cut, drill, grind, weld or perform similar operations on or near containers.Contains low boiling substance:Storage in sealed containers may result in pressure buildup causing violent rupture of containers not rated appropriately.- Check for bulging containers.- Vent periodically- Always release caps or seals slowly to ensure slow dissipation of vapours.- DO NOT allow clothing wet with material to stay in contact with skin.- Electrostatic discharge may be generated during pumping - this may result in fire.- Ensure electrical continuity by bonding and grounding (earthing) all equipment.- Restrict line velocity during pumping in order to avoid generation of electrostatic discharge (


CD 2011/3 Page 7 of 26Section 7 - HANDLING AND STORAGE

- Use good occupational work practice.- Observe manufacturer's storing and handling recommendations.- Atmosphere should be regularly checked against established exposure standards to ensure safe working conditions.

SUITABLE CONTAINER■ - CARE: Packing of high density product in light weight metal or plastic packages may result in container collapse with product release.- Heavy gauge metal packages / Heavy gauge metal drums.- Packing as supplied by manufacturer.- Plastic containers may only be used if approved for flammable liquid.- Check that containers are clearly labelled and free from leaks.- For low viscosity materials (i) : Drums and jerry cans must be of the non-removable head type. (ii) : Where a can is to be used as an inner package, the can must have a screwed enclosure.- For materials with a viscosity of at least 2680 cSt. (23 deg. C)- For manufactured product having a viscosity of at least 250 cSt. (23 deg. C)- Manufactured product that requires stirring before use and having a viscosity of at least 20 cSt (25 deg. C)(i) : Removable head packaging;(ii) : Cans with friction closures and(iii) : low pressure tubes and cartridges may be used.- Where combination packages are used, and the inner packages are of glass, there must be sufficient inert cushioning material in contact with inner and outer packages- In addition, where inner packagings are glass and contain liquids of packing group I there must be sufficient inert absorbent to absorb any spillage, unless the outer packaging is a close fitting moulded plastic box and the substances are not incompatible with the plastic.

STORAGE INCOMPATIBILITY■ - Avoid reaction with oxidising agents.- Reacts violently with caustic soda, other alkalies - generating heat, highly flammable hydrogen gas.- If alkali is dry, heat generated may ignite hydrogen - if alkali is in solution may cause violent foaming.- Reacts with acids producing flammable / explosive hydrogen (H2) gas.- Reacts slowly with water.- CAUTION contamination with moisture will liberate explosive hydrogen gas, causing pressure build up in sealed containers.

STORAGE REQUIREMENTS■ - Store in original containers in approved flame-proof area.- No smoking, naked lights, heat or ignition sources.- DO NOT store in pits, depressions, basements or areas where vapours may be trapped.- Keep containers securely sealed.- Store away from incompatible materials in a cool, dry well ventilated area.- Protect containers against physical damage and check regularly for leaks.- Observe manufacturer's storing and handling recommendations.

Section 8 - EXPOSURE CONTROLS / PERSONAL PROTECTION

EXPOSURE CONTROLSSource Material TWA ppm TWA mg/m³ STEL ppm STEL mg/m³ Notes___________ ___________ _______ _______ _______ _______ _______Australia Exposure DY- MARK ZINC- GAL 80 350 150 655Standards GALVANISING ZINC RICH

COATING (Xylene (o- ,m- , p- isomers))

Australia Exposure zinc (Inspirable dust 10Standards (not otherwise

classified))

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CD 2011/3 Page 8 of 26Section 8 - EXPOSURE CONTROLS / PERSONAL PROTECTION

Source Material TWA ppm TWA mg/m³ STEL ppm STEL mg/m³ Notes___________ ___________ _______ _______ _______ _______ _______

Australia Exposure naphtha petroleum, 900 (seeStandards light aromatic Chapter 16)

solvent (Petrol(gasoline))

Australia Exposure toluene (Toluene) 50 191 150 574 SkStandardsAustralia Exposure n- hexane (Hexane (n- 20 72Standards Hexane))

EMERGENCY EXPOSURE LIMITSMaterial Revised IDLHxylene 174 900toluene 84 500n- hexane 173 1, 100 [LEL]

NOTESValues marked LEL indicate that the IDLH was based on 10% of the lower explosive limit for safetyconsiderations even though the relevant toxicological data indicated that irreversible health effects orimpairment of escape existed only at higher concentrations.

MATERIAL DATAN-HEXANE:TOLUENE:XYLENE:

■ Exposure limits with "skin" notation indicate that vapour and liquid may be absorbed through intactskin. Absorption by skin may readily exceed vapour inhalation exposure. Symptoms for skin absorption are thesame as for inhalation. Contact with eyes and mucous membranes may also contribute to overall exposure andmay also invalidate the exposure standard.

DY-MARK ZINC-GAL GALVANISING ZINC RICH COATING:N-HEXANE:

■ For n-hexane:Odour Threshold Value: 65 ppmNOTE: Detector tubes for n-hexane, measuring in excess of 100 ppm, are available commercially.Occupational polyneuropathy may result from exposures as low as 500 ppm (as hexane), whilst nearly

continuous exposures of 250 ppm have caused neurotoxic effects in animals. Many literature reports havefailed to distinguish hexane from n-hexane and on the assumption that the commercial hexane contains 30% n-hexane, a worst case recommendation for TLV is assumed to reduce the risk of peripheral neuropathies (due tothe metabolites 2,5-heptanedione and 3,6-octanedione) and other adverse neuropathic effects.

Concurrent exposure to chemicals (including MEK) and drugs which induce hepatic liver oxidative metabolismcan reduce the time for neuropathy to appear.

Odour Safety Factor(OSF)OSF=0.15 (n-HEXANE).

DY-MARK ZINC-GAL GALVANISING ZINC RICH COATING:XYLENE:

■ for xylenes:IDLH Level: 900 ppmOdour Threshold Value: 20 ppm (detection), 40 ppm (recognition)NOTE: Detector tubes for o-xylene, measuring in excess of 10 ppm, are available commercially. (m-xylene

and p-xylene give almost the same response).Xylene vapour is an irritant to the eyes, mucous membranes and skin and causes narcosis at high

concentrations. Exposure to doses sufficiently high to produce intoxication and unconsciousness also producestransient liver and kidney toxicity. Neurologic impairment is NOT evident amongst volunteers inhaling up to

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400 ppm though complaints of ocular and upper respiratory tract irritation occur at 200 ppm for 3 to 5minutes.

Exposure to xylene at or below the recommended TLV-TWA and STEL is thought to minimise the risk ofirritant effects and to produce neither significant narcosis or chronic injury. An earlier skin notation wasdeleted because percutaneous absorption is gradual and protracted and does not substantially contribute tothe dose received by inhalation.

Odour Safety Factor(OSF)OSF=4 (XYLENE).

DY-MARK ZINC-GAL GALVANISING ZINC RICH COATING:TOLUENE:

■ For toluene:Odour Threshold Value: 0.16-6.7 (detection), 1.9-69 (recognition)NOTE: Detector tubes measuring in excess of 5 ppm, are available.High concentrations of toluene in the air produce depression of the central nervous system (CNS) in

humans. Intentional toluene exposure (glue-sniffing) at maternally-intoxicating concentration has alsoproduced birth defects. Foetotoxicity appears at levels associated with CNS narcosis and probably occurs onlyin those with chronic toluene-induced kidney failure. Exposure at or below the recommended TLV-TWA is thoughtto prevent transient headache and irritation, to provide a measure of safety for possible disturbances tohuman reproduction, the prevention of reductions in cognitive responses reported amongst humans inhalinggreater than 40 ppm, and the significant risks of hepatotoxic, behavioural and nervous system effects(including impaired reaction time and incoordination). Although toluene/ethanol interactions are wellrecognised, the degree of protection afforded by the TLV-TWA among drinkers is not known.

Odour Safety Factor(OSF)OSF=17 (TOLUENE).

NAPHTHA PETROLEUM, LIGHT AROMATIC SOLVENT:ZINC:

■ Sensory irritants are chemicals that produce temporary and undesirable side-effects on the eyes, nose orthroat. Historically occupational exposure standards for these irritants have been based on observation ofworkers' responses to various airborne concentrations. Present day expectations require that nearly everyindividual should be protected against even minor sensory irritation and exposure standards are establishedusing uncertainty factors or safety factors of 5 to 10 or more. On occasion animal no-observable-effect-levels (NOEL) are used to determine these limits where human results are unavailable. An additional approach,typically used by the TLV committee (USA) in determining respiratory standards for this group of chemicals,has been to assign ceiling values (TLV C) to rapidly acting irritants and to assign short-term exposurelimits (TLV STELs) when the weight of evidence from irritation, bioaccumulation and other endpoints combineto warrant such a limit. In contrast the MAK Commission (Germany) uses a five-category system based onintensive odour, local irritation, and elimination half-life. However this system is being replaced to beconsistent with the European Union (EU) Scientific Committee for Occupational Exposure Limits (SCOEL); thisis more closely allied to that of the USA.

OSHA (USA) concluded that exposure to sensory irritants can:- cause inflammation- cause increased susceptibility to other irritants and infectious agents- lead to permanent injury or dysfunction- permit greater absorption of hazardous substances and- acclimate the worker to the irritant warning properties of these substances thus increasing the risk of

overexposure.

ZINC:■ It is the goal of the ACGIH (and other Agencies) to recommend TLVs (or their equivalent) for all

substances for which there is evidence of health effects at airborne concentrations encountered in theworkplace.

At this time no TLV has been established, even though this material may produce adverse health effects (asevidenced in animal experiments or clinical experience). Airborne concentrations must be maintained as low asis practically possible and occupational exposure must be kept to a minimum.

NOTE: The ACGIH occupational exposure standard for Particles Not Otherwise Specified (P.N.O.S) does NOTapply.

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NAPHTHA PETROLEUM, LIGHT AROMATIC SOLVENT:■ Odour threshold: 0.25 ppm.The TLV-TWA is protective against ocular and upper respiratory tract irritation and is recommended for

bulk handling of gasoline based on calculations of hydrocarbon content of gasoline vapour. A STEL isrecommended to prevent mucous membrane and ocular irritation and prevention of acute depression of thecentral nervous system. Because of the wide variation in molecular weights of its components, the conversionof ppm to mg/m3 is approximate. Sweden recommends hexane type limits of 100 ppm and heptane and octane typelimits of 300 ppm. Germany does not assign a value because of the widely differing compositions and resultantdifferences in toxic properties.

Odour Safety Factor (OSF)OSF=0.042 (gasoline).REL TWA: 25-100 ppm*, 125 mg/m3* [Various Manufacturers]CEL TWA: 50 ppm, 125 mg/m3

PERSONAL PROTECTION

EYE■ - Safety glasses with side shields.- Chemical goggles.- Contact lenses may pose a special hazard; soft contact lenses may absorb and concentrate irritants. Awritten policy document, describing the wearing of lens or restrictions on use, should be created for eachworkplace or task. This should include a review of lens absorption and adsorption for the class of chemicalsin use and an account of injury experience. Medical and first-aid personnel should be trained in theirremoval and suitable equipment should be readily available. In the event of chemical exposure, begin eyeirrigation immediately and remove contact lens as soon as practicable. Lens should be removed at the firstsigns of eye redness or irritation - lens should be removed in a clean environment only after workers havewashed hands thoroughly. [CDC NIOSH Current Intelligence Bulletin 59], [AS/NZS 1336 or national equivalent].

HANDS/FEET■ - Wear chemical protective gloves, eg. PVC.- Wear safety footwear or safety gumboots, eg. Rubber.Suitability and durability of glove type is dependent on usage. Important factors in the selection of glovesinclude:- frequency and duration of contact,- chemical resistance of glove material,- glove thickness and- dexteritySelect gloves tested to a relevant standard (e.g. Europe EN 374, US F739, AS/NZS 2161.1 or nationalequivalent).- When prolonged or frequently repeated contact may occur, a glove with a protection class of 5 or higher(breakthrough time greater than 240 minutes according to EN 374, AS/NZS 2161.10.1 or national equivalent) isrecommended.- When only brief contact is expected, a glove with a protection class of 3 or higher (breakthrough timegreater than 60 minutes according to EN 374, AS/NZS 2161.10.1 or national equivalent) is recommended.- Contaminated gloves should be replaced.Gloves must only be worn on clean hands. After using gloves, hands should be washed and dried thoroughly.Application of a non-perfumed moisturiser is recommended.- Protective gloves eg. Leather gloves or gloves with Leather facing.

OTHER■ - Overalls.- PVC Apron.- PVC protective suit may be required if exposure severe.- Eyewash unit.- Ensure there is ready access to a safety shower.- Some plastic personal protective equipment (PPE) (e.g. gloves, aprons, overshoes) are not recommended asthey may produce static electricity.- For large scale or continuous use wear tight-weave non-static clothing (no metallic fasteners, cuffs or

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pockets), non sparking safety footwear.

RESPIRATOR•Type AX Filter of sufficient capacity. (AS/NZS 1716 & 1715, EN 143:2000 & 149:2001, ANSI Z88 or nationalequivalent)■ Cartridge respirators should never be used for emergency ingress or in areas of unknown vapourconcentrations or oxygen content. The wearer must be warned to leave the contaminated area immediately ondetecting any odours through the respirator. The odour may indicate that the mask is not functioning properly,that the vapour concentration is too high, or that the mask is not properly fitted. Because of theselimitations, only restricted use of cartridge respirators is considered appropriate.

The local concentration of material, quantity and conditions of use determine the type of personal protectiveequipment required. For further information consult site specific CHEMWATCH data (if available), or yourOccupational Health and Safety Advisor.

ENGINEERING CONTROLS■ Engineering controls are used to remove a hazard or place a barrier between the worker and the hazard. Well-designed engineering controls can be highly effective in protecting workers and will typically be independentof worker interactions to provide this high level of protection.The basic types of engineering controls are:Process controls which involve changing the way a job activity or process is done to reduce the risk.Enclosure and/or isolation of emission source which keeps a selected hazard "physically" away from the workerand ventilation that strategically "adds" and "removes" air in the work environment. Ventilation can removeor dilute an air contaminant if designed properly. The design of a ventilation system must match theparticular process and chemical or contaminant in use.Employers may need to use multiple types of controls to prevent employee overexposure.

Local exhaust ventilation usually required. If risk of overexposure exists, wear approved respirator. Correctfit is essential to obtain adequate protection. Supplied-air type respirator may be required in specialcircumstances. Correct fit is essential to ensure adequate protection.An approved self contained breathing apparatus (SCBA) may be required in some situations.Provide adequate ventilation in warehouse or closed storage area.

Section 9 - PHYSICAL AND CHEMICAL PROPERTIES

APPEARANCEFlammable liquid; does not mix with water.

PHYSICAL PROPERTIESLiquid.Does not mix with water.Sinks in water.

State Liquid Molecular Weight Not ApplicableMelting Range (°C) Not Available Viscosity Not AvailableBoiling Range (°C) >35 Solubility in water (g/L) ImmiscibleFlash Point (°C) - 23 (n- hexane) pH (1% solution) Not Applica bleDecomposition Temp (°C) Not Available pH (as supplied) Not A pplicableAutoignition Temp (°C) Not Available Vapour Pressure (kPa) Not AvailableUpper Explosive Limit (%) Not Available Specific Gravity (water=1) 1.0- 1.3Lower Explosive Limit (%) Not Available Relative Vapour Density Not Available

(air=1)Volatile Component (%vol) Not Available Evaporation Rate Not Available

xylenelog Kow (Prager 1995): 3.12- 3.20

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CD 2011/3 Page 12 of 26Section 9 - PHYSICAL AND CHEMICAL PROPERTIES

toluenelog Kow (Sangster 1997): 2.73n- hexanelog Kow (Sangster 1997): 3.9

Section 10 - STABILITY AND REACTIVITY

CONDITIONS CONTRIBUTING TO INSTABILITY■ - Presence of incompatible materials.- Product is considered stable.- Hazardous polymerisation will not occur.For incompatible materials - refer to Section 7 - Handling and Storage.

Section 11 - TOXICOLOGICAL INFORMATION

POTENTIAL HEALTH EFFECTS

ACUTE HEALTH EFFECTS

SWALLOWED■ Accidental ingestion of the material may be harmful; animal experiments indicate that ingestion of lessthan 150 gram may be fatal or may produce serious damage to the health of the individual.Ingestion of petroleum hydrocarbons can irritate the pharynx, oesophagus, stomach and small intestine, andcause swellings and ulcers of the mucous. Symptoms include a burning mouth and throat; larger amounts cancause nausea and vomiting, narcosis, weakness, dizziness, slow and shallow breathing, abdominal swelling,unconsciousness and convulsions. Damage to the heart muscle can produce heart beat irregularities,ventricular fibrillation (fatal) and ECG changes. The central nervous system can be depressed. Light speciescan cause a sharp tingling of the tongue and cause loss of sensation there. Aspiration can cause cough,gagging, pneumonia with swelling and bleeding.Soluble zinc salts produce irritation and corrosion of the alimentary tract with pain, and vomiting. Deathcan occur due to insufficiency of food intake due to severe narrowing of the oesophagus and pylorus.Not a likely route of entry into the body in commercial or industrial environments. The liquid may produceconsiderable gastrointestinal discomfort and be harmful or toxic if swallowed. Ingestion may cause nausea,pain and vomiting. Vomit entering the lungs by aspiration can cause inflammation of the lungs, which can leadto death.

EYE■ Direct eye contact with petroleum hydrocarbons can be painful, and the corneal epithelium may betemporarily damaged. Aromatic species can cause irritation and excessive tear secretion.The liquid produces a high level of eye discomfort and is capable of causing pain and severe conjunctivitis.Corneal injury may develop, with possible permanent impairment of vision, if not promptly and adequatelytreated.There is evidence that material may produce eye irritation in some persons and produce eye damage 24 hours ormore after instillation. Severe inflammation may be expected with pain. There may be damage to the cornea.Unless treatment is prompt and adequate there may be permanent loss of vision. Conjunctivitis can occurfollowing repeated exposure.

SKIN■ Skin contact with the material may be harmful; systemic effects may result following absorption.The material may cause moderate inflammation of the skin either following direct contact or after a delay ofsome time. Repeated exposure can cause contact dermatitis which is characterised by redness, swelling andblistering.Open cuts, abraded or irritated skin should not be exposed to this material.Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injurywith harmful effects. Examine the skin prior to the use of the material and ensure that any external damage

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CD 2011/3 Page 13 of 26Section 11 - TOXICOLOGICAL INFORMATION

is suitably protected.The material may accentuate any pre-existing dermatitis condition.Aromatic hydrocarbons may produce sensitivity and redness of the skin. They are not likely to be absorbedinto the body through the skin but branched species are more likely to.

INHALED■ Inhalation of aerosols (mists, fumes), generated by the material during the course of normal handling, maybe harmful.The material can cause respiratory irritation in some persons. The body's response to such irritation cancause further lung damage.Inhalation of vapours may cause drowsiness and dizziness. This may be accompanied by sleepiness, reducedalertness, loss of reflexes, lack of co-ordination, and vertigo.The acute toxicity of inhaled alkylbenzenes is best described by central nervous system depression. As a rule,these compounds may also act as general anaesthetics.Systemic poisoning produced by general anaesthesia is characterised by lightheadedness, nervousness,apprehension, euphoria, confusion, dizziness, drowsiness, tinnitus, blurred or double vision, vomiting andsensations of heat, cold or numbness, twitching, tremors, convulsions, unconsciousness and respiratorydepression and arrest. Cardiac arrest may result from cardiovascular collapse. Bradycardia, and hypotensionmay also be produced.Inhaled alkylbenzene vapours cause death in animals at air levels that are relatively similar (typicallyLC50s are in the range 5000 -8000 ppm for 4 to 8 hour exposures). It is likely that acute inhalation exposureto alkylbenzenes resembles that to general anaesthetics.Alkylbenzenes are not generally toxic other than at high levels of exposure. This may be because theirmetabolites have a low order of toxicity and are easily excreted. There is little or no evidence to suggestthat metabolic pathways can become saturated leading to spillover to alternate pathways. Nor is thereevidence that toxic reactive intermediates, which may produce subsequent toxic or mutagenic effects, areformed.Inhalation hazard is increased at higher temperatures.Inhalation of high concentrations of gas/vapour causes lung irritation with coughing and nausea, centralnervous depression with headache and dizziness, slowing of reflexes, fatigue and inco-ordination.Central nervous system (CNS) depression may include general discomfort, symptoms of giddiness, headache,dizziness, nausea, anaesthetic effects, slowed reaction time, slurred speech and may progress tounconsciousness. Serious poisonings may result in respiratory depression and may be fatal.Inhaling high concentrations of mixed hydrocarbons can cause narcosis, with nausea, vomiting andlightheadedness. Low molecular weight (C2-C12) hydrocarbons can irritate mucous membranes and causeincoordination, giddiness, nausea, vertigo, confusion, headache, appetite loss, drowsiness, tremors andstupor. Massive exposures can lead to severe central nervous system depression, deep coma and death.Convulsions can occur due to brain irritation and/or lack of oxygen. Permanent scarring may occur, withepileptic seizures and brain bleeds occurring months after exposure. Respiratory system effects includeinflammation of the lungs with oedema and bleeding. Lighter species mainly cause kidney and nerve damage; theheavier paraffins and olefins are especially irritant to the respiratory system. Alkenes produce pulmonaryoedema at high concentrations. Liquid paraffins may produce sensation loss and depressant actions leading toweakness, dizziness, slow and shallow respiration, unconsciousness, convulsions and death. C5-7 paraffins mayalso produce multiple nerve damage. Aromatic hydrocarbons accumulate in lipid rich tissues (typically thebrain, spinal cord and peripheral nerves) and may produce functional impairment manifested by nonspecificsymptoms such as nausea, weakness, fatigue, vertigo; severe exposures may produce inebriation orunconsciousness. Many of the petroleum hydrocarbons can sensitise the heart and may cause ventricularfibrillation, leading to death.

CHRONIC HEALTH EFFECTS■ Harmful: danger of serious damage to health by prolonged exposure through inhalation.Harmful: danger of serious damage to health by prolonged exposure through inhalation.This material can cause serious damage if one is exposed to it for long periods. It can be assumed that itcontains a substance which can produce severe defects. This has been demonstrated via both short- and long-term experimentation.Substance accumulation, in the human body, may occur and may cause some concern following repeated or long-term occupational exposure.Constant or exposure over long periods to mixed hydrocarbons may produce stupor with dizziness, weakness andvisual disturbance, weight loss and anaemia, and reduced liver and kidney function. Skin exposure may result

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in drying and cracking and redness of the skin. Chronic exposure to lighter hydrocarbons can cause nervedamage, peripheral neuropathy, bone marrow dysfunction and psychiatric disorders as well as damage the liverand kidneys.Welding or flame cutting of metals with zinc or zinc dust coatings may result in inhalation of zinc oxidefume; high concentrations of zinc oxide fume may result in "metal fume fever"; also known as "brass chills",an industrial disease of short duration. [I.L.O] Symptoms include malaise, fever, weakness, nausea and mayappear quickly if operations occur in enclosed or poorly ventilated areas.Chronic inhalation or skin exposure to n-hexane may cause damage to nerve ends in extremities, e.g. finger,toes with loss of sensation. Symptoms can progress for months even after removal of exposure, and recoverymay take years and may not be complete.Women exposed to xylene in the first 3 months of pregnancy showed a slightly increased risk of miscarriageand birth defects. Evaluation of workers chronically exposed to xylene has demonstrated lack of genetictoxicity. Exposure to xylene has been associated with increased rates of blood cancer, but this may becomplicated by exposure to other substances, including benzene. Animal testing found no evidence of cancer-causing activity.Metallic dusts generated by the industrial process give rise to a number of potential health problems. Thelarger particles, above 5 micron, are nose and throat irritants. Smaller particles however, may cause lungdeterioration. Particles of less than 1.5 micron can be trapped in the lungs and, dependent on the nature ofthe particle, may give rise to further serious health consequences.Chronic solvent inhalation exposures may result in nervous system impairment and liver and blood changes.[PATTYS].

TOXICITY AND IRRITATION■ unless otherwise specified data extracted from RTECS - Register of Toxic Effects of Chemical Substances.

XYLENE:TOLUENE:DY-MARK ZINC-GAL GALVANISING ZINC RICH COATING:■ The material may cause skin irritation after prolonged or repeated exposure and may produce on contact skinredness, swelling, the production of vesicles, scaling and thickening of the skin.

DY-MARK ZINC-GAL GALVANISING ZINC RICH COATING:■ Asthma-like symptoms may continue for months or even years after exposure to the material ceases. This maybe due to a non-allergenic condition known as reactive airways dysfunction syndrome (RADS) which can occurfollowing exposure to high levels of highly irritating compound. Key criteria for the diagnosis of RADSinclude the absence of preceding respiratory disease, in a non-atopic individual, with abrupt onset ofpersistent asthma-like symptoms within minutes to hours of a documented exposure to the irritant. Areversible airflow pattern, on spirometry, with the presence of moderate to severe bronchial hyperreactivityon methacholine challenge testing and the lack of minimal lymphocytic inflammation, without eosinophilia,have also been included in the criteria for diagnosis of RADS. RADS (or asthma) following an irritatinginhalation is an infrequent disorder with rates related to the concentration of and duration of exposure tothe irritating substance. Industrial bronchitis, on the other hand, is a disorder that occurs as result ofexposure due to high concentrations of irritating substance (often particulate in nature) and is completelyreversible after exposure ceases. The disorder is characterised by dyspnea, cough and mucus production.

ZINC:■ The material may cause skin irritation after prolonged or repeated exposure and may produce on contact skinredness, swelling, the production of vesicles, scaling and thickening of the skin.

XYLENE:TOXICITY IRRITATIONOral (human) LDLo: 50 mg/kg Skin (rabbit):500 mg/24h ModerateOral (rat) LD50: 4300 mg/kg Eye (human): 200 ppm IrritantInhalation (human) TCLo: 200 ppm Eye (rabbit): 87 mg MildInhalation (man) LCLo: 10000 ppm/6h Eye (rabbit): 5 mg/24h SEVEREInhalation (rat) LC50: 5000 ppm/4hOral (Human) LD: 50 mg/kg

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Inhalation (Human) TCLo: 200 ppm/4hIntraperitoneal (Rat) LD50: 2459 mg/kgSubcutaneous (Rat) LD50: 1700 mg/kgOral (Mouse) LD50: 2119 mg/kgIntraperitoneal (Mouse) LD50: 1548 mg/kgIntravenous (Rabbit) LD: 129 mg/kgInhalation (Guinea pig) LC: 450 ppm/4h■ The material may produce severe irritation to the eye causing pronounced inflammation. Repeated orprolonged exposure to irritants may produce conjunctivitis.The substance is classified by IARC as Group 3:

NOT classifiable as to its carcinogenicity to humans.Evidence of carcinogenicity may be inadequate or limited in animal testing.Reproductive effector in rats

NAPHTHA PETROLEUM, LIGHT AROMATIC SOLVENT:TOXICITY IRRITATIONOral (rat) LD50: >5000 mg/kg * Nil ReportedInhalation (rat) LC50: >3670 ppm/8 h *■ for petroleum:This product contains benzene which is known to cause acute myeloid leukaemia and n-hexane which has beenshown to metabolize to compounds which are neuropathic.This product contains toluene. There are indications from animal studies that prolonged exposure to highconcentrations of toluene may lead to hearing loss.This product contains ethyl benzene and naphthalene from which there is evidence of tumours in rodentsCarcinogenicity: Inhalation exposure to mice causes liver tumours, which are not considered relevant tohumans. Inhalation exposure to rats causes kidney tumours which are not considered relevant to humans.Mutagenicity: There is a large database of mutagenicity studies on gasoline and gasoline blending streams,which use a wide variety of endpoints and give predominantly negative results. All in vivo studies in animalsand recent studies in exposed humans (e.g. petrol service station attendants) have shown negative results inmutagenicity assays.Reproductive Toxicity: Repeated exposure of pregnant rats to high concentrations of toluene (around orexceeding 1000 ppm) can cause developmental effects, such as lower birth weight and developmentalneurotoxicity, on the foetus. However, in a two-generation reproductive study in rats exposed to gasolinevapour condensate, no adverse effects on the foetus were observed.Human Effects: Prolonged/ repeated contact may cause defatting of the skin which can lead to dermatitis andmay make the skin more susceptible to irritation and penetration by other materials.

Lifetime exposure of rodents to gasoline produces carcinogenicity although the relevance to humans has beenquestioned. Gasoline induces kidney cancer in male rats as a consequence of accumulation of the alpha2-microglobulin protein in hyaline droplets in the male (but not female) rat kidney. Such abnormal accumulationrepresents lysosomal overload and leads to chronic renal tubular cell degeneration, accumulation of celldebris, mineralisation of renal medullary tubules and necrosis. A sustained regenerative proliferation occursin epithelial cells with subsequent neoplastic transformation with continued exposure. The alpha2-microglobulin is produced under the influence of hormonal controls in male rats but not in females and, moreimportantly, not in humans.Inhalation (rat) TCLo: 1320 ppm/6h/90D-I* [Devoe]

TOLUENE:TOXICITY IRRITATIONOral (human) LDLo: 50 mg/kg Skin (rabbit):20 mg/24h- ModerateOral (rat) LD50: 636 mg/kg Skin (rabbit):500 mg - ModerateInhalation (human) TCLo: 100 ppm Eye (rabbit):0.87 mg - MildInhalation (man) TCLo: 200 ppm Eye (rabbit): 2mg/24h - SEVEREInhalation (rat) LC50: >26700 ppm/1h Eye (rabbit):100 mg/30sec - MildDermal (rabbit) LD50: 12124 mg/kg■ For toluene:Acute ToxicityHumans exposed to intermediate to high levels of toluene for short periods of time experience adverse central

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nervous system effects ranging from headaches to intoxication, convulsions, narcosis, and death. Similareffects are observed in short-term animal studies.Humans - Toluene ingestion or inhalation can result in severe central nervous system depression, and in largedoses, can act as a narcotic. The ingestion of about 60 mL resulted in fatal nervous system depression within30 minutes in one reported case.Constriction and necrosis of myocardial fibers, markedly swollen liver, congestion and haemorrhage of thelungs and acute tubular necrosis were found on autopsy.Central nervous system effects (headaches, dizziness, intoxication) and eye irritation occurred followinginhalation exposure to 100 ppm toluene 6 hours/day for 4 days.Exposure to 600 ppm for 8 hours resulted in the same and more serious symptoms including euphoria, dilatedpupils, convulsions, and nausea . Exposure to 10,000-30,000 ppm has been reported to cause narcosis and deathToluene can also strip the skin of lipids causing dermatitisAnimals - The initial effects are instability and incoordination, lachrymation and sniffles (respiratoryexposure), followed by narcosis. Animals die of respiratory failure from severe nervous system depression.Cloudy swelling of the kidneys was reported in rats following inhalation exposure to 1600 ppm, 18-20hours/day for 3 daysSubchronic/Chronic Effects:Repeat doses of toluene cause adverse central nervous system effects and can damage the upper respiratorysystem, the liver, and the kidney. Adverse effects occur as a result from both oral and the inhalationexposures. A reported lowest-observed-effect level in humans for adverse neurobehavioral effects is 88 ppm.Humans - Chronic occupational exposure and incidences of toluene abuse have resulted in hepatomegaly andliver function changes. It has also resulted in nephrotoxicity and, in one case, was a cardiac sensitiser andfatal cardiotoxin.Neural and cerebellar dystrophy were reported in several cases of habitual "glue sniffing." Anepidemiological study in France on workers chronically exposed to toluene fumes reported leukopenia andneutropenia. Exposure levels were not given in the secondary reference; however, the average urinaryexcretion of hippuric acid, a metabolite of toluene, was given as 4 g/L compared to a normal level of 0.6 g/LAnimals - The major target organs for the subchronic/chronic toxicity of toluene are the nervous system,liver, and kidney. Depressed immune response has been reported in male mice given doses of 105 mg/kg/day for28 days. Toluene in corn oil administered to F344 male and female rats by gavage 5 days/week for 13 weeks,induced prostration, hypoactivity, ataxia, piloerection, lachrymation, excess salivation, and body tremors atdoses 2500 mg/kg. Liver, kidney, and heart weights were also increased at this dose and histopathologiclesions were seen in the liver, kidneys, brain and urinary bladder. The no-observed-adverse effect level(NOAEL) for the study was 312 mg/kg (223 mg/kg/day) and the lowest-observed-adverse effect level (LOAEL) forthe study was 625 mg/kg (446 mg/kg/day) .Developmental/Reproductive ToxicityExposures to high levels of toluene can result in adverse effects in the developing human foetus. Severalstudies have indicated that high levels of toluene can also adversely effect the developing offspring inlaboratory animals.Humans - Variable growth, microcephaly, CNS dysfunction, attentional deficits, minor craniofacial and limbabnormalities, and developmental delay were seen in three children exposed to toluene in utero as a result ofmaternal solvent abuse before and during pregnancyAnimals - Sternebral alterations, extra ribs, and missing tails were reported following treatment of ratswith 1500 mg/m3 toluene 24 hours/day during days 9-14 of gestation. Two of the dams died during the exposure.Another group of rats received 1000 mg/m3 8 hours/day during days 1-21 of gestation. No maternal deaths ortoxicity occurred, however, minor skeletal retardation was present in the exposed fetuses. CFLP Mice wereexposed to 500 or 1500 mg/m3 toluene continuously during days 6-13 of pregnancy. All dams died at the highdose during the first 24 hours of exposure, however none died at 500 mg/m3. Decreased foetal weight wasreported, but there were no differences in the incidences of skeletal malformations or anomalies between thetreated and control offspring.Absorption - Studies in humans and animals have demonstrated that toluene is readily absorbed via the lungsand the gastrointestinal tract. Absorption through the skin is estimated at about 1% of that absorbed by thelungs when exposed to toluene vapor.Dermal absorption is expected to be higher upon exposure to the liquid; however, exposure is limited by therapid evaporation of toluene .Distribution - In studies with mice exposed to radiolabeled toluene by inhalation, high levels ofradioactivity were present in body fat, bone marrow, spinal nerves, spinal cord, and brain white matter.Lower levels of radioactivity were present in blood, kidney, and liver. Accumulation of toluene has generallybeen found in adipose tissue, other tissues with high fat content, and in highly vascularised tissues .

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Metabolism - The metabolites of inhaled or ingested toluene include benzyl alcohol resulting from thehydroxylation of the methyl group. Further oxidation results in the formation of benzaldehyde and benzoicacid. The latter is conjugated with glycine to yield hippuric acid or reacted with glucuronic acid to formbenzoyl glucuronide. o-cresol and p-cresol formed by ring hydroxylation are considered minor metabolitesExcretion - Toluene is primarily (60-70%) excreted through the urine as hippuric acid. The excretion ofbenzoyl glucuronide accounts for 10-20%, and excretion of unchanged toluene through the lungs also accountsfor 10-20%. Excretion of hippuric acid is usually complete within 24 hours after exposure.

N-HEXANE:TOXICITY IRRITATIONOral (rat) LD50: 28710 mg/kg Eye(rabbit): 10 mg - MildInhalation (human) TCLo: 190 ppm/8WInhalation (rat) LD50: 48000 ppm/4h■ The material may be irritating to the eye, with prolonged contact causing inflammation. Repeated orprolonged exposure to irritants may produce conjunctivitis.

CARCINOGENXylenes International Agency for Research on Cancer Group 3

(IARC) - Agents Reviewed by the IARCMonographs

Toluene International Agency for Research on Cancer Group 3(IARC) - Agents Reviewed by the IARCMonographs

REPROTOXINxylene ILO Chemicals in the Reduced fertility or

electronics industry sterilitythat have toxic effectson reproduction

SKINtoluene Australia Exposure Notes Sk

Standards - Skin

Section 12 - ECOLOGICAL INFORMATION

NAPHTHA PETROLEUM, LIGHT AROMATIC SOLVENT:N-HEXANE:ZINC:■ Do NOT allow product to come in contact with surface waters or to intertidal areas below the mean highwater mark. Do not contaminate water when cleaning equipment or disposing of equipment wash-waters.Wastes resulting from use of the product must be disposed of on site or at approved waste sites.

XYLENE:NAPHTHA PETROLEUM, LIGHT AROMATIC SOLVENT:TOLUENE:N-HEXANE:ZINC:■ DO NOT discharge into sewer or waterways.

N-HEXANE:NAPHTHA PETROLEUM, LIGHT AROMATIC SOLVENT:■ Toxic to aquatic organisms.

ZINC:■ Very toxic to aquatic organisms, may cause long-term adverse effects in the aquatic environment.

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CD 2011/3 Page 18 of 26Section 12 - ECOLOGICAL INFORMATION

For zinc and its compounds:Environmental fate:Zinc is capable of forming complexes with a variety of organic and inorganic groups (ligands). Biologicalactivity can affect the mobility of zinc in the aquatic environment, although the biota contains relativelylittle zinc compared to the sediments. Zinc bioconcentrates moderately in aquatic organisms; bioconcentrationis higher in crustaceans and bivalve species than in fish. Zinc does not concentrate appreciably in plants,and it does not biomagnify significantly through terrestrial food chains.However biomagnification may be of concern if concentration of zinc exceeds 1632 ppm in the top 12 inches ofsoil.Zinc can persist in water indefinitely and can be toxic to aquatic life. The threshold concentration for fishis 0.1 ppm. Zinc may be concentrated in the aquatic food chain; it is concentrated over 200,000 times inoysters. Copper is synergistic but calcium is antagonistic to zinc toxicity in fish. Zinc can accumulate infreshwater animals at 5 -1,130 times the concentration present in the water. Furthermore, although zincactively bioaccumulates in aquatic systems, biota appears to represent a relatively minor sink compared tosediments. Steady-state zinc bioconcentration factors (BCFs) for 12 aquatic species range from 4 to 24,000.Crustaceans and fish can accumulate zinc from both water and food. A BCF of 1,000 was reported for bothaquatic plants and fish, and a value of 10,000 was reported for aquatic invertebrates. The order ofenrichment of zinc in different aquatic organisms was as follows (zinc concentrations in µg/g dry weightappear in parentheses): fish (25), shrimp (50), mussel (60), periphyton (260), zooplankton (330), and oyster(3,300). The high enrichment in oysters may be due to their ingestion of particulate matter containing higherconcentrations of zinc than ambient water. Other investigators have also indicated that organisms associatedwith sediments have higher zinc concentrations than organisms living in the aqueous layer. With respect tobioconcentration from soil by terrestrial plants, invertebrates, and mammals, BCFs of 0.4, 8, and 0.6,respectively, have been reported. The concentration of zinc in plants depends on the plant species, soil pH,and the composition of the soil.Plant species do not concentrate zinc above the levels present in soil.In some fish, it has been observed that the level of zinc found in their bodies did not directly relate tothe exposure concentrations. Bioaccumulation of zinc in fish is inversely related to the aqueous exposure.This evidence suggests that fish placed in environments with lower zinc concentrations can sequester zinc intheir bodies.The concentration of zinc in drinking water may increase as a result of the distribution system and householdplumbing. Common piping materials used in distribution systems often contain zinc, as well as other metalsand alloys. Trace metals may enter the water through corrosion products or simply by the dissolution of smallamounts of metals with which the water comes in contact. Reactions with materials of the distribution system,particularly in soft low-pH waters, very often have produced concentrations of zinc in tap water much greaterthan those in the raw or treated waters at the plant of origin. Zinc gives water a metallic taste at lowlevels. Overexposures to zinc also have been associated with toxic effects. Ingestion of zinc or zinc-containing compounds has resulted in a variety of systemic effects in the gastrointestinal and hematologicalsystems and alterations in the blood lipid profile in humans and animals. In addition, lesions have beenobserved in the liver, pancreas, and kidneys of animals.Environmental toxicity of zinc in water is dependent upon the concentration of other minerals and the pH ofthe solution, which affect the ligands that associate with zinc.Zinc occurs in the environment mainly in the +2 oxidation state. Sorption is the dominant reaction, resultingin the enrichment of zinc in suspended and bed sediments. Zinc in aerobic waters is partitioned intosediments through sorption onto hydrous iron and manganese oxides, clay minerals, and organic material. Theefficiency of these materials in removing zinc from solution varies according to their concentrations, pH,redox potential (Eh), salinity, nature and concentrations of complexing ligands, cation exchange capacity,and the concentration of zinc. Precipitation of soluble zinc compounds appears to be significant only underreducing conditions in highly polluted water. Generally, at lower pH values, zinc remains as the free ion.The free ion (Zn+2) tends to be adsorbed and transported by suspended solids in unpolluted waters.Zinc is an essential nutrient that is present in all organisms. Although biota appears to be a minorreservoir of zinc relative to soils and sediments, microbial decomposition of biota in water can produceligands, such as humic acids, that can affect the mobility of zinc in the aquatic environment through zincprecipitation and adsorption.The relative mobility of zinc in soil is determined by the same factors that affect its transport in aquaticsystems (i.e., solubility of the compound, pH, and salinity)The redox status of the soil may shift zinc partitioning. Reductive dissolution of iron and manganese(hydr)oxides under suboxic conditions release zinc into the aqueous phase; the persistence of suboxicconditions may then lead to a repartitioning of zinc into sulfide and carbonate solids. The mobility of zinc

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in soil depends on the solubility of the speciated forms of the element and on soil properties such as cationexchange capacity, pH, redox potential, and chemical species present in soil; under anaerobic conditions,zinc sulfide is the controlling species.Since zinc sulfide is insoluble, the mobility of zinc in anaerobic soil is low. In a study of the effect ofpH on zinc solubility: When the pH is 7 in soils high in organic matter. This is a result of the release of organically complexed zinc,reduced zinc adsorption at higher pH, or an increase in the concentration of chelating agents in soil. Forcalcareous soils, the relationship between zinc solubility and pH is nonlinear. At a high pH, zinc insolution is precipitated as Zn(OH)2, zinc carbonate (ZnCO3), or calcium zincate. Clay and metal oxides arecapable of sorbing zinc and tend to retard its mobility in soil. Zinc was more mobile at pH 4 than at pH 6.5as a consequence of sorptionZinc concentrations in the air are relatively low, except near industrial sources such as smelters. Noestimate for the atmospheric lifetime of zinc is available at this time, but the fact that zinc istransported long distances in air indicates that its lifetime in air is at least on the order of days. Thereare few data regarding the speciation of zinc released to the atmosphere. Zinc is removed from the air by dryand wet deposition, but zinc particles with small diameters and low densities suspended in the atmospheretravel long distances from emission sources.The material is classified as an ecotoxin* because the Fish LC50 (96 hours) is less than or equal to 0.1 mg/l* Classification of Substances as Ecotoxic (Dangerous to the Environment)Appendix 8, Table 1Compiler's Guide for the Preparation of International Chemical Safety Cards: 1993 Commission of the EuropeanCommunities.The product has no effect on the environment unless in finely divided form. In this form it can be harmful toaquatic life.Acute toxicity data in the form of 96 hour LC50s for Australian freshwater animals are:fish - 0.14 to 38 mg Zn/L; and crustaceans 0.43 to 32 mg Zn/L.

XYLENE:■ Harmful to aquatic organisms.For xylenes :log Koc : 2.05-3.08Koc : 25.4-204Half-life (hr) air : 0.24-42Half-life (hr) H2O surface water : 24-672Half-life (hr) H2O ground : 336-8640Half-life (hr) soil : 52-672Henry's Pa m3 /mol: 637-879Henry's atm m3 /mol: 7.68E-03BOD 5 if unstated: 1.4,1%COD : 2.56,13%ThOD : 3.125BCF : 23log BCF : 1.17-2.41Environmental FateTerrestrial fate:: Measured Koc values of 166 and 182, indicate that 3-xylene is expected to have moderatemobility in soil. Volatilisation of p-xylene is expected to be important from moist soil surfaces given ameasured Henry's Law constant of 7.18x10-3 atm-cu m/mole. The potential for volatilisation of 3-xylene fromdry soil surfaces may exist based on a measured vapor pressure of 8.29 mm Hg. p-Xylene may be degraded duringits passage through soil). The extent of the degradation is expected to depend on its concentration,residence time in the soil, the nature of the soil, and whether resident microbial populations have beenacclimated. p-Xylene, present in soil samples contaminated with jet fuel, was completely degraded aerobicallywithin 5 days. In aquifer studies under anaerobic conditions, p-xylene was degraded, usually within severalweeks, with the production of 3-methylbenzylfumaric acid, 3-methylbenzylsuccinic acid, 3-methylbenzoate, and3-methylbenzaldehyde as metabolites.

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Aquatic fate: Koc values indicate that p-xylene may adsorb to suspended solids and sediment in water. p-Xylene is expected to volatilise from water surfaces based on the measured Henry's Law constant. Estimatedvolatilisation half-lives for a model river and model lake are 3 hours and 4 days, respectively. BCF valuesof 14.8, 23.4, and 6, measured in goldfish, eels, and clams, respectively, indicate that bioconcentration inaquatic organisms is low. p-Xylene in water with added humic substances was 50% degraded following 3 hoursirradiation suggesting that indirect photooxidation in the presence of humic acids may play an important rolein the abiotic degradation of p-xylene. Although p-xylene is biodegradable and has been observed to degradein pond water, there are insufficient data to assess the rate of this process in surface waters. p-Xylene hasbeen observed to degrade in anaerobic and aerobic groundwater in several studies; however, it is known topersist for many years in groundwater, at least at sites where the concentration might have been quite high.Atmospheric fate:Most xylenes released to the environment will occur in the atmosphere and volatilisation is the dominantenvironmental fate process. In the ambient atmosphere, xylenes are expected to exist solely in the vapourphase. Xylenes are degraded in the atmosphere primarily by reaction with photochemically-produced hydroxylradicals, with an estimated atmospheric lifetime of about 0.5 to 2 days. Xylenes' susceptibility tophotochemical oxidation in the troposphere is to the extent that they may contribute to photochemical smogformation.According to a model of gas/particle partitioning of semivolatile organic compounds in the atmosphere andfrom its vapour pressure, p-xylene, is expected to exist solely as a vapour in the ambient atmosphere. Vapour-phase p-xylene is degraded in the atmosphere by reaction with photochemically-produced hydroxyl radicals; thehalf-life for this reaction in air is estimated to be about 16 hours. A half-life of 1.0 hr in summer and 10hr in winter was measured for the reaction of p-xylene with photochemically-produced hydroxyl radicals. p-Xylene has a moderately high photochemical reactivity under smog conditions, higher than the other xyleneisomers, with loss rates varying from 9-42% per hr. The photooxidation of p-xylene results in the productionof carbon monoxide, formaldehyde, glyoxal, methylglyoxal, 3-methylbenzylnitrate, m-tolualdehyde, 4-nitro-3-xylene, 5-nitro-3-xylene, 2,6-dimethyl-p-benzoquinone, 2,4-dimethylphenol, 6-nitro-2,4-dimethylphenol, 2,6-dimethylphenol, and 4-nitro-2,6-dimethylphenol.Ecotoxicity:for xylenesFish LC50 (96 h) Pimephales promelas 13.4 mg/l; Oncorhyncus mykiss 8.05 mg/l; Lepomis macrochirus 16.1 mg/l(all flow through values); Pimephales promelas 26.7 (static)Daphnia EC50 948 h): 3.83 mg/lPhotobacterium phosphoreum EC50 (24 h): 0.0084 mg/lGammarus lacustris LC50 (48 h): 0.6 mg/l.

NAPHTHA PETROLEUM, LIGHT AROMATIC SOLVENT:■ May cause long-term adverse effects in the aquatic environment.For petroleum derivatives:Chemical analysis for all individual compounds in a petroleum bulk product released to the environment isgenerally unrealistic due to the complexity of these mixtures and the laboratory expense. Determining thechemical composition of a petroleum release is further complicated by hydrodynamic, abiotic, and bioticprocesses that act on the release to change the chemical character.The longer the release is exposed to the environment, the greater the change in chemical character and theharder it is to obtain accurate analytical results reflecting the identity of the release. After extensiveweathering, detailed knowledge of the original bulk product is often less valuable than current site-specificinformation on a more focused set of hydrocarbon components. Health assessment efforts are frequentlyfrustrated by three primary problems: (1) the inability to identify and quantify the individual compoundsreleased to the environment as a consequence of a petroleum spill; (2) the lack of information characterizingthe fate of the individual compounds in petroleum mixtures; and (3) the lack of specific health guidancevalues for the majority of chemicals present in petroleum products. To define the public health implicationsassociated with exposure to petroleum hydrocarbons, it is necessary to have a basic understanding ofpetroleum properties, compositions, and the physical, chemical, biological, and toxicological properties ofthe compounds most often identified as the key chemicals of concern.Environmental fate:Petroleum products released to the environment migrate through soil via two general pathways: (1) as bulk oilflow infiltrating the soil under the forces of gravity and capillary action, and (2) as individual compoundsseparating from the bulk petroleum mixture and dissolving in air or water. When bulk oil flow occurs, itresults in little or no separation of the individual compounds from the product mixture and the infiltrationrate is usually fast relative to the dissolution rate. Many compounds that are insoluble and immobile in

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water are soluble in bulk oil and will migrate along with the bulk oil flow. Factors affecting the rate ofbulk oil infiltration include soil moisture content, vegetation, terrain, climate, rate of release (e.g.,catastrophic versus slow leakage), soil particle size (e.g., sand versus clay), and oil viscosity (e.g.,gasoline versus motor oil).As bulk oil migrates through the soil column, a small amount of the product mass is retained by soilparticles. The bulk product retained by the soil particles is known as "residual saturation".Depending upon the persistence of the bulk oil, residual saturation can potentially reside in the soil foryears. Residual saturation is important as it determines the degree of soil contamination and can act as acontinuing source of contamination for individual compounds to separate from the bulk product and migrateindependently in air or groundwater. Residual saturation is important as it determines the degree of soilcontamination and can act as a continuing source of contamination for individual compounds to separate fromthe bulk product and migrate independently in air or groundwater. When the amount of product released to theenvironment is small relative to the volume of available soil, all of the product is converted to residualsaturation and downward migration of the bulk product usually ceases prior to affecting groundwaterresources. Adverse impacts to groundwater may still occur if rain water infiltrates through soil containingresidual saturation and initiates the downward migration of individual compounds. When the amount of productreleased is large relative to the volume of available soil, the downward migration of bulk product ceases aswater-saturated pore spaces are encountered. If the density of the bulk product is less than that of water,the product tends to "float" along the interface between the water saturated and unsaturated zones and spreadhorizontally in a pancake-like layer, usually in the direction of groundwater flow. Almost all motor andheating oils are less dense than water. If the density of the bulk product is greater than that of water, theproduct will continue to migrate downward through the water table aquifer under the continued influence ofgravity. Downward migration ceases when the product is converted to residual saturation or when animpermeable surface is encountered.As the bulk product migrates through the soil column, individual compounds may separate from the mixture andmigrate independently. Chemical transport properties such as volatility, solubility, and sorption potentialare often used to evaluate and predict which compounds will likely separate from the mixture. Since petroleumproducts are complex mixtures of hundreds of compounds, the compounds characterized by relatively high vaporpressures tend to volatilise and enter the vapor phase. The exact composition of these vapors depends on thecomposition of the original product. Using gasoline as an example, compounds such as butane, propane, benzene,toluene, ethylbenzene and xylene are preferentially volatilised. Because volatility represents transfer ofthe compound from the product or liquid phase to the air phase, it is expected that the concentration of thatcompound in the product or liquid phase will decrease as the concentration in the air phase increases.In general, compounds having a vapor pressure in excess of 10-2 mm Hg are more likely to be present in theair phase than in the liquid phase. Compounds characterized by vapor pressures less than 10-7 mm Hg are morelikely to be associated with the liquid phase. Compounds possessing vapor pressures that are less than 10-2mm Hg, but greater than 10-7 mm Hg, will have a tendency to exist in both the air and the liquid phases.Lighter petroleum products such as gasoline contain constituents with higher water solubility and volatilityand lower sorption potential than heavier petroleum products such as fuel oil.Data compiled from gasoline spills and laboratory studies indicate that these light-fraction hydrocarbonstend to migrate readily through soil, potentially threatening or affecting groundwater supplies. In contrast,petroleum products with heavier molecular weight constituents, such as fuel oil, are generally morepersistent in soils, due to their relatively low water solubility and volatility and high sorption capacity.Solubility generally decreases with increasing molecular weight of the hydrocarbon compounds. For compoundshaving similar molecular weights, the aromatic hydrocarbons are more water soluble and mobile in water thanthe aliphatic hydrocarbons and branched aliphatics are less water-soluble than straight-chained aliphatics.Aromatic compounds in petroleum fuels may comprise as much as 50% by weight; aromatic compounds in the C6-C13,range made up approximately 95% of the compounds dissolved in water.Indigenous microbes found in many natural settings (e.g., soils, groundwater, ponds) have been shown to becapable of degrading organic compounds. Unlike other fate processes that disperse contaminants in theenvironment, biodegradation can eliminate the contaminants without transferring them across media.The final products of microbial degradation are carbon dioxide, water, and microbial biomass. The rate ofhydrocarbon degradation depends on the chemical composition of the product released to the environment aswell as site-specific environmental factors. Generally the straight chain hydrocarbons and the aromatics aredegraded more readily than the highly branched aliphatic compounds. The n-alkanes, n-alkyl aromatics, and thearomatics in the C10-C22 range are the most readily biodegradable; n-alkanes, n-alkyl aromatics, andaromatics in the C5-C9 range are biodegradable at low concentrations by some microorganisms, but aregenerally preferentially removed by volatilisation and thus are unavailable in most environments; n-alkanesin the C1-C4 ranges are biodegradable only by a narrow range of specialized hydrocarbon degraders; and n-

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alkanes, n-alkyl aromatics, and aromatics above C22 are generally not available to degrading microorganisms.Hydrocarbons with condensed ring structures, such as PAHs with four or more rings, have been shown to berelatively resistant to biodegradation. PAHs with only 2 or 3 rings (e.g., naphthalene, anthracene) are moreeasily biodegraded. PAHs with only 2 or 3 rings (e.g., naphthalene, anthracene) are more easily biodegraded.A large proportion of the water-soluble fraction of the petroleum product may be degraded as the compounds gointo solution. As a result, the remaining product may become enriched in the alicyclics, the highly branchedaliphatics, and PAHs with many fused rings.In almost all cases, the presence of oxygen is essential for effective biodegradation of oil. Anaerobicdecomposition of petroleum hydrocarbons leads to extremely low rates of degradation. The ideal pH range topromote biodegradation is close to neutral (6-8). For most species, the optimal pH is slightly alkaline, thatis, greater than 7. The moisture content of the contaminated soil will affect biodegradation of oils due todissolution of the residual compounds, dispersive actions, and the need for microbial metabolism to sustainhigh activity. The moisture content in soil affects microbial locomotion, solute diffusion, substrate supply,and the removal of metabolic by-products. Biodegradation rates in soils are also affected by the volume ofproduct released to the environment. At concentrations of 0.5% of oil by volume, the degradation rate in soilis fairly independent of oil concentrations. However, as oil concentration rises, the first order degradationrate decreases and the oil degradation half-life increases. Ultimately, when the oil reaches saturationconditions in the soil (i.e., 30-50% oil), biodegradation virtually ceases.Excessive moisture will limit the gaseous supply of oxygen for enhanced decomposition of petroleumhydrocarbons. Most studies indicate that optimum moisture content is within 50-70% of the water holdingcapacity.All biological transformations are affected by temperature. Generally, as the temperature increases,biological activity tends to increase up to a temperature where enzyme denaturation occurs. The presence ofoil should increase soil temperature, particularly at the surface. The darker color increases the heatcapacity by adsorbing more radiation. The optimal temperature for biodegradation to occur ranges from 18 C to30 C. Minimum rates would be expected at 5 C or lower.

TOLUENE:■ For toluene:log Kow : 2.1-3log Koc : 1.12-2.85Koc : 37-260log Kom : 1.39-2.89Half-life (hr) air : 2.4-104Half-life (hr) H2O surface water : 5.55-528Half-life (hr) H2O ground : 168-2628Half-life (hr) soil :



biodegradation of toluene to carbon dioxide occurs with a typical half life of 1-7 days.Water - An important fate process for toluene is volatilization, the rate of which depends on the amount ofturbulence in the surface water .The volatilisation of toluene from static water has a half life of 1-16 days,whereas from turbulent water the half life is 5-6 hours. Degradation of toluene in surface water occursprimarily by biodegradation with a half life of less than one day under favorable conditions (presence ofmicroorganisms, microbial adaptation, and optimum temperature). Biodegradation also occurs in shallowgroundwater and in salt water at a reduced rate). No data are available on anaerobic degradation of toluenein deep ground water conditions where aerobic degradation would be minimal .Biota - Bioaccumulation in most organisms is limited by the metabolism of toluene into more polar compoundsthat have greater water solubility and a lower affinity for lipids. Bioaccumulation in the food chain ispredicted to be low.Ecotoxicity:Toluene has moderate acute toxicity to aquatic organisms; several toxicity values are in the range of greaterthan 1 mg/L and 100 mg/L.Fish LC50 (96 h): fathead minnow (Pimephales promelas) 12.6-72 mg/l; Lepomis macrochirus 13-24 mg/l;guppy (Poecilia reticulata) 28.2-59.3 mg/l; channel catfish (Ictalurus punctatus) 240 mg/l; goldfish(Carassius auratus): 22.8-57.68 mg/lCrustaceans LC50 (96 h): grass shrimp (Palaemonetes pugio) 9.5 ppm, crab larvae stage (Cancer magister) 28ppm; shrimp (Crangon franciscorum) 4.3 ppm; daggerblade grass shrimp (Palaemonetes pugio) 9.5 mg/lAlgae EC50 (24 h): green algae (Chlorella vulgaris) 245 mg/l (growth); (72 h) green algae (Selenastrumcapricornutum) 12.5 mg/l (growth).

N-HEXANE:/53#90hexane

EcotoxicityIngredient Persistence: Persistence: Air Bioaccumulation Mobility

Water/SoilDY- MARK ZINC- GAL GALVANISING No Data No DataZINC RICH COATING Available Availablezinc No Data No Data LOW

Available Availablexylene LOW LOW LOWnaphtha petroleum, light No Data No Dataaromatic solvent Available Availabletoluene LOW MED LOW MEDn- hexane LOW No Data MED MED

Available

Section 13 - DISPOSAL CONSIDERATIONS

■ - Containers may still present a chemical hazard/ danger when empty.- Return to supplier for reuse/ recycling if possible.Otherwise:- If container can not be cleaned sufficiently well to ensure that residuals do not remain or if the container cannot be used to store the same product, then puncture containers, to prevent re-use, and bury at an authorised landfill.- Where possible retain label warnings and MSDS and observe all notices pertaining to the product.Legislation addressing waste disposal requirements may differ by country, state and/ or territory. Each user must refer to laws operating in their area. In some areas, certain wastes must be tracked.A Hierarchy of Controls seems to be common - the user should investigate:- Reduction- Reuse- Recycling- Disposal (if all else fails)This material may be recycled if unused, or if it has not been contaminated so as to make it unsuitable for

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CD 2011/3 Page 24 of 26Section 13 - DISPOSAL CONSIDERATIONS

its intended use. If it has been contaminated, it may be possible to reclaim the product by filtration, distillation or some other means. Shelf life considerations should also be applied in making decisions of this type. Note that properties of a material may change in use, and recycling or reuse may not always be appropriate.- DO NOT allow wash water from cleaning or process equipment to enter drains.- It may be necessary to collect all wash water for treatment before disposal.- In all cases disposal to sewer may be subject to local laws and regulations and these should be considered first.- Where in doubt contact the responsible authority.- Recycle wherever possible.- Consult manufacturer for recycling options or consult local or regional waste management authority for disposal if no suitable treatment or disposal facility can be identified.- Dispose of by: burial in a land-fill specifically licenced to accept chemical and / or pharmaceutical wastes or Incineration in a licenced apparatus (after admixture with suitable combustible material).- Decontaminate empty containers. Observe all label safeguards until containers are cleaned and destroyed.

Section 14 - TRANSPORTATION INFORMATION

Labels Required: FLAMMABLE LIQUID

HAZCHEM: ●3YE (ADG7)

ADG7:Class or Division: 3 Subsidiary Risk: NoneUN No.: 1263 Packing Group: IISpecial Provision: 163 Limited Quantity: 5 LPortable Tanks & Bulk T4 Portable Tanks & Bulk TP1, TP8, TP28Containers - Containers - SpecialInstruction: Provision:Packagings & IBCs - PP1 Packagings & IBCs - P001, IBC02Packing Instruction: Special Packing

Provision:Name and Description: PAINT (including paint, lacquer, enamel, stain,

shellac, varnish, polish, liquid filler and liquid lacquer base) orPAINT RELATED MATERIAL (including paint thinning or reducingcompound) (see 3.2.5 for relevant [AUST.] entries)

Land Transport UNDG:Class or division: 3 Subsidiary risk: NoneUN No.: 1263 UN packing group: IIShipping Name:PAINT (including paint, lacquer, enamel, stain, shellac,

varnish, polish, liquid filler and liquid lacquer base)

Air Transport IATA:UN/ID Number: 1263 Packing Group: IISpecial provisions: A3Cargo OnlyPacking Instructions: 364 Maximum Qty/Pack: 60 LPassenger and Cargo Passenger and CargoPacking Instructions: Y341 Maximum Qty/Pack: 5 LPassenger and Cargo Passenger and CargoLimited Quantity Limited QuantityPacking Instructions: 353 Maximum Qty/Pack: 1 L

Shipping name:PAINT

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CD 2011/3 Page 25 of 26Section 14 - TRANSPORTATION INFORMATION

Maritime Transport IMDG:IMDG Class: 3 IMDG Subrisk: NoneUN Number: 1263 Packing Group: IIEMS Number: F- E, S- E Special provisions: 163Limited Quantities: 5 L Marine Pollutant: YesShipping Name: PAINT (including paint, lacquer, enamel, stain, shellac solutions, varnish, polish, liquidfiller and liquid lacquer bas

Section 15 - REGULATORY INFORMATION

POISONS SCHEDULE S6

REGULATIONS

Regulations for ingredients

ZINC STABILIZED (CAS: 7440-66-6) is found on the following regulatory lists;"Australia - Australian Capital Territory - Environment Protection Regulation: Ambient environmental standards (AQUA/1 to 6 - inorganic chemicals)","Australia -Australian Capital Territory - Environment Protection Regulation: Ambient environmental standards (Domestic water supply - inorganic chemicals)","Australia - Australian Capital Territory - Environment Protection Regulation: Ambient environmental standards (IRRIG - inorganic chemicals)","Australia - Australian Capital Territory - Environment Protection Regulation: Ambient environmental standards (STOCK - inorganic chemicals)","Australia

DY-MARK ZINC-GAL GALVANISING ZINC RICH COATING ……Issue Date: 3-Oct-2011 CHEMWATCH 4736-52...

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