C ENTRAL V ERMONT MEDICAL C ENTER Clinical Case Study Presented by: Tegan Bissell, KSC Dietetic...

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CENTRAL VERMONT MEDICAL CENTER Clinical Case Study Presented by: Tegan Bissell, KSC Dietetic Intern 2012-13

Transcript of C ENTRAL V ERMONT MEDICAL C ENTER Clinical Case Study Presented by: Tegan Bissell, KSC Dietetic...

Page 1: C ENTRAL V ERMONT MEDICAL C ENTER Clinical Case Study Presented by: Tegan Bissell, KSC Dietetic Intern 2012-13.

CENTRAL VERMONT MEDICAL CENTERClinical Case Study

Presented by: Tegan Bissell,

KSC Dietetic Intern 2012-13

Page 2: C ENTRAL V ERMONT MEDICAL C ENTER Clinical Case Study Presented by: Tegan Bissell, KSC Dietetic Intern 2012-13.

Objectives

• Discuss the role of alcoholism in the deterioration of

essential organs

• Understand the physiology of the Pancreas

• Determine the differences between Acute and Chronic

Pancreatitis

• Determine the clinical manifestations of malnutrition

• Meet the case study patient and follow his plan of care

and treatment

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Central Vermont Medical Center• Montpelier's Heaton Hospital and Barre City Hospital

merged in 1963 to form the Central Vermont Medical Center.

• 30 distinctive departments employing 1,400 full and part-time employees.

• Licensed for 122 inpatient beds

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Role of Dietitians

Responsible for:• Performing nutrition screening• Assessments• Developing and implementing nutrition care plans• Providing nutrition education to patients, patients’ families,

CVMC Medical staff, employees, and outside groups and agencies as required.

• Participating on interdisciplinary healthcare teams, departmental and interdepartmental work teams.

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Meet Mr. H• 56 year old Male admitted to IP on 7/11/13 with

depression, suicidal and EtOH detox

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Alcohol Metabolism

pubs.niaaa.nih.gov

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PMH• Alcohol dependency with alcoholic liver disease• Seizure disorder• HTN• HPLD• Chronic Hepatitis B• GERD• Alcohol induced Pancreatitis• Bipolar disorder• Depression• Anxiety• DJD• Hypothyroidism

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The Pancreas

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Anatomy of the Pancreas● Glandular tissue and system of ducts● Pancreatic duct merges with the bile duct to form

Ampulla of Vater● Leads to duodenum

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Pancreatitis

Inflammation of the pancreas• Characterized by edema, cellular exudate, and fat necrosis.

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Alcohol’s contribution• EtOH is responsible for 30% of AP cases in the US.• AP is common in men aged 35-45 years old from alcohol

abuse or gallstones.• Oxidative stress• Increased pressure within ducts• Auto-digestion

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Acute Pancreatitis – (AP)• Hyper-metabolic and Catabolic State• Signs of malnutrition: Reduced serum albumin, transferrin,

lymphocytes, and serum calcium

• Symptoms:

• Abdominal pain

• Nausea/Vomiting

• Abdominal distention

• Steatorrhea

• Hypotension

• Oliguria

• Dyspnea

• Shock

• Coma

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Chronic Pancreatitis – (CP)• Evolves slowly over time• Continual attacks of pain radiating into the back• Nausea, vomiting, diarrhea• Increased energy needs• Weight loss• Impaired immune function

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Medical Nutrition TherapyAcute Chronic

Withhold oral and enteral feeding -If oral nutrition cannot be initiated in 5-7 days, start nutrition support

Provide oral diet as in acute phase

Support with IV fluids TF can be used when oral diet is inadequate

Once oral nutrition is started, provide:

Supplement pancreatic enzymes

-Easily digestible foods -Low-fat diet -6 small meals -Adequate protein intake -Increased calories

Supplement fat-soluble vitamins and vitamin B12

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Medications• Antibiotics• Bile salts or fat-soluble vitamins• Diuretics• H2-receptor antagonists• Insulin• Octreotide• Opiates and other pain killers• Pancreatic enzymes• Vitamins and antioxidants

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Day 3: 7/13/13Nutrition consult was received for pt with need for increased Mg and K in diet, as levels are affected by EtOH detox.

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Social Hx• Pt is homeless and had been wandering between VT and

Maine.• Unable to access shelters while intoxicated and continues

to drink. • Has a tent and has been living in the woods at times.• Has been on a current binge for about 2 months.

Family hx:• Alcoholic father died at age 43 from suicide, stepfather who hung

himself and alcoholic paternal uncles. • Mother’s history unknown.

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Diet/Wt Hx• Drinks about 24 beers per day• No special diet

• Ht: 5’ 11” Wt: 130# or 59 kg BMI: 17.6

• Sept 2011: 145#• Oct 2011: 175#• June 2013: 137#

• 5% wt loss in 1 mo.• Clinical indicators of muscle wasting

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Malnutrition

ADA/ASPEN Clinical characteristics that the RD can obtain and document to support a diagnosis of malnutrition● Energy intake● Interpretation of weight loss● Physical findings

○ Body Fat○ Muscle Mass○ Fluid Accumulation

● Reduced grip strength

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SOAP Notes

• Subjective: • No specific complaints, feels he’s eating well. Discussed high potassium

foods and pt likes potatoes.

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Objective:• Dx: Depression, EtOH detox• 7# loss noted in last month per Meditech

• Calories needed: 25-35 cal/kg 1470-1770 cal/day• Protein needed: 1-1.2g/kg 59-71 g/day• Alternate Equation: Mifflin x 1.2

• Diet: Low Fat, Low Cholesterol Intake: 90% meals • Rx: Folic Acid, Thiamine, KCL, Pantoprozole, Mg, Multivitamins

Date Wt Alb Glu BUN CR K CL CA Phos Mg WBC HGB HCT

7/11 3.1

7/12 161 5 8.4 1.3

7/13 137 6 0.8 3.4 2.7 1.4

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• Assessment: • Unintentional wt loss 2’ mental health issues affecting self care,

appetite, and access to calories as evidenced by wt changes, underwt status, and H&P.

• Plan: • Provide 1-2 high K foods each meal per pt preferences, Mg not

readily repleted by diet – Rec supplement as needed• Not appropriate for diet ed at this time.• RD to follow

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Day 5: 7/15/13• Pt transferred to DSCU this AM with Chest Pain

• Put on telemetry monitoring, cardiac enzymes

• Nutrition follow up:• S: “I ate all my meals when on IP. I love fish, chicken, potatoes..”• O: Diet NPO this AM, advance to NAS, Low Fat Low Chol

• A: Tolerating PO diet with excellent intake, expecting transfer back to IP today.

• P: Continue current diet• To follow when admitted back to IP

Date Wt Alb Glu BUN CR K CL CA Phos Mg WBC HGB HCT

7/12 161 5 8.4 1.3

7/13 137 6 0.8 3.4 2.7 1.4

7/15 134 2.8 108 12 0.6 8.4 4.0 1.7 13.4

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Day 6: 7/16/13• New admission back to IP, requires new RD Assessment

• Wt: 135#• New PES: Underweight related to poor PO intake prior to

admission and EtOH intake as evidenced by BMI, Alb levels, and cachectic appearance.

• Current Plan:• Added NAS to current diet order of Low Fat, Low Chol • Nighttime nourishment of PB&J• Calorie count start x3 days

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Day 9: 7/19/13 • Nutrition Follow Up: Pt presenting with signs of refeeding

syndrome as evidenced by Phos and Mg labs.

Date Wt Alb Glu BUN CR K CL CA Phos Mg WBC HGB HCT

7/15 134 2.8 108 12 0.6 8.4 4.0 1.7 13.4

7/16 182 14 3.6 8.6 3.1 1.6

7/19 141 132 19 1.0 3.7 96 1.6 1.3

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Correcting Refeeding Syndrome

• Put on QID phos and IV mg• Wt: 141#• Results of Calorie count: 7/17- 2000cal

7/18- 2300cal

7/19- 2537cal ● 8% wt gain in 8 days

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Day 11: 7/21/13• Pt positive for pneumonia and emphysema changes.• Vomiting episode this AM• Tired and depressed.

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Day 14: 7/24/13• Improved lung sounds, pneumonia resolving.• Intake has been 100% at all meals.• Pt states the food here is great!• Wt: 142#

Date Wt Alb Glu BUN CR K CL CA Phos Mg WBC HGB HCT

7/19 141 132 19 1.0 3.7 96 1.6 1.3

7/22 2.9 130 11 0.9 4.4 3.8 1.4 12.4

7/23 141 145 13 0.9 4.5 99 9.3 1.6 11.7

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Day 16: 7/26/13• Unable to interview pt• Intake 100%

Date Wt Alb Glu BUN CR K CL CA Phos Mg WBC HGB HCT

7/23 141 145 13 0.9 4.5 99 9.3 1.6 11.7

7/26 2.5 123 25 0.9 4.4 104 9.0 3.4 1.7 12.0

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Day 20: 7/30/13• Discharge Plans

• Electrolytes within normal limits• To go to an assisted living facility in VT• Wt gain 10# over 2 weeks, BMI now 18.99• Discussed plan to maintain wt and pt confident with strategies to

prevent wt loss in the future.• Scheduled for mental health follow up within the next week.

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Questions?

Thank you!!

Tegan Bissell, KSC Dietetic Intern, 2012-2013

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References•Escott-Stump, S. Nutrition and diagnosis-related care. 7� th ed.

Lippincott Williams & Wilkin; 2012.

•Whitcomb DC. Clinical practice. Acute Pancreatitis. N Engl J Med 2006; 354:2142

•Mahan, K. Krause’s Food and Nutrition Therapy. 12th edition. Saunders Elsevier; 2008.

•Steer ML, Waxman 1, Freedman S. Chronic Pancreatitis.

•N Engl J Med 1995; 332: 1482.

•Anand P, Park JH, Wu BU. Modern management of acute pancreatitis. Gastroenterol Clin North America. 2012; 41:1-8.

•Gropper, S. Advanced Nutrition and Human Metabolism. 5th ed. Wadsworth, Cengage Learning; 2009.