Blast Injuries & Gunshot Wounds

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    PREET IGEHLAUT

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    BLAST:

    Explosions are physical, chemical,

    or nuclear reactions involving a

    large, rapid release of energy

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    BLAST PHYSIOLOGY:

    Blast injury

    Based on mechanism of tissue injury &physical tissue damage

    defined and broken into 4 categories

    Primary

    SecondaryTertiary

    quaternary

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    BLAST PHYSIOLOGY:

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    PRIMARY BLAST INJURY:

    Result of the physical properties of the blastwave

    occurs as a function of an increase in atmospheric

    pressure over time, referred to as blast overpreassure

    Measure of over pressure is dependent upon: energy of the explosion

    distance from detonation

    distance from detonation

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    PRIMARY BLAST INJURY:

    Molecules in air are constantly in a state ofmotion referred to as the state of the gas

    Due to blast / explosion this state is disturbed

    from normal conditions resulting in: escalation of molecular speed

    increase in the no of molecules occupying a definedspace

    density, pressure & temperature of gas increases a shock wave, or blast wave, develops moving at

    supersonic speeds (3000 to 8000 m/sec)

    loses its pressure and velocity as distance

    increases

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    PRIMARY BLAST INJURY:

    blast front- leading edge of the blast wave

    creates a high-pressure region, or positive phase,called blast wind

    Leaves behind a negative pressure area whichreverses the movement of gas

    damage can occur from both

    In nuclear blast- precursor shock wave isobserved.

    shock front near the ground of heated air and movesahead of the blast wave.6

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    PRIMARY BLAST INJURY:

    victim positioning relative to the primary waveresults in varied damage:

    perpendicular fashion to blast wave - greatest

    amount of impact and injury

    horizontal fashion - less direct surface contact forimpact

    underwater detonations: force of blast wavegreatest at the deepest depths & begins to dissipateas blast wave approaches the surface

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    PRIMARY BLAST INJURY:

    Essentially barotrauma little or no effect on

    solid or fluid-filled organs

    maximal destruction - air-containing organs

    Hallmarks : Perforation of eardrums (overpressure 15-50 psi)

    pneumothoraces (over[ressure 50-100 psi)

    At psi 65 fatality rate approaches 99%

    Other injuries: Intestinal /hollow viscus injury

    Brain injuries

    Paucity of obvious external signs

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    PRIMARY BLAST INJURY:

    Factors potentiating outcome to blast injuries magnitude of the explosion

    potential building collapse

    open air versus enclosed space (6 fold in pulmonary

    injuries in confines spaces) Enhanced wounding measures

    Ball bearings, nails, incendiary

    Other pathognomonic findings : presence of air emboli in pulmonary & coronary vessels

    Representing leading cause of death in victims of pulmonaryblast injury

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    SECONDARY INJURIES:

    Injury from flying debris

    Classic shrapnel injuries

    Of various velocities

    Primary bomb fragments

    Secondary fragments or missiles

    Inert from inanimate objects

    Biological allogenic bone fragments etc may beHIV, HBSAg infected

    Potential for pathogen transmission

    Cases seen in israel n iraq

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    SECONDARY INJURIES:

    Environmental debris such as glass, splinters,soil, and various structural particles may bemajor cause.

    Interface of debris with skin characteristic skin pattern called spalling

    More common than primary injury:

    Reason - Victim doesnt have to be near blastsite

    No 1 military killer in 20th century

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    TERTIARY BLAST INJURIES:

    Injuries from deceleration and structural collapse Axial load injuries

    Wide range blunt injuries:

    Spine Orthopedic

    Head

    Solid & hollow organ Crush syndrome - time delay

    to recovery and weight of

    falling debris

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    QUATERNARY BLAST INJURY:

    result of the byproducts of explosion

    Inhalational injuries:

    From dust and gases

    Burns : Thermal

    Radiation

    Chemical describes the sequela of reflective of dirty bomb

    in which chemical or radiologic-laden detonations mayoccur

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    HUMAN SUICIDE BOMB:

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    HUMAN SUICIDE BOMB:

    walking smart bomb. High grade explosive material used

    Ability to precisely time the explosion

    Ability to detonate in close proximity to victims

    Large load of heavy shrapnel as well as explosivematerial

    body parts acting as missile fragments and projectiles

    may carry with them HIV, hepatitis, and otherserious and yet to be identified threats

    Suicide Bomb victims suffer with the worst of both

    ie Explosion and Penetrating shrapnel

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    SPECIAL CONSIDERATIONS IN PREGNANCY

    Fetus' hollow organs are void of air offering protection from primary blast injury

    However, amniotic fluid, potentially amplifies 3-foldthe blast wave, as in underwater detonations

    concern of potential maternal-fetal injury

    Penetrating shrapnel (2 injuries)

    concern for possible fetal injury

    the closer to term greater potential for fetal injury

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    SPECIAL CONSIDERATIONS IN PREGNANCY

    Tertiary blast effects of deceleration - blunt traumain pregnancy

    Women with previous history of C-sections greater riskfor uterine rupture

    risk of (40-50% ) abruptio placenta

    Chemical injuries:

    Spontaneous abortions (about 4 fold in bhopaldisaster)

    Teratogenicity not proved

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    SPECIAL CONSIDERATIONS IN PREGNANCY

    Radiation injuries depend on: gestational age at time of the exposure

    period between the 2-8 weeks - extremely

    sensitive At significant risk is the central nervous system

    fetal dose of absorption.

    low birth weight

    best chance for fetal survival is that of the stabilityof the mother suffering traumatic injury

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    BLAST LUNG INJURY ( BLI ):

    Second most susceptible organ (1st TM) Direct consequence of blast wave on the body

    Overpressure needed - about 40 psi (40 psi being

    produced by 20Kg TNT exploding 6 meters away) Most common CRITICAL Injury in victims close to

    bomb

    Can be life threatening

    May not have obvious external injury to chest

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    BLAST LUNG INJURY ( BLI ):

    Other pulmonary injuries include:

    Pneumothorax

    Hemothorax

    Pneumomediastinum

    Subcutaneous emphysema

    Air emboli

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    BLAST LUNG INJURY ( BLI ):

    Results in tearing, hemorrhage, contusion andedema

    Micro -hemorrhages in alveoli

    Disruption and weakening of

    alveolar walls

    perivascular and peribronchial tissue Resultant Ventilation-Perfusion mismatch

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    BLAST LUNG INJURY ( BLI ):

    Symptoms: Dyspnoea, Haemoptysis, cough, chest pain

    Signs:

    Tachypnoeic, hypoxic, cynosis, wheezing X-Ray features

    similar to pulmonary contusion, bihilar (butterflypattern) shadows

    pneumothorax, haemothorax

    Can have bronchopleural fistula

    Air embolism from pulmonary disruption

    Other injuries may add to haemodynamic instability

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    Tension pneumothorax of the right lung afterblunt chest trauma

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    G I SYSTEM:

    Most commonly results in

    tissue tearing

    hemorrhage.

    GI blast injury more commonly occurs after blastwave propagation in water.

    GI hemorrhage and perforation is most common in

    lower small intestine or cecum, where gas accumulates.

    Perforations can be delayed

    May develop 24 to 48 hrs post blast

    Manifestations of peritonitis can occur hours or days later

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    HEAD INJURY:

    Loss of consciousness

    Headache, seizures, dizziness, memory problems

    Gait/balance problems, nausea/vomiting, difficultyconcentrating.

    Visual disturbances, tinnitus, slurred speech.

    Disoriented, irritability, confusion. Extremity weakness or numbness

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    CRUSH INJURY:

    results when muscle reperfusion injury occurs as aresult of the release of compressive forces on thetissues or compartment syndrome

    physiologic outcome - traumatic rhabdomyolysis

    myoglobin, potassium, and phosphorus leach intothe circulation

    Clinically, compression of large skeletal muscle is

    necessary for this syndrome About 33% of the patients with rhabdomyolysis will

    develop acute renal failure mortality rate of 30-50%

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    CRUSH INJURY:

    General cond. of pt with crush injury dictated by: other injuries

    delay in extrication

    environmental conditions SIGNS OF COMPARTMENT SYNDROME

    Pain, Pallor, Paresthesia, Paralysis, PulselessnessProgression of symptoms (the 6th P)

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    GUNSHOT WOUNDS:

    An explosive force is applied to a projectile that ispropelled down a tube to fly towards its target

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    GUNSHOT WOUNDS:

    Firearms Type of weapon

    Low-velocity- shot gun, pistol

    High-velocity- rifle

    Caliber

    Missile size

    Bullet construction

    Tumbling/yaw Distance traveled

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    PROJECTILES:

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    RANGE OF FIRE:

    Wound characteristics vary:

    Contact

    The most devastation

    Close Range

    Arms length

    Distant

    Most handguns: significant decrease in KE at 100 m

    Most military rounds: retain large KE at 500m

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    MECHANISM OF INJURY:

    2 areas of projectiletissue interaction in missile-causedwounds

    permanent cavity -localized area of cell necrosis,proportional to size of projectile

    temporary cavity- transient lateral displacement oftissue, after passage of the projectile.

    Elastictissue

    skeletal muscle, blood vessels, and skin, may be pushed

    aside but then rebound

    Inelastic tissue

    bone or liver, may fracture.

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    MECHANISM OF INJURY:

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    GUNSHOT WOUNDS: Entry wound Smaller

    May be darkened, burned Exit wound One, none, or many Larger May be ragged

    Imp to know to determine: Anatomy damaged

    Type of surgical procedure

    Entry and exit wounds can lie ! ! ! ! ! ! Projectiles do nothave to follow a straight line !

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    PATHOPHYSIOLOGY:

    Internal wound

    Tissue contact damage

    High-velocity transfer of energy Shock waves

    Temporary cavity

    Damage proportional to tissue density

    Highly dense tissue sustains more damage

    Distal embolization can occur when a projectile slowsenough and enters the vascular system

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    PATHOPHYSIOLOGY:

    50 % of deaths are due to exsanguination require rapid pressure application and evacuation

    exception Gun Shot Wound to head

    large bore IVs are needed for fluid replacement

    10 % from CNS injury

    Do not delay transport for ANY REASON ! ! ! ! ! ! !

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    GUNSHOT INJURIES:

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    MANAGEMENT:

    Prehospital trauma care:For severely injured patients,survival is time-dependent!

    Golden Hour

    From moment of injury To definitive treatment

    EMS platinum 10 minutes scoop & run OR stay & play

    Assessment and management Every action must have lifesaving purpose

    Organized, detail-oriented, selective, rapid

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    MANAGEMENT:

    Scene Size-up

    Standard precautions

    Scene safety

    Initial triage (total number of patients) Need for more help or equipment?

    Mechanism of injury?

    Expect combined injuries

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    TRAUMA SYSTEM:

    DEFINITION:

    an organized approach to acutely injuredpatients in a defined geographical area that

    provides full and optimal care and that isintegrated with the local or regionalEmergency Medical Service (EMS) system.

    provide the full range of care (from prehospital torehabilitation).

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    TRAUMA SYSTEM GOAL:

    To get the right patient

    to the right hospital

    at the right time

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    TRIAGE:

    French wordmeaning to separate, sort, sift orselect

    the sorting of allocation of treatment to pts esp.

    battle and disaster victims acc. to a system ofpriorities designed to maximize the no of survivors

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    TRIAGE UNIT:

    Determine location of triage areas Clear and assemble the walking wounded

    using verbal instructions

    Conduct Primary triage ensure all pts. are assessed & sorted

    Communicate resources required

    Secondary triage more in-depth assessment

    usually conducted in treatment Unit

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    TREATMENT UNIT:

    Determine location for treatment area

    Coordinate with the Triage unit

    to move patients from the triage treatmentareas

    Establish communication withIncident Command

    Reassess patients

    conduct secondary triage to matchpatient with resources

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    TRANSPORTATION UNIT:

    Management of patient movement from the sceneto the receiving Hospitals

    Establishes adequately sized, easily identifiablepatient loading area

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    STAGING AREA:

    Location designated to collect available resourcesnear incident area

    Several staging areas may be required

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    TRIAGE TAG:

    Alerts care providers to patient priority Prevents re-triage of the same patient

    Serves as a tracking system

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    TRIAGE TAG:

    carried with Diagnostic Equipment in all EMS kits

    should be considered on all calls involving 3 pts

    general placement location for tags

    one of the patients arms or hung around the patientsneck.

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    TRIAGE CATEGORIES:

    IMMEDIATE:Life-threatening buttreatable injuries

    requiring rapidmedical attention

    DELAYED:Potentially seriousinjuries, but are stable

    enough to wait a shortwhile for medicaltreatment

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    TRIAGE CATEGORIES:

    MINIMUM / MINOR:Minor injuries that canwait for longer period

    of time prior totreatment

    MORGUE/EXPECTANT:Death or lack ofspontaneous respiration

    after airway is opened

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    TRIAGE SCORING SYSTEMS

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    START TRIAGE METHOD:

    Simple Triage And Rapid Transport Triage assessment based on 3 criteria

    RPM

    Respirations ( > or < 30/min) Perfusion (Capillary Refill > or< 2/ sec)

    Mental Status (Follow ssimple commands)

    MNEMONIC: 30 2 CAN DO

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    REVERSE TRIAGE: conditions where less wounded are treated in

    preference to more severely wounded such as:

    war - where military setting may require soldiers bereturned to combat as quickly as possible

    disaster situations - where medical resources arelimited

    where significant numbers of medical personnel are

    among the affected patients

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    CAUTION:

    Personal protective equipment is alwaysneeded at trauma scenes

    Do not approach until Scene Size-up iscomplete!

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    ASK SOME QUESTIONS.

    What type of explosive and how much?

    Where was victim located with respect to theblast?

    Were fire/fumes present to cause inhalationalinjury?

    What was orientation of head and torso to theblast?

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    You see what you look for Stephen Sondheim

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    MANAGEMENT:

    Primary survey

    Many components are assessed simultaneously

    Airway- maintenance with cervical spine protection

    Breathing and ventilation

    Circulation with hemorrhage control

    Disability; neurologic status

    Exposure/Environment (completely undress the pt

    and prevent hypothermia)

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    MANAGEMENT: AIRWAY Assume Cervical Spine Injury Maintain inline cervical spine stabilization

    Airway Suction Blood

    Mucus Dental fragments

    Open Airway Head Tilt-Chin Lift

    Jaw Thrust (if Cervical Spine Injury is suspected)

    Maintain Airway Oropharyngeal Airway

    Nasopharyngeal Airway

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    MANAGEMENT: BREATHING

    Assess Breathing Ventilation

    Oxygenation

    Awake with spontaneous breathing Supplemental 100% Oxygen Delivery

    Infants under 1 year old: Oxygen Hood

    Children/Adults: Non rebreather mask with reservoir

    Conscious with respiratory failure Bag Valve Mask with 100% Oxygen

    Unresponsive or respiratory failure

    Orotracheal intubation

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    MANAGEMENT: BREATHING

    Nasotracheal intubation: in breathing patient without major facial trauma

    Surgical airways

    jet insufflation Retrograde intubation

    cricothyrotomy

    tracheostomy

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    BREATHING: PITFALLS

    Tension Pneumothorax

    Rib Fractures

    High risk injury if Fractured ribs 1-3

    Associated with significant cardiopulmonaryinjury

    Flail chest

    Pulmonary Contusion

    Open Pneumothorax

    Massive hemothorax

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    MANAGEMENT: CIRCULATION

    Level of Consciousness Skin color

    Central Pulse

    Child or adult: Carotid pulse or femoral pulse Infant: Brachial Pulse

    Sites of rapid blood loss Chest Injury

    Abdominal Injury (especially retroperitoneal) Pelvic Injury

    Extremity Injury (especially femur)

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    MANAGEMENT: CIRCULATION

    Correct Hypovolemia: Fluid Replacement in Trauma Two large bore IVs (14 or 16 gauge)

    Shorter tubing provides faster IV rate

    Replace fluid deficit

    Infuse Lactated Ringers 2-3 Liters until response

    Consider blood transfusion Unmatched Type-specific blood may be used OR

    Low titer O, or Rh- O if other not available

    Hemorrhage Evaluation

    Avoid potentially harmful measures Vasopressors

    Steroids

    Sodium Bicarbonate

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    MANAGEMENT: DISABILITY

    Level of Consciousness: (AVPU system)

    Alert

    Vocal Stimuli

    Painful stimuli Unresponsive

    GCS

    Pupil response

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    EXPOSURE:

    Undress patient do complete visual inspection

    Keep spine immobilized and log roll

    Prevent Hypothermia Warm crystalloid in microwave or bath to 39C

    Do not microwave Blood, Plasma or Dextrose

    Minimize 2 injury

    RAPID TRANSPORT to Surgical Facility

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    MANAGEMENT:

    Resuscitation of vital functions Detailed secondary survey Definitive care

    Consider abdominal films in all pts with significant blast

    injury Auscultation of chest & chest X rays DPL / FAST- for unstable pts. CT Scan Abdomen/Pelvis for patients with appropriate signs

    and symptoms.

    Hearing in both ears should be tested at bedside. Limb X rays & examination

    Avoid tunnel vision on one injury.

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    ABDOMINAL INJURIES:

    Laparotomy - main stay of investigation andmanagement

    Priorities of the Trauma Laparotomy

    Hemorrhage control

    Contamination control

    Detection of all injuries

    Missed injury - high mortality and morbidity

    Tetanus toxoid and Antibiotics

    initial blood loss on opening abdomen- can be brisk

    patient rapidly can become unstable

    Volume resuscitation & blood transfusion

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    RADIATION MANAGEMENT: Radiation deaths are delayed. Management of conventional injuries and acute life

    threats takes precedence over radiation exposure.

    Treat injury first, then decontaminate.

    Contamination issues No medical personnel have ever received an

    exposure anywhere near the degree to cause

    radiation effects 95% of decontamination occurs with:

    Removal of patients clothing

    Soap & water

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    ENTRAPPED PATIENT TREATMENT

    Volume resuscitation before extrication maintain a euvolemic state with brisk urine output (2ml/kg/hr)

    Limb Stabilization

    Vital signs, oxygen, EKG, IVAdditional Rx &transport

    IS CRUSH SYNDROME OR COMPARTMENTSYNDROME SUSPECTED?

    Look for injuries on Limbs, Pelvis, Gluteal region &Abdominal muscles

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    ENTRAPPED PATIENT TREATMENT:

    Crush / compartment injury signs & symptoms ifpresent:

    TREATMENT PREHOSPITAL ABCs

    Treat other injuries Immobilize affected part-dont use constricting bandages

    TREATMENT HOSPITAL Fluid resuscitation - Brisk diuresis (2 ml/kg/hr)

    Diagnose and treat other metabolic derangementsHyperkalemia, Hypocalcemia

    Pain control

    Anxiolysis

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    ENTRAPPED PATIENT TREATMENT: Mannitol - a nonosmotic diuretic

    Help augments diuresis effective radical scavenger

    Use of bicarbonate:

    alkalization of the urine cast formation

    direct toxic effects ofmyoglobin upon nephrons Hyperkalemia, severe acidosis, and hypervolemia

    continuous renal replacement therapy

    If injury is open: Antibiotics, tetanus, jet irrigation.

    Debridement of nonviable tissues.

    Early amputation for severely injured limbs may be required

    Fasciotomy

    BLAST LUNG INJURY

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    BLAST LUNG INJURYWas the Bombing in Open or Closed Space?

    higher incidence of blast lung injury in enclosed spaces Signs/symptoms suggestive of BLI or resp distress??

    NO

    NO YES

    Management

    ventilatory failure- intubate

    Caution with PPV- BLI, embolism

    Appropriate Rx & transport

    BLAST LUNG INJURY

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    BLAST LUNG INJURY

    HOSPITAL DIAGNOSTIC EVALUATION

    Chest radiography

    Arterial blood gases

    computed tomography

    doppler ultrasound to help diagnose BLI and air emboli.

    Testing conducted per resuscitation protocols

    Acc to nature of explosion (eg. confined space,fire etc)

    OXYGENATION High flow O2 via non-rebreather mask, CPAP,or

    endotracheal intubation.

    BLAST LUNG INJURY

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    BLAST LUNG INJURY:

    CLOSE OBSERVATION Chest decompression- clinical presentation of tension

    pneumothorax.

    Fluid administration

    enough fluid to ensure tissue perfusion & avoiding volumeoverload.

    AIR EMBOLISM

    Position in prone, semi-left lateral, or left lateral

    transport to a facility with a hyperbaric chamber.

    TENSION PNEUMOTHORAX

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    TENSION PNEUMOTHORAX:

    Tension pneumothorax is not an x-raydiagnosis

    it MUST be recognized clinically

    Treatment is decompression needle into 2nd intercostal space of mid-clavicular line -

    followed by thoracotomy tube

    Insert needle here

    MASSIVE HEMOTHORAX TREATMENT

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    MASSIVE HEMOTHORAX: TREATMENT

    Large-bore (32 to 36 F) tube to drain blood

    If moderate sized (500 to 1500 ml) and stops bleeding

    closed drainage usually sufficient

    If initial drainage >1500 ml OR continuous bleeding>200 ml / hr

    OPEN THORACOTOMY

    FLAIL CHEST

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    FLAIL CHEST: Free-floating chest segment- multiple ribs #

    Pain and restricted movement

    paradoxical movement of chest wall with

    respiration

    Treatment :

    Ventilate well

    Humidify oxygen

    Resuscitate with fluids Manage pain

    Stabilize chest

    ventilator

    DISPOSITION AND OUTCOME

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    DISPOSITION AND OUTCOME: No definitive guidelines for observation, admission, discharge

    Patients diagnosed with BLI may need complex management

    should be admitted to an intensive care unit

    suspicious for BLI should be observed in the hospital.

    Discharge decisions will also depend on:

    associated injuries

    other issues related to the event, including the patients

    current social & mental situation.

    Patients with normal chest X Rays, blood gases & pulse oximetry

    no complaints suggesting a BLI

    can be considered for discharge after 4-6 hours of

    observation.

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    BURN INJURY:

    Fluid resuscitation while avoiding fluid overload toprevent further pulmonary injury

    guided by urine output. Consider monitoring central venous pressure

    systemic vascular resistance when indicated.

    Standard burn management

    WOUND MANAGEMENT

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    WOUND MANAGEMENT: Tetanus status.

    Local exploration.

    Delayed primary closure.

    IV followed by oral antibiotics for all but the mosttrivial wounds

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