April 30, 2015Dr. Plechner A.J. Plechner DVM Lecture Two Lecture Two.
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Transcript of April 30, 2015Dr. Plechner A.J. Plechner DVM Lecture Two Lecture Two.
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April 18, 2023 Dr. Plechner
A.J. Plechner DVMLecture Two
Lecture Two
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A modern day animal with an EI imbalance.
Demodectic mange is a common disease.
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Inflammatory Bowel syndrome
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After hormone supplementation and immune regulation
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Irish setter with EI imbalance
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Irish setter with EI imbalance
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Two weeks after adrenal-thyroid supplementation and nomallizing IgA, IgG and IgM.
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Genetic EI imbalance passed from mother to son
SonMother
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3 weeks after correction of adrenal-thyroid immune system. (mother)
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In 1971, clinical studies show an 8 week old golden retriever male puppy with a severe, generalized pyoderma.
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Blood tests indicated a low cortisol, low T3T4, deregulated IgA, IgG, IgM
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In one week.
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In two weeks, T3T4 and IgA,IgG & IgM were remeasured. All had increased and the puppy looked better.
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What happened? Why would a steroid increase the IgG, IgA, IgM?
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After researching endocrine cycles, a biochemist friend was asked to develop a total estrogen test.
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Upon developing this test it demonstrated that high adrenal estrogen, causes deregulation of IgA, IgG & IgM with the case studies tested.
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After adrenal thyroid immune regulation, the puppy appeared like this!
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Lecture One showed this basset hound puppy with an EI imbalance
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This is the puppy after adrenal-thyroid-immune regulation
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Animal cancer patients have an EI imbalance with loss of control of the immune system.
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A cancer patient
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Splenic mass
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Splenic mass – 5 days later.
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Excised mass – Hemangiosarcoma
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As with the hemangiosarcoma patient, a patient with a typical cancer EI makeup has:
Deficient or bound cortisol. High adrenal estrogen. Deficient or bound T3T4 Deregulated IgA, IgG & IgM
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Three other related canines developed Hemangiocarsomas.
The lesions were excised and EI replacement done.
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All three are still alive three years later which has never happened before.
One to six month of life before death usually occurs.
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Six of their offspring had the exact same EI imbalance.
They were corrected with EI therapy to help avoid the same cancer.
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Replacement EI therapy usually avoids cancer development or at least slows down its development until very late in life.
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Can you predict an EI imbalance?Yes! A six to eight week old animal
can be tested for Cortisol, T3T4 and total estrogen! The IgG, IgA & IgM will vary due to vaccination procedures.
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At this time, a new owner may decide to either not take the animal or fund its EI imbalance for life.
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Can you prevent the EI imbalance?
Yes!
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It is not to say that you cannot breed two animals, it only says they may not have an EI compatibility.
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You can mix and match and breed parents with different EI imbalances and produce an average without an imbalance.
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Example – a female Brittany spaniel, a national field trial champion named CeCe, had low cortisol, high adrenal estrogen, suppressed T3T4 and deregulated B & T cells, not only did she stop running and could not become pregnant, she developed “Valley Fever” due to her suppressed T cells.
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EI therapy corrected the above.
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CeCe was good enough as a national field trial champion that the owners decided to breed her.
The male that was picked was “Sundance”, he had a high cortisol, more normal adrenal estrogen, and a normal thyroid.
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CeCe had a litter – the offspring appeared normal, I was given one of the puppies and chose to name her Murphy’s law!
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At 7 months her EI levels and littermates levels were normal.
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If you identify the EI imbalance in parents you can breed it out. The imbalance is in their off-spring.
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We need to have healthy animals so that young people like this can become Veterinarians.