AKU KEGANASAN 191111.ppt

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    KeganasanKeganasanRongga MulutRongga Mulut

    goeno subagyo

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    Speaking

    Chewing

    Tasting

    Swallowing

    Laughing

    Smilling

    Kissing

    Happiness

    Socializing

    C.Everett Koop: You are not healthy without good oral health.

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    Inte!Relationships "etween #al

    an$ S%stemic Health an$ &isease

    SIST'MI

    K

    #R(

    L

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    dokter gigi mempunyai tanggung jawab

    profesional dan moral untuk mempelajarikesehatan umum pasien sebelum

    memberikan perawatan dental

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    Wanita usia 65 tahun dengan

    polimedikasi untuk penyakit

    kardiovaskuler, dengan rampant aries!karies yang "erkem"ang epat

    meli"atkan "e"erapa gigi#

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    Mani,estasi Keganasan $i Mulut

    Signs S%mptoms

    Keganasan

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    Mani,estasi Keganasan $i Mulut

    Sistemik

    Keganasan

    Lokal

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    Mani,estasi #al keganasan

    $on%&odgkin lymphoma

    !large uler on the so't

    palate#

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    Mani,estasi #al Keganasan

    Chroni lymphoyti leukemia

    !uler on the palate#

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    Mani,estasi #al Keganasan

    Chroni lymphoyti leukemia!severe gingival enlargement and

    ulerations#

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    Mani,estasi #al Keganasan

    (lkus Kronis )))))))))

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    Mani,estasi #al Keganasan

    Spontaneous gingial lee$ing o,

    same patient with cihosis

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    Mani,estasi #al Keganasan

    Swollen shin% lee$ing gingia o,a patient with acute m%elogenous leukemia

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    Mani,estasi #al Keganasan

    KE*+$++$

    +$E-+

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    Mani,estasi #al Keganasan

    /aki%laki 60 tahun dengan

    ulkus kronis pada sisi

    lateral lidah kiri.

    -empunyai ke"ia% saan

    merokok dan minum

    minuman keras.

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    /aki%laki perokok 51th.

    dg nonpain'ul lesion

    pd dasar mulut

    selama 1 "ln.

    /eukoplakia

    Candidiasis

    /ihen

    2lanus

    3ral Caner

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    #R(L C(4C'RSilent Kille

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    #al Cance

    5 #R#!6H(R748'(L C(4C'R

    IS TH' 'L'9'4TH

    M#ST C#MM#4 C(4C'R :#RL&:I&'

    ; :ol$ Health #ganization 200*

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    #al Cance

    30000 new cases $iagnose$ %eal%

    .000 $eaths each %ea

    * %ea suial ate< *0=

    Oral cancer is the sixth most common cancer among

    men

    Inci$ence an$ Suial o, #al

    o 6ha%ngeal Cance

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    #al Cance

    4 he overall survival rate 'or oral

    aner in the general population is

    50.

    4 + survival rate 7orse than prostate,

    uterine, "reast, "ladder and olonaners.

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    #al Cance

    5 ( * !%ea suial ate ,o (,ican (meican

    men with oal cance is appo>imatel%

    3)=?

    5 :ith eal% $etection an$ teatment the *!

    %ea %ea suial ate can incease up to

    .*=?

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    #al Cance

    'al% &etection Saes Lies

    5%year survival 'or loali8ed disease is95

    5%year survival 'or metastati disease is;

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    'al% Cance &etection

    'al% #al Cance &etection is #,ten6ossile

    issue hanges in the mouth that may

    signal aner an o'ten "e seen and 'elt

    easily

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    Early detectionEarly detection ChemopreventionChemoprevention

    High-risk:P53; ploidy

    BDJ

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    ( 6ecanceous Lesion

    ( mophologicall% altee$ tissue in which

    cance is moe likel% to occu than in itsappaentl% nomal countepat

    @:H# pulication1//-AA

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    Clinial /esions

    +ssoiated 7ith 2remalignany

    Leukoplakia'%thoplakiaLichen planus

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    ( pe$ominantl% white lesion o, the oalmucosa that cannot e chaacteize$ as an%

    othe $e,inale lesion?

    Leukoplakia

    !W&3, ;;1#

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    :hite Lesions

    /ihen planus

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    Leukoplakia< Clinical Beatues

    5 Leukoplakia usuall% a,,ects people oe

    the age o, )0 %eas @aeage age is +0

    %easA?

    5 6ealence inceases api$l% with age

    paticulal% in males?

    5 (ppo>imatel% . = o, the males oe the

    age o, -0 %eas ae epote$l% a,,ecte$?

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    Leukoplakia< Clinical Beatues

    Continue$

    5 (ppo>imatel% -0 = o, the oal

    leukoplakias ae ,oun$ on the lip emilion

    uccal mucosa an$ gingia?

    5 4ote< Lesions o, the tongue lip emilion

    an$ ,loo o, the mouth account ,o moe

    than /0 = o, those that show $%splasia o

    cacinoma upon histologic e>amination?

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    Leukoplakia< Clinical Beatues

    Continue$

    5 In$ii$ual lesions a% in clinical appeaancean$ ten$ to change oe time?

    5 'al%mil$ lesions usuall% appea as slightl%eleate$ ga% o ga%!white plaDues which ma%

    appea tanslucent ,issue$ o winkle$ an$ aet%picall% so,t an$ ,lat?

    5 'al%mil$ lesions ae usuall% well $emacate$ut ma% len$ into the suoun$ing nomal

    mucosa?

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    Leukoplakia< Clinical Beatues

    Continue$

    5 'al%mil$ thin leukoplakia which sel$om shows$%splasia on iops% ma% $isappea o continueunchange$?

    5I, the cause @sA o, the lesion ae not emoe$man% lesions will ga$uall% ecome thicke an$lage?

    5 The clinical appeaance @sA o, leukoplakia an$

    the anticipate$ un$el%ing histopathologicchanges ae pesente$ in the ,ollowing $iagam?

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    3*

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    3+

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    3.

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    )0

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    Homogeneous leukoplakia

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    + predominantly 7hite lesion 7ithverruous,nodular, ulerated or erythematous

    'eatures.

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    4#4!H#M#8'4'#ES L'EK#6L(KI(

    4o$ula @4A an$ eucous @9A leukoplakia

    49

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    Leukoplakia

    5 Tans,omation to $%splasia ates

    ! Thick leukoplakia F 1!-=

    ! 8anula leukoplakia F )!1*=

    5 Tans,omation to SCC ates

    ! Seee $%splasia F 20!3*=

    ! Esuall% 2!) %eas a,te onset o, leukoplakia

    ! Risk incease$ i,persistent lesion, female patient,

    nonsmoker, FOM or ventral tongue site

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    Leukoplakia5 Teatmentamination

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    Sceening '>amination

    ,o #al Cance

    Sceening e>am eDuies