age change in dental hard tissue

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AGE CHANGES IN DENTAL HARD TISSUES PR ESENTED BY JigyashaTimsina BDS 2 nd year

Transcript of age change in dental hard tissue

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AGE CHANGES IN DENTAL HARD TISSUES

PR ESENTED BY

JigyashaTimsina

BDS 2nd year

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AGING

Definition : Refers to irreversible and inevitable change that occurs with time

It is also defined as sum of all morphological and functional alteration in an organism that leads to functional impairement

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BIOLOGICAL AGING PROCESS IS :

Universal : detectable in all members of the species

Progressive: develop gradually and irreversibly

Deleterious : harmful to the survival of individual

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AGING AND THE HARD TISSUES

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ENAMEL

DENTIN

CEMENTUM

ALVEOLAR BONE

DENTALHARD

TISSUES

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ENAMEL CHANGES

Attrition

Change in permeability

Discoloration

Chemical and surface changes

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ATTRITION

Physiological wear of occlusal or incisal and proximal surfaces of teeth resulting in a decrease in vertical dimension

In severe attrition dentin may be exposed

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CHANGE IN PERMEABILITY

Young enamel acts as a semi-permeable membrane and permits slow passage of water and molecular substances through the pores between the crystals.

With age the enamel crystals grow in size and the pores between them is obliterated resulting in reduced permeability of the enamel.

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DISCOLORATION Normal: white to

yellowish white. With age darkening seen. Thought to be because of: I. Loss of enamel rods-

this loss alters the light reflection of enamel and results tooth color change.

II. Deepening of dentin color seen through progressively thinning layer of enamel.

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CHEMICAL AND SURFACE CHANGES

Increase in fluoride and nitrogen content.

Water and organic content decrease with age.

Reduction in organic content reduces chance of caries in teeth with age.

Loss of Perikymata

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DENTIN

The main changes in dentin associated with aging are

Increase in sclerotic dentin. Increase in the number of

dead tracts. Increase in formation of

reparative and reactive dentin.

Vitality of dentin

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DEAD TRACTS In normal dentin the odontoblastic

processes may disintegrate and the empty tubules get filled with air. These are called dead tracts.

They appear black in transmitted light and white in reflected light.

In narrow pulpal horns degeneration of odontoblast seen due to crowding of odontoblasts.

thought to be the initial step in the formation of sclerotic dentin.

Dead tract

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SCLEROTIC DENTIN\TRANSPARENT DENTIN Refers to the dentinal tubules that have

become occluded with calcified materials. It may be result of the aging process and

called physiologic dentin sclerosis or may occur due to some irritation like caries, attrition, abrasion and called reactive dentin sclerosis

When this occurs in several tubules in the same area , the dentin assumes a glassy appearance and become transparent

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Most common in apical 3rd of the root.

It appears transparent or light in transmitted light and dark in reflected light.

Sclerotic dentin

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REPARATIVE –REACTIVE DENTIN If the provoking stimulus cause destruction

of the original odontoblasts, the new, less tubular dentin formed by newly differentiated odontoblast like cells is called Reparative dentin.

However if the odontoblast survive the provoking stimuli the dentin produced by them is called Reactionary dentin

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Reparative dentin

Reactive dentin

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VITALITY OF DENTIN

Since the odontoblasts do not degenerate normally, dentin is laid down throughout life.

Although after the teeth have erupted and become functional dentinogenesis slows and further dentin formation is at much slower rate

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CEMENTUM Hypercementosis . Cementicles. Change in permeability. Cementum re-absorption and

repair

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HYPERCEMENTOSIS

It is an abnormal thickening of the cementum. may be generalized or localized ,diffused or

limited. Hypercementosis is termed cementum

hypertrophy if the overgrowth improves the functional qualities of the cementum and is termed cementum hyperplasia if it is not correlated with increased function.

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FIG : A

FIG : B

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CEMENTICLES They are ovoid or round

calcified structure that are formed as a result of calcification of the degenerated periodontal tissue or the epithelial rests of Malssez.

Cementicles may be: 1. Free in the periodontal

ligament. 2. Attached the cementum 3. Embedded in the

cementum

FC-free cementicleSC-sessile cementicle

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Permeability : The permeability of cementum decreases gradually by age.

The permeability from the periodontal side is lost except in the most recently formed layer of cementum, while that from the dentine side remains only in the apical region.

Cementum Resorption and Repair: Cementum resorption can occur after trauma or excessive occlusal forces .

After resorption ceases, the damage is usually repaired.

If the repair establishes the former outline of the root surface it is called anatomic repair. However if only a thin layer of cementum is deposited and the root outline is not constructed it is called functional repair.

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ALVEOLAR BONE

Bone resorption is the major age change in alveolar bone.

Common site of resorption- labial aspect of alveolar crest.

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Bone reabsorp

tion

Decrease in height and width of the jaw

Distance between the crest of the

alveolar bone and cemento-enamel junction increase

Alveolar sockets

appear jagged and uneven

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OTHER CHANGES

The marrow space have fatty infiltration.

Loss of maxillary bone is accompanied by increase in size of maxillary sinus.

Internal trabecular arrangement is more open which indicate bone loss.

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REFERENCES

Orban’s oral histology and embryology

Ten Cate’s oral histology

Oral anatomy ,histology and embryology

Berkovitz , Holland , Moxham

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THANK YOU